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showing 10 items of 4356 documents

Lack of Hypothalamus Polysialylation Inducibility Correlates With Maladaptive Eating Behaviors and Predisposition to Obesity

2018

This original research article (6 p.) is part of the research topic . Specialty section: This article was submitted to Neuroenergetics, Nutrition and Brain Health, a section of the journal Frontiers in Nutrition.; International audience; High variability exists in individual susceptibility to develop overweight in an obesogenic environment and the biological underpinnings of this heterogeneity are poorly understood. In this brief report, we show in mice that the vulnerability to diet-induced obesity is associated with low level of polysialic acid-neural cell adhesion molecule (PSA-NCAM), a factor of neural plasticity, in the hypothalamus. As we previously shown that reduction of hypothalami…

0301 basic medicineobesityfood intakePSA-NCAMEndocrinology Diabetes and Metabolismmedia_common.quotation_subject[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiologybrainmaladaptive eating behaviorlcsh:TX341-641BiologyOverweightEnergy homeostasis03 medical and health sciencesNeuroplasticitymedicineFood and Nutritionhypothalamusmedia_commonNutritionOriginal ResearchNutrition and Dieteticssynaptic plasticitycomportement alimentairepolysialylation[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyNeurosciencesAppetitemedicine.diseaseprise alimentaireObesity3. Good health[SDV] Life Sciences [q-bio]food intake;obesity;maladaptive eating behavior;synaptic plasticity;PSA-NCAM;polysialylation;brain;hypothalamusEating disordersobésité030104 developmental biologynervous systemHypothalamusNeurons and CognitionSynaptic plasticityAlimentation et Nutritionplasticité synaptiquecerveaumedicine.symptomlcsh:Nutrition. Foods and food supplyNeuroscienceFood ScienceFrontiers in Nutrition
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Betaine and Choline Improve Lipid Homeostasis in Obesity by Participation in Mitochondrial Oxidative Demethylation

2018

We investigated the metabolic effects of betaine (Bet) supplementation on CTP:phosphoethanolamine cytidylyltransferase/Pcyt2 heterozygous mice (HET). HET received either no treatment or were allowed access to 1% Bet supplemented water for 8 weeks. As we previously showed with choline (Cho), Bet improved hypertriglyceridemia, and hepatic steatosis in HET. The protection from obesity associated with reduced hepatic steatosis and increased lipid breakdown in adipocytes was attributed to increased energy requirements for metabolism and elimination of supplemented Bet and Cho. 1H-NMR-based profiling revealed metabolic changes caused by Bet and Cho supplementation. Cho increased the citric acid c…

0301 basic medicineobesitymedicine.medical_specialtyTaurineEndocrinology Diabetes and Metabolismlcsh:TX341-641chemical and pharmacologic phenomena7. Clean energy03 medical and health scienceschemistry.chemical_compoundBetainecholineValineInternal medicinemedicineLipolysisbetainemouse modelsNutritionOriginal ResearchNutrition and Dietetics030102 biochemistry & molecular biologyChemistryCatabolismhemic and immune systemsMetabolismmedicine.diseasemethyl donors3. Good healthCitric acid cycle030104 developmental biologyEndocrinologySteatosislcsh:Nutrition. Foods and food supplyFood ScienceFrontiers in Nutrition
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HSP60 activity on human bronchial epithelial cells

2017

HSP60 has been implicated in chronic inflammatory disease pathogenesis, including chronic obstructive pulmonary disease (COPD), but the mechanisms by which this chaperonin would act are poorly understood. A number of studies suggest a role for extracellular HSP60, since it can be secreted from cells and bind Toll-like receptors; however, the effects of this stimulation have never been extensively studied. We investigated the effects (pro- or anti-inflammatory) of HSP60 in human bronchial epithelial cells (16-HBE) alone and in comparison with oxidative, inflammatory, or bacterial challenges. 16-HBE cells were cultured for 1–4 h in the absence or presence of HSP60, H2O2, lipopolysaccharide (…

