Search results for "patho"

showing 10 items of 10772 documents

Oral manifestations, salivary flow rates and Candida species in Thai HIV-infected patients

2019

Background Effects of various Highly Active Antiretroviral Therapy (HAART) regimens on oral heath are unclear. Objectives: We aimed to evaluate effects of HAART on oral manifestations, salivary flow rates (SFR) and Candida species in HIV-infected patients who took mostly non-protease inhibitor-based HAART regimens. Material and methods A cross-sectional study was performed on HIV-infected patients taking and never taken HAART who attended Thai Red Cross AIDS Research Centre (n = 48). Non-HIV subjects were recruited as control (n = 20). Oral conditions and salivary flow rates were evaluated using oral examination and measurement of unstimulated and stimulated saliva. In addition, Candida col…

0301 basic medicineSalivamedicine.medical_specialty030106 microbiology03 medical and health sciences0302 clinical medicineAcquired immunodeficiency syndrome (AIDS)immune system diseasesInternal medicineHiv infected patientsMedicine030212 general & internal medicineGeneral DentistryColony-forming unitOral Medicine and Pathologybusiness.industryResearchvirus diseases:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseAntiretroviral therapyHyperpigmentationCorpus albicansSensitivity testUNESCO::CIENCIAS MÉDICASmedicine.symptombusiness
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Cellular Injuries in Cronobacter sakazakii CIP 103183T and Salmonella enterica Exposed to Drying and Subsequent Heat Treatment in Milk Powder

2018

International audience; Because of the ability of foodborne pathogens to survive in low-moisture foods, their decontamination is an important issue in food protection. This study aimed to clarify some of the cellular mechanisms involved in inactivation of foodborne pathogens after drying and subsequent heating. Individual strains of Salmonella Typhimurium, Salmonella Senftenberg, and Cronobacter sakazakii were mixed into whole milk powder and dried to different water activity levels (0.25 and 0.58); the number of surviving cells was determined after drying and subsequent thermal treatments in closed vessels at 90 and 100 degrees C, for 30 and 120 s. For each condition, the percentage of unc…

0301 basic medicineSalmonellalcsh:QR1-502medicine.disease_causelcsh:Microbiologyperméabilité membranairechemistry.chemical_compound[SDV.IDA]Life Sciences [q-bio]/Food engineeringFood sciencedryingOriginal Researchpropidium iodidebiologyChemistryMicrobiology and Parasitologyplasma-membraneSalmonella entericainfant formulaMicrobiologie et ParasitologieSalmonella entericaAlimentation et Nutritionsaccharomyces-cerevisiaeenterobacter-sakazakiitraitement thermiqueséchageMicrobiology (medical)Water activityMembrane permeabilitydesiccation tolerance030106 microbiologylow-water activityMicrobiologyrespiratory activity03 medical and health sciencesCronobacter sakazakiimedicineFood and NutritionPropidium iodideactivation respiratoireEscherichia colifoodborne pathogensheat treatmentbiology.organism_classificationCronobacter sakazakii030104 developmental biologymembrane permeabilitythermal inactivationSalmonella enterica;Cronobacter sakazakii;membrane permeability;respiratory activity;heat treatment;dryingescherichia-coliBacteria
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SPARC regulation of PMN clearance protects from pristane-induced lupus and rheumatoid arthritis

2021

Summary The secreted protein acidic and rich in cysteine (SPARC) is a matricellular protein with unexpected immunosuppressive function in myeloid cells. We investigated the role of SPARC in autoimmunity using the pristane-induced model of lupus that, in mice, mimics human systemic lupus erythematosus (SLE). Sparc−/− mice developed earlier and more severe renal disease, multi-organ parenchymal damage, and arthritis than the wild-type counterpart. Sparc+/- heterozygous mice showed an intermediate phenotype suggesting Sparc gene dosage in autoimmune-related events. Mechanistically, reduced Sparc expression in neutrophils blocks their clearance by macrophages, through defective delivery of don'…

0301 basic medicineScienceImmunologyArthritis02 engineering and technologySettore MED/08 - Anatomia Patologicamedicine.disease_causeArticleAutoimmunityPathogenesis03 medical and health sciencesmedicineSettore MED/05 - Patologia ClinicaMacrophageMolecular physiologyMultidisciplinarySystemic lupus erythematosusbusiness.industryQMatricellular proteinDendritic cell021001 nanoscience & nanotechnologymedicine.diseaseBiological sciences Immunology Molecular physiologyBiological sciences030104 developmental biologyRheumatoid arthritisCancer research0210 nano-technologybusinessiScience
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Paracrine effects of human adipose-derived mesenchymal stem cells in inflammatory stress-induced senescence features of osteoarthritic chondrocytes

2016

Aging and exposure to stress would determine the chondrocyte phenotype in osteoarthritis (OA). In particular, chronic inflammation may contribute to stress-induced senescence of chondrocytes and cartilage degeneration during OA progression. Recent studies have shown that adipose-derived mesenchymal stem cells exert paracrine effects protecting against degenerative changes in chondrocytes. We have investigated whether the conditioned medium (CM) from adipose-derived mesenchymal stem cells may regulate senescence features induced by inflammatory stress in OA chondrocytes. Our results indicate that CM down-regulated senescence markers induced by interleukin-1β including senescence-associated β…

