Search results for "pathogen"

showing 10 items of 1657 documents

Current drugs in early development for hereditary angioedema: potential for effective treatment

2014

Hereditary angioedema (HAE) through C1 inhibitor deficiency is a rare but important disease. It is characterized by recurrent episodes of angioedema, which commonly affects the skin (in the form of swelling in the extremities, face and genitals) as well as the gastrointestinal tract (abdominal pain attacks). In approximately 1% of cases of angiodema-related swelling, there is obstruction of the upper airway, which is potentially life-threatening. Therefore, HAE due to C1 inhibitor deficiency may be associated with significant morbidity and mortality. Recent research has added to our ever-increasing understanding of the pathogenesis of HAE, which has, in addition, new clinical trials with ne…

Pharmacologymedicine.medical_specialtyGastrointestinal tractAbdominal painAngioedemabusiness.industryAngioedemas HereditaryGeneral MedicineDiseasemedicine.diseaseDermatologySurgeryClinical trialPathogenesisTreatment OutcomeHereditary angioedemamedicineAnimalsHumansPharmacology (medical)medicine.symptomAirwaybusinessComplement C1 Inhibitor ProteinExpert Opinion on Investigational Drugs
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Inhibitors of antibiotic resistance mechanisms: clinical applications and future perspectives

2020

Bacterial strains responsible for antibiotic resistant infections are increasing in an alarming way and the evolution of resistance mechanisms seems to be unstoppable. In the past decade, many efforts have been made in order to counteract this phenomenon but very few compounds have reached clinical trials. The development of new classes of antibiotics able to overcome the main bacterial drug resistance mechanisms is urgently required to counter the imminent danger of a postantibiotic era.

Pharmacologymedicine.medical_specialtyantibiotic resistanceBacteriaChemistryantivirulence strategyDrug Resistance MicrobialMicrobial Sensitivity TestsAnti-Bacterial Agentsanti-biofilm agentAntibiotic resistanceDrug DiscoveryESKAPE pathogensmedicineMolecular MedicineIntensive care medicine
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Antisense Phosphorothioate Oligonucleotides to the p65 Subunit of NF-κB Abrogate Fulminant Septic Shock Induced byS. typhimuriumin Mice

2001

The aim of this study was to characterize the functional relevance of the transcription factor NF-κB in the pathogenesis of septic shock. BALB/c mice were infected with two wild-type (WT 1, WT 2) strains of S. typhimurium that induce NF-κB or an escape variant that lacks this ability (P21) at a dose of 1 × 109/animal, respectively. Furthermore, wild-type infected mice were treated with antisense oligonucleotides directed against NF-κB 24 h before and 3 or 6 h after infection, while mismatched oligonucleotides were used as controls. Subsequently, the clinical course, histological and immunological alterations were monitored. Infection with WT 1 and WT 2 strains led to lethal septic shock wit…

Phosphorothioate OligonucleotidesNecrosisSeptic shockFulminantImmunologyInterleukinGeneral MedicineBiologymedicine.diseaseVirologyMolecular biologyPathogenesisInterferonmedicineTumor necrosis factor alphamedicine.symptommedicine.drugScandinavian Journal of Immunology
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Complement and Atherogenesis

1999

Abstract —Complement activation occurs in temporal correlation with the subendothelial deposition of LDL during early atherogenesis, and complement also plays a pathogenetic role in promoting lesion progression. Two lesion components have been identified that may be responsible for complement activation. First, enzymatic degradation of LDL generates a derivative that can spontaneously activate complement, and enzymatically degraded LDL (E-LDL) has been detected in the lesions. Second, C-reactive protein (CRP) colocalizes with complement C5b-9, as evidenced by immunohistological studies of early atherosclerotic lesions, so the possibility exists that this acute phase protein also fulfills a…

PhosphorylcholineNeuraminidaseComplement Membrane Attack ComplexCoronary Artery DiseaseBiologyPhospholipaseLesionPathogenesismedicineHumansElectrophoresis Gel Two-DimensionalTrypsinComplement Activationchemistry.chemical_classificationPhosphorylcholineC-reactive proteinAcute-phase proteinCholesterol LDLComplement C3Coronary VesselsMolecular biologyComplement systemC-Reactive ProteinEnzymeBiochemistrychemistryType C Phospholipasesbiology.proteinCalciummedicine.symptomCardiology and Cardiovascular MedicineProtein BindingArteriosclerosis, Thrombosis, and Vascular Biology
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Survival strategies and pathogenicity of Ralstonia solanacearum phylotype II subjected to prolonged starvation in environmental water microcosms

