Search results for "pero"

showing 10 items of 3365 documents

PGC-1α, Inflammation, and Oxidative Stress: An Integrative View in Metabolism

2020

Peroxisome proliferator-activated receptor-γ coactivator (PGC)-1α is a transcriptional coactivator described as a master regulator of mitochondrial biogenesis and function, including oxidative phosphorylation and reactive oxygen species detoxification. PGC-1α is highly expressed in tissues with high energy demands, and it is clearly associated with the pathogenesis of metabolic syndrome and its principal complications including obesity, type 2 diabetes mellitus, cardiovascular disease, and hepatic steatosis. We herein review the molecular pathways regulated by PGC-1α, which connect oxidative stress and mitochondrial metabolism with inflammatory response and metabolic syndrome. PGC-1α regula…

AgingThioredoxin reductaseReview ArticleOxidative phosphorylationmedicine.disease_causeBiochemistryAntioxidantsCoactivatormedicineAnimalsHumansInflammationMetabolic Syndromechemistry.chemical_classificationReactive oxygen speciesOrganelle BiogenesisQH573-671ChemistryCell BiologyGeneral MedicinePeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaMitochondriaCell biologyOxidative StressMitochondrial biogenesisOrgan SpecificityThioredoxinCytologyPeroxiredoxinOxidative stressOxidative Medicine and Cellular Longevity
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Age-Related Changes of Liver Antioxidant Enzymes and 8-Hydroxy-2-Deoxyguanosine During Fetal–Neonate Transition and Early Rat Development

2000

We have studied the pro-antioxidant status of the rat liver on the last day of gestation and at 1, 15, and 30 days of extrauterine life. Representative variables, such as activities of superoxide dismutase (SOD), catalase, and glutathione peroxidase and concentrations of reduced glutathione and 8-hydroxy-2'-deoxyguanosine, were determined in liver to assess the degree of birth-associated oxidative stress during the fetal-neonatal transition and early development of the rat. Percentages by which liver Cu/ZnSOD activity increased over the basal value of the fetal liver were 54%, 95%, and 127% at neonatal days 1, 15, and 30, respectively. There was a lack of induction in the development profil…

Agingmedicine.medical_specialtyAntioxidantmedicine.medical_treatmentClinical Biochemistrymedicine.disease_causeBiochemistryAntioxidantsSuperoxide dismutasechemistry.chemical_compoundFetusPregnancyInternal medicineGeneticsmedicineAnimalsDeoxyguanosineRats WistarMolecular Biologychemistry.chemical_classificationGlutathione PeroxidaseFetusbiologySuperoxide DismutaseGlutathione peroxidaseDeoxyguanosineCell BiologyGlutathioneCatalaseGlutathioneRatsOxidative StressEndocrinologyAnimals NewbornLiverchemistry8-Hydroxy-2'-DeoxyguanosineCatalasebiology.proteinFemaleOxidative stressDNA DamageIUBMB Life (International Union of Biochemistry and Molecular Biology: Life)
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Age-related changes in linoleic acid bioconversion by isolated hepatocytes from spontaneously hypertensive and normotensive rats

1994

This study points out the hepatocyte interconversion of the linoleic acid family during hypertension. Hepatocyte delta 6 desaturase activity was higher in 1 month-old spontaneously hypertensive rats than in normotensive controls. A similar tendency was observed in 6 month-old SHR. delta 5 desaturase activity was higher only in 1 month-old spontaneously hypertensive rats as compared to controls. Desaturase activities were particularly high at the age of 6 months. The hepatocyte fatty acid composition showed an impairment of n-6 polyunsaturated fatty acid metabolism in spontaneously hypertensive animals. Changes were greater in the young prehypertensive rats than in adults. A storage of n-3 l…

Agingmedicine.medical_specialtyClinical chemistryLinoleic acidClinical BiochemistryProstaglandinRats Inbred WKYLinoleic Acidchemistry.chemical_compoundRats Inbred SHRInternal medicinemedicineAnimalsMolecular Biologychemistry.chemical_classificationKidneyFatty AcidsCell BiologyGeneral MedicineMetabolismPeroxisomeRatsEndocrinologymedicine.anatomical_structureLinoleic AcidsLiverchemistryHepatocyteHypertensionPolyunsaturated fatty acidMolecular and Cellular Biochemistry
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Physiological changes in glutathione metabolism in foetal and newborn rat liver

1991

Glutathione metabolism was studied in isolated hepatocytes from foetal, newborn and adult rats. The GSH/GSSG ratio decreased 15-20-fold through the foetal-neonatal-adult transition. This was mainly due to an increase in GSSG. All enzyme activities involved in the glutathione redox cycle tend to increase during that transition, but the relative increases in glutathione peroxidase and glutathione S-transferase were 3-5 times those of glutathione reductase or glucose-6-phosphate dehydrogenase. GSH synthesis from methionine as a sulphur source was 6 times lower in foetal than in adult hepatocytes. However, when N-acetylcysteine was used as a sulphur donor to by-pass the cystathionine pathway, t…

