Search results for "pathogen"

showing 10 items of 1657 documents

Cutting Edge: IL-23 Cross-Regulates IL-12 Production in T Cell-Dependent Experimental Colitis

2006

Abstract Although IL-12 and IL-23 share the common p40 subunit, IL-23, rather than IL-12, seems to drive the pathogenesis of experimental autoimmune encephalomyelitis and arthritis, because IL-23/p19 knockout mice are protected from disease. In contrast, we describe in this study that newly created LacZ knockin mice deficient for IL-23 p19 were highly susceptible for the development of experimental T cell-mediated TNBS colitis and showed even more severe colitis than wild-type mice by endoscopic and histologic criteria. Subsequent studies revealed that dendritic cells from p19-deficient mice produce elevated levels of IL-12, and that IL-23 down-regulates IL-12 expression upon TLR ligation. …

T-LymphocytesTransgeneT cellImmunologyDown-RegulationMice TransgenicInterleukin-23PathogenesisMiceInterleukin 23AnimalsImmunology and AllergyMedicineColitisCells Culturedbusiness.industryInterleukinsExperimental autoimmune encephalomyelitisColitismedicine.diseaseInterleukin-12Survival RateDisease Models AnimalProtein Subunitsmedicine.anatomical_structureImmunologyKnockout mouseInterleukin-23 Subunit p19Interleukin 12Disease SusceptibilitybusinessThe Journal of Immunology
researchProduct

Lights and shadows of the Taenia asiatica life cycle and pathogenicity.

2013

Humans are definitive hosts of two well-known species of the Taenia genus, Taenia solium (the pig tapeworm) and Taenia saginata (the cattle tapeworm). In the 1990s, a third species, Taenia asiatica, was discovered, sharing features with the other two since the adult morphology is similar to that of T. saginata, but its life cycle is like that of T. solium. Human taeniasis usually is asymptomatic or displays mild symptoms, and only T. solium can cause other sometimes serious disorders when humans accidentally ingest the eggs and develop the larval stage in different organs (cysticercosis). In this review, we expose what we currently know (lights) and what we do not yet know (shadows) about t…

Taenia asiaticaPathologymedicine.medical_specialtySymposiumbiologyHuman liverLife cyclepathogenesisZoologyCysticercosisbiology.organism_classificationmedicine.diseasePathogenicityTaenia asiaticamedicine.drug_formulation_ingredientTaenia soliumparasitic diseasesmedicineTaeniaTaeniasisTropismTropical parasitology
researchProduct

Extracellular vesicles in airway homeostasis and pathophysiology

2021

The epithelial–mesenchymal trophic unit (EMTU) is a morphofunctional entity involved in the maintenance of the homeostasis of airways as well as in the pathogenesis of several diseases, including asthma and chronic obstructive pulmonary disease (COPD). The “muco-microbiotic layer” (MML) is the innermost layer of airways made by microbiota elements (bacteria, viruses, archaea and fungi) and the surrounding mucous matrix. The MML homeostasis is also crucial for maintaining the healthy status of organs and its alteration is at the basis of airway disorders. Nanovesicles produced by EMTU and MML elements are probably the most important tool of communication among the different cell types, inclu…

TechnologyCell typenanovesiclesQH301-705.5QC1-999Asthma Chronic obstructive pulmonary diseaseCOPDEpithelial–mesenchymal trophic unitExosomesMicrobiota Muco-microbiotic layer nanovesicles Outer membrane vesicles.Biologychronic obstructive pulmonary diseasePathogenesismedicineCOPDGeneral Materials ScienceBiology (General)QD1-999InstrumentationAsthmaFluid Flow and Transfer ProcessesCOPDSettore BIO/16 - Anatomia UmanaTPhysicsProcess Chemistry and TechnologyGeneral EngineeringasthmaEngineering (General). Civil engineering (General)medicine.diseasemuco-microbiotic layerMicrovesiclesPathophysiologyrespiratory tract diseasesComputer Science ApplicationsChemistryepithelial–mesenchymal trophic unitImmunologyTA1-2040AirwayHomeostasis
researchProduct

