Search results for "pero"

showing 10 items of 3365 documents

Acute methionine load‐induced hyperhomocysteinemia enhances platelet aggregation, thromboxane biosynthesis, and macrophage‐derived tissue factor acti…

1997

A moderate elevation of plasma homocysteine is a risk factor for atherosclerosis and arterial and veinous thrombosis. However, the mechanisms leading to vascular disorders are poorly understood because studies that have investigated the potential atherothrombogenicity of hyperhomocysteinemia in vivo are scarce. Using a rat model, we were the first to show that dietary folic acid deficiency, a major cause of basal hyperhomocysteinemia, is associated with enhanced macrophage-derived tissue factor and platelet activities. We proposed that an homocysteine-induced oxidative stress may account for this hypercoagulable state. To determine the true thrombogenicity of moderate hyperhomocysteinemia a…

medicine.medical_specialtyHyperhomocysteinemiaMethioninebiologybusiness.industryThromboxanemedicine.disease_causemedicine.diseaseBiochemistryLipid peroxidationchemistry.chemical_compoundTissue factorEndocrinologychemistryInternal medicineGeneticsbiology.proteinmedicinePlateletThromboxane-A synthasebusinessMolecular BiologyOxidative stressBiotechnologyThe FASEB Journal
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Effects of Long-Term Nitroglycerin Treatment on Endothelial Nitric Oxide Synthase (NOS III) Gene Expression, NOS III–Mediated Superoxide Production, …

2000

Abstract —Long-term nitroglycerin (NTG) treatment has been shown to be associated with cross-tolerance to endothelium-dependent vasodilators. It may involve increased production of reactive oxygen species (such as superoxide, O 2 ·− ) that rapidly inactivate the nitric oxide (NO) released from the endothelial cells. It remains to be elucidated, however, whether long-term treatment with NTG alters the activity and expression of the endothelial NO synthase (NOS III) and whether this enzyme can contribute to O 2 ·− formation. We studied the influence of long-term NTG treatment on the expression of NOS III as assessed by RNase protection assay and Western blot. Tolerance was measured ex vivo i…

medicine.medical_specialtyIndolesNitric Oxide Synthase Type IIIPhysiologyCarbazolesBiological AvailabilityVasodilationArginineNitric OxideGene Expression Regulation EnzymologicTimeNitric oxideNitroglycerinchemistry.chemical_compoundAlkaloidsSuperoxidesInternal medicinemedicineAnimalsRNA MessengerLucigeninCloning MolecularEnzyme InhibitorsRats WistarCalcimycinProtein Kinase CProtein kinase CBenzophenanthridineschemistry.chemical_classificationReactive oxygen speciesSuperoxideAcetylcholinePhenanthridinesRatsVasodilationEndocrinologychemistryBiochemistryEndothelium VascularNitric Oxide SynthaseCardiology and Cardiovascular MedicineEx vivoAcetylcholinemedicine.drugCirculation Research
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Islet beta-cell apoptosis triggered in vivo by interleukin-1beta is not related to the inducible nitric oxide synthase pathway: evidence for mitochon…

2003

IL-1beta is recognized as an effector cytokine contributing to islet beta-cell destruction during diabetes. We have previously shown in vitro that IL-1beta induces nitric oxide (NO) and beta-cell damage. Here, we show that IL-1beta administration in vivo to Wistar rats transiently increases manganese superoxide dismutase activity, whereas inducible NO synthase is not detected, and the levels of nitrate+nitrate do not change. Moreover, a significant decrease of mitochondrial aconitase, leading to a rise of hydroperoxides, and islet beta-cell apoptosis, involving caspase-3 and -8, is observed. Analysis of adhesion molecules in beta-cells showed that intercellular adhesion molecule-1 is highly…

medicine.medical_specialtyLipid PeroxidesNitric Oxide Synthase Type IIApoptosisBiologyMitochondrionIn Vitro TechniquesAconitaseNitric oxidechemistry.chemical_compoundIslets of LangerhansEndocrinologyIn vivoInternal medicinemedicineAnimalsRats WistarNitritesAconitate HydratasegeographyCaspase 8geography.geographical_feature_categoryNitratesCell adhesion moleculeCaspase 3Superoxide DismutaseIsletIntercellular Adhesion Molecule-1Caspase 9Cell biologyMitochondriaRatsNitric oxide synthaseEndocrinologyBiochemistrychemistryApoptosisCaspasesbiology.proteinNitric Oxide SynthaseInterleukin-1Endocrinology
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Oxidative tissue damage after phacoemulsification: influence of ophthalmic viscosurgical devices.

