Search results for "mmunopathology"

showing 10 items of 156 documents

Does dietary salt induce autoimmunity?

2013

Two recent publications suggest that dietary salt may polarize TH17 cells and therefore increase the risk of developing autoimmune disease. Where low salt diets can readily be tested for their therapeutic effects in autoimmune disease, more work is needed to connect dietary salts with the development of immunopathology.

610 Medicine & healthBiology10263 Institute of Experimental Immunologymedicine.disease_causeImmediate early proteinAutoimmunity1307 Cell Biology03 medical and health sciences0302 clinical medicineLow-salt dietsImmunopathology1312 Molecular BiologymedicineMolecular Biology030304 developmental biologyAutoimmune disease0303 health sciencesCell Biologymedicine.diseaseResearch Highlight3. Good healthImmunology570 Life sciences; biologyInterleukin 17030215 immunologyDietary saltCell research
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Celiac disease and selective immunoglobulin A deficiency

1997

Selective IgA deficiency was observed in 12 of 688 (1.7%) patients with celiac disease who were clinically undistinguishable from patients with celiac disease with normal IgA levels. This high prevalence of IgA deficiency in patients with celiac disease makes serum IgA assay advisable when screening for celiac disease is performed by measurement of antigliadin antibodies or anti-IgA endomysium antibodies. Similarly, subjects with IgA deficiency should be considered at risk of celiac disease.

AdolescentGlutensCross-sectional studyMuscle Fibers SkeletalDiseaseSelective IgA deficiencyImmunoglobulin EGliadinCoeliac diseaseMyofibrilsRisk FactorsImmunopathologyConfidence IntervalsDiet Protein-RestrictedPrevalencemedicineHumansChildChi-Square Distributionbiologybusiness.industryAge FactorsIgA DeficiencyInfantnutritional and metabolic diseasesmedicine.diseaseEndomysiumdigestive system diseasesImmunoglobulin ACeliac DiseaseIntestinal DiseasesCross-Sectional Studiesmedicine.anatomical_structureImmunoglobulin MChild PreschoolImmunoglobulin GPediatrics Perinatology and Child HealthImmunologybiology.proteinAntibodybusinessFollow-Up StudiesThe Journal of Pediatrics
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Lack of requirement for CD8+ cells in recovery from and resistance to experimental autoimmune encephalomyelitis.

1995

Abstract Experimental autoimmune encephalomyelitis (EAE) is a model of T-cell mediated autoimmune disease. Active disease is mediated by myelin basic protein specific CD4+T-cells, whose adoptive transfer can also induce passive disease. In the Lewis rat EAE is a transient disease inducing lasting resistance to rechallenge. The mechanisms of recovery and resistance are poorly understood. CD8+suppressor T-cells have mostly been thought to be central, especially in resistance to reinduction of the disease. In this study we showed by complete depletion of CD8+cells that this subset does not influence either recovery or resistance to EAE in the Lewis rat. This was further confirmed by depleting …

Adoptive cell transferEncephalomyelitis Autoimmune Experimentalmedicine.drug_classEncephalomyelitisImmunologyCD4-CD8 RatioCD8-Positive T-LymphocytesMonoclonal antibodyLymphocyte DepletionImmunopathologymedicineImmunology and AllergyAnimalsAutoimmune diseasebiologyExperimental autoimmune encephalomyelitisAntibodies Monoclonalmedicine.diseaseImmunity InnateMyelin basic proteinRatsRats Inbred LewImmunologybiology.proteinFemaleCD8Journal of autoimmunity
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Humoral immunity and retrobulbar fibroblasts in endocrine ophthalmopathy

1992

The exact role of retrobulbar fibroblasts in the immunopathogenesis of endocrine ophthalmopathy still remains to be elucidated. To evaluate the in vitro influence of humoral immunity on retrobulbar fibroblasts, the effects of immunoglobulin G as well as of the sera of 50 euthyroid patients with endocrine ophthalmopathy and 30 controls on both porcine and human (patients' and controls') retrobulbar fibroblasts were measured by means of several assays: a colorimetric test involving a heterocyclic chemical, a tetrazolium bromide, was applied to quantify the activity of mitochondrial dehydrogenases; the incorporation of 3H-thymidine was determined as a sensitive parameter for cell proliferation…

