Search results for "pero"

showing 10 items of 3365 documents

Peroxynitrite generated from constitutive nitric oxide synthase mediates the early biochemical injury in short-term cultured hepatocytes

2000

AbstractEarly loss of P450 in rat hepatocyte cultures appears directly related to nitric oxide (NO) overproduction. This study provides experimental evidence for the induction – shortly after isolation through the classical procedure – of strong oxidative stress that involves both oxygen-derived and NO-derived species. NO formation at this stage is due to the early activation of liver constitutive NO synthase (cNOS). Immunodetection of nitrated proteins provides direct evidence of endogenous peroxynitrite (PN) formation upon hepatocyte isolation. On the basis of the combined use of dihydrorhodamine 123 and NOS inhibitors, the analysis of the amount, time course and nature of the species inv…

CultureBiophysicsEndogenyNitric Oxidemedicine.disease_causeBiochemistryPeroxynitriteNitric oxideP450 contentchemistry.chemical_compoundStructural BiologyGeneticsmedicineAnimalsViability assayOverproductionMolecular BiologyCells CulturedNitratesHepatocyte isolationbiologyNitric oxide synthaseProteinsCell BiologyOxidantsRatsNitric oxide synthaseKineticsmedicine.anatomical_structureLiverchemistryBiochemistryOxidative stressHepatocytebiology.proteinReactive Oxygen SpeciesProtein nitrationPeroxynitriteOxidative stressFEBS Letters
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Insights into the Mechanism of Cumene Peroxidation Using Supported Gold and Silver Nanoparticles

2013

Due to the considerable industrial implications, an in-depth study of cumene peroxidation using supported gold and silver nanoparticles was carried out to gain more insight into the mechanism of this reaction. Supported gold nanoparticles were found to efficiently catalyze the decomposition of cumene hydroperoxide with a selectivity of 25% at 80 °C when using gold supported on hydrotalcite (AuNP@HT), and 2-phenyl-2-propanol (i.e., cumyl alcohol) was the main product. Further, silver nanoparticles supported on hydrotalcite (AgNP@HT) converted cumene to cumene hydroperoxide at 80 °C with 80% selectivity. Both benchtop and oxygen-uptake experiments were used to probe the reaction mechanism and…

CumeneReaction mechanismHydrotalcite010405 organic chemistryChemistryGeneral Chemistry010402 general chemistryPhotochemistry01 natural sciencesCatalysisSilver nanoparticle0104 chemical sciences3. Good healthCatalysischemistry.chemical_compoundCumene hydroperoxideColloidal goldSelectivityACS Catalysis
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Nachweis des di-2-cyanoisopropylperoxides bei der zersetzung von azoisobuttersäuredinitril in gegenwart von sauerstoff

1970

Das Di-2-cyanoisopropylperoxid entsteht in etwa 5-proz. Ausbeute bei der Zersetzung von Azoisobuttersaredinitril unter Sauerstoff in Isobutyronitril als Losungsmittel. Thermisch zerfallt das Peroxid est oberhalb von 120°C mit mesbarer Geschwindigkeit in einer Reaktion erster Ordnung. Die Aktivierungsenergie, in Cumol als Losungsmittel, wurde zu 37,9 kcal · mol−1 bestimmt. Werte fur die Zerfallskonstante bei 140°C sind in Cumol 0,057 h−1, in tert-Butylbenzol 0,073 h−1, in Chlorbenzol 0,18 h−1 und in o-Dichlorbenzol 0,23 h−1. Di-2-cyanoisopropylperoxide is formed when azobisisobutyronitrile is decomposed under oxygen in isobutyronitrile as a solvent. The yield amounts to ca. 5%. Thermal decom…

CumeneSolventchemistry.chemical_compoundReaction rate constantchemistryChlorobenzenePolymer chemistryThermal decompositionAzobisisobutyronitrilePeroxideChemical decompositionDie Makromolekulare Chemie
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Fabrication of quercetin and curcumin bionanovesicles for the prevention and rapid regeneration of full-thickness skin defects on mice

2013

In the present work biocompatible quercetin and curcumin nanovesicles were developed as a novel approach to prevent and restore skin tissue defects on chronic cutaneous pathologies. Stable and suitable quercetin- and curcumin-loaded phospholipid vesicles, namely liposomes and penetration enhancer-containing vesicles (PEVs), were prepared. Vesicles were made from a highly biocompatible mixture of phospholipids and alternatively a natural polyphenol, quercetin or curcumin. Liposomes were obtained by adding water, while PEVs by adding polyethylene glycol 400 and Oramix®CG110 to the water phase. Transmission electron microscopy, cryogenic-transmission electron microscopy and small- and wide-ang…