Evaluation of dermal toxicity of antibacterial cotton textile coated by sol-gel technology

2017

AbstractThis paper reports about cotton textile modification by sol-gel technology with the purpose of obtaining antibacterial properties, evaluation of antibacterial properties and dermal toxicity tests of cotton textile with Zn and Si coating. Antibacterial properties evaluation against pathogenic microorganisms Pseudomonas aeruginosa, Staphylococcus aureus, and Escherichia coli made using the Parallel streak method in accordance with ATCC147 standard. For more specific evaluation of the coated textile, in vitro cytotoxicity test with epidermal HaCat cells was done. It is concluded that the coatings containing Zn and Si obtained by the sol-gel technology can impart antibacterial propertie…

TextilePolymers and PlasticsMaterials Science (miscellaneous)Microorganism02 engineering and technologyengineering.materialmedicine.disease_cause01 natural sciencesIndustrial and Manufacturing EngineeringCoating0103 physical sciencesmedicineFood scienceComposite materialEscherichia coli010302 applied physicsintegumentary systembiologyChemistrybusiness.industryPathogenic bacteria021001 nanoscience & nanotechnologybiology.organism_classificationHaCaTStaphylococcus aureusengineering0210 nano-technologyGeneral Agricultural and Biological SciencesbusinessBacteriaThe Journal of The Textile Institute
researchProduct

Assessment of the antioxidant and antibacterial activities of different olive processing wastewaters.

2017

Olive processing wastewaters (OPW), namely olive mill wastewater (OMW) and table-olive wastewaters (TOW) were evaluated for their antibacterial activity against five Gram-positive and two Gram-negative bacteria using the standard disc diffusion and thin layer chromatography (TLC)-bioautography assays. Disc diffusion screening and bioautography of OMW were compared to the phenolic extracts of table-olive brines. Positive activity against S. aureus was demonstrated. The optimization of chromatographic separation revealed that hexane/acetone in the ratio of 4:6 was the most effective for phenolic compounds separation. A HPLC-MS analysis was performed showing that only two compounds, hydroxytyr…

Thin-Layer Chromatography0301 basic medicineDPPHStaphylococcuslcsh:MedicineWastewaterPathology and Laboratory MedicineBiochemistryAntioxidantsMass Spectrometrychemistry.chemical_compoundPlant ProductsMedicine and Health SciencesStaphylococcus Aureuslcsh:ScienceChromatography High Pressure LiquidMultidisciplinaryBioautographyAntimicrobialsChromatographic TechniquesOlivesDrugsAgriculture04 agricultural and veterinary sciencesPlants040401 food scienceThin-layer chromatographyBacterial PathogensAnti-Bacterial AgentsHexaneChemistryBioassays and Physiological AnalysisMedical MicrobiologyPhysical SciencesPathogensAntibacterial activityResearch ArticleMicrobial Sensitivity TestsResearch and Analysis MethodsMicrobiologyVegetable OilsFruitsInhibitory Concentration 5003 medical and health sciences0404 agricultural biotechnologyPhenolsPicratesMicrobial ControlOleaAcetonePhenolsMicrobial PathogensPharmacologyChromatographyBacteriaBiphenyl Compoundslcsh:RChemical CompoundsOrganismsBiology and Life SciencesAgronomyTyrosolPlanar Chromatography030104 developmental biologychemistryAntibacterialsHydroxytyrosollcsh:QChromatography Thin LayerBiochemical AnalysisCrop SciencePLoS ONE
researchProduct

Inhibition of giant cell formation by compound 48/80 after infection with herpesvirus hominis

1974

Choline kinase has been found to be a soluble enzyme with a molecular weight of 105,000 in the cytoplasm of primary rabbit kidney cells. It has been purified 150-fold. It was investigated whether the inhibiting effect of Cpd 48/80 on virus-induced giant cell formation is due to interference with this enzyme. Cpd 48/80-dimer was shown to inhibit the choline kinase activityin vitro without a concomitant inhibition of giant cell formation. Likewise, another competitive inhibitor of choline kinase, purinyl-6-histamine, does not prevent giant cell formation. This finding suggests that there is no correlation between choline kinase activity and giant cell formation.