AdultDNA Replicationmedicine.medical_specialtygenetic structuresEye DiseasesEndocrinology Diabetes and MetabolismEndocrine System DiseasesImmunoglobulin GEndocrinologyInternal medicineImmunopathologymedicineHumansFibroblastAgedAutoimmune diseasebiologybusiness.industryGeneral MedicineFibroblastsMiddle Agedmedicine.diseaseeye diseasesIn vitroPathophysiologymedicine.anatomical_structureEndocrinologyConnective TissueImmunoglobulin GImmunologyHumoral immunityAntibody Formationbiology.proteinsense organsAntibodybusinessEnergy MetabolismOrbit
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Chronic Intestinal Infection due to Subgenus F Type 40 Adenovirus in a Patient with AIDS

1997

A case of chronic intestinal infection due to adenovirus type 40 lasting for 13 months in a patient with AIDS is described. Adenovirus particles were detected by electron microscopy in biopsy samples taken from the duodenum 3 months after the onset of diarrhoea. The virus was identified as adenovirus type 40 in stool samples by ELISA monoclonal antibodies to adenovirus group antigen (MAd-g2) and types 40 and 41 (MA 40-1 and MA 41-1). No other enteropathogens were found. These data support a causal relationship between adenovirus 40 and the gastrointestinal symptoms of the patient. This is the first reported case of intestinal infection caused by adenovirus type 40 in a patient with AIDS.

AdultDiarrheaMaleMicrobiology (medical)DuodenumOpportunistic infectionvirusesBiologymedicine.disease_causeVirusAdenovirus Infections HumanFecesAntigenImmunopathologyBiopsymedicineHumansDuodenal DiseasesIntestinal MucosaAIDS-Related Opportunistic InfectionsGeneral Immunology and Microbiologymedicine.diagnostic_testAdenoviruses HumanGeneral Medicinemedicine.diseaseVirologyAdenoviridaeIntestinal DiseasesMicroscopy ElectronDiarrheaInfectious DiseasesChronic DiseaseImmunologyViral diseasemedicine.symptomScandinavian Journal of Infectious Diseases
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Localized crusted scabies in the acquired immunodeficiency syndrome

1992

Summary Crusted scabies (CrS) is an uncommon occurrence among patients with AIDS. Indeed to date only five cases have been described, all with widespread lesions. A case of localized CrS appearing as a yellowish and crusted plaque on the second right toe is reported in a woman with AIDS. Scraping off the verrucous surface, as well as punch-biopsy revealed many mites within the horny layer. The infestation is related to the cutaneous immune response and thus CrS should be considered an opportunistic infestation in AIDS. The importance of the early diagnosis of CrS in order to prevent disseminated lesions and involvement of other health-care workers is underlined.

AdultFoot DermatosesPathologymedicine.medical_specialtyAIDS-Related Opportunistic Infectionsintegumentary systembusiness.industryHorny layerDermatologyCrusted scabiesToesmedicine.diseaseScabiesAcquired immunodeficiency syndrome (AIDS)Immunopathologyparasitic diseasesotorhinolaryngologic diseasesmedicineScabiesHumansFemaleViral diseasebusinesssaRNASkinClinical and Experimental Dermatology
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Cytokine genotyping (TNF and IL-10) in patients with celiac disease and selective IgA deficiency

2003

Selective IgA deficiency (IgAD) and celiac disease (CD) are frequently associated and share the ancestral haplotype human leukocyte antigen (HLA)-8.1, which is characterized by a peculiar cytokine profile. The aim of this study was to evaluate the role of tumor necrosis factor (TNF) and interleukin (IL)-10 alleles in CD and CD-IgAD.The distribution of some biallelic polymorphisms of both cytokine promoters (-308G--A and -863C--A at TNF promoter sequence and -1082G--A, -819C--A, and -592C--T at IL-10 promoter) were typed using biotilinated specific probes in 32 celiac patients, in 34 CD-IgAD patients, and in 96 healthy controls.In CD and CD-IgAD, the -308A allele was significantly more frequ…