CurcuminMaterials scienceStatic ElectricitySus scrofaBiomedical EngineeringPolyethylene glycolBiochemistryBiomaterialsMicechemistry.chemical_compoundX-Ray DiffractionScattering Small AnglePEG ratioAnimalsEdemaRegenerationParticle SizeMolecular BiologyPeroxidaseSkinMice Inbred ICRLiposomeVesicleGeneral MedicineIn vitroDisease Models AnimalchemistryBiochemistryLiposomesCurcuminBiophysicsNanoparticlesFemaleQuercetinQuercetinWound healingBiotechnologyActa Biomaterialia
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Grain-size-induced relaxor properties in nanocrystalline perovskite films

2004

Thin films of ${\mathrm{Pb}}_{0.76}{\mathrm{Ca}}_{0.24}{\mathrm{TiO}}_{3}$ (PTC), which is a classical ferroelectric as a bulk material and of the relaxor material $\mathrm{Pb}({\mathrm{Sc}}_{0.5}{\mathrm{Nb}}_{0.5}){\mathrm{O}}_{3}$, have been produced to find out whether nanocrystalline ferroelectric films show a grain-size-induced relaxor behavior. Amorphous films were deposited onto a $\mathrm{Ti}∕\mathrm{Pt}$ coated silicon (100) wafer by reactive $\text{rf}$ sputtering. Different grain sizes were prepared by a controlled annealing process and they were determined by profile analysis of x-ray diffraction spectra. Temperature dependent Raman spectroscopy was used to look for phase trans…

Curie–Weiss lawMaterials scienceAnnealing (metallurgy)DielectricCondensed Matter PhysicsFerroelectricityNanocrystalline materialElectronic Optical and Magnetic MaterialsAmorphous solidCondensed Matter::Materials ScienceCrystallographyCalcium titanatechemistry.chemical_compoundchemistryPerovskite (structure)Physical Review B
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Elimination of cyanobacteria and microcystins in irrigation water—effects of hydrogen peroxide treatment

2020

AbstractCyanobacterial blooms pose a risk to wild and domestic animals as well as humans due to the toxins they may produce. Humans may be subjected to cyanobacterial toxins through many routes, e.g., by consuming contaminated drinking water, fish, and crop plants or through recreational activities. In earlier studies, cyanobacterial cells have been shown to accumulate on leafy plants after spray irrigation with cyanobacteria-containing water, and microcystin (MC) has been detected in the plant root system after irrigation with MC-containing water. This paper reports a series of experiments where lysis of cyanobacteria in abstracted lake water was induced by the use of hydrogen peroxide and…

CyanobacteriaIrrigationAgricultural IrrigationMicrocystinsHydrogenHealth Toxicology and Mutagenesis0208 environmental biotechnologychemistry.chemical_element02 engineering and technologyMicrocystin010501 environmental sciencesCyanobacteriaWaste Disposal Fluid01 natural scienceschemistry.chemical_compoundPhytoplanktonAnimalsHumansEnvironmental ChemistryEcotoxicologyHydrogen peroxideFinland0105 earth and related environmental scienceschemistry.chemical_classificationbiologyfungiSpinachHydrogen PeroxideGeneral MedicineIrrigation waterbiology.organism_classificationPollution6. Clean water020801 environmental engineeringLakeschemistryEnvironmental chemistrySpinachWater MicrobiologyResearch ArticleEnvironmental Science and Pollution Research
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The structural plasticity of the C terminus of p21Cip1 is a determinant for target protein recognition.

2003

The cyclin-dependent kinase inhibitory protein p21(Cip1) might play multiple roles in cell-cycle regulation through interaction of its C-terminal domain with a defined set of cellular proteins such as proliferating cell nuclear antigen (PCNA), calmodulin (CaM), and the oncoprotein SET. p21(Cip1) could be described as an intrinsically unstructured protein in solution although the C-terminal domain adopts a well-defined extended conformation when bound to PCNA. However, the molecular mechanism of the interaction with CaM and the oncoprotein SET is not well understood, partly because of the lack of structural information. In this work, a peptide derived from the C-terminal domain of p21(Cip1) …

Cyclin-Dependent Kinase Inhibitor p21Models MolecularMagnetic Resonance SpectroscopyCalmodulinChromosomal Proteins Non-HistoneProtein ConformationPeptideBiologyLigandsBiochemistryBinding CompetitiveDomain (software engineering)Molecular recognitionCalmodulinCyclinsProliferating Cell Nuclear AntigenEscherichia coliHumansHistone ChaperonesMolecular Biologychemistry.chemical_classificationC-terminusCircular DichroismOrganic ChemistryCell CycleProteinsPeptide FragmentsCell biologyDNA-Binding ProteinschemistryBiochemistrybiology.proteinMolecular MedicineTarget proteinAlpha helixBinding domainTranscription FactorsChembiochem : a European journal of chemical biology
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Neuronal Bioenergetics and Acute Mitochondrial Dysfunction: A Clue to Understanding the Central Nervous System Side Effects of Efavirenz

