Search results for "pathogenesi"

showing 10 items of 764 documents

FUS mutations in sporadic amyotrophic lateral sclerosis

2011

Mutations in the FUS gene have recently been described as a cause of familial amyotrophic lateral sclerosis (ALS), but their role in the pathogenesis of sporadic ALS is unclear. We undertook mutational screening of all coding exons of FUS in 228 sporadic ALS cases, and, as previous reports suggest that exon 15 represents a mutational hotspot, we sequenced this exon in an additional 1295 sporadic cases. Six variants in six different cases were found, indicating that FUS mutations can underlie apparently sporadic ALS, but account for less than 1% of this form of disease. © 2010 .

AdultMaleAgingAmyotrophic lateral sclerosis; FUS; Italy; Sporadic disease; United States of America;AdolescentGenotypesporadic patientsDNA Mutational AnalysisALS; FUS mutations; sporadic patientsBiologymedicine.disease_causeArticlePathogenesisExonYoung AdultDNA Mutational AnalysisGenotypemedicineHumansFUS mutationsAmyotrophic lateral sclerosisChildGeneAgedGeneticsAged 80 and overMutationGeneral NeuroscienceAmyotrophic Lateral Sclerosisamyotrophic lateral sclerosis FUS geneticsExonsMiddle Agedmedicine.diseaseUnited StatesSettore MED/26 - NEUROLOGIAItalyMutationRNA-Binding Protein FUSFemaleNeurology (clinical)Geriatrics and GerontologyALSDevelopmental BiologyRNA-Binding Protein FUS
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Acute Myocardial Infarction and Proinflammatory Gene Variants

2007

We identified four genetic risk sets for acute myocardial infarction (AMI) from information on functional gene variants that favor inflammation or modulate cholesterol metabolism: IL6 -174 G/C, TNF -308 G/A, IL10 -1082 G/A, SERPINA3 -51 G/T, IFNG +874 T/A, HMGCR -911 C/A, and APOE ε2/3/4; 316 patients and 461 healthy subjects, all Italian. Putative risk alleles are shown underlined. The sets were identified using grade-of-membership analysis. Membership scores in the sets are automatically generated for individuals. The ''low intrinsic risk'' set had alleles that downregulate inflammation and cholesterol synthesis (IL6, TNF, ILl0, HMGCR). ''AMI across a broad age range'' carried multiple pr…

AdultMaleApolipoprotein EAdolescentMyocardial InfarctionGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokinePathogenesischemistry.chemical_compoundApolipoproteins EHistory and Philosophy of SciencemedicineHumansGenetic Predisposition to DiseaseMyocardial infarctionAge of OnsetAlleleAllelesSerpinsAgedAged 80 and overbusiness.industryCholesterolGeneral NeuroscienceMiddle Agedmedicine.diseaseMiddle ageInterleukin 10CholesterolchemistryAMI Grade of Membership Genetic profile IL6 -174 G/C TNF -308 G/A IL10 -1082 G/A SERPINA3 -51 G/T IFNG +874 T/A HMGCR -911 C/A APOE ε2/3/4Acute DiseaseImmunologyCytokinesFemaleHydroxymethylglutaryl CoA Reductaseslipids (amino acids peptides and proteins)businessAnnals of the New York Academy of Sciences
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Individual and common antigen-recognition sites of liver-derived T cells in patients with autoimmune hepatitis.

2003

Autoimmune hepatitis (AIH) is characterized by dense T-cell infiltrations in the liver tissue, but little is known how T cells influence the pathogenesis. To address this question, the distribution of T-cell receptor variable beta-chain (TCR Vbeta) transcripts of peripheral blood and liver-infiltrating T cells from previously untreated patients with newly diagnosed acute exacerbated AIH was investigated. Furthermore, the lengths and sequences of complementary-determining region 3 (CDR3) were studied. Reverse transcriptase-polymerase chain reaction (RT-PCR) analysis and CDR3 spectratyping revealed multiple clonal expansions of liver-infiltrating T cells but not peripheral T cells within vari…

AdultMaleBiopsyT-LymphocytesImmunologyMolecular Sequence DataReceptors Antigen T-CellEpitopes T-Lymphocytechemical and pharmacologic phenomenaInflammationAutoimmune hepatitisBiologyCDR3 SpectratypingEpitopePathogenesismedicineHumansAmino Acid SequenceRNA MessengerReceptorAgedBase SequenceReverse Transcriptase Polymerase Chain ReactionT-cell receptorhemic and immune systemsGeneral MedicineMiddle Agedmedicine.diseaseComplementarity Determining RegionsClone CellsHepatitis AutoimmuneGene Expression RegulationImmunologyFemalemedicine.symptomNested polymerase chain reactionScandinavian journal of immunology
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GDF 15 as an anti-apoptotic, diagnostic and prognostic marker in oral squamous cell carcinoma