0301 basic medicinep38αSettore BIO/17 - IstologiaLipopolysaccharidep38 mitogen-activated protein kinasesImmunologyStimulationBronchip38 Mitogen-Activated Protein KinasesERK1Cell LinePathogenesisMitochondrial Proteins03 medical and health scienceschemistry.chemical_compound0302 clinical medicineOriginal Research ArticlesHumansImmunology and AllergyCOPDInterleukin 8Protein kinase AReceptor16-HBE; COPD; CREB1; ERK1; HSP60; IL-10; IL-8; JNK1; MyD88; NF-κB p65 subunit; TLR-4; p38αPharmacologyIL-8Settore BIO/16 - Anatomia UmanaInterleukin-8JNK1NF-κB p65 subunitEpithelial CellsTLR-4Chaperonin 60MyD88Interleukin-1016-HBEToll-Like Receptor 416-HBE; COPD; CREB1; ERK1; HSP60; IL-10; IL-8; JNK1; MyD88; NF-κB p65 subunit; p38α; TLR-4; Immunology and Allergy; Immunology; PharmacologyInterleukin 10030104 developmental biologychemistry030220 oncology & carcinogenesisIL-10Cancer researchCREB1NF-κB p65 subunitHSP60p38α
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Anti-proliferative and apoptosis-inducing effect of theabrownin against non-small cell lung adenocarcinoma A549 cells

2016

With the highest cancer incidence rate, lung cancer, especially non-small cell lung cancer (NSCLC), is the leading cause of cancer death in the world. Tea (leaves of Camellia sinensis) has been widely used as a traditional beverage beneficial to human health, including anti-NSCLC activity. Theabrownin (TB) is one major kind of tea pigment responsible for the beneficial effects of tea liquor. However, its effect on NSCLC is unknown. The aim of the present study was to evaluate anti-proliferative and apoptosis-inducing effect of TB on NSCLC (A549) cells, using MTT assay, morphological observation (DAPI staining), in situ terminal deoxynucleotidyl transferase dUTP nick end labeling (TUNEL) ass…

0301 basic medicinep53ApoptosisBiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineA549 cellsPharmacology (medical)MTT assayDAPIOriginal ResearchPharmacologyA549 cellTUNEL assayCell growthTheabrowninlcsh:RM1-950Molecular biologylung cancer030104 developmental biologyReal-time polymerase chain reactionlcsh:Therapeutics. PharmacologyTerminal deoxynucleotidyl transferasechemistryApoptosis030220 oncology & carcinogenesisA549 cells;Frontiers in Pharmacology
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Multifaceted Mechanisms of WY-14643 to Stabilize the Blood-Brain Barrier in a Model of Traumatic Brain Injury

2017

The blood-brain barrier (BBB) is damaged during ischemic insults such as traumatic brain injury or stroke. This contributes to vasogenic edema formation and deteriorate disease outcomes. Enormous efforts are pursued to understand underlying mechanisms of ischemic insults and develop novel therapeutic strategies. In the present study the effects of PPARα agonist WY-14643 were investigated to prevent BBB breakdown and reduce edema formation. WY-14643 inhibited barrier damage in a mouse BBB in vitro model of traumatic brain injury based on oxygen/glucose deprivation in a concentration dependent manner. This was linked to changes of the localization of tight junction proteins. Furthermore, WY-1…

0301 basic medicinepirinixic acidTraumatic brain injuryp38 mitogen-activated protein kinasesIschemiaischemiaPharmacologyBlood–brain barrierPPARαlcsh:RC321-57103 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineDownregulation and upregulationmedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMolecular BiologyOriginal ResearchTight junctionbusiness.industryKinasetraumatic brain injuryblood-brain barriermedicine.diseasestroke030104 developmental biologymedicine.anatomical_structureKnockout mousebusinessNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Molecular Neuroscience
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Oxidation-Induced Increase In Photoreactivity of Bovine Retinal Lipid Extract