2008

Survival strategies exhibited over 4 years by Ralstonia solanacearum phylotype (ph) II biovar (bv) 2 in environmental water microcosms were examined. The bacterium is a devastating phytopathogen whose ph II bv 2 causes bacterial wilt in solanaceous crops and ornamental plants. Outbreaks of the disease may originate from dissemination of the pathogen in watercourses, where it has to cope with prolonged nutrient limitation. To ascertain the effect of long-term starvation on survival and pathogenicity of R. solanacearum in natural water microcosms, survival experiments were conducted. Microcosms were prepared from different sterile river water samples, inoculated separately with two European s…

Phylotypeeducation.field_of_studyBacilliRalstonia solanacearumMicrobial ViabilityBacterial wiltPopulationfood and beveragesBiologybiology.organism_classificationMicrobiologyMicrobiologySolanum lycopersicumRalstonia solanacearumWater MicrobiologyeducationMicrocosmPathogenBacteriaPlant Diseases
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PCR for the detection of pathogens in neonatal early onset sepsis.

2020

Background A large proportion of neonates are treated for presumed bacterial sepsis with broad spectrum antibiotics even though their blood cultures subsequently show no growth. This study aimed to investigate PCR-based methods to identify pathogens not detected by conventional culture. Methods Whole blood samples of 208 neonates with suspected early onset sepsis were tested using a panel of multiplexed bacterial PCRs targeting Streptococcus pneumoniae, Streptococcus agalactiae (GBS), Staphylococcus aureus, Streptococcus pyogenes (GAS), Enterobacteriaceae, Enterococcus faecalis, Enterococcus faecium, Ureaplasma parvum, Ureaplasma urealyticum, Mycoplasma hominis and Mycoplasma genitalium, a …

PhysiologyArtificial Gene Amplification and ExtensionPathology and Laboratory Medicinemedicine.disease_causePolymerase Chain ReactionUreaplasmaUreaplasmaMycoplasma0302 clinical medicineAntibioticsRNA Ribosomal 16SMedicine and Health Sciences030212 general & internal medicineAge of OnsetCandidaMultidisciplinaryNeonatal sepsisAntimicrobialsQCandidiasisRDrugsPneumococcusBacterial InfectionsBacterial PathogensBody FluidsBloodMedical MicrobiologyInfant Extremely PrematureMedicinePathogensNeonatal SepsisAnatomyInfant PrematureResearch ArticleStaphylococcus aureusScienceMycoplasma hominisBiologyResearch and Analysis MethodsReal-Time Polymerase Chain ReactionMicrobiologyDNA RibosomalSensitivity and SpecificityMicrobiology03 medical and health sciencesSigns and SymptomsEnterobacteriaceaeDiagnostic MedicineSepsisMicrobial Control030225 pediatricsStreptococcus pneumoniaemedicineHumansMolecular Biology TechniquesMicrobial PathogensMolecular BiologyPharmacologyBacteriaOrganismsInfant NewbornBiology and Life SciencesNeonatesStreptococcusMycoplasmamedicine.diseasebiology.organism_classificationEarly DiagnosisStreptococcus agalactiaeMultiplex Polymerase Chain ReactionEnterococcusDevelopmental BiologyUreaplasma urealyticumEnterococcus faecium
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The MAVS Immune Recognition Pathway in Viral Infection and Sepsis.

2021

Significance: It is estimated that close to 50 million cases of sepsis result in over 11 million annual fatalities worldwide. The pathognomonic feature of sepsis is a dysregulated inflammatory response arising from viral, bacterial, or fungal infections. Immune recognition of pathogen-associated molecular patterns is a hallmark of the host immune defense to combat microbes and to prevent the progression to sepsis. Mitochondrial antiviral signaling protein (MAVS) is a ubiquitous adaptor protein located at the outer mitochondrial membrane, which is activated by the cytosolic pattern recognition receptors, retinoic acid-inducible gene I (RIG-I) and melanoma differentiation associated gene 5 (M…

PhysiologyClinical BiochemistryBiologyBiochemistrySepsisImmune systemInterferonSepsismedicineAnimalsHumansMolecular BiologyGeneral Environmental ScienceMitochondrial antiviral-signaling proteinAdaptor Proteins Signal TransducingImmune EvasionLGP2Pattern recognition receptorSignal transducing adaptor proteinMDA5Cell Biologymedicine.diseaseForum Review ArticlesVirus DiseasesImmunologyHost-Pathogen InteractionsGeneral Earth and Planetary Sciencesmedicine.drugSignal TransductionAntioxidantsredox signaling
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Activin A and bone metastasis

