Search results for "mir"

showing 10 items of 1117 documents

miRNAs and their potential for use against cancer and other diseases

2007

miRNAs are 19–24 nucleotide long noncoding RNAs found in almost all genetically dissected species, including viruses, plants, nematodes, flies, fish, mice and humans. Rapid advances have been made in understanding their physiological functions, while abnormal patterns of miRNA expression have been found in many disease states, most notably human cancer. It is now clear that miRNAs represent a class of genes with a great potential for use in diagnosis, prognosis and therapy. In this review we will focus on the discoveries that elucidate their crucial role in mammalian diseases, particularly in cancer, and propose that miRNA-based gene therapy might become the potential technology of choice …

Cancer ResearchGenetic enhancementGenetic TherapyGeneral MedicineDiseaseComputational biologyBiologyPrognosisBioinformaticsMicroRNAsOncologyMirna expressionNeoplasmsmicroRNAAnimalsHumansPersonalized therapyGeneHuman cancerFuture Oncology
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MiRNA profiling by NGS in resectable non-small cell lung cancer: Prognostic implications.

2014

7559 Background: MicroRNAs (miRNA) regulate gene expression, and are implicated in several processes like tumorogenesis. Here, we applied a multiplexed NGS approach to study differentially expressed miRNAs (tumor/ normal) in a cohort of resectable NSCLC patients and its correlation with clinical outcome. Methods: RNA was isolated from frozen lung specimens (tumor/ normal) from 33 patients. High-quality samples were analyzed and enriched in the miRNA fraction. Libraries were prepared according to manufactured instruction (SOLID), and miRNAs were sequenced. Data analysis was carried out using CLCbio software. First, raw data were annotated using miRBase and normalized by total reads followed …

Cancer ResearchIn silicoRNAComputational biologyBiologyBioinformaticsmedicine.diseaseMiRBaseOncologymicroRNAGene expressionmedicineMirna profilingNon small cellLung cancer
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miR-155expression in antitumor immunity: The higher the better?

2019

MicroRNAs are small noncoding RNAs that modulate gene expression either directly, by impairing the stability and/or translation of transcripts that contain their specific target sequence, or indirectly through the targeting of transcripts that encode transcription factors, factors implicated in signal transduction pathways, or epigenetic regulators. Abnormal expression of micro-RNAs has been found in nearly all types of pathologies, including cancers. MiR-155 has been the first microRNA to be implicated in the regulation of the innate and adaptative immune responses, and its expression is either increased or decreased in a variety of liquid and solid malignancies. In this review, we examine…

Cancer ResearchLeukemiaCarcinogenesisBiologymiR-155MicroRNAs03 medical and health sciences0302 clinical medicineImmune system030220 oncology & carcinogenesisGene expressionmicroRNAGeneticsCancer researchAnimalsHumansCytotoxic T cellTumor EscapeImmunotherapyEpigeneticsDown SyndromeSignal transductionTranscription factorGenes, Chromosomes and Cancer
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miRNA detection methods and clinical implications in lung cancer

2014

[EN] Lung cancer is the leading cause of cancer death worldwide. Therefore, advances in the diagnosis and treatment of the disease are urgently needed. miRNAs are a family of small, noncoding RNAs that regulate gene expression at the transcriptional level. miRNAs have been reported to be deregulated and to play a critical role in different types of cancer, including lung cancer. Thus, miRNA profiling in lung cancer patients has become the core of several investigations. To this end, the development of a multitude of platforms for miRNA profiling analysis has been essential. This article focuses on the different technologies available for assessing miRNAs and the most important results obtai…

Cancer ResearchLung NeoplasmsDiseaseBioinformaticsmicroRNADiagnosismedicineBiomarkers TumorMirna profilingHumansGenetic TestingLung cancerCancer deathmiRNAbusiness.industryCancerMICROBIOLOGIAGeneral MedicineBiomarkermedicine.diseasePrognosishumanitiesBiomarker (cell)MicroRNAsOncologymiRNA profilingLung cancerbusiness
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Newly-Discovered Neural Features Expand the Pathobiological Knowledge of Blastic Plasmacytoid Dendritic Cell Neoplasm

2021

Simple Summary For the first time, neuronal features are described in blastic plasmacytoid dendritic cell neoplasm (BPDCN) by a complex array of molecular techniques, including microRNA and gene expression profiling, RNA and Chromatin immunoprecipitation sequencing, and immunohistochemistry. The discovery of unexpected neural features in BPDCN may change our vision of this disease, leading to the designing of a new BPDCN cell model and to re-thinking the relations occurring between BPDCN and nervous system. The observed findings contribute to explaining the extreme tumor aggressiveness and also to propose novel therapeutic targets. In view of this, the identification, in this work of new po…

Cancer ResearchNeurogenesisNeoplasms. Tumors. Oncology. Including cancer and carcinogensMicroRNA Expression ProfilesequencingBiologySettore MED/08 - Anatomia PatologicaBPDCN MiRNA Network Neurogenesis SequencingBPDCNArticleChromatinGene expression profilingBPDCN; MiRNA; Network; Neurogenesis; SequencingneurogenesisOncologyDownregulation and upregulationmicroRNAnetworkCancer researchImmunohistochemistrySettore MED/05 - Patologia ClinicaNeurogenesiRC254-282ProgenitormiRNACancers
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Key lncRNAs involved in ischemic strokes

