Search results for "pathogenesi"

showing 10 items of 764 documents

Increased Vascularity in Cervicovaginal Mucosa with Schistosoma haematobium Infection

2011

Background Close to 800 million people in the world are at risk of schistosomiasis, 85 per cent of whom live in Africa. Recent studies have indicated that female genital schistosomiasis might increase the risk of human immunodeficiency virus (HIV) infection. The aim of this study is to quantify and analyse the characteristics of the vasculature surrounding Schistosoma haematobium ova in the female genital mucosa. Methodology/Principal Findings Cervicovaginal biopsies with S. haematobium ova (n = 20) and control biopsies (n = 69) were stained with immunohistochemical blood vessel markers CD31 and von Willebrand Factor (vWF), which stain endothelial cells in capillary buds and established blo…

PathologyAnatomy and PhysiologyBiopsyVDP::Medical disciplines: 700::Clinical medical disciplines: 750::Tropical medicine: 761Gynecologic InfectionsPathogenesisCervix UteriCardiovascularSchistosomiasis haematobiaVascularitySchistosomiasisSchistosoma haematobiumMicroscopyNeovascularization PathologicHistocytochemistrylcsh:Public aspects of medicineMucous membraneMiddle AgedImmunohistochemistryPlatelet Endothelial Cell Adhesion Molecule-1Infectious Diseasesmedicine.anatomical_structureMedical MicrobiologyVaginaSchistosoma haematobiumVaginaNeglected tropical diseasesMedicineFemalemedicine.symptomImmunohistochemical AnalysisResearch ArticleNeglected Tropical DiseasesAdultmedicine.medical_specialtylcsh:Arctic medicine. Tropical medicineHistologyAdolescentlcsh:RC955-962ImmunologySchistosomiasisBiologyMicrobiologyYoung AdultVascular Biologyvon Willebrand FactorParasitic DiseasesmedicineAnimalsHumansBiologySchistosomaMucous MembraneReproductive SystemParasite PhysiologyPublic Health Environmental and Occupational Healthlcsh:RA1-1270biology.organism_classificationmedicine.diseaseSchistosoma haematobium infectionAfricaImmunologic TechniquesWomen's HealthParasitologyGenital Diseases FemalePLoS Neglected Tropical Diseases
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Ultramicro-trauma in the endometrial-myometrial junctional zone and pale cell migration in adenomyosis

2015

Objective To determine if ultrastructural tissue trauma occurs in the junctional zone in uteri in adenomyosis. Design A case-control experimental study. Setting Endometriosis research center. Patient(s) Twelve uteri with adenomyosis, and 9 uteri without adenomyosis, were gained during laparoscopy-assisted vaginal hysterectomy. Intervention(s) Transmission electron microscopic study of the junctional zone, as well as immunohistochemical staining for epithelial cadherin, and van Gieson staining and immunofluorescence for CD45 and CD68. Main Outcome Measure(s) Analysis of the electron microscopy photos and the immunoreactive scores of the staining. Result(s) The inner myometrial muscle fibers …

PathologyEndometriosisFluorescent Antibody TechniqueEndometriumBasal (phylogenetics)EndometriumMyofibrilsCell MovementVan Gieson's stainHysterectomy VaginalMedicine (all)MyometriumObstetrics and GynecologyAntigens CD45AnatomyMiddle AgedCadherinsultrastructureImmunohistochemistrymedicine.anatomical_structurePhenotypeMyometriumImmunohistochemistryFemaleAdenomyosis pathogenesiCase-Control StudieAdenomyosisHumanAdultmedicine.medical_specialtyAntigens Differentiation MyelomonocyticBiologypale cellMicroscopy Electron TransmissionAntigens CDmedicineHumansAdenomyosisendometrial-myometrial junctional zoneEpithelial CellCadherinEpithelial CellsBiomarkermedicine.diseaseReproductive MedicineCase-Control StudiesCadherinLeukocyte Common AntigensLaparoscopyAdenomyosiBiomarkersMyofibril
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Neuroimmunomodulatory Alterations in Non-Lesional Peritoneum Close to Peritoneal Endometriosis

2012

<b><i>Objectives:</i></b> An imbalance in the ratio of sensory to sympathetic nerve fibre (NF) density in peritoneal endometriotic lesions (pEL) has recently been demonstrated and leads to the assumption that this preponderance of the sensory pro-inflammatory milieu is a major cause of pain in endometriosis. Therefore, the density of sensory and sympathetic NFs was determined in distal unaffected peritoneum of endometriosis patients to be able to detect possible alterations in unaffected peritoneum. <b><i>Methods:</i></b> In serial pEL sections (n = 40), lesional and matching unaffected peritoneum as well as healthy peritoneum (HP) from patien…

