Search results for "microenvironment."

showing 10 items of 363 documents

SPIONs embedded in polyamino acid nanogels to synergistically treat tumor microenvironment and breast cancer cells.

2018

Abstract The extremely complex tumor microenvironment (TME) in humans is the major responsible for the therapeutic failure in cancer nanomedicine. A new concept of disease-driven nanomedicine, henceforth named “Theranomics”, which attempts to target cancer cells and TME on the whole, represents an attractive alternative. Herein, a nanomedicine able to co-deliver doxorubicin and a tumor suppressive proteolytic protein such as collagenase-2 was developed. We successfully obtained superparamagnetic nanogels (SPIONs/Doco@Col) via the intermolecular azide-alkyne Huisgen cycloaddition. We demonstrated that a local ECM degradation and remodeling in solid tumors by means of collagenase-2 could enha…

Polyamino acidPolyamino acidsCollagenasePharmaceutical ScienceBreast Neoplasms02 engineering and technology030226 pharmacology & pharmacy03 medical and health sciences0302 clinical medicineBreast cancerBreast cancerDrug Delivery SystemsCell Line TumormedicineTumor MicroenvironmentHumansDoxorubicinTargeted cancer therapyAmino AcidsMagnetite NanoparticlesTumor microenvironmentAntibiotics AntineoplasticChemistrySPIONCancerTheranomicDrug Synergism021001 nanoscience & nanotechnologymedicine.diseasenanomedicineNanomedicinesDrug LiberationSPIONsMatrix Metalloproteinase 8DoxorubicinCancer cellCancer researchNanomedicineTheranomicsFemaleBreast cancer cellspolyamino acid0210 nano-technologyGelsmedicine.drugInternational journal of pharmaceutics
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Molecular insights and novel approaches for targeting tumor metastasis

2020

In recent years, due to the effective drug delivery and preciseness of tumor sites or microenvironment, the targeted drug delivery approaches have gained ample attention for tumor metastasis therapy. The conventional treatment approaches for metastasis therapy have reported with immense adverse effects because they exhibited maximum probability of killing the carcinogenic cells along with healthy cells. The tumor vasculature, comprising of vasculogenic impressions and angiogenesis, greatly depends upon the growth and metastasis in the tumors. Therefore, various nanocarriers-based delivery approaches for targeting to tumor vasculature have been attempted as efficient and potential approaches…

PolymersAngiogenesisMetal NanoparticlesPharmaceutical ScienceAntineoplastic Agents02 engineering and technologyTumor vasculature030226 pharmacology & pharmacyMetastasis03 medical and health sciencesDrug Delivery Systems0302 clinical medicineNeoplasmsTumor MicroenvironmentHumansMedicineNeoplasm MetastasisAdverse effectDrug CarriersNeovascularization Pathologicbusiness.industryConventional treatmentPhototherapy021001 nanoscience & nanotechnologymedicine.diseaseLipidsTargeted drug deliveryDrug deliveryCancer researchNanoparticlesRNANanocarriersPeptides0210 nano-technologybusinessInternational Journal of Pharmaceutics
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Iron Metabolism in the Tumor Microenvironment-Implications for Anti-Cancer Immune Response

2021

New insights into the field of iron metabolism within the tumor microenvironment have been uncovered in recent years. Iron promotes the production of reactive oxygen species, which may either trigger ferroptosis cell death or contribute to malignant transformation. Once transformed, cancer cells divert tumor-infiltrating immune cells to satisfy their iron demand, thus affecting the tumor immunosurveillance. In this review, we highlight how the bioavailability of this metal shapes complex metabolic pathways within the tumor microenvironment and how this affects both tumor-associated macrophages and tumor-infiltrating lymphocytes functions. Furthermore, we discuss the potentials as well as th…

Programmed cell deathIronReviewMalignant transformationImmune systemNeoplasmsTumor MicroenvironmentmedicineHumanscanceriron metabolismlcsh:QH301-705.5chemistry.chemical_classificationReactive oxygen speciesTumor microenvironmentInnate immune systemImmunityCancerGeneral Medicinemedicine.diseaseferroptosisferroptosiadaptive immune response tumor microenvironmentlcsh:Biology (General)chemistryCancer cellCancer researchinnate immune response
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Hypoxia inducible factor-1alpha inactivation unveils a link between tumor cell metabolism and hypoxia-induced cell death.

