Search results for "neuron"

showing 10 items of 2611 documents

''Deferoxamine blocks death induced by glutathione depletion in PC 12 cells''

2013

Chouraqui, E. | Leon, A. | Repesse, Y. | Prigent-Tessier, A. | Bouhallab, S. | Bougle, D. | Marie, C. | Duval, D.; International audience; ''The purpose of the present work was to investigate the mechanisms by which glutathione depletion induced by treatment with buthionine sulfoximine (BSO) led within 24-30 h to PC 12 cells apoptosis. Our results showed that treatment by relatively low concentrations (10-30 mu M) of deferoxamine (DFx), a natural iron-specific chelator, almost completely shielded the cells from BSO-induced toxicity and that DFx still remained protective when added up to 9-12 h after BSO treatment. On the other hand, phosphopeptides derived from milk casein and known to carr…

Time FactorsIronApoptosisDeferoxaminePharmacologyIron Chelating AgentsToxicologymedicine.disease_causePC12 Cellschemistry.chemical_compoundOXIDATIVE-STRESSPARKINSONS-DISEASECaseinmedicineAnimalsHomeostasisButhionine sulfoximineButhionine SulfoximineNeuronsCELLULAR IRONDose-Response Relationship DrugbiologyChemistryGeneral NeuroscienceGlutathioneGlutathioneIRON CHELATORRatsDeferoxamineFerritinSYMPATHETIC NEURONSISCHEMIC-STROKEBiochemistryBRAIN IRONCELLULAR IRON''CytoprotectionApoptosisToxicity[ SCCO.NEUR ] Cognitive science/Neurosciencebiology.proteinSERUM DEPRIVATIONHEME OXYGENASE-1NEURODEGENERATIVE DISORDERSOxidative stress''OXIDATIVE-STRESSmedicine.drug
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Medullary respiratory-related neurons with axonal connections to rostral pons and their function in termination of inspiration.

1985

In urethane-anaesthetized, paralyzed and artificially ventilated rabbits, medullary respiration-related neurons (RRU) were classified according to the phase relation of their burst discharge to phrenic nerve activity. Phase-bound inspiratory (I) or expiratory (E) neurons were discriminated from phase-spanning expiratory-inspiratory (EI) or inspiratory-expiratory (IE) units. Mechanisms of termination of inspiration by electrical stimulation of rostral pontine nuclei (Nc. parabrachialis medialis; Lc. coeruleus) were examined firstly to demonstrate whether RRU receive descending excitatory and inhibitory afferents as well as ascending efferents and secondly to analyse the time course of the ne…

Time FactorsPhysiologyClinical BiochemistryStimulationInhibitory postsynaptic potentialPhysiology (medical)PonsNeural PathwaysMedicineAnimalsNeurons AfferentPhrenic nerveNeuronsMedulla Oblongatabusiness.industryRespirationPontine nucleiPons VaroliiAnatomyPonsAxonsElectric Stimulationnervous systemMedulla oblongataExcitatory postsynaptic potentialRabbitsbusinessNeurosciencePflugers Archiv : European journal of physiology
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Multipotent Neural Stem Cells Reside into the Rostral Extension and Olfactory Bulb of Adult Rodents

2002

The lateral walls of the forebrain lateral ventricles are the richest source of stem cells in the adult mammalian brain. These stem cells give rise to new olfactory neurons that are renewed throughout life. The neurons originate in the subventricular zone (SVZ), migrate within the rostral extension (RE) of the SVZ along the rostral migratory stream (RMS) within tube-like structures formed of glial cells, to eventually reach the olfactory bulb (OB). We demonstrate that, contrary to the current view, multipotential (neuronal-astroglial-oligodendroglial) precursors with stem cell features can be isolated not only from the SVZ but also from the entire RE, including the distal portion within the…

Time FactorsRostral migratory streamanimal diseasesCell Culture TechniquesSubventricular zoneCell SeparationBiologyCell LineMiceCell MovementLateral VentriclesSpheroids CellularNeurospheremedicineAnimalsARTICLEGrowth SubstancesCells CulturedNeuronsNeurotransmitter AgentsStem CellsGeneral NeuroscienceNeurogenesisCell DifferentiationOlfactory BulbNeural stem cellClone CellsNeuroepithelial cellOligodendrogliaPhenotypemedicine.anatomical_structurenervous systemAstrocytesStem cellNeuroscienceCell DivisionAdult stem cellThe Journal of Neuroscience
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D2R striatopallidal neurons inhibit both locomotor and drug reward processes.

