Search results for "peripheral nervous system"

showing 10 items of 90 documents

Neurophysiological studies of pain pathways in peripheral and central nervous system disorders.

2003

Standard clinical neurophysiological assessment of somatosensory pathways by sensory evoked potentials (SEPs) is limited to the tactile and proprioceptive systems consisting of large fibers in the peripheral nerve, the dorsal columns of the spinal cord and the medial lemniscus in the brainstem. This limitation means that about half of the lesions in the somatosensory system will not be detectable. In recent years, many clinical studies have confirmed that laser evoked potentials (LEPs) allow the assessment of the other half of the somatosensory system. Rapid heating of the skin by infrared laser pulses specifically activates the nociceptive and thermoreceptive pathways consisting of small f…

Spinothalamic tractLaser-Evoked Potentialsbusiness.industryLasersTemperaturePainPeripheral Nervous System DiseasesSensory systemAnatomySpinal cordSomatosensory systemmedicine.anatomical_structureNociceptionNeurologyDermatomeSomatosensory evoked potentialCentral Nervous System DiseasesTouchEvoked Potentials SomatosensorymedicineHumansNeurology (clinical)businessNeuroscienceJournal of neurology
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Vulnerability of peripheral catecholaminergic neurons to MPTP is not regulated by alpha-synuclein.

2010

Although generally considered a prototypical movement disorder, Parkinson's disease is commonly associated with a broad-spectrum of non-motor symptoms, including autonomic dysfunctions caused by significant alterations in catecholaminergic neurons of the peripheral sympathetic nervous system. Here we present evidence that alpha-synuclein is highly expressed by sympathetic ganglion neurons throughout embryonic and postnatal life and that it is found in tyrosine hydroxylase-positive sympathetic fibers innervating the heart of adult mice. However, mice deficient in alpha-synuclein do not exhibit any apparent alterations in sympathetic development. Sympathetic neurons isolated from mouse embryo…

Sympathetic nervous system1-Methyl-4-phenylpyridiniumα-Synuclein knockoutTyrosine 3-MonooxygenaseNeurotoxinsNeurotrophic factorSubstantia nigraBiologylcsh:RC321-571chemistry.chemical_compoundMiceCatecholaminesSympathetic Fibers PostganglionicParkinsonian DisordersNeurotrophic factorsmedicineNeurotoxinAutonomic gangliaAnimalslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCells CulturedNeuronsGanglia SympatheticCell DeathMPTPSympathetic ganglionMice Mutant Strainsnervous system diseasesMPP+medicine.anatomical_structureNeurologychemistrynervous system1-Methyl-4-phenyl-1236-tetrahydropyridinePeripheral nervous systemSympathetic nervous systemNerve Degenerationalpha-SynucleinCatecholaminergic cell groupsPeripheral nervous systemNeuroscienceNeurobiology of disease
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Electrophysiologic evaluation of phrenic nerve and diaphragm function after coronary bypass surgery: Prospective study of diabetes and other risk fac…

2006

Objective Phrenic neuropathy after coronary artery bypass grafting has been related to various risk factors with conflicting results. The aim of this study was to assess the incidence, characteristics, and clinical consequences of phrenic neuropathy and the influence of diabetes and other risk factors. Methods We conducted an observational, prospective study of parallel groups including 94 consecutive patients subjected to coronary artery bypass grafting, half of them with diabetes and associated polyneuropathy . Electrophysiologic study of phrenic nerve conduction as the reference method, chest radiography, diaphragm ultrasound, and functional respiratory tests were performed 24 to 48 hour…

ThoraxMalePulmonary and Respiratory MedicineDiaphragmInternal thoracic arteryPhrenic Nerve InjuryDiabetes ComplicationsRisk Factorsmedicine.arteryDiabetes mellitusmedicineDiabetes MellitusHumansProspective StudiesRisk factorCoronary Artery BypassPhrenic nervebusiness.industryPeripheral Nervous System DiseasesMiddle Agedmedicine.diseaseElectrophysiologyPhrenic NerveBypass surgeryAnesthesiaFemaleSurgerybusinessCardiology and Cardiovascular MedicinePolyneuropathyThe Journal of Thoracic and Cardiovascular Surgery
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Efficacy and tolerability of a fixed dose combination of cortex phospholipid liposomes and cyanocobalamin for intramuscular use in peripheral neuropa…