2008

Hypoxia and the acquisition of a glycolytic phenotype are intrinsic features of the tumor microenvironment. The hypoxia inducible factor-1alpha (HIF-1alpha) pathway is activated under hypoxic conditions and orchestrates a complex transcriptional program that enhances cell survival. Although the consequences of HIF-1alpha inactivation in cancer cells have been widely investigated, only a few studies have addressed the role of HIF-1alpha in the survival of cancer cells endowed with different glycolytic capacities. In this study, we investigated this aspect in ovarian cancer cells. Hypoxia-induced toxicity was increased in highly glycolytic cells compared with poorly glycolytic cells; it was a…

Programmed cell deathMice SCIDBiologyPathology and Forensic MedicineMiceCell Line TumormedicineAnimalsHumansGene SilencingRNA Small InterferingCell ProliferationOvarian NeoplasmsTumor microenvironmentCell DeathCell growthLentivirusHypoxia (medical)Hypoxia-Inducible Factor 1 alpha SubunitCell HypoxiaCell biologyPhenotypeHypoxia-inducible factorsApoptosisCell cultureCancer cellFemalemedicine.symptomRegular ArticlesThe American journal of pathology
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Contribution of proteomics to understanding the role of tumor-derived exosomes in cancer progression: State of the art and new perspectives

2013

Exosomes are nanometer-sized vesicles (40-100 nm diameter) of endocytic origin released from different cell types under both normal and pathological conditions. They function as cell free messengers, playing a relevant role in the cell-cell communication that is strongly related to the nature of the molecules (proteins, mRNAs, miRNAs, and lipids) that they transport. Tumor cells actively shed exosomes into their surrounding microenvironment and growing evidence indicates that these vesicles have pleiotropic functions in the regulation of tumor progression, promoting immune escape, tumor invasion, neovascularization, and metastasis. During the last few years remarkable efforts have been made…

ProteomicsCell signalingProteomeEndocytic cycleCell CommunicationBiologyExosomesProteomicsBiochemistryRNA TransportCell biology / Tumor-derived exosome / Tumor progressionSettore BIO/13 - Biologia ApplicataNeoplasmsmicroRNABiomarkers TumorTumor MicroenvironmentAnimalsHumansMolecular BiologyTumor microenvironmentTumor-DerivedMicrovesiclesCell biologyTumor progressionDisease ProgressionPROTEOMICS
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Role of Extracellular Vesicles in Hematological Malignancies.

2015

In recent years the role of tumor microenvironment in the progression of hematological malignancies has been widely recognized. Recent studies have focused on how cancer cells communicate within the microenvironment. Among several factors (cytokines, growth factors, and ECM molecules), a key role has been attributed to extracellular vesicles (EV), released from different cell types. EV (microvesicles and exosomes) may affect stroma remodeling, host cell functions, and tumor angiogenesis by inducing gene expression modulation in target cells, thus promoting cancer progression and metastasis. Microvesicles and exosomes can be recovered from the blood and other body fluids of cancer patients a…

ProteomicsCell typeImmunology and Microbiology (all)lcsh:MedicineReview ArticleBiologyProteomicsExosomesGeneral Biochemistry Genetics and Molecular BiologyMetastasisExtracellular VesiclesStromaSettore BIO/13 - Biologia ApplicatamedicineTumor MicroenvironmentHumansTumor microenvironmentBiochemistry Genetics and Molecular Biology (all)General Immunology and MicrobiologyNeovascularization Pathologiclcsh:RCancerGeneral Medicinemedicine.diseaseMicrovesiclesCell biologyGene Expression Regulation NeoplasticHematologic NeoplasmsCancer cellBiochemistry Genetics and Molecular Biology (all); Immunology and Microbiology (all)BioMed research international
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Multiple changes induced by fibroblasts on breast cancer cells.

2010

It is now widely recognised that the cross-talk between cancer and stromal cells may play a crucial role in cancer progression. However little is known about the complex underlying molecular mechanisms that occur within the tumor microenvironment. Fibroblasts are the major stromal cells with multiple roles, especially towards both the extracellular matrix and the neighbouring cell population, including neoplastic cells. Consequently, proteomic analyses would provide a wider resource for a better understanding of the potential modulating effects exerted by fibroblasts on cancer cells. In this report we describe the effects of fibroblast stimulation on the breast cancer cell line (8701-BC) pr…

ProteomicsStromal cellProteomeCellGenes mycBreast NeoplasmsCell CommunicationBiologyBiochemistryProto-Oncogene Proteins c-mycRheumatologyCell MovementCell Line TumormedicineHumansOrthopedics and Sports MedicineNeoplasm InvasivenessSettore BIO/06 - Anatomia Comparata E CitologiaFibroblastMolecular BiologyCell ProliferationTumor microenvironmentOncogeneCancerCell BiologyFibroblastsmedicine.diseaseCoculture TechniquesCell biologyUp-RegulationGene Expression Regulation NeoplasticCytoskeletal Proteinsmedicine.anatomical_structureCulture Media ConditionedSpectrometry Mass Matrix-Assisted Laser Desorption-IonizationCancer cellNeoplastic cellproteomics breast cancer cells fibroblasts invasion assay cell proliferation.FemaleStromal CellsConnective tissue research
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The GRP94 Inhibitor PU-WS13 Decreases M2-like Macrophages in Murine TNBC Tumors: A Pharmaco-Imaging Study with 99mTc-Tilmanocept SPECT