2009

The specific functions of dopamine D(2) receptor-positive (D(2)R) striatopallidal neurons remain poorly understood. Using a genetic mouse model, we found that ablation of D(2)R neurons in the entire striatum induced hyperlocomotion, whereas ablation in the ventral striatum increased amphetamine conditioned place preference. Thus D(2)R striatopallidal neurons limit both locomotion and, unexpectedly, drug reinforcement.

Time FactorsstriatumParkinson's diseaseStriatumNeurons -- drug effectsEnkephalins -- metabolism10263 Institute of Experimental ImmunologyMiceDopamine Uptake InhibitorsTyrosine 3-Monooxygenase -- geneticsCorpus Striatum -- cytologyDiphtheria ToxinGlutamate Decarboxylase -- metabolismstriatum; indirect opathway; A2A receptors; D2 receptors; locomotion; amphetamine addiction; Parkinson's diseaseNeuronsamphetamine addictionGlutamate DecarboxylaseGeneral NeuroscienceAmphetamine -- pharmacologyNeurodegeneration2800 General NeuroscienceEnkephalinsSciences bio-médicales et agricoleslocomotionmedicine.anatomical_structureA2A receptorsIntercellular Signaling Peptides and ProteinsReceptors Dopamine D2 -- metabolismPsychologyLocomotionmedicine.drugHeparin-binding EGF-like Growth FactorProtein BindingGlobus Pallidus -- cytologyReceptors Dopamine D2 -- deficiencyReinforcement ScheduleTyrosine 3-MonooxygenaseGlutamate Decarboxylase -- geneticsLocomotion -- geneticsIntercellular Signaling Peptides and Proteins -- genetics610 Medicine & healthMice TransgenicNerve Tissue ProteinsDiphtheria Toxin -- pharmacologyGlobus PallidusNeurons -- physiologyLocomotion -- drug effectsRewardDopamineDopamine receptor D2medicineNerve Tissue Proteins -- metabolismAnimalsGene Expression Regulation -- geneticsAmphetamineD2 receptorsReceptors Adenosine A2Receptors Dopamine D2indirect opathwayVentral striatumReceptors Adenosine A2 -- geneticsDopamine Uptake Inhibitors -- pharmacologymedicine.diseaseConditioned place preferenceCorpus StriatumMice Inbred C57BLGene Expression Regulation -- drug effectsAmphetaminenervous systemGene Expression RegulationProtein Binding -- drug effectsTyrosine 3-Monooxygenase -- metabolism570 Life sciences; biologyAutoradiographyConditioning OperantNeuronConditioning Operant -- physiologyNeuroscienceEnkephalins -- geneticsNature neuroscience
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Detection of Fibroblast Growth Factor Receptor 1 (FGFR1) Transactivation by Muscarinic Acetylcholine Receptors (mAChRs) in Primary Neuronal Hippocamp…

2018

In addition to their canonical intracellular signals involved in the regulation of neuronal plasticity, G-protein coupled receptors can also rapidly transactivate tyrosine kinase receptors and their downstream intracellular signaling in absence of specific ligands. Here we describe our protocol for dissociating and maintaining hippocampal primary neurons in high- and low-density culture, followed by a description of methods employed to evaluate neurite outgrowth and protein phosphorylation associated with fibroblast growth factor receptor 1 transactivation by muscarinic acetylcholine receptors. Our goal was to provide the reader with detailed protocols of the abovementioned techniques and t…

TransactivationChemistryFibroblast growth factor receptor 1Tyrosine kinase receptorHippocampal formationHippocampusSettore BIO/09 - FisiologiaFibroblast growth factor receptorWestern blottingCell biologyMuscarinic acetylcholine receptorPrimary neuronal cultureTransactivationNeurite growthMuscarinic acetylcholine receptorPhosphorylationReceptor–receptor interactions
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Nuclear aggregation of olfactory receptor genes governs their monogenic expression.

