Search results for "phosphatidylserine"

showing 10 items of 55 documents

Anti-proliferative effect of main dietary phytosterols and β-cryptoxanthin alone or combined in human colon cancer Caco-2 cells through cytosolic Ca+…

2015

β-cryptoxanthin (β-Cx) and phytosterols (Ps) have potential against different cancer types, including colon cancer. However, their combined action has not been reported so far. Human colon cancer Caco-2 cells were treated 24 h with β-Cx and/or main dietary Ps (β-sitosterol, campesterol and stigmasterol), alone or in combination, at concentrations compatible with physiological human serum levels. A decrease in cell viability due to apoptosis (rise in sub-G1 population and exposure of membrane phosphatidylserine) was accompanied with dephosphorylation of BAD, mitochondrial depolarization and caspase 3-dependent PARP cleavage, with intracellular Ca2+ influx and increase of RONS levels as initi…

PopulationMedicine (miscellaneous)ApoptosisPharmacologymedicine.disease_causeβ-Cryptoxanthinchemistry.chemical_compoundSettore BIO/10 - BiochimicamedicineTX341-641educationCaco-2 cellsCaspaseeducation.field_of_studyNutrition and DieteticsbiologyNutrition. Foods and food supplyCancerPhytosterolsPhosphatidylserinemedicine.diseaseAnti-proliferation Apoptosis β-Cryptoxanthin Caco-2 cells PhytosterolsBiochemistrychemistryCaco-2ApoptosisAnti-proliferationbiology.proteinIntracellularOxidative stressFood Science
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Short-term exposure to cadmium affects the expression of stress response and apoptosis-related genes in immortalized epithelial cells from the human …

2009

Abstract It is known that cadmium (Cd) evokes cell responses that not only involve protective reactions against toxicity but also induces cell death. Increasing interest has been recently focused on the elucidation of the cellular and molecular aspects of Cd-dependent regulation of gene expression in different model systems. Here, we examined the effects of short-term (24 h) exposure of immortalized non-tumoral HB2 cells from human breast epithelium to CdCl2 at 50 μM concentration, corresponding to the IC50 for this time of incubation. The possible occurrence of apoptosis-related events was evaluated via analysis of the physical state of the DNA and of the membrane localization of phosphaty…

Programmed cell deathCellApoptosisPhosphatidylserinesBiologyToxicologyCell LineInhibitory Concentration 50Heat shock proteinGene expressionmedicineHumansSettore BIO/06 - Anatomia Comparata E CitologiaGeneRegulation of gene expressionCell DeathCell MembraneEpithelial CellsDNAGeneral MedicineCell biologyGene Expression Regulation Neoplasticmedicine.anatomical_structurecadmium gene expression apoptosis stress response epithelial cellsCell cultureApoptosisFemaleCadmiumToxicology in Vitro
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Apoptotic Cell Debris and Phosphatidylserine-Containing Lipid Vesicles Induce Apolipoprotein J (Clusterin) Gene Expression in Vital Fibroblasts

2001

The molecular events in cells undergoing programmed cell death (apoptosis) are well studied; however, the response of the surviving neighbor cells to local cell death is largely uncharacterized. Apolipoprotein J (clusterin) is an 80-kDa glycoprotein that has been implied in cytoprotection of the vital cells, presumably by assisting in the clearance of apoptotic vesicles and membrane remnants. Its mRNA is specifically up-regulated in the vital cells of apoptotic tissues. The molecular mechanisms, however, leading to this response are not known. We here show that exposure of vital fibroblasts to apoptotic vesicles, disrupted vital cells, and trypsin-treated membrane remnants induces apoJ mRNA…

Programmed cell deathEndocytic cycleGene ExpressionApoptosisPhosphatidylserinesCell Linechemistry.chemical_compoundCricetinaeAnimalsTrypsinGlycoproteinsClusterinbiologyVesicleCell BiologyPhosphatidylserinePhosphatidic acidFibroblastsLipid MetabolismMolecular biologyCytoprotectionRatsCell biologyClusterinchemistryApoptosisbiology.proteinMolecular ChaperonesExperimental Cell Research
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Characterization of cells with different mitochondrial membrane potential during apoptosis.