2022

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Cancer Researchmedicine.medical_specialtyIschemic strokesBiologymedicine.diseaseBrain Ischemiaischemic stroke lncRNAs miRNAs RNA transcripts stroke Humans Brain Ischemia Ischemic Stroke RNA Long NoncodingInternal medicineIschemic strokeGeneticsKey (cryptography)medicineCardiologyHumansRNA Long NoncodingStrokeIschemic StrokeEpigenomics
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MYC dosage compensation is mediated by miRNA-transcription factor interactions in aneuploid cancer

2021

Summary We hypothesize that dosage compensation of critical genes arises from systems-level properties for cancer cells to withstand the negative effects of aneuploidy. We identified several candidate genes in cancer multiomics data and developed a biocomputational platform to construct a mathematical model of their interaction network with micro-RNAs and transcription factors, where the property of dosage compensation emerged for MYC and was dependent on the kinetic parameters of its feedback interactions with three micro-RNAs. These circuits were experimentally validated using a genetic tug-of-war technique to overexpress an exogenous MYC, leading to overexpression of the three microRNAs …

Candidate geneMultidisciplinaryDosage compensationColorectal cancerBioinformaticsScienceQCancerMycBiologymedicine.diseaseArticleDownregulation and upregulationCancer cellmicroRNATranscription factorsmedicineCancer researchcancerDosage compensationaneuploidyMathematical biosciencesSystems biologyTranscription factormiRNAiScience
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WIN55,212-2-induced expression of Mir-29b1 favours the suppression of osteosarcoma cell migration in a SPARC-independent manner

2019

WIN55,212-2 (WIN) is a synthetic agonist of cannabinoid receptors that displays promising antitumour properties. The aim of this study is to demonstrate that WIN is able to block the migratory ability of osteosarcoma cells and characterize the mechanisms involved. Using wound healing assay and zymography, we showed that WIN affects cell migration and reduces the activity of the metalloproteases MMP2 and MMP9. This effect seemed to be independent of secreted protein acidic and rich in cysteine (SPARC), a matricellular protein involved in tissue remodeling and extracellular matrix deposition. SPARC release was indeed prevented by WIN, and SPARC silencing by RNA interference did not influence …

Cannabinoid receptorMorpholinesAntineoplastic AgentsMMP9NaphthalenesCatalysisArticlelcsh:ChemistryInorganic ChemistryExtracellular matrixExtracellular VesiclescannabinoidsDownregulation and upregulationCell MovementCell Line TumorSettore BIO/10 - BiochimicaGene silencingHumansOsteonectinCell migrationPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologyCannabinoidSpectroscopyCell ProliferationOsteosarcomaChemistryCell growthOrganic ChemistryMatricellular proteinCell migrationSPARCGeneral MedicineComputer Science ApplicationsCell biologyBenzoxazinesMiR-29b1MicroRNAslcsh:Biology (General)lcsh:QD1-999
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Carbapenem-Susceptible OXA-23-Producing Proteus mirabilis in the French Community

2019

International audience; Nineteen Proteus mirabilis isolates producing the carbapenemase OXA-23 were recovered over a 2-year period in 19 French hospitalized patients, of whom 12 had community onset infections. The isolates exhibited a slightly reduced susceptibility to carbapenems. Whole-genome analysis revealed that all 19 isolates formed a cluster compared to 149 other P. mirabilis isolates. Because of its susceptibility to carbapenems, this clone may be misidentified as a penicillinase producer while it constitutes a reservoir of the OXA-23-encoding gene in the community.

CarbapenemHospitalized patientsspreadclonalityMicrobial Sensitivity Testsbeta-LactamasesEpidemiology and SurveillanceMicrobiology03 medical and health sciencescarbapenemasemedicinepolycyclic compoundsHumansPharmacology (medical)Proteus mirabilis030304 developmental biologyCommunity onsetPharmacology0303 health sciencesbiologyOXA-23030306 microbiologybiochemical phenomena metabolism and nutritionbiology.organism_classificationProteus mirabilisAnti-Bacterial AgentsInfectious DiseasesReduced susceptibility[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyCarbapenemsbacteriaFranceProteus Infectionsmedicine.drug
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miR-133a Enhances the Protective Capacity of Cardiac Progenitors Cells after Myocardial Infarction

2014

Summary miR-133a and miR-1 are known as muscle-specific microRNAs that are involved in cardiac development and pathophysiology. We have shown that both miR-1 and miR-133a are early and progressively upregulated during in vitro cardiac differentiation of adult cardiac progenitor cells (CPCs), but only miR-133a expression was enhanced under in vitro oxidative stress. miR-1 was demonstrated to favor differentiation of CPCs, whereas miR-133a overexpression protected CPCs against cell death, targeting, among others, the proapoptotic genes Bim and Bmf. miR-133a-CPCs clearly improved cardiac function in a rat myocardial infarction model by reducing fibrosis and hypertrophy and increasing vasculari…

Cardiac function curveProgrammed cell deathMyocardial InfarctionGene ExpressionCardiomegalyBiologyBiochemistryArticleMuscle hypertrophyParacrine signallingDownregulation and upregulationmiR-133a; Cardiac Progenitors Cells; Myocardial InfarctionFibrosisREGENERATIONmicroRNAGeneticsmedicineMyocyteAnimalsRNA MessengerOXIDATIVE STRESSlcsh:QH301-705.5ENGINEERED HEART-TISSUElcsh:R5-920Gene Expression ProfilingMICRORNAComputational BiologyCell BiologyMUSCLEmedicine.disease3. Good healthCell biologyRatsAPOPTOSISHYPERTROPHYMicroRNAsDIFFERENTIATIONlcsh:Biology (General)ImmunologyGROWTHRNA Interferencelcsh:Medicine (General)EMBRYONIC STEM-CELLSMyoblasts CardiacDevelopmental BiologyStem Cell Reports
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