2012

SummaryGene positioning and regulation of nuclear architecture are thought to influence gene expression. Here, we show that, in mouse olfactory neurons, silent olfactory receptor (OR) genes from different chromosomes converge in a small number of heterochromatic foci. These foci are OR exclusive and form in a cell-type-specific and differentiation-dependent manner. The aggregation of OR genes is developmentally synchronous with the downregulation of lamin b receptor (LBR) and can be reversed by ectopic expression of LBR in mature olfactory neurons. LBR-induced reorganization of nuclear architecture and disruption of OR aggregates perturbs the singularity of OR transcription and disrupts the…

Transcription GeneticCytoplasmic and NuclearChromosomal Proteins Non-HistoneDown-RegulationReceptors Cytoplasmic and NuclearLamin B receptorBiologyReceptors OdorantMedical and Health SciencesGeneral Biochemistry Genetics and Molecular BiologyFluorescenceOlfactory Receptor NeuronsArticle03 medical and health sciencesMice0302 clinical medicineDownregulation and upregulationGeneticTranscription (biology)HeterochromatinGene expressionReceptorsmedicineGeneticsAnimalsGeneIn Situ HybridizationIn Situ Hybridization Fluorescence030304 developmental biologyRegulation of gene expressionCell Nucleus0303 health sciencesOlfactory receptorBiochemistry Genetics and Molecular Biology(all)Neurosciencesta1182Non-HistoneBiological SciencesCell biologyChromosomal Proteinsmedicine.anatomical_structureOdorantGene Expression RegulationEctopic expressionTranscription030217 neurology & neurosurgeryDevelopmental BiologyCell
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Rapid nucleus-scale reorganization of chromatin in neurons enables transcriptional adaptation for memory consolidation

2020

AbstractThe interphase nucleus is functionally organized in active and repressed territories defining the transcriptional status of the cell. However, it remains poorly understood how the nuclear architecture of neurons adapts in response to behaviorally relevant stimuli that trigger fast alterations in gene expression patterns. Imaging of fluorescently tagged nucleosomes revealed that pharmacological manipulation of neuronal activity in vitro and auditory cued fear conditioning in vivo induce nucleus-scale restructuring of chromatin within minutes. Furthermore, the acquisition of auditory fear memory is impaired after infusion of a drug into auditory cortex which blocks chromatin reorganiz…

Transcription GeneticPhysiologySensory PhysiologyGene ExpressionSocial SciencesMiceCognitionLearning and MemoryAnimal CellsBehavioral ConditioningMedicine and Health SciencesPsychologyPremovement neuronal activityFear conditioningNeuronsMultidisciplinaryChromosome BiologyQRBrainAnimal ModelsAdaptation PhysiologicalChromatinSensory SystemsChromatinIn Vivo ImagingHistonemedicine.anatomical_structureAuditory SystemExperimental Organism SystemsMedicineEpigeneticsMemory consolidationCellular TypesAnatomyResearch ArticleImaging TechniquesScienceMouse ModelsBiologyResearch and Analysis MethodsAuditory cortexModel OrganismsMemoryFluorescence ImagingGeneticsmedicineAnimalsNucleosomeMemory ConsolidationCell NucleusAuditory CortexBehaviorBiology and Life SciencesCell BiologyCellular NeuroscienceAnimal Studiesbiology.proteinCognitive ScienceFear ConditioningNeuroscienceNucleusNeuroscience
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mTOR Driven Gene Transcription Is Required for Cholesterol Production in Neurons of the Developing Cerebral Cortex