2010

Activin A, is a multifunctional cytokine of the transforming growth factor-b superfamily of growth factors. This molecule has been shown to be implicated in the regulation of a broad range of important biological functions including bone remodelling. Therefore, a deregulation in the activin signalling pathway may result in disturbances of normal bone metabolism and, eventually, in the onset of severe pathological conditions associated with an altered bone resorption. These observations support the concept that Act A might also be implicated in the pathogenesis of bone metastasis. This review provides insight into the most recent advances in understanding the role of this growth factor in th…

PhysiologyGrowth factormedicine.medical_treatmentClinical BiochemistryBone metastasisBone NeoplasmsCell BiologyBiologymedicine.diseaseHedgehog signaling pathwayBone resorptionBone remodelingActivinsActivin bone metastasisPathogenesisGene Expression Regulation NeoplasticCytokineImmunologymedicineCancer researchAnimalsBone RemodelingActivin type 2 receptorsSignal Transduction
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Analysis of the Molecular Dialogue Between Gray Mold (Botrytis cinerea) and Grapevine (Vitis vinifera) Reveals a Clear Shift in Defense Mechanisms Du…

2015

Mature grapevine berries at the harvesting stage (MB) are very susceptible to the gray mold fungus Botrytis cinerea, while veraison berries (VB) are not. We conducted simultaneous microscopic and transcriptomic analyses of the pathogen and the host to investigate the infection process developed by B. cinerea on MB versus VB, and the plant defense mechanisms deployed to stop the fungus spreading. On the pathogen side, our genome-wide transcriptomic data revealed that B. cinerea genes upregulated during infection of MB are enriched in functional categories related to necrotrophy, such as degradation of the plant cell wall, proteolysis, membrane transport, reactive oxygen species (ROS) genera…

Physiology[SDV]Life Sciences [q-bio]Defence mechanismsVeraisonCell WallGene Expression Regulation PlantGene Expression Regulation FungalStilbenesPlant defense against herbivoryVitisPathogenComputingMilieux_MISCELLANEOUSDisease ResistanceOligonucleotide Array Sequence AnalysisBotrytis cinerea2. Zero hungerchemistry.chemical_classificationVirulencebiologyReverse Transcriptase Polymerase Chain ReactionPhytoalexinGene Expression Regulation Developmentalfood and beveragesGeneral MedicineSalicylatesPlant disease[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyHost-Pathogen Interactions[SDE]Environmental SciencesBotrytisSesquiterpenesPlant DiseaseVirulenceCyclopentanesMicrobiologyPhytoalexinsBotany[SDV.BV]Life Sciences [q-bio]/Vegetal BiologyOxylipinsPlant DiseasesPhytopathologyGene Expression Profilingfungibiology.organism_classificationGene OntologychemistryResveratrolFruitReactive Oxygen SpeciesAgronomy and Crop Science[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/Symbiosis
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Possible Role of Glymphatic System of the Brain in the Pathogenesis of High-Altitude Cerebral Edema

2018

Simka, Marian, Paweł Latacz, and Joanna Czaja. Possible role of glymphatic system of the brain in the pathogenesis of high-altitude cerebral edema. High Alt Med Biol. 19:394–397, 2018.—In this article, we suggest that the glymphatic system of the brain can play an important role in the pathogenesis of high-altitude cerebral edema (HACE). Water enters the intercellular space of the brain primarily through aquaporin-4 (AQP-4) water channels, the main component of the glymphatic system, whereas acetazolamide, pharmacological agent used in the prevention of HACE, is the blocker of the AQP-4 molecule. In animal experiments, cerebral edema caused by hypobaric hypoxia was associated with an increa…

Physiologyglymphatic systemaquaporin-4Brain EdemaAltitude Sickness030204 cardiovascular system & hematologyCerebral edemaPathogenesis03 medical and health sciences0302 clinical medicinemedicineAnimalsHumansAquaporin 4business.industryAltitudePublic Health Environmental and Occupational HealthastrocytesBrainGeneral MedicineEffects of high altitude on humansmedicine.diseaseSleep in non-human animalsAcetazolamideAquaporin 4Glymphatic systemAcetazolamidebusinessNeuroscience030217 neurology & neurosurgeryHigh-altitude cerebral edemamedicine.drug
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