Search results for "mito"

showing 10 items of 2513 documents

Interplay of oxidants and antioxidants during exercise: Implications for muscle health

2010

Muscle contraction results in generation of reactive oxygen and nitrogen species (RONS) at a rate determined by the intensity, frequency, and duration of the exercise protocols. Strenuous exercise causes oxidation of protein, lipid, and DNA, release of cytosolic enzymes, and other signs of cell damage; however, only exhaustive exercise is detrimental. Indeed, the regulation of vascular tone, the excitation-contraction coupling, growth, and differentiation in skeletal muscle, are governed in part by RONS. This is accomplished by RONS interaction with redox-sensitive transcription factors, leading to increased gene expression of antioxidant enzymes, cytoprotective proteins, and other enzymes …

Cell signalingmedicine.medical_specialtyFree RadicalsHealth StatusGene ExpressionPhysical Therapy Sports Therapy and Rehabilitationmedicine.disease_causeAntioxidantsInternal medicinemedicineHumansOrthopedics and Sports MedicineExercise physiologyMuscle SkeletalExerciseTranscription factorCell damageExercise ToleranceChemistryNF-kappa BSkeletal musclemedicine.diseaseAdaptation PhysiologicalReactive Nitrogen SpeciesOxidative Stressmedicine.anatomical_structureEndocrinologyMitogen-Activated Protein KinasesSignal transductionmedicine.symptomReactive Oxygen SpeciesOxidation-ReductionOxidative stressMuscle ContractionSignal TransductionMuscle contraction
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Predetermined embryonic glial cells form the distinct glial sheaths of the Drosophila peripheral nervous system

2013

International audience; One of the numerous functions of glial cells in Drosophila is the ensheathment of neurons to isolate them from the potassium-rich haemolymph, thereby establishing the blood-brain barrier. Peripheral nerves of flies are surrounded by three distinct glial cell types. Although all embryonic peripheral glia (ePG) have been identified on a single-cell level, their contribution to the three glial sheaths is not known. We used the Flybow system to label and identify each individual ePG in the living embryo and followed them into third instar larva. We demonstrate that all ePG persist until the end of larval development and some even to adulthood. We uncover the origin of al…

Cell typeCell tracingCellular differentiation[SDV]Life Sciences [q-bio]Biology03 medical and health sciences0302 clinical medicineFlybowmedicineAnimalsDrosophila ProteinsGlial sheathsMolecular BiologyMitosis[SDV.BDD]Life Sciences [q-bio]/Development BiologyResearch Articles030304 developmental biologyProgenitorHomeodomain Proteins0303 health sciencesMicroscopy ConfocalHyperplasiafungiEmbryoCell DifferentiationAnatomyHypertrophyEmbryonic stem cellImmunohistochemistryCell biology[SDV] Life Sciences [q-bio]medicine.anatomical_structurePeripheral nervous systemNeurogliaDrosophilaPeripheral nervous systemNeuroglia030217 neurology & neurosurgeryCell-specific mitotic abilitiesDevelopmental Biology
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Terminally differentiated postmitotic tumor cells in a rat rhabdomyosarcoma cell line.

1988

A permanent rat rhabdomyosarcoma cell line (BA-HAN-1C) has been established, the phenotype of which is characterized by the coexistence of undifferentiated mononuclear cells and differentiated multinuclear myotube-like giant cells. The failure of attempts to separate these two cell types by repeated recloning procedures indicates their close histogenetic relationship and suggests that differentiation in this tumor proceeds in a similar manner to that in normal striated muscle where postmitotic myotubes arise from mononuclear myoblasts by fusion. The morphologically undifferentiated mononuclear tumor cells were shown to be actively proliferating and to incorporate thymidine methyl-3H(3H-TdR)…

Cell typePathologymedicine.medical_specialtyCellular differentiationCell DifferentiationNeoplasms ExperimentalBiologyCell cyclePeripheral blood mononuclear cellPathology and Forensic MedicineCell biologyRatsGiant cellCell cultureRhabdomyosarcomamedicineMitotic IndexTumor Cells CulturedAnimalsClonogenic assayFloxuridineMitosisCell DivisionVirchows Archiv. B, Cell pathology including molecular pathology
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Primary Neural Precursors and Intermitotic Nuclear Migration in the Ventricular Zone of Adult Canaries