Time FactorsCholine kinaseeducationGalactosamineOleic AcidsBiologyKidneyTritiumCholinechemistry.chemical_compoundCytopathogenic Effect ViralBiosynthesisVirologyAnimalsSimplexvirusp-Methoxy-N-methylphenethylamineCarbon RadioisotopesCells Culturedchemistry.chemical_classificationGlucosamineBinding SitesPhosphotransferasesGeneral MedicineCompound 48/80LipidsVirologyMolecular biologyIn vitroEnzymechemistryEthanolaminesCytoplasmGiant cellDepression ChemicalPhosphatidylcholinesTritiumChromatography Thin LayerRabbitsArchiv f�r die gesamte Virusforschung
researchProduct

Effects of salinity and temperature on long-term survival of the eel pathogen Vibrio vulnificus biotype 2 (serovar E)

1999

ABSTRACT Vibrio vulnificus biotype 2 (serovar E) is a primary eel pathogen. In this study, we performed long-term survival experiments to investigate whether the aquatic ecosystem can be a reservoir for this bacterium. We have used microcosms containing water of different salinities (ranging from 0.3 to 3.8%) maintained at three temperatures (12, 25, and 30°C). Temperature and salinity significantly affected long-term survival: (i) the optimal salinity for survival was 1.5%; (ii) lower salinities reduced survival, although they were nonlethal; and (ii) the optimal temperature for survival was dependent on the salinity (25°C for microcosms at 0.3 and 0.5% and 12°C for microcosms at 1.5 to 3.…

Time FactorsColony Count MicrobialVibrio vulnificusSodium ChlorideApplied Microbiology and BiotechnologyMicrobiologyMiceVibrionaceaeAnimalsSeawaterSurvival ratePathogenBacterial CapsulesEcosystemVibrioInfectivityEelsVirulenceEcologybiologyTemperaturebiology.organism_classificationVibrioCulture MediaSalinityMicroscopy ElectronEnvironmental and Public Health MicrobiologyVibrio InfectionsWater MicrobiologyMicrocosmFood ScienceBiotechnology
researchProduct

Anti-fetal immune response mechanisms may be involved in the pathogenesis of placental abruption

2003

Placental abruption is an unpredictable severe complication in pregnancy. In order to investigate the possibility that the activation of the fetal nonadaptive immune system may be involved in the pathogenesis of this disease, IL-6 release from cord blood monocytes was examined by intracellular cytokine staining and flow cytometric analysis. Our results demonstrate that preterm placental abruption (n = 15) in contrast to uncontrollable preterm labor (n = 33) is associated with significantly (P < 0.001) increased release of IL-6 from the fetal monocytes. The same holds true for rhesus disease (n = 9, P < 0.001) that is characterized by a maternal production of antibodies against the rhesus-D …

Time FactorsImmunologyAntibodiesMonocytesPreeclampsiaPathogenesisFetusObstetric Labor PrematureImmune systemHLA AntigensPregnancyHumansImmunology and AllergyMedicineAbruptio PlacentaeFetusPregnancybiologyPlacental abruptionbusiness.industryImmunityFetal Bloodmedicine.diseaseFetal circulationImmunologybiology.proteinFemaleAntibodybusinessClinical Immunology
researchProduct

The aryl hydrocarbon receptor modulates acute and late mast cell responses.

