Search results for "pathogen"

showing 10 items of 1657 documents

Prevention of ischemic-type biliary lesions by arterial back-table pressure perfusion

2003

Abstract Ischemic-type biliary lesions (ITBLs) lead to considerable morbidity after orthotopic liver transplantation (OLT). The exact pathogenesis is unknown. We tested the hypothesis that insufficient perfusion of biliary arterial vessels might be responsible for ITBLs. This could be prevented by improved perfusion techniques. Since February 2000, we performed a controlled study using arterial back-table pressure perfusion (AP) to achieve reliable perfusion of the biliary-tract capillary system, which may be impaired by the high viscosity of University of Wisconsin solution. We retrospectively analyzed 190 OLTs performed between September 1997 and July 2002 with regard to ITBLs. One hundre…

AdultGraft Rejectionmedicine.medical_specialtyAdenosineOrthotopic liver transplantationAllopurinolmedicine.medical_treatmentOrgan Preservation SolutionsUrologyLiver transplantationPathogenesisPostoperative ComplicationsRaffinosePressuremedicineHumansInsulinViaspanAlanine aminotransferaseAgedRetrospective StudiesTransplantationHepatologyViscositybusiness.industryLiver DiseasesGraft SurvivalImmunosuppressionMiddle AgedGlutathioneStandard techniqueLiver TransplantationSurgeryPerfusionSurvival RateReperfusion InjurySurgeryBile DuctsbusinessPerfusionImmunosuppressive AgentsLiver Transplantation
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Correlation of " in vivo" microcirculation and degeneration of the basal cells of the epithelium in lingual lichen planus

2010

Background: Oral lichen planus is an inflammatory chronic disease with an autimmune pathogenesis and unknown etiology that affects oral mucosa, with or without the involvement of the skin and other mucous membranes. The principal histological characteristics are the degeneration of the basal cell layer and the abnormal infiltration of inflammatory cells into the subepithelial layer of connective tissue. Objectives: This study is aimed to appraise if lingual lichen planus (LLP) is sustained by alteration of the oral microcirculation and if this abnormal vascularisation increases the degeneration of basal keratinocytes and the disruption of the basement membrane. Materials and Methods: Fiftee…

AdultKeratinocytesMalePathologymedicine.medical_specialtyConnective tissueBiologyBasement MembraneEpitheliumMicroscopic AngioscopyTongue DiseasesMicrocirculationPathogenesisoralSettore MED/28 - Malattie OdontostomatologicheTonguetongueCapillaroscopy lichen planus oral tonguemedicineHumansOral mucosaGeneral DentistryAgedBasement membraneMicroscopy VideoCapillaroscopylichen planusMicrocirculationMouth MucosaEpithelial CellsGeneral MedicineMiddle Agedmedicine.diseaseEpitheliumCapillarieslcsh:RK1-715medicine.anatomical_structureConnective Tissuelcsh:DentistryFemaleOral lichen planusLichen Planus OralIndian Journal of Dental Research
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Leptin and leptin receptor expression in asthma.

2009

Background The adipokine leptin is a potential new mediator for bronchial epithelial homeostasis. Asthma is a chronic inflammatory disease characterized by airway remodeling that might affect disease chronicity and severity. TGF-β is a tissue growth factor the dysregulation of which is associated with airway remodeling. Objective We sought to determine whether a bronchial epithelial dysfunction of the leptin/leptin receptor pathway contributes to asthma pathogenesis and severity. Methods We investigated in vitro the presence of leptin/leptin receptor on human bronchial epithelial cells. Then we studied the effect of TGF-β and fluticasone propionate on leptin receptor expression. Finally, th…

AdultLeptinMalemedicine.medical_specialtyPyridinesMorpholinesImmunologyAdipokineBronchiRespiratory MucosaSettore MED/10 - Malattie Dell'Apparato RespiratorioSettore BIO/09 - FisiologiaCell LinePathogenesisTransforming Growth Factor beta1Leptin leptin receptor severe asthma epithelium TGF-b remodelingInternal medicineImmunology and AllergyMedicineHumansEnzyme InhibitorsReceptorCell ProliferationLeptin receptorbusiness.industryTumor Necrosis Factor-alphaLeptindigestive oral and skin physiologyImidazolesMiddle AgedEpitheliumAsthmaRecombinant Proteinsrespiratory tract diseasesAndrostadienesmedicine.anatomical_structureEndocrinologyChromonesImmunologyFluticasoneReceptors LeptinFemalebusinesshormones hormone substitutes and hormone antagonistsEx vivoTransforming growth factorThe Journal of allergy and clinical immunology
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Inflammatory markers and cardiovascular risk in the metabolic syndrome