1998

New neurons continue to be born in the ventricular zone (VZ) of the lateral ventricles in the brain of adult birds. On the basis of serial section reconstruction and electron microscopy, we determined that the VZ of the adult canary brain is composed of three main cell types (A, B, and E). Type A cells were never found in contact with the ventricle and had microtubule-rich processes typical of young migrating neurons. Type B cells were organized as a pseudostratified epithelium, all contacted the ventricle, and most had a characteristic single cilium. Type E cells, also in contact with ventricle, were ultrastructurally similar to the mammalian multiciliated ependymal cells. After six inject…

Cell typePathologymedicine.medical_specialtyEpendymal CellCanariesCell SurvivalMitosisCell CountBiologyTritiumArticleCerebral VentriclesLateral ventriclesCell MovementEpendymamedicineAnimalsCiliaB cellCell NucleusNeuronsGeneral NeuroscienceStem CellsAge FactorsMolecular biologyMicroscopy Electronmedicine.anatomical_structureVentricleCerebral ventricleFemaleStem cellEpendymaCell DivisionThymidine
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U937 variant cells as a model of apoptosis without cell disintegration

2012

AbstractThe variant cell line U937V was originally identified by a higher sensitivity to the cytocidal action of tumor necrosis factor alpha (TNFα) than that of its reference cell line, U937. We noticed that a typical morphological feature of dying U937V cells was the lack of cellular disintegration, which contrasts to the formation of apoptotic bodies seen with dying U937 cells. We found that both TNFα, which induces the extrinsic apoptotic pathway, and etoposide (VP-16), which induces the intrinsic apoptotic pathway, stimulated U937V cell death without cell disintegration. In spite of the distinct morphological differences between the U937 and U937V cells, the basic molecular events of ap…

Cell typeProgrammed cell deathBlotting WesternCellApoptosisU937 cellsDNA FragmentationBiologyModels BiologicalBiochemistrymedicineHumansCell ShapeMolecular BiologyU937 cellCytochrome cCytochromes chemic and immune systemsCell BiologyApoptotic bodyCaspase 9MitochondriaCell biologyEnzyme Activationmedicine.anatomical_structureApoptosisCell culturebiology.proteinApoptotic bodiesLymphoma Large B-Cell DiffuseCell disintegrationSignal TransductionResearch ArticleCellular and Molecular Biology Letters
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Timing of identity: spatiotemporal regulation of hunchback in neuroblast lineages of Drosophila by Seven-up and Prospero.

2006

Neural stem cells often generate different cell types in a fixed birth order as a result of temporal specification of the progenitors. In Drosophila, the first temporal identity of most neural stem cells(neuroblasts) in the embryonic ventral nerve cord is specified by the transient expression of the transcription factor Hunchback. When reaching the next temporal identity, this expression is switched off in the neuroblasts by seven up (svp) in a mitosis-dependent manner, but is maintained in their progeny (ganglion mother cells). We show that svpmRNA is already expressed in the neuroblasts before this division. After mitosis, Svp protein accumulates in both cells, but the downregulation of h…

Cell typeReceptors Steroidanimal structuresTranscription GeneticMitosisNerve Tissue ProteinsNeuroblastAnimalsDrosophila ProteinsCell LineageProgenitor cellMolecular BiologyMitosisGeneticsNeuronsbiologyStem CellsfungiGene Expression Regulation DevelopmentalNuclear ProteinsProsperobiology.organism_classificationEmbryonic stem cellNeural stem cellCell biologyDNA-Binding ProteinsDrosophila melanogasterGanglion mother cellDevelopmental BiologyTranscription FactorsDevelopment (Cambridge, England)
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Electron microscopic demonstration of intracelluar promethazine accumulation sites by a precipitation technique: application to the cerebellar cortex…

1996

A method is described that allows electron microscopic identification of the phenothiazine neuroleptic promethazine after supravital intracardiac injection of high drug concentrations (greater than or equal to 3 %). The cerebellar cortex of the mouse was used for the investigation. This procedure is based on simultaneous fixation of drug and tissue by immersion in a paraformaldehyde-glutaraldehyde solution with the addition of phosphomolybdic acid. The electron microscopic investigation revealed that the drug could easily be identified as an electron-dense precipitate. Subpopulations of neurons exhibited a higher affinity for the drug than others, but no preference for any nerve cell type …

Cell typeTissue FixationHistologyChromatographyChemistryEndoplasmic reticulumMitochondrionPromethazinePromethazineCerebellar CortexMiceMicroscopy Electronchemistry.chemical_compoundCytoplasmCerebellar cortexPhenothiazineUltrastructureBiophysicsmedicineAnimalsAnatomymedicine.drugJournal of Histochemistry & Cytochemistry
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Subcellular targeting of multiligand-binding protein gC1qR.