2011

Growth-differentiation factor 15 (GDF 15) is involved in tumor pathogenesis and its expression is increased in many types of cancers. Functional effects of GDF 15 on oncogenesis of oral squamous cell carcinoma (OSCC) remain unclear. Therefore, the aim of this study was to examine the apoptotic characteristics of GDF 15 in OSCC cell lines in vitro and to analyze serum GDF 15 concentrations as a diagnostic and prognostic tumor marker for OSCC in vivo. Caspase activity was assessed in OSCC cell lines with the Caspase-Glo 3/7 system. Serum GDF 15 concentrations from 64 patients with histopathological proven OSCC and from 30 healthy volunteers were measured using an enzyme-linked immunosorbent a…

AdultMaleCancer ResearchPathologymedicine.medical_specialtyGrowth Differentiation Factor 15Enzyme-Linked Immunosorbent AssayCaspase 3Biologymedicine.disease_causePathogenesisIn vivoCell Line TumorBiomarkers TumorCarcinomamedicineHumansAgedTumor markerAged 80 and overMiddle AgedPrognosismedicine.diseasestomatognathic diseasesOncologyApoptosisCase-Control StudiesCaspasesembryonic structuresCarcinoma Squamous CellCancer researchFemaleMouth NeoplasmsGDF15Oral SurgeryCarcinogenesisOral Oncology
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Cyclosporin- and nifedipine-induced gingival overgrowth in renal transplant patients: correlations with periodontal and pharmacological parameters, a…

1996

The factors associated with cyclosporin A (CsA)- and nifedipine (Nif)-induced gingival overgrowth were investigated in 113 renal transplant recipients receiving CsA alone (Group 1) [n = 61], CsA and Nif (Group 2) [n = 28], or azathioprine (Aza) (Control Group) [n = 24]. Periodontal and pharmacological parameters were assessed for each patient. The patients with a gingival overgrowth index (GOI) score1 were considered responders (R); those with a score/= 1 were non-responders (NR). Gingival overgrowth occurred in 33.7% of the patients in Groups 1 and 2; 60% of the responders were receiving CsA+Nif. In R, no relationship was found between the GOI and the periodontal and pharmacological parame…

AdultMaleCancer Researchmedicine.medical_specialtyAdolescentNifedipineAzathioprineGastroenterologyPathology and Forensic MedicinePathogenesisNifedipineHLA AntigensInternal medicineCyclosporin aAzathioprinePrevalencemedicineHumansPeriodontal PocketDental CalculusChildSalivaKidney transplantationKidneyHLA-A AntigensGingival Overgrowthbusiness.industryDental Plaque IndexMiddle AgedCalcium Channel Blockersmedicine.diseaseGingivitisKidney TransplantationTransplantationEndocrinologymedicine.anatomical_structureOtorhinolaryngologyToxicityCyclosporineIrritantsPeriodonticsFemaleDisease SusceptibilityPeriodontal IndexOral SurgerybusinessImmunosuppressive Agentsmedicine.drugJournal of Oral Pathology and Medicine
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In Lysinuric Protein Intolerance system y+L activity is defective in monocytes and in GM-CSF-differentiated macrophages

2010

Abstract Background In the recessive aminoaciduria Lysinuric Protein Intolerance (LPI), mutations of SLC7A7/y+LAT1 impair system y+L transport activity for cationic amino acids. A severe complication of LPI is a form of Pulmonary Alveolar Proteinosis (PAP), in which alveolar spaces are filled with lipoproteinaceous material because of the impaired surfactant clearance by resident macrophages. The pathogenesis of LPI-associated PAP remains still obscure. The present study investigates for the first time the expression and function of y+LAT1 in monocytes and macrophages isolated from a patient affected by LPI-associated PAP. A comparison with mesenchymal cells from the same subject has been a…

AdultMaleCellular differentiationlcsh:MedicinePulmonary Alveolar ProteinosisBiologyMonocytesPathogenesisYoung AdultMacrophages AlveolarmedicineHumansGenetics(clinical)Pharmacology (medical)Amino Acid Metabolism Inborn ErrorsCells CulturedGenetics (clinical)Medicine(all)chemistry.chemical_classificationResearchFusion Regulatory Protein 1 Light ChainsLysinelcsh:RMesenchymal stem cellAmino Acid Transport System y+LGranulocyte-Macrophage Colony-Stimulating FactorCell DifferentiationGeneral Medicinemedicine.diseaseLysinuric protein intoleranceMolecular biologyAmino acidGranulocyte macrophage colony-stimulating factorchemistryAminoaciduriaImmunologyPulmonary alveolar proteinosismedicine.drugOrphanet Journal of Rare Diseases
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Chloroquine Stimulates the Mitogen-Driven Lymphocyte Proliferation in Patients with Psoriasis

1993

Chloroquine is known to exacerbate psoriasis. Since immunological stimuli are considered to be important for the pathogenesis of psoriasis, we compared the effects of chloroquine on cell-mediated immunity in 15 healthy control individuals and 15 patients with psoriasis. We employed the spontaneous and phytohemagglutin (PHA)-induced uptake of 3H-thymidine to measure lymphocyte proliferation. Chloroquine was added to the cultures at concentrations ranging from 0.022 to 220 microM. We found that both spontaneous and PHA-driven lymphocyte proliferations were significantly lower in patients with psoriasis (p0.002). The spontaneous blastogenesis in both controls and patients remained stable under…

AdultMaleCellular immunityT-LymphocytesDermatologyLymphocyte proliferationLymphocyte ActivationPathogenesisChloroquinePsoriasismedicineHumansPsoriasisPhytohemagglutininsCells CulturedAgedAged 80 and overbiologyCell growthChloroquineT lymphocyteMiddle Agedmedicine.diseaseMitogen-activated protein kinaseImmunologybiology.proteinFemaleCell Divisionmedicine.drugDermatology
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Flow cytometric analysis of glyoxalase-1 expression in human leukocytes.