0301 basic medicineSenescenceAgingPathologymedicine.medical_specialtyadipose-derived mesenchymal stem cells conditioned mediumsenescenceCaveolin 1chondrocytesAdipose tissueInflammationmedicine.disease_cause03 medical and health sciencesParacrine signalling0302 clinical medicineOsteoarthritisParacrine CommunicationmedicineHumansCellular SenescenceInflammation030203 arthritis & rheumatologybiologySirtuin 1KinaseMesenchymal stem cellMesenchymal Stem CellsCell Biologybeta-GalactosidaseCell biologyOxidative Stress030104 developmental biologyAdipose Tissuebiology.proteinmedicine.symptomOxidative stressResearch PaperAging
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Early life infection and host senescence

2018

IF 3.224 (2017); International audience; Advanced age is often associated with a chronic inflammatory status and inflammatory diseases. It has been suggested that exposure to infectious agents that stimulate the inflammatory response at early ages might have carry over effects in terms of accelerated senescence and increased mortality at late ages. However, not all pathogens and parasites have pro-inflammatory effects. In particular, parasitic nematodes have been shown to dampen the inflammatory response and to prevent or alleviate the symptoms of inflammatory diseases. We, therefore, tentatively predicted that early infection with a parasite that has anti-inflammatory properties might post…

0301 basic medicineSenescenceMaleAgingLPSRodent[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]LongevityInflammationHeligmosomoides polygyrusBiochemistryHost-Parasite Interactions03 medical and health sciencesMice0302 clinical medicineEndocrinologyBiomarkers of aging[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesbiology.animalGeneticsmedicine[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]AnimalsMolecular BiologyStrongylida InfectionsNematodeInflammationNematospiroides dubiusbiology[SDV.MHEP.GEG] Life Sciences [q-bio]/Human health and pathology/Geriatry and gerontologyHost (biology)[SDV.MHEP.GEG]Life Sciences [q-bio]/Human health and pathology/Geriatry and gerontologyCell Biologybiology.organism_classificationPhenotype3. Good healthDisease Models Animal030104 developmental biologyNematodeImmunology[SDV.MHEP.MI] Life Sciences [q-bio]/Human health and pathology/Infectious diseasesMice Inbred CBAFemaleHeligmosomoides polygyrusmedicine.symptomBiomarkers[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology030215 immunology
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Mediterranean nutraceutical foods: Strategy to improve vascular ageing.

2015

Ageing is characterized by a decline in all systemic functions. A greater susceptibility to apoptosis and senescence may contribute to proliferative and functional impairment of endothelial progenitor cells. They play an important role in neo-angiogenesis and endothelial repair. Vascular ageing is associated with changes in the structure and functions of vessels' wall. There are many possible causes of this damage. For sure, inflammation and oxidative stress play a fundamental role in the pathogenesis of endothelial dysfunction, commonly attributed to a reduced availability of nitric oxide. Inflammageing, the chronic low-grade inflammation that characterizes elderly people, aggravates vascu…

0301 basic medicineSenescencePathologymedicine.medical_specialtyAgingInflammationDisease030204 cardiovascular system & hematologyBioinformaticsmedicine.disease_causeDiet MediterraneanEndothelial progenitor cellPathogenesis03 medical and health sciences0302 clinical medicineNutraceuticalmedicineHumansVascular DiseasesEndothelial dysfunctionEndothelial Progenitor CellsSettore MED/04 - Patologia Generalebusiness.industryVascular ageingmedicine.diseaseInflammageing030104 developmental biologyAgeingNutraceuticalEndothelium Vascularmedicine.symptombusinessOxidative stressDevelopmental BiologyMechanisms of ageing and development
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Secondary Progression in Multiple Sclerosis: Neuronal Exhaustion or Distinct Pathology?

2016

Prevention of progression in neurological diseases, particularly in multiple sclerosis (MS) but also in neurodegenerative diseases, remains a significant challenge. MS patients switch from a relapsing-remitting to a progressive disease course, but it is not understood why and how this conversion occurs and why some patients never experience disease progression. Do aging and accumulation of neuronal damage induce progression, or do cognitive symptoms and accelerated grey matter (GM) atrophy point to distinct processes affecting networks? This review weighs accepted dogma against real data on the secondary progressive phase of the disease, highlighting current challenges in this important fie…

0301 basic medicineSenescencePathologymedicine.medical_specialtyMultiple SclerosisDiseaseGrey matter03 medical and health sciences0302 clinical medicineAtrophymedicineHumansSecondary progressiveNeuronsbusiness.industryGeneral NeuroscienceMultiple sclerosisNeurodegenerationBrainmedicine.diseaseMagnetic Resonance Imaging030104 developmental biologymedicine.anatomical_structureDisease ProgressionbusinessNeuroscience030217 neurology & neurosurgeryProgressive diseaseTrends in Neurosciences
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Renal Programming by Transient Postnatal Overfeeding: The Role of Senescence Pathways