Agingmedicine.medical_specialtyGPX1GPX3Glutathione reductaseBiochemistrychemistry.chemical_compoundFetusInternal medicinemedicineAnimalsAmino AcidsMolecular Biologychemistry.chemical_classificationMethioninebiologyGlutathione peroxidaseCystathionine gamma-LyaseRats Inbred StrainsCell BiologyGlutathioneGlutathioneCystathionine beta synthaseRatsEndocrinologyAnimals NewbornLiverBiochemistrychemistryembryonic structuresbiology.proteinResearch ArticleCysteineBiochemical Journal
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Impaired calcium homeostasis in aged hippocampal neurons

2009

Abstract Development of neurodegenerative diseases such as Alzheimer's and Parkinson's disease is strongly age-associated. The impairment of calcium homeostasis is considered to be a key pathological event leading to neuronal dysfunction and cell death. However, the exact impact of aging on calcium homeostasis in neurons remains largely unknown. In the present work we have investigated intracellular calcium levels in cultured primary hippocampal neurons from young (2 months) and aged (24 months) rat brains. Upon stimulation with glutamate or hydrogen peroxide aged neurons in comparison to young neurons demonstrated an increased vulnerability to these disease-related toxins. Measurement of c…

Agingmedicine.medical_specialtyNeurotoxinsGlutamic Acidchemistry.chemical_elementHippocampusBiologyCalciumHippocampusCalcium in biologyRats Sprague-DawleyInternal medicinemedicineAnimalsCalcium SignalingOrganic ChemicalsCells CulturedCellular SenescenceNeuronsCalcium metabolismCalpainGeneral NeuroscienceNeurodegenerationGlutamate receptorCalpainHydrogen PeroxideOxidantsmedicine.diseaseRatsOxidative Stressmedicine.anatomical_structureEndocrinologynervous systemchemistryNerve Degenerationbiology.proteinCalciumNeuronNeuroscienceNeuroscience Letters
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The atypical cannabinoid O-1602 protects against experimental colitis and inhibits neutrophil recruitment.

2011

Background: Cannabinoids are known to reduce intestinal inflammation. Atypical cannabinoids produce pharmacological effects via unidentified targets. We were interested in whether the atypical cannabinoid O-1602, reportedly an agonist of the putative cannabinoid receptor GPR55, reduces disease severity of dextran sulfate sodium (DSS) and trinitrobenzene sulfonic acid (TNBS)-induced colitis in C57BL/6N and CD1 mice. Methods: DSS (2.5% and 4%) was supplied in drinking water for 1 week while TNBS (4 mg) was applied as a single intrarectal bolus. Results: Both treatments caused severe colitis. Injection of O-1602 (5 mg/kg intraperitoneally) significantly reduced macroscopic and histological col…

AgonistMaleCannabinoid receptormedicine.drug_classColonNeutrophilsmedicine.medical_treatmentPharmacologyMotor ActivityInflammatory bowel diseaseArticleReceptors G-Protein-CoupledReceptor Cannabinoid CB2chemistry.chemical_compoundMiceReceptor Cannabinoid CB1CyclohexanesmedicineImmunology and AllergyAnimalsCannabidiolColitisReceptorReceptors CannabinoidPeroxidaseMice KnockoutAnalysis of VarianceO-1602business.industryCannabinoidsDextran SulfateGastroenterologyResorcinolsmedicine.diseaseColitisMice Inbred C57BLChemotaxis LeukocyteDisease Models AnimalchemistryGPR55Neutrophil InfiltrationTrinitrobenzenesulfonic AcidImmunologylipids (amino acids peptides and proteins)CannabinoidbusinessInflammatory bowel diseases
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Telmisartan as metabolic modulator: a new perspective in sports doping?

2011

The World Antidoping Agency (WADA) has introduced some changes in the 2012 prohibited list. Among the leading innovations to the rules are that both 5-aminoimidazole-4-carboxamide-1-[beta]-D-ribofuranoside (peroxisome proliferator�activated receptor-[delta] [PPAR-[delta]]-5' adenosine monophosphate-activated protein kinase [AMPK] agonist) and GW1516 (PPAR-[delta]-agonist) are no longer categorized as gene doping substances in the new 2012 prohibited list but as metabolic modulators in the class �Hormone and metabolic modulators.� This may also be valid for the angotensin II receptor blocker telmisartan. It has recently been shown that telmisartan might induce similar biochemical, biological…