PathologyEndometriosisSympathetic nerveSubstance PSensory nerve fibreSubstance PPeritoneal Diseaseschemistry.chemical_compoundNerve growth factorPelvic painEndocrinologyhemic and lymphatic diseasesEndometriosiintegumentary systemvirus diseasesMiddle AgedInterleukin-1βImmunohistochemistrymedicine.anatomical_structureNeurologyImmunohistochemistryFemalemedicine.symptomPeritoneal diseasesPeritoneumHumanAdultmedicine.medical_specialtyAdolescentImmunologyEndometriosisEndocrine and Autonomic SystemYoung AdultPeritoneumAdrenergic FiberInternal medicinemedicineHumansPathogenesis endometriosiEndocrine and Autonomic Systemsbusiness.industryPelvic painmedicine.diseaseNerve growth factorEndocrinologychemistrySympathetic nerve fibrePeritoneal DiseasebusinessAdrenergic Fibers
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IL-6 Regulates Neutrophil Microabscess Formation in IL-17A-Driven Psoriasiform Lesions

2014

The lack of a generally accepted animal model for human psoriasis has hindered progress with respect to understanding the pathogenesis of the disease. Here we present a model in which transgenic IL-17A expression is targeted to the skin in mice, achievable after crossing our IL-17A(ind) allele to the K14-Cre strain. K14-IL-17A(ind/+) mice invariably develop an overt skin inflammation bearing many hallmark characteristics of human psoriasis including dermal infiltration of effector T cells, formation of neutrophil microabscesses, and hyperkeratosis. IL-17A expression in the skin results in upregulated granulopoiesis and migration of IL-6R-expressing neutrophils into the skin. Neutralization …

Pathologymedicine.medical_specialty1303 BiochemistryNeutrophilsT-LymphocytesHyperkeratosisGene Expression610 Medicine & healthInflammationDermatology10263 Institute of Experimental ImmunologyBiochemistryGranulopoiesis2708 Dermatology1307 Cell BiologyPathogenesisMicePsoriasis1312 Molecular BiologymedicineAnimalsPsoriasisMicroabscessMolecular BiologyMice Knockoutintegumentary systemInterleukin-6business.industryMacrophagesInterleukin-17Cell Biologymedicine.diseaseReceptors Interleukin-6AbscessDisease Models AnimalImmunology570 Life sciences; biologyEpidermismedicine.symptombusinessInfiltration (medical)GranulocytesSignal TransductionEpidermal thickeningJournal of Investigative Dermatology
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Trägt leukozytäres Acetylcholin zur chronischen Abstoßung von Nierentransplantaten bei?

2010

Einleitung: Akute Abstoßungsepisoden prädisponieren für die chronische Abstoßung von Nierentransplantaten. Für die fatale akute Abstoßung haben wir experimentell gezeigt, dass Monozyten, die sich in Transplantatgefäßen massiv ansammeln, Acetylcholin (ACh) produzieren.[for full text, please go to the a.m. URL]

Pathologymedicine.medical_specialtyAllogeneic transplantationbusiness.industryIsograft610 Medical sciences; MedicineCholine acetyltransferaseBlotCholine transporterPathogenesissurgical procedures operativeddc: 610MedicineImmunohistochemistrybusinessAcetylcholinemedicine.drug
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Hypoxia in Breast Cancer: Pathogenesis, Characterization and Biological/Therapeutic Implications

2002

Nearly 50% of locally advanced breast cancers exhibit hypoxic and/or anoxic tissue areas that are heterogeneously distributed within the tumour mass. Hypoxia is predominantly caused by structural and functional abnormalities of the newly formed tumour vessels arising from neovascularization, by a disturbed microcirculation, by enlarged diffusion distances, and by tumour-associated or therapy-induced anaemia. The extent of pretherapeutically measured hypoxic tissue areas is independent of clinical tumour size and stage, and histological type and grade. Anaemia can substantially worsen tumour O2 depletion. Hypoxia is known to directly or indirectly confer resistance to irradiation and some ch…

Pathologymedicine.medical_specialtyBreast NeoplasmsDisease-Free SurvivalTreatment failureMicrocirculationPathogenesisNeovascularizationBreast cancerHumansMedicineTreatment FailureNeovascularization Pathologicbusiness.industryMicrocirculationGeneral MedicineHypoxia (medical)Prognosismedicine.diseaseCell HypoxiaClinical trialCancer researchFemalemedicine.symptomMalignant progressionbusinessWiener Medizinische Wochenschrift
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Intermediate-filament expression in ocular tissue.