2020

Background: Early nutrition influences the risk of Chronic Kidney Diseases development in adulthood. Mechanisms underlying the early programming of altered renal function remain incompletely understood. This study aims at characterizing the role of cell senescence pathways in early programming of Chronic Kidney Disease after transient postnatal overfeeding. Material and Methods: Reduced litters of 3 mice pups and standard litters of 10 mice pups were obtained to induce overfed animals during lactation and control animals, respectively. Animals were sacrificed at 24 days (weaning) or at 7 months of life (adulthood). Body weight, blood pressure, kidney weight, and glomerular count were assess…

0301 basic medicineSenescencekidneymedicine.medical_specialtyPhysiologyRenal function030204 cardiovascular system & hematologyprogramming[SDV.MHEP.UN]Life Sciences [q-bio]/Human health and pathology/Urology and Nephrologylcsh:Physiology03 medical and health sciencesovernutrition0302 clinical medicineOvernutritionPhysiology (medical)Internal medicineLactationmedicineWeaningOriginal Research2. Zero hungerKidneypostnatal overfeedinglcsh:QP1-981biologySirtuin 1medicine.disease[SDV.MHEP.UN] Life Sciences [q-bio]/Human health and pathology/Urology and Nephrologychronic kidney disease; developmental origins of health and disease; kidney; overnutrition; postnatal overfeeding; programming[SDV.AEN] Life Sciences [q-bio]/Food and Nutrition030104 developmental biologymedicine.anatomical_structureEndocrinologybiology.proteindevelopmental origins of health and disease[SDV.AEN]Life Sciences [q-bio]/Food and Nutritionchronic kidney diseaseKidney diseaseFrontiers in Physiology
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Innate and Adaptive Immunity in Aging and Longevity: The Foundation of Resilience

2020

The interrelation of the processes of immunity and senescence now receives an unprecedented emphasis during the COVID-19 pandemic, which brings to the fore the critical need to combat immunosenescence and improve the immune function and resilience of older persons. Here we review the historical origins and the current state of the science of innate and adaptive immunity in aging and longevity. From the modern point of view, innate and adaptive immunity are not only affected by aging but also are important parts of its underlying mechanisms. Excessive levels or activity of antimicrobial peptides, C-reactive protein, complement system, TLR/NF-κB, cGAS/STING/IFN 1,3 and AGEs/RAGE pathways, mye…

0301 basic medicineSenescencemedia_common.quotation_subjectBiologyPathology and Forensic Medicine03 medical and health sciences0302 clinical medicineImmune systemBiomarkers of aginglongevityImmunityinnate immunityresiliencemedia_commonSettore MED/04 - Patologia GeneraleInnate immune systemagingLongevityCell BiologyImmunosenescenceadaptive immunityOpinion ArticleAcquired immune system030104 developmental biologyImmunologyNeurology (clinical)Geriatrics and Gerontology030217 neurology & neurosurgeryAging and Disease
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Genetically predicted longer telomere length is associated with increased risk of B-cell lymphoma subtypes

2016

International audience; Evidence from a small number of studies suggests that longer telomere length measured in peripheral leukocytes is associated with an increased risk of non-Hodgkin lymphoma (NHL). However, these studies may be biased by reverse causation, confounded by unmeasured environmental exposures and might miss time points for which prospective telomere measurement would best reveal a relationship between telomere length and NHL risk. We performed an analysis of genetically inferred telomere length and NHL risk in a study of 10 102 NHL cases of the four most common B-cell histologic types and 9562 controls using a genetic risk score (GRS) comprising nine telomere length-associa…

0301 basic medicineSerumMaleLymphomaanalysisChronic lymphocytic leukemiaFollicular lymphomaGlobal Health[ SDV.CAN ] Life Sciences [q-bio]/Cancerimmunologysurgery0302 clinical medicineEndocrinologyimmune system diseasessingle nucleotide polymorphismGermanyhemic and lymphatic diseasesLondon80 and overOdds RatiogeneticsProspective StudiesB-cell lymphomaAssociation Studies ArticleGenetics (clinical)Aged 80 and overeducation.field_of_studytelomereGenomeLeukemiaAge FactorsGeneral MedicineEnvironmental exposureGenomicsMiddle Agedb-cell lymphomasmall cell lymphomaItaly030220 oncology & carcinogenesisMedicineepidemiologyFemaleFranceRisk of B-cell lymphoma subtypesRiskAdultCanadaChinaLymphoma B-CellGenotypeAdolescentleukocytesetiologyPopulationPopulation[SDV.CAN]Life Sciences [q-bio]/CancerBiologyEnvironmentRisk AssessmentmethodsTime03 medical and health sciencesmedicineHumansFamilyGenetic Predisposition to DiseaseeducationMolecular BiologyAllelesOccupational HealthGenetic Association StudiesAgedB-CellInternational AgenciesOdds ratioEnvironmental Exposuremedicine.diseaseTelomereNon-Hodgkin's lymphoma030104 developmental biologyImmunologyphysiologyChronic DiseasepathologyLaboratoriesmetabolism
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