Search results for "phytoestrogen"

showing 10 items of 43 documents

Sex Differences in Mitochondrial Antioxidant Gene Expression

2020

Females live longer than males. This could be in part due to the higher levels of estrogens in females, which protect them against aging. Physiological concentrations of estrogens have antioxidant effects as they induce the expression of manganese superoxide dismutase and glutathione peroxidase by stimulating estrogen receptors and the mitogen-activated protein kinase and nuclear factor kappa B pathways. However, estrogens can have undesirable effects such as they are feminizing to males, so other alternatives need to be searched. Phytoestrogens are good candidates as they can also bind to estrogens receptors, and in fact, they are able to mimic the antioxidant properties of estrogens. It i…

chemistry.chemical_classificationmedicine.medical_specialtyAntioxidantmedicine.medical_treatmentGlutathione peroxidaseEstrogen receptorBiologyManganese Superoxide Dismutasechemistry.chemical_compoundEndocrinologychemistryInternal medicineGene expressionmedicinePhytoestrogensProtein kinase AReceptorhormones hormone substitutes and hormone antagonists
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Acute effects of three isoflavone class phytoestrogens and a mycoestrogen on cerebral microcirculation.

2007

Phytoestrogens and mycoestrogens are naturally occurring plant and fungus secondary metabolites with estrogen-like structure and/or actions. We aimed to check the hypothesis that phytoestrogens and mycoestrogens, due to their ability to elicit cerebral vasodilation, can induce acute increases in brain blood perfusion. For this purpose, we continuously recorded cerebrocortical perfusion by laser-Doppler flowmetry in anesthetized rats receiving intracarotid infusions (1 mg/kg) of one of the following estrogenic compounds: biochanin A, daidzein, genistein or zearalanone. We have shown the ability of two isoflavone class phytoestrogens (daidzein and biochanin A) and the mycoestrogen zearalanone…

Maleendocrine systemmedicine.medical_specialtyPharmaceutical SciencePhytoestrogensPharmacologyBiochanin Achemistry.chemical_compoundCerebral circulationInternal medicineDrug DiscoverymedicineAnimalsZearalanoneRats WistarPharmacologyChemistryMicrocirculationDaidzeinfood and beveragesMycoestrogenBrainIsoflavonesIsoflavonesRatsEndocrinologyComplementary and alternative medicineCerebral blood flowCerebrovascular CirculationMolecular MedicineZearalenonePhytoestrogensPhytomedicine : international journal of phytotherapy and phytopharmacology
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Effects of soy isoflavones on endothelial function in healthy postmenopausal women.

2005

Objective: To evaluate the effects of soy isoflavone administration on endothelial function in healthy postmenopausal women. Design: Sixty naturally postmenopausal women were randomly assigned to receive isoflavone or placebo tablets for 6 months. Endothelium-dependent vasodilatation was measured by brachial reactivity technique along with levels of plasma soluble intercellular adhesion molecule-1 (ICAM-1), vascular cell adhesion molecule-1 (VCAM-1), E-selectin, P-selectin and soluble thrombomodulin, von Willebrand factor, and tissue plasminogen activator. Differences between endothelium-dependent and endothelium-independent vasodilatation were assessed by evaluating brachial reactivity par…

medicine.medical_specialtyAdhesion moleculeEndotheliumBrachial ArteryGenisteinVascular Cell Adhesion Molecule-1PhytoestrogensFibrinogenAdhesion molecule; Endothelial function; Postmenopausal womenchemistry.chemical_compoundVon Willebrand factorInternal medicineMedicineHumansProspective StudiesReactive hyperemiabiologybusiness.industryObstetrics and GynecologyEndothelial functionIsoflavonesMiddle AgedIntercellular Adhesion Molecule-1Postmenopausal womenIsoflavoneGenisteinIsoflavonesDietPostmenopauseVasodilationProspective StudieP-SelectinEndocrinologymedicine.anatomical_structurechemistryPhytoestrogencardiovascular systembiology.proteinFemaleEndothelium VascularSoybeansSoybeanbusinessE-SelectinPlasminogen activatorHumanLipoproteinmedicine.drugMenopause (New York, N.Y.)
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Resveratrol, a polyphenolic phytoalexin present in red wine, enhances expression and activity of endothelial nitric oxide synthase.