2002

Intermediate-filament proteins (IFPs) occur in the intracellular cytoskeleton of eukaryotic cells, and their expression in diverse tissues is related both to embryology as well as to differentiation. Although the available information concerning their functional properties in vivo is still incomplete, antibodies against individual IFPs are commonly used in immunohistochemical procedures as markers for differentiation, and these antibodies are of outstanding value in the routine histopathological evaluation of tumor specimens. This review presents a compilation of the currently available data concerning IFP expression in normal and diseased ocular tissues. Representatives of every known clas…

Pathologymedicine.medical_specialtyCell typebiologyEye DiseasesMesenchymal stem cellVimentinEyeSensory SystemsPathogenesisOphthalmologyCytokeratinmedicine.anatomical_structureIntermediate Filament ProteinsReference Valuesmedicinebiology.proteinImmunohistochemistryAnimalsHumansTissue DistributionIntermediate filamentCorneal epitheliumProgress in retinal and eye research
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Blood Brain Barrier Compromise with Endothelial Inflammation may Lead to Autoimmune Loss of Myelin during Multiple Sclerosis

2009

Multiple sclerosis is an autoimmune disease characterized by multifocal areas of inflammation and demyelination within the central nervous system. The mechanism that triggers the disease remains elusive. However, recent findings may indicate that multiple sclerosis, at its source, could be a hemodynamic disorder. It has been found that multiple sclerosis patients exhibit significant stenoses in extracranial veins draining the central nervous system (in azygous and internal jugular veins), which are associated with significant pressure gradients measured across strictures. Such anatomic venous abnormalities were not found in the control group of healthy subjects. In this review, it is hypoth…

Pathologymedicine.medical_specialtyCentral nervous systemInflammationBlood–brain barriermultiple sclerosisProinflammatory cytokinePathogenesisCellular and Molecular NeuroscienceMyelinDevelopmental Neurosciencevenous insufficiencyMedicineAnimalsHumansEndotheliumMyelin SheathAutoimmune diseaseInflammationbusiness.industryMultiple sclerosisblood-brain barriermedicine.diseasemedicine.anatomical_structureNeurologyImmunologymedicine.symptombusinessAdhesion moleculesCurrent Neurovascular Research
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Chronic cerebrospinal venous insufficiency is unlikely to be a direct trigger of multiple sclerosis

2013

Abstract Background Chronic cerebrospinal venous insufficiency, a vascular pathology affecting the veins draining the central nervous system can accompany multiple sclerosis and is suspected to be involved in its pathogenesis. Objective This study was aimed at exploring a potential role for chronic cerebrospinal venous insufficiency in triggering multiple sclerosis. If it were venous abnormalities responsible for neurological pathology, one should expect negative correlation, i.e. more severe vascular lesions in the patients with early onset of multiple sclerosis. Methods Localization and degree of venous blockages in 350 multiple sclerosis patients were assessed using catheter venography. …

Pathologymedicine.medical_specialtyCentral nervous systemVenographyDiseaseAzygous veinPathogenesisMultiple sclerosisVenous malformationInternal medicinemedicinePathologicalJugular veinsmedicine.diagnostic_testbusiness.industryMultiple sclerosisGeneral MedicinePhlebographymedicine.diseaseVenous insufficiencyChronic cerebrospinal venous insufficiencymedicine.anatomical_structureNeurologyCardiologyNeurology (clinical)businessVenous malformationMultiple Sclerosis and Related Disorders
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Assessment of Tumor Development and Wound Healing Using Endoscopic Techniques in Mice

2010

Mouse models of intestinal inflammation and colon cancer are valuable tools to gain insights into the pathogenesis of the corresponding human diseases. Recently, in vivo mouse endoscopy has been developed, allowing not only the high-resolution monitoring and scoring of experimental disease development, but also enables the investigator to perform manipulations, including local injection of reagents or the taking of biopsies for molecular and histopathologic analyses. Chromoendoscopic staining with methylene blue enables visualization of the crypt structure and allows discrimination between inflammatory and neoplastic changes. The development of endoscopic techniques in live mice opened new …

Pathologymedicine.medical_specialtyColorectal cancerBiopsyDiseasePathogenesisMiceMicromanipulation03 medical and health sciences0302 clinical medicineIn vivoIntestinal inflammationmedicineAnimalsHumans030304 developmental biologyWound Healing0303 health sciencesMiniaturizationHepatologymedicine.diagnostic_testbusiness.industryDisease mechanismsGastroenterologyEndoscopyColitismedicine.disease3. Good healthEndoscopyEndoscopes GastrointestinalDisease Models AnimalColonic Neoplasms030211 gastroenterology & hepatologybusinessWound healingGastroenterology
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