2003

To quantify the oxidative tissue damage after phacoemulsification, correlate the damage to the energy applied, and investigate the influence of ophthalmic viscosurgical devices (OVDs).Department of Ophthalmology, University of Mainz, Mainz, Germany.The study comprised 130 eyes operated on by 1 surgeon using the same phacoemulsification machine. Some eyes received an OVD before phacoemulsification and some did not. Energy values were expressed as phaco time; that is, ultrasound (US) time (seconds) after conversion to 100% phaco power. Patients were grouped as follows: Group 1, phaco time less than 20 seconds and no OVD; Group 2, phaco time 20 to 40 seconds and no OVD; Group 3, phaco time mor…

medicine.medical_specialtyLipid Peroxidesgenetic structuresFree Radicalsmedicine.medical_treatmentSodium hyaluronateAqueous humorLactoseMethylcelluloseThiobarbituric Acid Reactive SubstancesAqueous Humorchemistry.chemical_compoundPostoperative ComplicationsOphthalmologyTissue damageOxazinesMedicineHumansHyaluronic AcidAgedPhacoemulsificationbusiness.industryUltrasoundPhacoemulsificationFree Radical ScavengersSensory SystemsOphthalmologyOxidative StresschemistrySurgeryLipid PeroxidationbusinessJournal of cataract and refractive surgery
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Guinea pig Kupffer cells can be activated in vitro to an enhanced superoxide response

1988

Summary In the preceding paper it was shown that Kupffer cells isolated by digestion of the liver and purified by centrifugal elutriation can be activated in vitro by lipopolysaccharide and muramyl dipeptide to an enhanced superoxide response upon zymosan phagocytosis. Lipopolysaccharide and muramyl dipeptide also led to a strongly increased prostaglandin E 2 release during the phagocytosis of zymosan. This activation was accompanied by an increased production of prostaglandin E 2 during the incubation with the stimuli. Prostaglandin E 2 synthesis was inhibited by the cyclooxygenase inhibitor indomethacin, reduced by dexamethasone, but only slightly decreased by the lipoxygenase inhibitor n…

medicine.medical_specialtyLipopolysaccharidemedicine.medical_treatmentPhagocytosisPronaseBiologyLipoxygenasechemistry.chemical_compoundInternal medicinemedicineHepatologySuperoxideZymosanMolecular biologyIn vitroNordihydroguaiaretic acidEndocrinologymedicine.anatomical_structurechemistryBiochemistryHepatocytebiology.proteinlipids (amino acids peptides and proteins)CyclooxygenaseMuramyl dipeptideProstaglandin EJournal of Hepatology
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Type 2 diabetes, dyslipidemia, and vascular risk: rationale and evidence for correcting the lipid imbalance.

2004

Type 2 diabetes is an important cardiovascular risk factor. A significant component of the risk associated with type 2 diabetes is thought to be because of its characteristic lipid "triad" profile of raised small dense low-density lipoprotein levels, lowered high-density lipoprotein, and elevated triglycerides (TGs). Trials of statins and fibrates have included substantial numbers of patients with diabetes and indicate that lipid lowering reduces cardiovascular event rates in these patients. However, statins alone do not always address all the lipid abnormalities of diabetes. Fibrates, which have low affinity for peroxisome proliferator-activated receptor alpha (PPARalpha), improve most asp…

medicine.medical_specialtyLipoproteinsPeroxisome proliferator-activated receptorType 2 diabetesInsulin resistanceRisk FactorsInternal medicineDiabetes mellitusmedicineLipolysisHumansPPAR alphaDyslipidemiaschemistry.chemical_classificationbusiness.industrynutritional and metabolic diseasesLipid metabolismmedicine.diseaseLipid MetabolismEndocrinologychemistryDiabetes Mellitus Type 2Cardiovascular Diseaseslipids (amino acids peptides and proteins)Hydroxymethylglutaryl-CoA Reductase InhibitorsCardiology and Cardiovascular MedicinebusinessDyslipidemiaLipoproteinAmerican heart journal
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PGC-1α: a master gene that is hard to master

2012

Peroxisome proliferator-activated receptor-γ coactivator-1α (PGC-1α) is a transcriptional coactivator that favorably affects mitochondrial function. This concept is supported by an increasing amount of data including studies in PGC-1α gene-deleted mice, suggesting that PGC-1α is a rescue factor capable of boosting cell metabolism and promoting cell survival. However, this view has now been called into question by a recent study showing that adeno-associated virus-mediated PGC-1α overexpression causes overt cell degeneration in dopaminergic neurons. How is this to be understood, and can these seemingly conflicting findings tell us something about the role of PGC-1α in cell stress and in cont…