2005

Background Until now, the simultaneous analysis of several parameters during apoptosis, including DNA content and mitochondrial membrane potential (ΔΨ), has not been possible because of the spectral characteristics of the commonly used dyes. Using polychromatic flow cytometry based upon multiple laser and UV lamp excitation, we have characterized cells with different ΔΨ during apoptosis. Methods U937 cells were treated with the flavonoid quercetin (Qu) and stained with JC-1 to detect ΔΨ, propidium iodide (PI) for cell viability, Hoechst 33342 for DNA content, Annexin V conjugated with Alexa Fluor-647 for detection of phosphatidilserine (PS) exposure, marker of early apoptosis, or Mitotracke…

Programmed cell deathHistologyCell Membrane PermeabilityCell Survivalpolychromatic flow cytometry • mitochondrial membrane potential • apoptosis • JC-1 • propidium iodide • Hoechst • Annexin-VPopulationApoptosisHL-60 CellsDNA FragmentationPhosphatidylserinesBiologyPathology and Forensic MedicineFlow cytometryMembrane Potentialschemistry.chemical_compoundAnnexinCell Line TumormedicineHumansViability assayPropidium iodideeducationFluorescent Dyeseducation.field_of_studymedicine.diagnostic_testDaunorubicinCell BiologyDNAIntracellular MembranesU937 CellsCarbocyaninesFlow CytometryMolecular biologyMitochondriachemistryApoptosisCell cultureDoxorubicinLeukocytes MononuclearBenzimidazolesQuercetinCytometry. Part A : the journal of the International Society for Analytical Cytology
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Phytochemical indicaxanthin suppresses 7-ketocholesterol-induced THP-1 cell apoptosis by preventing cytosolic Ca(2+) increase and oxidative stress.

2012

7-Ketocholesterol (7-KC)-induced apoptosis of macrophages is considered a key event in the development of human atheromas. In the present study, the effect of indicaxanthin (Ind), a bioactive pigment from cactus pear fruit, on 7-KC-induced apoptosis of human monocyte/macrophage THP-1 cells was investigated. A pathophysiological condition was simulated by using amounts of 7-KC that can be reached in human atheromatous plaque. Ind was assayed within a micromolar concentration range, consistent with its plasma level after dietary supplementation with cactus pear fruit. Pro-apoptotic effects of 7-KC were assessed by cell cycle arrest, exposure of phosphatidylserine at the plasma membrane, varia…

Programmed cell deathPyridinesCellMedicine (miscellaneous)Apoptosismedicine.disease_causeMonocytesCell Linechemistry.chemical_compoundCytosolmedicineHumansSulfhydryl CompoundsKetocholesterolsNutrition and DieteticsChemistryPlant ExtractsMonocyteMacrophagesNF-kappa BNADPH OxidasesOpuntiaPhosphatidylserineAtherosclerosisPlaque AtheroscleroticCell biologyBetaxanthinsMitochondriaCytosolOxidative Stressmedicine.anatomical_structureApoptosisNADPH Oxidase 4FruitDietary SupplementsCalciumReactive Oxygen SpeciesIndicaxanthinOxidative stressPhytotherapyThe British journal of nutrition
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Macrophage-mediated clearance of cells undergoing caspase-3-independent death

2003

Little is known of the functions of caspases in mediating the surface changes required for phagocytosis of dying cells. Here we investigate the role played by the effector caspase, caspase-3 in this process using the caspase-3-defective MCF-7 breast carcinoma line and derived caspase-3-expressing transfectants. Our results indicate that, while certain typical features of apoptosis induced by etoposide - namely classical morphological changes and the ability to degrade DNA into oligonucleosomal fragments - are caspase-3-dependent, loss of cell adhesion to plastic and the capacity to interact with, and to be phagocytosed by, human monocyte-derived macrophages - both by CD14-dependent and CD14…

Programmed cell deathTime FactorsBlotting WesternGreen Fluorescent ProteinsLipopolysaccharide ReceptorsApoptosisCaspase 3PhosphatidylserinesDNA FragmentationTransfectionCaspase 7Proinflammatory cytokinePhagocytosisCell Line TumorSettore BIO/10 - BiochimicaHumansMacrophageAnnexin A5Cell adhesionCytokineMolecular BiologyCells CulturedCaspaseEtoposideCaspase 7InflammationCell DeathbiologyCaspase 3MacrophagesDNACell BiologyCaspaseCell biologyEnzyme ActivationLuminescent ProteinsApoptosisCaspasesbiology.proteinCytokinesElectrophoresis Polyacrylamide GelCell Death & Differentiation
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Pyrrolo[3,4-h]quinolinones a new class of photochemotherapeutic agents

2011

Abstract Pyrrolo[3,4- h ]quinolin-2-ones were synthesized as nitrogen isosters of the angular furocoumarin angelicin, with the aim of obtaining new photochemotherapeutic agents with increased antiproliferative activity and lower undesired toxic effects. A versatile synthetic pathway was approached to allow the isolation of derivatives of the new ring system with a good substitution pattern on the pyrrole moiety. Photobiological screenings of the new compounds revealed a potent phototoxic effect and a great UVA dose dependence, reaching IC 50 values at submicromolar level. The induced cellular photocytotoxicity was related to apoptosis with the involvement of mitochondria and lysosomes, alte…