AdultGenotypemedicine.medical_treatmentDiseaseSelective IgA deficiencyCoeliac diseaseGene FrequencyImmunopathologyHumansMedicineChildPromoter Regions GeneticGenotypingPolymorphism GeneticHepatologyTumor Necrosis Factor-alphabusiness.industryIgA DeficiencyGastroenterologynutritional and metabolic diseasesmedicine.diseasedigestive system diseasesInterleukin-10Celiac DiseaseInterleukin 10Cross-Sectional StudiesCytokineImmunologyCytokinesTumor necrosis factor alphabusinessSequence AnalysisThe American Journal of Gastroenterology
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Monosodium benzoate hypersensitivity in subjects with persistent rhinitis

2004

Background:  Very few data are available from the literature on whether nonatopic subjects affected by persistent rhinitis may show the appearance of objective symptoms of rhinitis after the ingestion of food additives such as tartrazine (E102), erythrosine (E127), monosodium benzoate (E211), p-hydroxybenzoate (E218), sodium metabisulphite (E223), and monosodium glutamate (E620). It is still unclear whether the ingestion of food additive may cause, as well, a consensual reduction of nasal peak inspiratory flow (NPIFR). Therefore, we used a double-blind placebo-controlled (DBPC) study to evaluate this hypothesis. Patients and methods:  Two hundred and twenty-six consecutive patients (76 male…

AdultHypersensitivity ImmediateMaleAllergymedicine.medical_specialtySettore MED/09 - Medicina InternaAdolescentMonosodium glutamateImmunologyGastroenterologychemistry.chemical_compoundVasomotor RhinitisInternal medicineImmunopathologySodium BenzoatemedicineImmunology and AllergyIngestionHumansMonosodium benzoate; hypersensitivity; persistent rhinitisChildRhinitisbusiness.industryMonosodium benzoatedouble-blind placebo-controlledMiddle Agednasal peak inspiratory flowmedicine.diseasefood additivesRegimenchemistryImmunologyChronic DiseaseEtiologyFood PreservativesFemalehypersensitivityNasal CavitybusinessPulmonary Ventilationpersistent rhinitisTartrazine
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Metastatic Anterior Chamber Non-Hodgkin Lymphoma in a Patient With Acquired Immunodeficiency Syndrome

1997

Purpose To alert ophthalmologists to the possibility of metastatic anterior chamber intraocular non-Hodgkin lymphoma in patients with acquired immunodeficiency syndrome (AIDS). Methods We examined a 19-year-old man with AIDS who had an anterior uveitis with pseudohypopyon and a history of 2 months of malaise. A specimen of the lesion was obtained with a 25-gauge needle for pathologic examination. RESULTS: Systemic exploration showed hepatomegaly. Abdominal computed tomography showed multiple lesions in the liver and spleen. Fine-needle aspiration disclosed a high-grade non-Hodgkin lymphoma. The result of a fine-needle aspiration obtained from a lesion in the anterior chamber was consistent …

AdultMaleAcquired Immunodeficiency SyndromePathologymedicine.medical_specialtyAnterior Chamberbusiness.industryEye NeoplasmsLymphoma Non-HodgkinEye diseaseBiopsy Needlemedicine.diseaseLymphomaMetastasisMalaiseLesionOphthalmologyImmunopathologymedicineHumansmedicine.symptombusinessComplicationUveitisAmerican Journal of Ophthalmology
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Increased prevalence of autoimmune diseases in first-degree relatives of patients with celiac disease.

2003

The prevalence of autoimmune disorders is increased in patients with celiac disease (CD), and it is unknown whether their first-degree relatives also have a high risk of autoimmune disorders.To assess the prevalence of autoimmune diseases in first-degree relatives of CD patients, the authors looked for autoimmune disorders in 225 first-degree relatives of 66 children with CD (group A) and in 232 first-degree relatives of 68 healthy children (group B). For both groups, serologic screening for CD was performed through antiendomysium (EMA) and tissue transglutaminase autoantibodies (tTGAA). EMA- and tTGAA-positive subjects were offered an intestinal biopsy. The age at onset of autoimmune disea…

AdultMaleAdolescentBiopsyDiseaseGenetic determinismCoeliac diseaseAutoimmune DiseasesImmunopathologyOdds RatioMedicineHumansFamilyFirst-degree relativesRisk factorIntestinal MucosaChildAutoimmune diseaseHyperplasiabusiness.industryGastroenterologyInfantmedicine.diseaseIntestinesCeliac DiseaseEl NiñoChild PreschoolPediatrics Perinatology and Child HealthImmunologyFemaleAtrophybusinessJournal of pediatric gastroenterology and nutrition
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