2011

Elevated blood glucose, obesity, high blood pressure, elevated triglycerides and low high density lipoprotein (HDL) cholesterol are well accepted risk factors in the development of coronary artery disease. Clustering of at least three of these factors in an individual is defined as metabolic syndrome (MetS). Obesity is a central pathological mechanism in the disease and it is expected that the incidence of this condition will increase dramatically within the next years. The visceral adipose tissue is not only an energy depot but also an endocrine organ which produces a large number of bioactive molecules, the so called adipokines. In the setting of obesity, the over-production of proinflamm…

AdultLeptinRiskAdolescentAngiotensinogenAdipokineAdipose tissueDiseaseIntra-Abdominal FatBioinformaticsCoronary artery diseasePathogenesisInsulin resistancePlasminogen Activator Inhibitor 1medicineHumansResistinObesityLife StyleChemokine CCL2InflammationMetabolic SyndromeInterleukin-6Tumor Necrosis Factor-alphabusiness.industryAtherosclerosisPrognosismedicine.diseaseObesityPlaque AtheroscleroticCardiovascular DiseasesAdiponectinEndothelium VascularInsulin ResistanceMetabolic syndromebusinessBiomarkersFrontiers in Bioscience
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Evaluation of serum levels of soluble CD4, CD8 and β2-microglobulin in visceral human leishmaniasis

1994

SUMMARY The levels of soluble CD4 (sCD4), sCD8 and β2-microglobulin (β2-M) were measured in sera from patients with visceral leishmaniasis during the course of infection. Levels of sCD4. sCD8 and β2-M were raised significantly above levels In normal sera and returned to the normal range after recovery. The decrease in the levels of CDS was related to a reduction of anaemia, leukopenia and thrombocytopenia. In contrast, sCD4 levels fluctuated during the period of infection. β2-M returned within normal range more rapidly than sCD8 secretion. Our results suggest that T cells are activated during infection, and that it is also possible that the raised levels of these soluble molecules play a ro…

AdultLeukopeniaBeta-2 microglobulinCD8 AntigensImmunologyLeishmaniasisBiologymedicine.diseaseImmunophenotypingPathogenesisVisceral leishmaniasisT-Lymphocyte SubsetsCD4 AntigensImmunologymedicineHumansLeishmaniasis VisceralImmunology and AllergySecretionmedicine.symptombeta 2-MicroglobulinBeta (finance)CD8Research ArticleClinical and Experimental Immunology
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Release of transforming growth factor-beta (TGF-β) and fibronectin by alveolar macrophages in airway diseases

1996

Abstract Asthma and chronic bronchitis are associated with airway remodelling, and airway macrophages are present in bronchial inflammation. TGF-β and fibronectin released by alveolar macrophages possess a fibrogenic potency. The potential role of alveolar macrophages in airway remodelling was studied in asthma and chronic bronchitis by the release of TGF-β and fibronectin. Alveolar macrophages were isolated by bronchoalveolar lavage in 14 control subjects, 14 asthmatics and 14 chronic bronchitics. The spontaneous and lipopolysaccharide (LPS)- or concanavalin A (Con A)-induced release of TGF-β and fibronectin was measured by ELISA. Alveolar macrophages from chronic bronchitics spontaneously…

AdultLipopolysaccharidesMaleChronic bronchitisLipopolysaccharideImmunologyPathogenesischemistry.chemical_compoundFibrosisTransforming Growth Factor betaMacrophages AlveolarConcanavalin AImmunology and AllergyMacrophageMedicineHumansBronchitisbiologymedicine.diagnostic_testbusiness.industryOriginal Articlesrespiratory systemMiddle Agedmedicine.diseaseAsthmarespiratory tract diseasesFibronectinsFibronectinmedicine.anatomical_structureBronchoalveolar lavagechemistryImmunologyChronic Diseasebiology.proteinFemalePulmonary alveolusbusiness
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Evidence that the interaction between circulating IgA and fibronectin is a normal process enhanced in primary IgA nephropathy

1991

A solid-phase ELISA was set up to measure the direct binding capacity (BC) of different, commercially available, purified human IgA preparations to plates coated with human fibronectin (FN). It was found that secretory, polymeric, and, to a much lesser extent, monomeric IgA exhibited elevated FN-BC as compared to their BC to plates coated with bovine serum albumin. This binding was specific since not observed with human IgG or IgM antibodies. In addition, we noted that this interaction was dose dependent, Ca2+ dependent, saturable, and not covalent, was inhibited by soluble FN, but not by a prior incubation of FN-coated plates with anti-human fibronectin antibodies, and appeared to involve …