2019

Peripheral neuropathies are frequently encourtered in clinical practice and are assiociated with a major impairment in quality of life . Howewvwr, their management reamans poor, and current therapies are often burdened with major side effects and can present poor efficacy on pain and functionality. Therefore, it has been suggested that the combination of two or more different drugs may improve analgesis efficacy and reduce side effects. Tricortin 1000 is formulated with 12 mg of Brain cortex phospholipid liposomes + 1000 microgrammi of Cyanocobalamin injectable (PL+ CNCb1) for intramuscolar use and is indicated in the tratment of poly-algo-neuropathic syndromes. This combination exerts a ma…

Viitamin B 12Fixed-dose combinationAnalgesicPharmacologyInjections IntramuscularInjections03 medical and health sciences0302 clinical medicinePharmacotherapyPeripheral nervous system diseasemedicineHumansCyanocobalaminphospholipidPhospholipidsCerebral CortexIntramuscularAnalgesicsClinical Trials as TopicNeck painLiposomebiologybusiness.industrySettore MED/34 - Medicina Fisica E RiabilitativaBack painPeripheral Nervous System DiseasesGeneral MedicineBack pain; Liposomes; Neuralgia; Peripheral nervous system diseases; Phospholipids; Vitamin B 12; Analgesics; Cerebral Cortex; Clinical Trials as Topic; Drug Combinations; Humans; Injections Intramuscular; Liposomes; Neuroprotective Agents; Peripheral Nervous System Diseases; Phospholipids; Vitamin B 12Drug CombinationsVitamin B 12Neuroprotective AgentsTolerability030220 oncology & carcinogenesisLiposomesbiology.proteinNeuralgia030211 gastroenterology & hepatologyIiposomemedicine.symptombusinessNeurotrophinMinerva Medica
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Number, identity, and sequence of the Drosophila head segments as revealed by neural elements and their deletion patterns in mutants.

1994

The development of the insect head tagma involves massive rearrangements and secondary fusions of segment anlagen during embryogenesis. Due to the lack of reliable morphological markers, the number, identity, and sequence of the head segments, particularly in the pregnathal region, are still a matter of ongoing debates. We examined the complex array of internal structures of the embryonic Drosophila melanogaster head such as the sensory structures and nerves of the peripheral and stomatogastric nervous systems, and we used embryonic head mutations causing a lack of overlapping segment anlagen to unravel the segmental identity and the sequence of the neural elements. Our results provide evid…

animal structuresHead (linguistics)media_common.quotation_subjectMorphogenesisInsectPeripheral Nervous SystemMorphogenesisAnimalsDrosophila ProteinsDrosophila (subgenus)TagmaSequence (medicine)media_commonHomeodomain ProteinsGeneticsMultidisciplinarybiologyPhylogenetic treeGenes Homeoboxbiology.organism_classificationDrosophila melanogasterInsect HormonesImmunologic TechniquesDrosophila melanogasterHeadResearch ArticleTranscription FactorsProceedings of the National Academy of Sciences
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Imaging photoplethysmography for evaluation of cutaneous sensory nerve fiber function

2020

Peripheral neuropathy refers to peripheral nervous system dysfunction which affects up to 2% of the world's population. This condition is caused by damage to the small nerve fibers, hence its assessment is still challenging due to the lack of simple, non-invasive and objective diagnostic techniques. The present study aimed to develop and evaluate a simple, objective and non-invasive technique for assessment of small cutaneous sensory nerve fiber function. Our approach is based on utilization of imaging photoplethysmography and local skin heating. The effectiveness of the method was evaluated on young, healthy volunteers (n = 14) after 10 minutes of 45-degree local skin heating, while record…

education.field_of_studybusiness.industryPopulationNerve fibermedicine.diseaseCutaneous sensory nervemedicine.anatomical_structurePeripheral neuropathyPeripheral nervous systemPhotoplethysmogramHealthy volunteersmedicineeducationbusinessPerfusionBiomedical engineeringBiophotonics—Riga 2020
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Pathomorphology of hereditary sensory neuropathies

1995

Currently, the hereditary sensory neuropathies (HSN) — because of the involvement of the autonomic system recently called HSAN — comprise types I–V predicated on clinical differences. This classification of HSAN I–V seems to be uncontested, at the present time. Morphologically, individual forms I–V only differ in the non-specific loss or lack of myelinated and unmyelinated nerves in varying degrees in that in HSAN II large myelinated axons are most affected, in HSAN IV unmyelinated axons are almost absent; but each HSAN is considered an axonal type of neuropathy. Early onset, slow or no progression of the neuropathic process, and little or no evidence of ongoing degeneration suggest maldeve…

medicine.anatomical_structureHereditary Sensory Neuropathiesbusiness.industryMaldevelopmentPeripheral nervous systemNeuropathologistMedicineSural nerveSensory systembusinessNeuroscienceCombined approachEarly onset
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Tachykinin-, calcitonin gene-related peptide-, and protein gene product 9.5-immunoreactive nerve fibers in alveolar walls of mammals.