1999

Abstract gC1q receptor, a protein originally described as the cell surface receptor for the globular heads of complement factor C1q, has been found to bind human H-kininogen with high affinity and specificity. Therefore, gC1qR has been considered candidate kininogen docking site on the surfaces of platelets, neutrophils and endothelial cells. Recent work demonstrating that gC1qR is an intracellular protein that is tightly associated with mitochondria rather than targeted to the cell surface has challenged this view. To further probe cellular trafficking routes of gC1qR, we overexpressed human gC1qR in a mammalian cell and monitored cell surface exposure of recombinant gC1qR by virtue of its…

CellComplement factor IBiologyLigandsMitochondrial ProteinsCell surface receptormedicineAnimalsHumansBinding siteReceptorPharmacologyBinding SitesMembrane GlycoproteinsBinding proteinComplement C1qBiological TransportTransfectionMolecular biologyCell biologyReceptors Complementmedicine.anatomical_structureHyaluronan ReceptorsCell cultureCOS CellsCarrier ProteinsProtein Processing Post-Translationalcirculatory and respiratory physiologySubcellular FractionsImmunopharmacology
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Role of humanin, a mitochondrial-derived peptide, in cardiovascular disorders

2020

The mitochondria produce specific peptides-mitochondrial-derived peptides-that mediate the transcriptional stress response by their translocation into the nucleus and interaction with deoxyribonucleic acid. Mitochondrial-derived peptides are regulators of metabolism. This class of peptides comprises humanin, mitochondrial open reading frame of the 12S ribosomal ribonucleic acid type c (MOTS-c) and small humanin-like peptides (SHLPs). Humanin inhibits mitochondrial complex 1 activity and limits the level of oxidative stress in the cell. Data show that mitochondrial-derived peptides have a role in improving metabolic diseases, such as type 2 diabetes. Perhaps humanin can be used as a marker f…

CellPeptide030204 cardiovascular system & hematologyMitochondrionmedicine.disease_causeCardiovascular System03 medical and health sciences0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemAnimalsHumansMedicine030212 general & internal medicineEndothelial dysfunctionComputingMilieux_MISCELLANEOUSHumaninchemistry.chemical_classificationbusiness.industryIntracellular Signaling Peptides and ProteinsGeneral Medicinemedicine.diseaseMitochondriaUp-RegulationCell biologyOxidative StressOpen reading framemedicine.anatomical_structurechemistryCardiovascular DiseasesInflammation MediatorsCardiology and Cardiovascular MedicinebusinessFunction (biology)Oxidative stressSignal TransductionArchives of Cardiovascular Diseases
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Cadmium and mitochondria

2009

The heavy metal cadmium (Cd) a pollutant associated with several modern industrial processes, is absorbed in significant quantities from cigarette smoke, water, food and air contaminations. It is known to have numerous undesirable effects on health in both experimental animals and humans, targeting kidney, liver and vascular system. The molecular mechanism accounting for most of the biological effects of Cd are not well-understood and the toxicity targets are largely unidentified. The present review focuses on important recent advances about the effects of cadmium on mitochondria of mammalian cells. Mitochondria are the proverbial powerhouses of the cell, running the fundamental biochemical…

Cellchemistry.chemical_elementMitochondrionBiologyModels BiologicalmedicineAnimalsHumansSettore BIO/06 - Anatomia Comparata E CitologiaCytotoxicityMolecular BiologyMembrane potentialMammalsPollutantCadmiumMitochondrial gene expressionApoptosiROSCell BiologyMitochondriamedicine.anatomical_structurechemistryBiochemistryApoptosisToxicityMolecular MedicineEnergy MetabolismIntracellularInner membrane ion permeabilityCadmiumMitochondrion
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