AdultLiver CirrhosisMaleImmunoglobulin Amedicine.medical_specialtyHot TemperatureAdolescentImmunologyEnzyme-Linked Immunosorbent AssayAntigen-Antibody ComplexPathogenesisAntibody SpecificityInternal medicinemedicineHumansImmunology and AllergyBovine serum albuminEdetic AcidDose-Response Relationship DrugbiologyChemistryGlomerulonephritis IGAGlomerulonephritisMiddle Agedmedicine.diseaseFibronectinsFibronectinsImmunoglobulin AFibronectinDose–response relationshipEndocrinologyChromatography Gelbiology.proteinCalciumFemaleAntibody
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Generalized Linear Model (GLM) framework for the association of host variables and viral strains with liver fibrosis in HCV/HIV coinfected patients

2012

Chronic hepatitis C virus (HCV) infection is the main cause of advanced and end-stage liver disease world-wide, and an important factor of morbidity and mortality in Human Immunodeficiency virus-1 (HIV-1) co-infected individuals. Whereas the genetic variability of HCV has been studied extensively in monoinfected patients, comprehensive analyses of both patient and virus characteristics are still scarce in HCV/HIV co-infection. In order to find correlates for liver damage, we sought to analyze demographic, epidemiological and clinical features of HCV/HIV co-infected patients along with the genetic makeup of HCV (viral subtypes and lineage studied by nucleotide sequencing and phylogenetic ana…

AdultLiver CirrhosisMaleMicrobiology (medical)medicine.medical_specialtyHepatitis C virusHIV InfectionsHepacivirusViral Nonstructural ProteinsBiologymedicine.disease_causeModels BiologicalMicrobiologyViruschemistry.chemical_compoundLiver diseaseFibrosisEpidemiologyGeneticsmedicineHumansGenetic variabilityMolecular BiologyNS5BPhylogenyEcology Evolution Behavior and SystematicsRetrospective StudiesPhylogenetic treeCoinfectionvirus diseasesHepatitis C ChronicMiddle Agedmedicine.diseaseVirologyInfectious DiseaseschemistryHost-Pathogen InteractionsImmunologyLinear ModelsFemaleInfection, Genetics and Evolution
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Bilateral cystic pulmonary glial heterotopia and palatinal teratoma causing respiratory distress in an infant

2009

We report on a male infant with extensive, bilateral cystic and solid lung lesions who presented postnatally with respiratory distress caused by bilateral cystic lung lesions. Parenchyma-sparing resections were performed. Histology revealed the presence of neuroglial cell-lined cysts and glial nodules. In addition, a neural element containing palatinal teratoma was detected and excised. Based on previously published cases, the pathogenesis and clinical features of pulmonary neuroglial heterotopia are discussed.

AdultLung DiseasesMalePathologymedicine.medical_specialtyChoristomaFunctional LateralityPathogenesisGlial nodulesmedicineHumansAbnormalities MultipleRespiratory Distress Syndrome NewbornPalatal NeoplasmsLungRespiratory distressCystsbusiness.industryInfant NewbornTeratomaNeuroglial HeterotopiaBrainInfantHistologyGeneral Medicinemedicine.diseaseHeterotopia (medicine)medicine.anatomical_structurePediatrics Perinatology and Child HealthFemaleSurgeryTeratomaTomography X-Ray ComputedbusinessNeurogliaJournal of Pediatric Surgery
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Lung Compartmentalization of Increased TNF Releasing Ability by Mononuclear Phagocytes in Pulmonary Sarcoidosis

1989

The TNF is a monokine with cytotoxic and tumor-necrosing activities; in addition, TNF may play a role in inflammatory processes. The present study evaluates spontaneous and LPS-mediated release of TNF by AMs and autologous peripheral BMs of normal subjects and patients with pulmonary sarcoidosis. A recently developed cytotoxicity assay, specific for detection of TNF activity, was applied. This study demonstrates that (1) unstimulated mononuclear phagocytes released low levels of TNF with no differences between groups; (2) when effector cells were stimulated with LPS, AMs from patients with active pulmonary sarcoidosis released more TNF than AMs recovered from normal subjects and from patien…

AdultLung DiseasesMalePulmonary and Respiratory MedicineSarcoidosisCritical Care and Intensive Care MedicinePathogenesisHumansMedicineMacrophageLungLungTumor Necrosis Factor-alphabusiness.industryMacrophagesRespiratory diseaseMononuclear phagocyte systemCytotoxicity Tests Immunologicmedicine.diseasePulmonary AlveoliMonokinemedicine.anatomical_structureImmunologyLeukocytes MononuclearFemaleTumor necrosis factor alphaSarcoidosisCardiology and Cardiovascular MedicinebusinessBronchoalveolar Lavage FluidChest
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