2012

Abstract The aryl hydrocarbon receptor (AhR) is a ligand-dependent transcription factor whose activity is modulated by xenobiotics as well as physiological ligands. These compounds may modulate inflammatory responses and contribute to the rising prevalence of allergic diseases observed in industrialized countries. Mast cells (MCs), located within tissues at the boundary of the external environment, represent a potential target of AhR ligands. In this study, we report that murine and human MCs constitutively express AhR, and its activation by the high-affinity ligand 6-formylindolo[3,2-b]carbazole (FICZ) determines a boost in degranulation. On the contrary, repeated exposure to FICZ inhibits…

Time FactorsInbred C57BLLigandsCell DegranulationPathogenesischemistry.chemical_compoundMiceAnaphylaxiReceptorsMast CellImmunology and AllergyMast CellsReceptorMice KnockoutbiologyInterleukin-17DegranulationMast cellUp-RegulationImmunology Mast Cell Aryl Receptormedicine.anatomical_structureAryl HydrocarbonBone Marrow Celldeficiency/metabolism/physiologyIgEmedicine.symptomimmunology/metabolism/pathologyHistamineHumanReceptorTime FactorKnockoutImmunologyDown-RegulationLigandInflammationBone Marrow CellsSettore MED/08 - Anatomia PatologicaCell LinebiosynthesiAnaphylaxis; immunology/metabolism/pathology Animals Bone Marrow Cells; immunology/metabolism/pathology Cell Degranulation; genetics/immunology Cell Line Down-Regulation; genetics/immunology Humans Interleukin-17; biosynthesis Interleukin-6; biosynthesis Ligands Mast Cells; immunology/metabolism/pathology Mice Mice; Inbred C57BL Mice; Knockout Receptors; Aryl Hydrocarbon; deficiency/metabolism/physiology Receptors; IgE; physiology Time Factors Up-Regulation; genetics/immunologymedicineAnimalsHumansTranscription factorAnaphylaxisAnimalInterleukin-6Receptors IgEAryl hydrocarbon receptorgenetics/immunologyMice Inbred C57BLMAST CELL; ARYL HYDROCARBON RECEPTORchemistryReceptors Aryl HydrocarbonImmunologyphysiologybiology.proteinbiosynthesisJournal of immunology (Baltimore, Md. : 1950)
researchProduct

Role of tir and intimin in the virulence of rabbit enteropathogenic Escherichia coli serotype O103:H2.

2000

ABSTRACT Attaching and effacing (A/E) rabbit enteropathogenic Escherichia coli (REPEC) strains belonging to serogroup O103 are an important cause of diarrhea in weaned rabbits. Like human EPEC strains, they possess the locus of enterocyte effacement clustering the genes involved in the formation of the A/E lesions. In addition, pathogenic REPEC O103 strains produce an Esp-dependent but Eae (intimin)-independent alteration of the host cell cytoskeleton characterized by the formation of focal adhesion complexes and the reorganization of the actin cytoskeleton into bundles of stress fibers. To investigate the role of intimin and its translocated coreceptor (Tir) in the pathogenicity of REPEC, …

Time Factors[SDV]Life Sciences [q-bio]MutantAdministration OralPATHOGENICITEmedicine.disease_causeBacterial AdhesionMICROSCOPIE ELECTRONIQUE A TRANSMISSIONFecesCytoskeleton0303 health sciencesVirulenceEscherichia coli ProteinsEnterobacteriaceae3. Good health[SDV] Life Sciences [q-bio]IntestinesInfectious DiseasesMolecular and Cellular PathogenesisRabbitsLocus of enterocyte effacementBacterial Outer Membrane ProteinsImmunologyMolecular Sequence DataVirulenceReceptors Cell SurfaceBiologyMicrobiologydigestive systemMicrobiologyCell Line03 medical and health sciencesBacterial ProteinsIleummedicineEscherichia coliAnimalsHumansEnteropathogenic Escherichia coliAdhesins BacterialEscherichia coli030304 developmental biologyIntiminModels Genetic030306 microbiologyGenetic Complementation TestEpithelial Cellsbiochemical phenomena metabolism and nutritionbiology.organism_classificationActin cytoskeleton[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyActinsKineticsMicroscopy ElectronMicroscopy FluorescenceMutagenesisParasitologyCarrier ProteinsHeLa CellsInfection and immunity
researchProduct