Pyrrolo[3; 4-h]quinolinones; Angelicin heteroanalogues; Photochemotherapeutic agents; PhototoxicityStereochemistryClinical BiochemistryMembrane lipid peroxidationPharmaceutical ScienceHL-60 CellsPhosphatidylserinesQuinolonesMitochondrionBiochemistryPhototoxicityJurkat CellsStructure-Activity Relationshipchemistry.chemical_compoundPhotochemotherapeutic agentsAngelicinCell Line TumorDrug DiscoveryHumansMoietyFluorometryPyrrolesPyrrolo[3Molecular BiologyPyrrolePyrrolo[34-h]quinolinoneFurocoumarinCell CycleOrganic Chemistry4-h]quinolinonesDNAPhotochemical ProcessesSettore CHIM/08 - Chimica FarmaceuticaAngelicin heteroanaloguesPhotochemotherapeutic agentPhotochemotherapychemistryApoptosisMolecular MedicineLipid PeroxidationPhototoxicityAngelicin heteroanalogueSubcellular FractionsBioorganic & Medicinal Chemistry
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Phosphatidylserine liposomes reduce inflammatory response, mycobacterial viability and HIV replication in coinfected human macrophages

2021

AbstractChronic immune activation is the key pathogenetic event of Mycobacterium tuberculosis-human immunodeficiency virus (HIV) coinfection. We assessed the therapeutic value of phosphatidylserine-liposome (PS-L) in an in vitro model of M. tuberculosis-HIV coinfection. PS-L reduced nuclear factor-κB activation and the downstream production of tumor necrosis factor-α (TNF-α), interleukin-1β (IL-1β), and IL-6 in bacille Calmette-Guérin-infected macrophages and of TNF-α and IL-1β in M. tuberculosis-infected and M. tuberculosis-HIV–coinfected macrophages. Importantly, a significant reduction of intracellular M. tuberculosis viability and HIV replication were also observed. These results suppor…

Settore MED/04 - Patologia GeneraleTumor Necrosis Factor-alphaMacrophagesHIVHIV InfectionsMycobacterium tuberculosisPhosphatidylserinesVirus ReplicationSettore BIO/19Host-Directed TherapycoinfectionInfectious DiseasesLiposomesliposomeImmunology and AllergyHumansTuberculosisPhosphatidylserine
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Scrutiny of annexin A1 mediated membrane-membrane interaction by means of a thickness shear mode resonator and computer simulations.

2004

The dissipational quartz crystal microbalance (D-QCM) technology was applied to monitor the adsorption of vesicles to membrane-bound annexin A1 by simultaneously reading out the shifts in resonance frequency and dissipation. Solid-supported membranes (SSMs) composed of a chemisorbed octanethiol monolayer and a physisorbed 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine/1-palmitoyl-2-oleoyl-sn-glycero-3-phosphoserine monolayer were immobilized on the gold electrode of a 5 MHz quartz plate. Adsorption and desorption of annexin A1 to the SSM was followed by means of the QCM technique. After nonbound annexin A1 was removed from solution, the second membrane binding was monitored by the D-QCM t…

Time FactorsSurface PropertiesAnalytical chemistryBiosensing TechniquesPhosphatidylserinesAdsorptionAnnexinDesorptionMonolayerElectrochemistryGeneral Materials ScienceComputer SimulationSulfhydryl CompoundsSpectroscopyAnnexin A1ChemistryVesicleMembranes ArtificialSurfaces and InterfacesQuartz crystal microbalanceQuartzCondensed Matter PhysicsMembranePhosphatidylcholinesCalciumAdsorptionGoldAnnexin A1Langmuir : the ACS journal of surfaces and colloids
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Partially Reversible Adsorption of Annexin A1 on POPC/POPS Bilayers Investigated by QCM Measurements, SFM, and DMC Simulations

2005

The kinetics of annexin A1 binding to solid-supported lipid bilayers consisting of 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC)/1-palmitoyl-2-oleoyl-sn-glycero-3-phosphoserine (POPS; 4:1) has been investigated as a function of the calcium ion concentration in the bulk phase. Quartz crystal microbalance measurements in conjunction with scanning force microscopy, fluorescence microscopy, and computer simulations indicate that at a given Ca2+ concentration annexin A1 adsorbs irreversibly on membrane domains enriched in POPS. By contrast, annexin A1 adsorbs reversibly on the POPC-enriched phase, which is composed of single POPS molecules embedded within a POPC matrix. The overall are…

Time FactorsSurface PropertiesLipid BilayersKineticsAnalytical chemistryBiosensing TechniquesPhosphatidylserinesMicroscopy Atomic ForceBiochemistrychemistry.chemical_compoundAdsorptionPhase (matter)MicroscopyComputer SimulationParticle SizeLipid bilayerMolecular BiologyPOPCAnnexin A1ChemistryOrganic Chemistrytechnology industry and agricultureMembranes ArtificialQuartz crystal microbalanceMembraneMicroscopy FluorescencePhosphatidylcholinesMolecular Medicinelipids (amino acids peptides and proteins)AdsorptionStress MechanicalMonte Carlo MethodChemBioChem
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