2002

Background— Estrogens can upregulate endothelial nitric oxide synthase (eNOS) in human endothelial cells by increasing eNOS promoter activity and enhancing the binding activity of the transcription factor Sp1. Resveratrol, a polyphenolic phytoalexin found in grapes and wine, has been reported to act as an agonist at the estrogen receptor. Therefore, we tested the effect of this putative phytoestrogen on eNOS expression in human endothelial cells. Methods and Results— Incubation of human umbilical vein endothelial cells (HUVEC) and HUVEC-derived EA.hy 926 cells with resveratrol for 24 to 72 hours upregulated eNOS mRNA expression in a time- and concentration-dependent manner (up to 2.8-fold)…

PolymersRNA StabilityElectrophoretic Mobility Shift AssayWineResveratrolUmbilical veinchemistry.chemical_compoundEnosStilbenesPromoter Regions GeneticCells Culturedchemistry.chemical_classificationbiologyPhytoalexinEstrogen Antagonistsfood and beveragesNitric Oxide Synthase Type IIIUp-RegulationNitric oxide synthasemedicine.anatomical_structureReceptors EstrogenEnzyme InductionCardiology and Cardiovascular MedicineSesquiterpenesmedicine.medical_specialtyEndotheliumNitric Oxide Synthase Type IIINuclease Protection AssaysEnzyme ActivatorsPhytoestrogensNitric OxidePhenolsPhytoalexinsPhysiology (medical)Internal medicinemedicineHumansEstrogens Non-SteroidalRNA MessengerFlavonoidsSp1 transcription factorPlant ExtractsTerpenesPolyphenolsbiology.organism_classificationMolecular biologyIsoflavonesEnzyme ActivationEndocrinologychemistryResveratrolbiology.proteinEndothelium VascularPlant PreparationsNitric Oxide SynthaseCirculation
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Characterization of particle morphology of biochanin A molecularly imprinted polymers and their properties as a potential sorbent for solid-phase ext…

2014

Abstract Molecularly imprinted polymers (MIPs) with biochanin A as a template were obtained using a bulk polymerization with non-covalent imprinting approach. The polymers were prepared in acetonitrile as porogen, using ethylene glycol dimethacrylate (EDMA) as cross-linking agent. The synthesis, with an application of 1′,1′-azobis(cyclohexanecarbonitrile) (ACHN) as an initiator, has been performed thermally. During the synthesis process the effect of different functional monomers such as methacrylic acid (MAA), acrylamide (AA) and 4-vinylpyridine (4-VP) was investigated. The application of nitrogen sorption porosimetry, scanning electron microscopy (SEM), and Fourier transform infrared spec…

AcetonitrilesMaterials scienceNitrogenPolymersPyridinesEthylene glycol dimethacrylateBioengineeringPolymerizationBiochanin ABiomaterialschemistry.chemical_compoundSpectroscopy Fourier Transform Infraredsolid-phase extractionSolid phase extractionFourier transform infrared spectroscopyChromatography High Pressure Liquidchemistry.chemical_classificationphytoestrogensAcrylamideChromatographySolid Phase ExtractionMolecularly imprinted polymerPolymerGenisteinIsoflavonesCross-Linking ReagentschemistryMethacrylic acidMechanics of MaterialsadsorptionMicroscopy Electron ScanningMethacrylatesmolecular imprintingMolecular imprintingNuclear chemistryMaterials Science & Engineering C-Materials for Biological Applications
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Role of mitochondrial oxidative stress to explain the different longevity between genders. Protective effect of estrogens

2006

Females live longer than males. Work from our laboratory has shown that this may be due to the up-regulation of longevity-associated genes by estrogens. Estrogens bind to the estrogen receptors and subsequently activate the mitogen activated protein kinase and nuclear factor kappa B signalling pathways, resulting in an up-regulation of antioxidant enzymes. Estrogen administration, however, has serious undesirable effects and of course, cannot be administered to males because of its powerful feminizing effects. Thus, we tested the effect of genistein, a phytoestrogen of high nutritional importance whose structure is similar to estradiol, on the regulation of the expression of antioxidant, lo…

Malemedicine.medical_specialtymedicine.drug_classmedia_common.quotation_subjectLongevityEstrogen receptorGenisteinPhytoestrogensBiologymedicine.disease_causeBiochemistryAntioxidantschemistry.chemical_compoundSex FactorsDownregulation and upregulationInternal medicinemedicineAnimalsEstrogen Receptor betaHumansmedia_commonSuperoxide DismutaseLongevityEstrogensGeneral MedicineMitochondriaRatsUp-RegulationOxidative StressEndocrinologychemistryEstrogenFemalePhytoestrogensOxidative stressHormoneFree Radical Research
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Oestradiol or genistein rescues neurons from amyloid beta-induced cell death by inhibiting activation of p38.

2007

Oestrogenic compounds have been postulated as neuroprotective agents. This prompted us to investigate their mechanism action in neurons in primary culture. Cells were pretreated with physiological concentrations of 17-beta estradiol (0.2 nm) or with nutritionally relevant concentrations of genistein (0.5 microm), and 48 h later treated with 5 microm of amyloid beta (Abeta) for 24 h. We found that Abeta increased oxidative stress, measured as peroxide levels or oxidized glutathione/reduced glutathione ratio, which in turn, caused phosphorylation of p38 MAP kinase. Amyloid beta subsequently induced neuronal death. Inhibiting the MAP kinase pathway prevented cell death, confirming the role of …

MAPK/ERK pathwayAgingProgrammed cell deathmedicine.medical_specialtyAmyloid betaCell Survivalp38 mitogen-activated protein kinasesGenisteinPhytoestrogensIn Vitro Techniquesmedicine.disease_causeNeuroprotectionp38 Mitogen-Activated Protein Kinaseschemistry.chemical_compoundInternal medicinemedicineAnimalsCells CulturedCerebral CortexNeuronsAmyloid beta-PeptidesbiologyCell DeathEstradiolEstrogensCell BiologyGlutathioneGenisteinMitochondriaRatsOxidative StressEndocrinologychemistrybiology.proteinOxidation-ReductionOxidative stressAging cell
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Modulation of longevity-associated genes by estrogens or phytoestrogens.