2021

Triple-negative breast cancer (TNBC) is the most aggressive subtype of breast cancers and is not eligible for hormone and anti-HER2 therapies. Identifying therapeutic targets and associated biomarkers in TNBC is a clinical challenge to improve patients’ outcome and management. High infiltration of CD206+ M2-like macrophages in the tumor microenvironment (TME) indicates poor prognosis and survival in TNBC patients. As we previously showed that membrane expression of GRP94, an endoplasmic reticulum chaperone, was associated with the anti-inflammatory profile of human PBMC-derived M2 macrophages, we hypothesized that intra-tumoral CD206+ M2 macrophages expressing GRP94 may represent innovative…

QH301-705.5GRP94M2-like macrophages03 medical and health sciences0302 clinical medicineBreast cancerIn vivoSpect imagingmedicineBiology (General)Triple-negative breast cancerGRP94; M2-like macrophages; triple-negative breast cancer; PU-WS13; SPECT imaging; biomarker; CD206; Tilmanocept030304 developmental biology0303 health sciencesTumor microenvironmentSPECT imagingbusiness.industryGeneral Medicinemedicine.diseasePU-WS133. Good health030220 oncology & carcinogenesisCancer researchtriple-negative breast cancerBiomarker (medicine)biomarkerbusinessCD8HormoneCells
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Targeted Therapy Modulates the Secretome of Cancer-Associated Fibroblasts to Induce Resistance in HER2-Positive Breast Cancer

2021

The combination of trastuzumab plus pertuzumab plus docetaxel as a first-line therapy in patients with HER2-positive metastatic breast cancer has provided significant clinical benefits compared to trastuzumab plus docetaxel alone. However, despite the therapeutic success of existing therapies targeting HER2, tumours invariably relapse. Therefore, there is an urgent need to improve our understanding of the mechanisms governing resistance, so that specific therapeutic strategies can be developed to provide improved efficacy. It is well known that the tumour microenvironment (TME) has a significant impact on cancer behaviour. Cancer-associated fibroblasts (CAFs) are essential components of the…

Receptor ErbB-2Cancer-associated fibroblastQH301-705.5breast cancer; HER2-positive; tumour microenvironment; targeted therapy; trastuzumab; resistance; cancer-associated fibroblast; label-free proteomics; miRNABreast NeoplasmsDocetaxelAntibodies Monoclonal HumanizedArticleCatalysisInorganic ChemistryresistanceDrug Delivery SystemsLabel-free proteomicsbreast cancerCancer-Associated FibroblastsCell Line TumorAntineoplastic Combined Chemotherapy ProtocolsHumansPhysical and Theoretical ChemistryBiology (General)skin and connective tissue diseasesMolecular BiologyQD1-999SpectroscopymiRNAOrganic ChemistryGeneral Medicinetargeted therapyHER2-positiveComputer Science ApplicationstrastuzumabChemistryDrug Resistance NeoplasmFemaletumour microenvironmentInternational Journal of Molecular Sciences
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Endothelial cells and normal breast epithelial cells enhance invasion of breast carcinoma cells by CXCR-4-dependent up-regulation of urokinase-type p…

2008

Here we show the increase of invasion of three breast cancer cell lines (8701-BC, MDA-MB-231 and SKBR3) upon long-term co-incubation with culture medium of normal microvascular endothelial cells (MVEC) and normal breast epithelial cells (HB2). The enhancement of invasion relied on the interaction of microvascular endothelial cell and normal breast epithelial cell CXCL12 (SDF1) chemokine, whose expression by breast cancer cells was very low, with the cognate CXCR4 receptor of malignant cells, which resulted in over-expression of the urokinase-type plasminogen activator receptor (uPAR) on their surfaces. uPAR over-expression, showed by RT-PCR and Western blotting, was paralleled by increased …

Receptors CXCR4MAP Kinase Kinase 4AngiogenesisCellBreast NeoplasmsReceptors Cell SurfaceCell CommunicationBiologyCell LineReceptors Urokinase Plasminogen ActivatorPathology and Forensic MedicineMetastasisangiogenesisbreast cancerTumor Cells CulturedmedicineHumansNeoplasm InvasivenessBreastSettore BIO/06 - Anatomia Comparata E CitologiaPhosphorylationskin and connective tissue diseasesCXCR4Settore MED/04 - Patologia GeneraleNeovascularization PathologicReverse Transcriptase Polymerase Chain ReactionFibrinolysisEpithelial CellsCXCL12invasionmedicine.diseasemicroenvironmentChemokine CXCL12Neoplasm ProteinsUp-RegulationEndothelial stem cellUrokinase receptormedicine.anatomical_structureCulture Media ConditionedCancer cellCancer researchFemaleJNKEndothelium VascularBreast diseaseSDF1uPARPlasminogen activatorThe Journal of Pathology
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