2017

Open access original paper Source : 10th EPR Workshop on Applications of EPR in Biology and Medicine, 2016 in Krakow, POLAND.; International audience; The mammalian retina contains a high level of polyunsaturated fatty acids, including docosahexaenoic acid (22:6) (DHA), which are highly susceptible to oxidation. It has been shown that one of the products of DHA oxidation-carboxyethylpyrrole (CEP), generated in situ, causes modifications of retinal proteins and induces inflammation response in the outer retina. These contributing factors may play a role in the development of age-related macular degeneration (AMD). It is also possible that some of the lipid oxidation products are photoreactiv…

0301 basic medicineretinaépithéliumLightOrganes des sensalpha-TocopherolBiochemistrysinglet oxygenchemistry.chemical_compoundspectrométrie de masseChromatography High Pressure Liquidrod outer segmentschemistry.chemical_classificationLiposomePhotorSuperoxideSinglet oxygenvitamine ephotorGeneral MedicineLipidsvertebrate retinaBiochemistryDocosahexaenoic acidAlimentation et Nutritionoxidized phospholipidsfatty-acid compositionRetina;Lipids;Polyunsaturated fatty acids;Oxidation;Photor;ROD OUTER SEGMENTS;GLYCATION END-PRODUCTS;SINGLET OXYGEN;PIGMENT EPITHELIUM;VITAMIN-E;MACULAR DEGENERATION;OXIDIZED PHOSPHOLIPIDS;MASS-SPECTROMETRY;VERTEBRATE RETINAOxidation-Reductionpolyunsaturated fatty acidsphospholipideSpectrometry Mass Electrospray IonizationChromatography Gasmacular degenerationoxidationrétineElectrospray ionizationvitamin-eSensory OrgansBiophysicsMédecine humaine et pathologieRetinalipids03 medical and health sciencesLipid oxidationZeaxanthinsacide gras polyinsaturé[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyOxidationAnimalsFood and NutritionlipideOriginal PaperReactive oxygen speciesChromatography030102 biochemistry & molecular biologyElectron Spin Resonance SpectroscopyoxydationRetinaldégénérescence maculaireCell Biologymass-spectrometryvertébrépigment epitheliumOxygen030104 developmental biologychemistryLiposomesglycation end-productsQuantum TheoryCattleSpin LabelsHuman health and pathologyPolyunsaturated fatty acidssense organs[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Effects of Infant Formula With Human Milk Oligosaccharides on Growth and Morbidity: A Randomized Multicenter Trial

2017

Objectives:The aim of the study was to evaluate the effects of infant formula supplemented with 2 human milk oligosaccharides (HMOs) on infant growth, tolerance, and morbidity. Methods:Healthy infants, 0 to 14 days old, were randomized to an intact-protein, cow's milk-based infant formula (control, n=87) or the same formula with 1.0g/L 2fucosyllactose (2FL) and 0.5g/L lacto-N-neotetraose (LNnT) (test, n=88) from enrollment to 6 months; all infants received standard follow-up formula without HMOs from 6 to 12 months. Primary endpoint was weight gain through 4 months. Secondary endpoints included additional anthropometric measures, gastrointestinal tolerance, behavioral patterns, and morbidit…

0301 basic medicinesafetyMalePediatricsmedicine.medical_specialtyMEDLINEOligosaccharidesWeight Gainlaw.inventionbronchitis03 medical and health scienceschemistry.chemical_compound2'-FucosyllactoseRandomized controlled trialDouble-Blind Method2fucosyllactose; bronchitis; lacto-N-neotetraose; safety; tolerancelawMulticenter trialmedicineAnimalsHumansLacto-N-neotetraoseRespiratory Tract Infectionslacto-N-neotetraose2′fucosyllactose030109 nutrition & dieteticstoleranceMilk Humanbusiness.industryGastroenterologyOriginal Articles: NutritionInfant Newbornfood and beveragesInfantProtective Factorsmedicine.diseaseInfant Formula030104 developmental biologyMilkchemistryInfant formulaPediatrics Perinatology and Child Health2'fucosyllactose bronchitis lacto-N-neotetraose safety toleranceBronchitisFemalemedicine.symptombusinessWeight gainFollow-Up Studies
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PNPLA3 and TLL-1 Polymorphisms as Potential Predictors of Disease Severity in Patients With COVID-19