1991

The presence and distribution of the presumed pan-neural marker protein gene product 9.5 (PGP)- and peptide-immunoreactive (ir) nerve fibers in alveolar walls of various species was investigated by light microscopic single and double staining immunohistochemistry. PGP-, tachykinin (TK)-, and calcitonin gene-related peptide (CRGP)-ir fibers were sparsely distributed in a similar pattern in alveolar walls of all species investigated. No vasoactive intestinal peptide-, peptide histidine isoleucine-, galanin-, and opioid-ir nerve fibers could be detected. PGP-ir fibers outnumbered those staining for TKs and CGRP. There was partial coexistence of PGP and TK as well as of TK and CRGP. PGP-, TK-, …

medicine.medical_specialtyAlveolar EpitheliumCalcitonin Gene-Related PeptideVasoactive intestinal peptideGuinea PigsCalcitonin gene-related peptideBiologyAlveolar cellsDogsNerve FibersSpecies SpecificityInternal medicineCricetinaeTachykininsmedicineAnimalsGalaninMammalsintegumentary systemMesocricetusGeneral NeuroscienceNeuropeptidesrespiratory systemMolecular biologyRatsPulmonary Alveolimedicine.anatomical_structureEndocrinologyCalcitoninPeripheral nervous systemCatsPulmonary alveolusUbiquitin ThiolesteraseBiomarkersNeuroscience letters
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Cisplatin-induced peripheral neuropathy: neuroprotection by erythropoietin without affecting tumour growth

2007

This study examined the dose-dependent efficacy of erythropoietin (EPO) for preventing and/or treating cisplatin (CDDP) induced peripheral neurotoxicity (CINP), and its influence on tumour treatment and growth. Rats received eight intraperitoneal (ip) injections of 2 mg/kg CDDP twice weekly. EPO co-administered (50 or 10 microg/kg ip, three times/week) had a dose-dependent effect, partially preventing CINP, but 0.5 microg/kg ip was not effective. The neuroprotective effect lasted at least 5 weeks after the last dose of EPO and CDDP. In addition, EPO (50 microg/kg ip three times/week) after the last injection of CDDP still induced a significant recovery of CINP. In a separate experiment in r…

medicine.medical_specialtyCancer ResearchPeripheral neuropathyNeural ConductionNeurophysiologyAntineoplastic AgentsHindlimbHematocritNeuroprotectionAntineoplastic AgentInternal medicinemedicinePathologyAnimalsRats WistarErythropoietinCisplatincisplatin; Erythropoietin; peripheral neuropathy; tumor growthmedicine.diagnostic_testDose-Response Relationship Drugbusiness.industryAnimalNeurotoxicityPeripheral Nervous System DiseasesMammary Neoplasms ExperimentalTumour growthHematologymedicine.diseaseRatsHindlimbDose–response relationshipPeripheral neuropathyEndocrinologyOncologyHematocritErythropoietinRatFemalePeripheral Nervous System DiseaseCisplatinbusinessCell Divisionmedicine.drug
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Effects of nicotine receptor agonists on acetylcholine release from the isolated motor nerve, small intestine and trachea of rats and guinea-pigs

1992

The effects of nicotine receptor agonists on the release of [3H]acetylcholine from the phrenic nerve, the small intestine and the trachea were investigated to characterize neuronal nicotine receptors within the peripheral nervous system. Contraction of the indirectly-stimulated hemidiaphragm was recorded to investigate desensitization of the postsynaptic muscular nicotine receptors. Nicotine, cytisine, 1,1-dimethyl-4-phenylpiperazinium and 2-(4-aminophenyl)-ethyl-trimethyl-ammoniumiodide caused a concentration-dependent (0.1-30 microM) increase in evoked [3H]acetylcholine release from the phrenic nerve, whereby bell-shaped concentration-response curves were obtained. The rank order of decre…

medicine.medical_specialtyDiaphragmGuinea PigsMyenteric PlexusMotor nerveReceptors NicotinicMotor EndplateNicotineCytisinechemistry.chemical_compoundPostsynaptic potentialInternal medicineIntestine SmallDrug DiscoverymedicineAnimalsGenetics (clinical)Phrenic nerveDose-Response Relationship DrugMuscle SmoothGeneral MedicineAcetylcholineStimulation ChemicalRatsPhrenic NerveTracheaEndocrinologymedicine.anatomical_structureParasympathomimeticschemistryPeripheral nervous systemMolecular Medicinemedicine.symptomSecretory RateAcetylcholineMuscle Contractionmedicine.drugMuscle contractionThe Clinical Investigator
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