2014

Background. Neurological pathogenesis is associated with mitochondrial dysfunction and differences in neuronal/glial handling of oxygen and glucose. The main side effects attributed to efavirenz involve the CNS, but the underlying mechanisms are unclear. Methods. Human cell lines and rat primary cultures of neurons and astrocytes were treated with clinically relevant efavirenz concentration. Results. Efavirenz alters mitochondrial respiration, enhances reactive oxygen species generation, undermines mitochondrial membrane potential, and reduces adenosine triphosphate (ATP) levels in a concentration-dependent fashion in both neurons and glial cells. However, it activates adenosine monophospha…

CyclopropanesCell SurvivalCell RespirationPharmacologyMitochondrionBiologymedicine.disease_causechemistry.chemical_compoundOxygen ConsumptionHIV-associated neurocognitive disordersSuperoxidesnitric oxideCell Line TumorneurotoxicitymedicineAnimalsHumansImmunology and AllergyGlycolysisRats WistarMembrane Potential MitochondrialNeuronsMembrane potentialDose-Response Relationship DrugNeurotoxicityHIVefavirenzmedicine.diseasecentral nervous systemAdenosineBenzoxazinesMitochondriaRatsmitochondriaInfectious Diseasesmedicine.anatomical_structurechemistrynervous systemAlkynesAstrocytesReverse Transcriptase InhibitorsNeurogliaEnergy MetabolismNeurogliaAdenosine triphosphateOxidative stressmedicine.drug
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ER stress in human hepatic cells treated with Efavirenz: Mitochondria again

2013

Background & Aims ER stress is associated with a growing number of liver diseases, including drug-induced hepatotoxicity. The non-nucleoside analogue reverse transcriptase inhibitor Efavirenz, a cornerstone of the multidrug strategy employed to treat HIV1 infection, has been related to the development of various adverse events, including metabolic disturbances and hepatic toxicity, the mechanisms of which remain elusive. Recent evidence has pinpointed a specific mitochondrial effect of Efavirenz in human hepatic cells. This study assesses the induction of ER stress by Efavirenz in the same model and the implication of mitochondria in this process. Methods Primary human hepatocytes and Hep3B…

CyclopropanesEfavirenzXBP1Anti-HIV AgentsMitochondria LiverMitochondrionBiologyPharmacologyModels BiologicalCell Linechemistry.chemical_compoundMicroscopy Electron TransmissionDownregulation and upregulationHumansSide effectsEndoplasmic Reticulum Chaperone BiPCells CulturedHepatologyEndoplasmic reticulumHepatotoxicityATF4HIVEndoplasmic Reticulum StressHIV Reverse TranscriptaseBenzoxazinesMitochondriachemistryAlkynesHepatocytesHepatic stellate cellUnfolded protein responseReverse Transcriptase InhibitorsThapsigarginCalciumEfavirenzER stressBiomarkersJournal of Hepatology
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Enhanced oxidative stress and increased mitochondrial mass during Efavirenz-induced apoptosis in human hepatic cells

2010

BACKGROUND AND PURPOSE Efavirenz (EFV) is widely used in the treatment of HIV-1 infection. Though highly efficient, there is growing concern about EFV-related side effects, the molecular basis of which remains elusive. EXPERIMENTAL APPROACH In vitro studies were performed to address the effect of clinically relevant concentrations of EFV (10, 25 and 50 mu M) on human hepatic cells. KEY RESULTS Cellular proliferation and viability were reduced in a concentration-dependent manner. Analyses of the cell cycle and several cell death parameters (chromatin condensation, phosphatidylserine exteriorization, mitochondrial proapoptotic protein translocation and caspase activation) revealed that EFV tr…

CyclopropanesMalehepatotoxicityCarcinoma HepatocellularTime FactorsAnti-HIV AgentsCell SurvivalApoptosisMitochondria LiverPhosphatidylserinesAntioxidantsSuperoxidesHumansChromansantiretroviral drugsCell Proliferationreactive oxygen speciesDose-Response Relationship DrugCell CycleLiver NeoplasmsChromatin Assembly and DisassemblyResearch PapersGlutathioneBenzoxazinesmitochondriaOxidative Stressside effectscell deathLiverAlkynesFemaleEfavirenzApoptosis Regulatory ProteinsHeLa Cells
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