2021

Albeit the pathogenesis of COVID-19 remains unclear, host’s genetic polymorphisms in genes involved in infection and reinfection, inflammation, or immune stimulation could play a role in determining the course and outcome. We studied in the early phase of pandemic consecutive patients (N = 383) with SARS-CoV-2 infection, whose subsequent clinical course was classified as mild or severe, the latter being characterized by admission to intensive therapy unit or death. Five host gene polymorphisms (MERTK rs4374383, PNPLA3 rs738409, TLL-1 rs17047200, IFNL3 rs1297860, and INFL4 rs368234815) were assessed by using whole nucleic acids extracted from nasopharyngeal swabs. Specific protease cleavage …

0301 basic medicineseverity of diseaseQH301-705.5InflammationPathogenesisCell and Developmental Biology03 medical and health sciences0302 clinical medicineDownregulation and upregulationGenotypemedicinegenetic polymorphismBiology (General)GeneOriginal Researchbusiness.industryConfoundingCOVID-19InflammasomeCell BiologyMERTK030104 developmental biology030220 oncology & carcinogenesisImmunologymedicine.symptombusinessPNPLA3 I148MTLL-1Developmental Biologymedicine.drugFrontiers in Cell and Developmental Biology
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Repeated, Intermittent Social Defeat across the Entire Juvenile Period Resulted in Behavioral, Physiological, Hormonal, Immunological, and Neurochemi…

2016

The developing brain is vulnerable to social defeat during the juvenile period. As complements of human studies, animal models of social defeat provide a straightforward approach to investigating the functional and neurobiological consequences of social defeats. Taking advantage of agonist behavior and social defeat in male golden hamster, a set of 6 experiments was conducted to investigate the consequences at multiple levels in young adulthood resulting from repeated, intermittent social defeats or “social threats” across the entire juvenile period. Male hamsters at postnatal day 28 (P28) were randomly assigned to either the social defeat, “social threat”, or arena control group, and they …

0301 basic medicinesocial threatCognitive NeuroscienceHippocampusContext (language use)cortisolpro-inflammatory cytokinesrepeated intermittent social defeatDevelopmental psychologySocial defeat03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineNeurochemicalmonoamine neurotransmittersJuvenileYoung adultOriginal Researchmale golden hamstersSocial relationadolescent bullying030104 developmental biologyjuvenileNeuropsychology and Physiological PsychologyPsychology030217 neurology & neurosurgeryGolden hamsterNeuroscienceFrontiers in Behavioral Neuroscience
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Structural and functional insights into lysostaphin–substrate interaction

2018

Lysostaphin from Staphylococcus simulans and its family enzymes rapidly acquire prominence as the next generation agents in treatment of S. aureus infections. The specificity of lysostaphin is promoted by its C-terminal cell wall targeting domain selectivity towards pentaglycine bridges in S. aureus cell wall. Scission of these cross-links is carried out by its N-terminal catalytic domain, a zinc-dependent endopeptidase. Understanding the determinants affecting the efficiency of catalysis and strength and specificity of interactions lies at the heart of all lysostaphin family enzyme applications. To this end, we have used NMR, SAXS and molecular dynamics simulations to characterize lysostap…

0301 basic medicinestaphylococcus aureusentsyymitStaphylococcus aureusSH3b domain030106 microbiologyPeptidePeptidoglycanProtein dynamicspeptidoglycanCleavage (embryo)PentaglycineBiochemistry Genetics and Molecular Biology (miscellaneous)Biochemistry03 medical and health scienceschemistry.chemical_compoundHydrolaseMolecular Biosciencessubstrate bindingmolekyylidynamiikkaBinding siteNMR-spektroskopiaMolecular Biologylcsh:QH301-705.5Original Researchchemistry.chemical_classificationantimikrobiset yhdisteetSubstrate InteractionLysostaphinProtein dynamicsta1182030104 developmental biologychemistrylcsh:Biology (General)Substrate bindingprotein dynamicsBiophysicsLysostaphin1182 Biochemistry cell and molecular biologyNMR structurelysostaphinpentaglycinePeptidoglycanFrontiers in Molecular Biosciences
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