2021

AbstractDysregulated mammalian target of rapamycin (mTOR) activity is associated with various neurodevelopmental disorders ranging from idiopathic autism spectrum disorders to syndromes caused by single gene defects. This suggests that maintaining mTOR activity levels in a physiological range is essential for brain development and functioning. Upon activation, mTOR regulates a variety of cellular processes such as cell growth, autophagy and metabolism. On a molecular level, however, the consequences of mTOR activation in the brain are not well understood.Low levels of cholesterol are associated with a wide variety of neurodevelopmental disorders. We here describe numerous genes of the stero…

Transcription GeneticQH301-705.5Primary Cell CulturemTORC1Mechanistic Target of Rapamycin Complex 1BiologySREBPCatalysisArticleInorganic ChemistryMiceAutophagyTranscriptional regulationmedicineAnimalsPhysical and Theoretical ChemistryBiology (General)Molecular BiologyTranscription factorQD1-999mTORC1SpectroscopyPI3K/AKT/mTOR pathwayCerebral CortexNeuronsSterol Regulatory Element Binding ProteinsCell growthTOR Serine-Threonine KinasesOrganic Chemistrycholesterol ; NF-Y ; neurogenesis ; mTOR ; mTORC1 ; SP1 ; SREBPAutophagyGene Expression Regulation DevelopmentalcholesterolGeneral MedicineComputer Science ApplicationsSterol regulatory element-binding proteinCell biologySP1Chemistryneurogenesismedicine.anatomical_structureCCAAT-Binding FactorCerebral cortexmTORNF-YProtein KinasesSignal TransductionInternational Journal of Molecular Sciences
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Transcriptional Mechanisms of Proneural Factors and REST in Regulating Neuronal Reprogramming of Astrocytes

2015

Summary Direct lineage reprogramming induces dramatic shifts in cellular identity, employing poorly understood mechanisms. Recently, we demonstrated that expression of Neurog2 or Ascl1 in postnatal mouse astrocytes generates glutamatergic or GABAergic neurons. Here, we take advantage of this model to study dynamics of neuronal cell fate acquisition at the transcriptional level. We found that Neurog2 and Ascl1 rapidly elicited distinct neurogenic programs with only a small subset of shared target genes. Within this subset, only NeuroD4 could by itself induce neuronal reprogramming in both mouse and human astrocytes, while co-expression with Insm1 was required for glutamatergic maturation. Cu…

Transcription GeneticRepressorNerve Tissue ProteinsCell fate determinationBiologyDNA-binding proteinArticleMiceGlutamatergicBasic Helix-Loop-Helix Transcription FactorsGeneticsAnimalsHumansPromoter Regions GeneticTranscription factorCells CulturedNeuronsCell BiologyCellular ReprogrammingMolecular biologyCell biologyDNA-Binding ProteinsRepressor ProteinsASCL1Astrocytesembryonic structuresMolecular MedicineGABAergicReprogrammingTranscription FactorsCell Stem Cell
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Sea urchin neural alpha 2 tubulin gene: isolation and promoter analysis

2004

Abstract Expression of Tα2 gene, during sea urchin Paracentrotus lividus development, is spatially and temporally regulated. In order to characterize this gene, we isolated the relevant genomic sequences and scanned the isolated 5 ′ -flanking region in searching for cis -regulatory elements required for proper expression. Gel mobility shift and footprinting assays, as well as reporter gene (CAT and β-gal) expression assays, were used to address cis -regulatory elements involved in regulation. Here we report that an upstream 5 ′ -flanking fragment of PlTα2 gene drives temporal expression of reporter genes congruent with that of endogenous Tα2 gene. The fragment contains cis -elements able to…

Transcriptional ActivationMolecular Sequence DataResponse elementBiophysicsPair-rule geneSettore BIO/11 - Biologia MolecolareBiochemistryParacentrotus lividusTubulinConsensus sequenceAnimalsCloning MolecularPromoter Regions GeneticMolecular BiologyGeneTranscription factorNeuronsGeneticsReporter geneBase SequencebiologyCell Biologybiology.organism_classificationGene ComponentsGenesSea UrchinsTubulin genes Neurogenesis Paracentrotus lividus Promoter Ectopic expressionEctopic expressionTranscription Initiation Site
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