Agonistmedicine.medical_specialtymedicine.drug_classPeroxisome proliferator-activated receptorPhysical Therapy Sports Therapy and RehabilitationdopingBenzoatesMiceGene dopingInternal medicinemedicineAnimalsHumansOrthopedics and Sports MedicineTelmisartanMuscle SkeletalDoping in Sportschemistry.chemical_classificationFiber typeTelmisartan; doping; sport.business.industryAMPKGeneral MedicineRatssport.EndocrinologyMitochondrial biogenesischemistryBenzimidazolesTelmisartanbusinessAngiotensin II Type 1 Receptor Blockersmedicine.drug
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LXR antagonists induce ABCD2 expression

2014

X-linked adrenoleukodystrophy (X-ALD) is a rare neurodegenerative disorder characterized by the accumulation of very-long-chain fatty acids resulting from a beta-oxidation defect. Oxidative stress and inflammation are also key components of the pathogenesis. X-ALD is caused by mutations in the ABCDI gene, which encodes for a peroxisomal half ABC transporter predicted to participate in the entry of VLCFA-CoA into the peroxisome, the unique site of their beta-oxidation. Two homologous peroxisomal ABC transporters, ABCD2 and ABCD3 have been proven to compensate for ABCD1 deficiency when overexpressed. Pharmacological induction of these target genes could therefore represent an alternative ther…

Agonistx-ald;very-long-chain fatty acid;lxr;hydroxycholesterol;abcd2medicine.medical_specialtymedicine.drug_classx-aldEndogenyContext (language use)ATP-binding cassette transporterBiologyATP Binding Cassette Transporter Subfamily DInternal medicinemedicineHumanslxr[ SDV.BDD ] Life Sciences [q-bio]/Development BiologyhydroxycholesterolLiver X receptorAdrenoleukodystrophyMolecular Biology[SDV.BDD]Life Sciences [q-bio]/Development BiologyLiver X ReceptorsFatty AcidsBiologie du développementNeurosciencesCell BiologyHep G2 CellsPeroxisomemedicine.diseaseOrphan Nuclear ReceptorsDevelopment BiologyHydroxycholesterolsvery-long-chain fatty acidOxidative StressEndocrinologyGene Expression RegulationCell cultureabcd2Neurons and Cognition[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Cancer researchlipids (amino acids peptides and proteins)AdrenoleukodystrophyATP-Binding Cassette Transporters[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]
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An imperial town in a time of transition. Life, environment, and decline of early Byzantine Caričin Grad

2016

The site of Caricin Grad in south-eastern Serbia – currently listed on UNESCO’s tentative list – has been the subject of archaeological investigations for more than 100 years. For the last decades it has been the focus of a joint project of the Archaeological Institute in Belgrade and the Ecole Francaise de Rome. A reconstruction of the economic, environmental and social history of the city is the main objective of a cooperative project started in 2014 with the Romisch-Germanisches Zentralmuseum Mainz. The remains of the early Byzantine complex of Caricin Grad show the features of a city built in the classical, Hellenistic-Roman tradition, combined with ecclesiastical Christian architecture…

Agrarian societyGeographybiologyUrban archaeologyAntiqueGeoarchaeologyEconomic historyEmperorHuman ecologyHousehold archaeologybiology.organism_classificationArchaeologyByzantine architectureMulti-, inter- and transdisciplinary research in Landscape Archaeology
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Glutathione-dependent formaldehyde dehydrogenase (ADH3) and low km mitochondrial aldehyde dehydrogenase (ALDH2). New evidence for differential expres…

2011

Epidemiological and experimental studies support the involvement of lipid peroxidation (LPO) in retinal diseases. In addition to other pathogenic mechanisms not fully understood, the possibility remains that peroxidic aldehydes, acting as cytotoxic chemicals, mediate in the progression of chronic ocular disorders.To test proper mechanisms involved in removing peroxidic aldehydes from the retina, in an attempt to understand long-lasting changes induced by LPO, the oxidative and antioxidant enzymatic activities, as well as the retinal distribution and activity of glutathione-dependent formaldehyde dehydrogenase (ADH3) and low km mitochondrial aldehyde dehydrogenase (ALDH2), were studied and c…

Aldehyde dehydrogenaseBiologymedicine.disease_causeBiochemistryRetinaLipid peroxidationMitochondrial Proteinschemistry.chemical_compoundRetinal DiseasesmedicineAnimalsRats WistarFormaldehyde dehydrogenaseALDH2Alcohol dehydrogenaseAldehyde Dehydrogenase MitochondrialAlcohol DehydrogenaseRetinalGeneral MedicineGlutathioneAldehyde DehydrogenaseMolecular biologyGlutathioneImmunohistochemistryRatsOxidative StresschemistryBiochemistrybiology.proteinFemaleLipid PeroxidationOxidative stressFree radical research
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