2008

Abstract Females live longer than males. We have shown that the higher levels of estrogens in females protect them against aging, by up-regulating the expression of antioxidant, longevity-related genes, such as that of selenium-dependent glutathione peroxidase (GPx) and Mn-superoxide dismutase (Mn-SOD). Both estradiol and genistein (the most abundant phytoestrogen in soybeans) share chemical properties which confer antioxidant features to these compounds. However, the low concentration of estrogens and phytoestrogens make it unlikely that they exhibit significant antioxidant capacity in the organism. Physiological concentrations of estrogens and nutritionally relevant concentrations of geni…

MAPK/ERK pathwayMalemedicine.medical_specialtyAntioxidantFree Radicalsmedicine.medical_treatmentClinical BiochemistryLongevityGenisteinPhytoestrogensmedicine.disease_causeBiochemistryAntioxidantschemistry.chemical_compoundInternal medicinemedicineAnimalsHumansMolecular BiologyEstrogen receptor betachemistry.chemical_classificationSex CharacteristicsSuperoxide DismutaseGlutathione peroxidasefood and beveragesEstrogensMitochondriaUp-RegulationEndocrinologychemistryPhytoestrogensFemaleSignal transductionReactive Oxygen SpeciesOxidative stressSignal TransductionBiological chemistry
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Daidzein has neuroprotective effects through ligand-binding-independent PPARγ activation.

2011

Phytoestrogens are a group of plant-derived compounds that include mainly isoflavones like daidzein. Phytoestrogens prevent neuronal damage and improve outcome in experimental stroke; however, the mechanisms of this neuroprotective action have not been fully elucidated. In this context, it has been postulated that phytoestrogens might activate the peroxisome proliferator-activated receptor-γ (PPARγ), which exerts neuroprotective effects in several settings. The aim of this study was to determine whether the phytoestrogen daidzein elicits beneficial actions in neuronal cells by mechanisms involving activation of PPARγ. Our results show that daidzein (0.05-5 μM) decreases cell death induced b…

endocrine systemmedicine.drug_classPyridinesPeroxisome proliferator-activated receptorPharmacologyLigandsNeuroprotectionCellular and Molecular Neurosciencechemistry.chemical_compoundmedicineSynaptic vesicle recyclingAnimalsReceptorCells Culturedchemistry.chemical_classificationNeuronsDaidzeinfood and beveragesCell BiologyIsoflavonesReceptor antagonistIsoflavonesRatsOxygenPPAR gammaGlucoseNeuroprotective AgentschemistryBenzamidesPhytoestrogensNeurochemistry international
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Abnormal peripubertal development of the rat mammary gland following exposure in utero and during lactation to a mixture of genistein and the food co…

2011

The impact of early exposure to endocrine disruptor mixtures on mammary gland development is poorly known. Here, we identify the effects of a conception to weaning exposure of rats to the phytoestrogen genistein (G) and/or the antiandrogen vinclozolin (V) at 1 mg/kg-d, alone or in association. Using several approaches, we found that G- and GV-exposed rats displayed significantly greater epithelial branching and proliferation, wider terminal end buds than controls at PND35, as well as ductal hyperplasia and periductal fibrosis. Focal branching defects were present in V-exposed rats. An increased ER and AR expression was observed in G- and CV- as compared to V-exposed rats at PND35. Surprisin…

[SDV.BA] Life Sciences [q-bio]/Animal biology[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionreceptorGenisteinmammary gland developmentsprague-dawley ratsToxicologyAntiandrogen[ SDV.BA ] Life Sciences [q-bio]/Animal biologychemistry.chemical_compound0302 clinical medicineLactationSexual MaturationVinclozolinReceptorOxazolesfemale mice0303 health sciences[SDV.BA]Life Sciences [q-bio]/Animal biologyendocrine disruptiondifferentiationGenisteinDrug Combinationsmedicine.anatomical_structuregestational and lactational exposureEndocrine disruptorMaternal ExposureIn utero030220 oncology & carcinogenesisVaginaphytoestrogenFemalemedicine.medical_specialtyanti-androgenbreast-cancer riskmedicine.drug_classgrowthFood ContaminationPhytoestrogensandrogenBiologytransgenic mice03 medical and health sciencesMammary Glands AnimalInternal medicinemedicineAnimalsLactationRats Wistar030304 developmental biologyHyperplasiaBody WeightAndrogen AntagonistsAndrogenRats[SDV.AEN] Life Sciences [q-bio]/Food and Nutritionbisphenol-a alterstumorigenesisEndocrinologychemistrycells[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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