medicine.medical_specialtyModels NeurologicalSettore BIO/11 - Biologia MolecolareRNA-binding proteinBiologyMitochondrionSettore BIO/09 - FisiologiaMiceCellular and Molecular NeuroscienceHeat shock proteinInternal medicinemedicineAnimalsHomeostasisHumansReceptorMolecular BiologyTranscription factorHeat-Shock ProteinsMice KnockoutPharmacologyPGC-1α Mitochondria Dopaminergic neurons Transgenic animal Adenovirus Parkinson’s diseaseDopaminergic NeuronsDopaminergicRNA-Binding ProteinsParkinson DiseaseCell BiologyPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaMitochondriaEndocrinologyCell metabolismNerve DegenerationTrans-ActivatorsMolecular MedicineNeuroscienceHomeostasisTranscription FactorsCellular and Molecular Life Sciences
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New approaches in the treatment of Adamantiades-Behçet's disease.

2005

Purpose of review To update clinicians on the recent advances in the treatment of Adamantiades-Behcet's disease. Recent findings Interferon-α-2a and infliximab have proved able to induce prompt remission in the vast majority of Adamantiades-Behcet's patients with DMARD-resistant uveoretinitis. Efficacy of interferon-α-2a has also been reported for mucocutaneous lesions, arthritis, and (more anecdotally) for neuro-Behcet, while results from small case series suggest that infliximab is beneficial for mucocutaneous lesions and (more anecdotally) for arthritis and gastro-intestinal manifestations. Two cases of neuro-Behcet treated with infliximab showed a complete resolution. Finally, in a rand…

medicine.medical_specialtyMucocutaneous zoneArthritisBehcet's diseaseDiseaseInterferon alpha-2Receptors Tumor Necrosis Factorlaw.inventionEtanerceptEtanerceptRheumatologyRandomized controlled triallawAntibodies Monoclonal; Behcet Syndrome; Chaperonin 60; Humans; Immunoglobulin G; Immunologic Factors; Interferon-alpha; Receptors Tumor Necrosis Factor; Recombinant ProteinsmedicineHumansImmunologic FactorsIFN-α2abusiness.industryBehcet SyndromeAntibodies MonoclonalInterferon-alphaChaperonin 60medicine.diseaseDermatologyeye diseasesInfliximabAdamantiades-Behçet's diseaseInfliximabRecombinant Proteinsstomatognathic diseasesImmunoglobulin GImmunologyTumour necrosis factorbusinessTolerizationUveitismedicine.drugCurrent opinion in rheumatology
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Endotheliale Dysfunktion: Pathophysiologie, Diagnostik und prognostische Bedeutung

2008

The endothelium plays a crucial role in the regulation of vascular tone. Recent studies have indicated that endothelial dysfunction develops in the presence of cardiovascular risk factors such as hypertension, diabetes mellitus, hypercholesterolemia and in chronic smokers, as well as in patients with a family history of cardiovascular disease. It has now been established that endothelial dysfunction represents the first indicator of vascular damage. Endothelial function can be assessed in coronary and peripheral conductance and resistance vessels by means of invasive and noninvasive (ultrasound-guided) methods such as intracoronary infusion of acetylcholine, the endothelium-dependent vasodi…

medicine.medical_specialtyNADPH oxidaseEndotheliumbiologybusiness.industrySuperoxideVasodilationGeneral Medicinemedicine.diseasemedicine.disease_causeNitric oxide synthasechemistry.chemical_compoundEndocrinologymedicine.anatomical_structurechemistryInternal medicinebiology.proteinMedicineEndothelial dysfunctionbusinessXanthine oxidaseOxidative stressDMW - Deutsche Medizinische Wochenschrift
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Nitric Oxide Synthesis in Vascular Physiology and Pathophysiology

2015

Nitric oxide (NO) is produced by three NO synthase (NOS) isoforms: neuronal NOS (nNOS), inducible NOS (iNOS), and endothelial NOS (eNOS). Under physiological conditions, vascular NO is produced by eNOS and nNOS, with both playing atheroprotective roles. Under pathological conditions, iNOS can be induced and eNOS may become uncoupled. iNOS produces a large amount of NO, induces vascular dysfunction, and promotes atherogenesis. Uncoupled eNOS generates superoxide instead of NO and contributes significantly to endothelial dysfunction and atherogenesis. Major mechanisms of eNOS uncoupling include depletion of tetrahydrobiopterin, an essential co-factor for the eNOS enzyme, and deficiency of L-a…

medicine.medical_specialtyNADPH oxidasebiologyChemistrySuperoxideNitric Oxide Synthase Type IIITetrahydrobiopterinmedicine.diseasebiology.organism_classificationEndothelial NOSNitric oxidechemistry.chemical_compoundEndocrinologyEnosInternal medicinemedicinebiology.proteinEndothelial dysfunctionmedicine.drug
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