2011

Altered glyoxalase-1 (GLO-1) activity and expression is associated with the development of late diabetic complications, malignancy and oxidative stress- and aging-related diseases. In the present study, we developed a flow cytometry method for GLO-1 detection in human leukocytes isolated from peripheral blood samples to investigate GLO-1 expression in leukocyte subsets from type 1 and 2 diabetes mellitus patients (n = 11) and healthy subjects (n = 8). The flow cytometry analysis of GLO-1 in leukocytes showed that expression index of GLO-1-positive cells was slightly increased in mononuclear leukocytes from diabetic patients. This result correlated with the increase in GLO-1 activity in the …

AdultMaleClinical BiochemistryType 2 diabetesBiochemistryGene Expression Regulation EnzymologicFlow cytometryPathogenesisYoung AdultGlycationDiabetes mellitusDiabetes MellitusMedicineHumansCells CulturedWhole bloodAgedmedicine.diagnostic_testbusiness.industryExpression indexLactoylglutathione LyaseCell BiologyGeneral MedicineMiddle Agedmedicine.diseaseFlow CytometryCase-Control StudiesImmunologyLeukocytes MononuclearFemalebusinessGlyoxalase systemCell biochemistry and function
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Intestinal permeability and genetic determinants in patients, first-degree relatives, and controls in a high-incidence area of Crohn's disease in Sou…

2005

1. Am J Gastroenterol. 2005 Dec;100(12):2730-6. Intestinal permeability and genetic determinants in patients, first-degree relatives, and controls in a high-incidence area of Crohn's disease in Southern Italy. Fries W, Renda MC, Lo Presti MA, Raso A, Orlando A, Oliva L, Giofré MR, Maggio A, Mattaliano A, Macaluso A, Cottone M. Dipartimento di Medicina Interna e Terapia Medica, Università di Messina, Messina, Italy. OBJECTIVE: A defect of gastrointestinal barrier function is considered to represent an important step in the pathogenesis of Crohn's disease (CD) but the mechanisms leading to an increased intestinal permeability (IP) are poorly understood. Since IP is influenced by pro-inflammat…

AdultMaleEndemic DiseasesRisk AssessmentStatistics NonparametricPathogenesisCapillary PermeabilityCohort StudiesIntestinal mucosaCrohn DiseaseReference ValuesMedicineHumansGenetic Predisposition to DiseaseFirst-degree relativesIntestinal Mucosapermeability.crohn's disease.NOD2Allele frequencyProbabilityCrohn's diseaseIntestinal permeabilityHepatologybusiness.industryIncidenceGastroenterologyCase-control studyMiddle Agedmedicine.diseasedigestive system diseasesPedigreeToll-Like Receptor 4Genetics PopulationItalyGenetic markerCase-Control StudiesImmunologyMutationFemalebusiness
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Is Epstein-Barr virus infection associated with the pathogenesis of microscopic colitis?

2017

Abstract Background Epstein-Barr virus (EBV) has been associated with inflammation in the colon, particularly in patients with inflammatory bowel disease (IBD). Even if a relevant plasmocytosis, similar to IBD, is present in microscopic colitis (MC), the frequency of EBV infection in this setting is unknown. Objectives We aimed to compare the frequency of colonic EBV infection in patients with MC, ulcerative colitis (UC), and irritable bowel syndrome (IBS). Study design The frequency of colonic EBV infection in biopsies of 30 patients with MC, 30 patients with UC, and 30 controls with IBS was retrospectively assessed. PCR was performed to detect viral EBV DNA in colonic biopsies. In situ hy…

AdultMaleEpstein-Barr Virus InfectionsHerpesvirus 4 HumanColonBiopsymedicine.disease_causeInflammatory bowel diseasePolymerase Chain ReactionPathogenesisIrritable Bowel Syndrome03 medical and health sciences0302 clinical medicineMicroscopic colitishemic and lymphatic diseasesVirologyBiopsymedicineHumansEpstein–Barr virus infectionIrritable bowel syndromeIn Situ HybridizationAgedRetrospective Studiesmedicine.diagnostic_testbusiness.industryMiddle Agedmedicine.diseaseUlcerative colitisEpstein–Barr virusdigestive system diseasesColitis MicroscopicInfectious Diseases030220 oncology & carcinogenesisImmunologyDNA ViralRNA Viral030211 gastroenterology & hepatologyColitis UlcerativeFemalebusinessJournal of clinical virology : the official publication of the Pan American Society for Clinical Virology
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