Search results for "poisoning"

showing 10 items of 76 documents

Transglutaminase Type II Plays a Protective Role in Hepatic Injury

2003

The up-regulation of "tissue" transglutaminase (TG2) gene has been shown to occur in various pathologies and can lead to severe liver injury; however, its role in the onset of liver damage has not yet been clarified. To address this issue, we have used two experimental settings: carbon tetrachloride (CCl(4))-induced liver injury in wild-type and TG2 knockout mice; and liver biopsies obtained from a large cohort of hepatitis C virus (HCV)-infected patients. Mice lacking TG2 failed to clear the hepatic necrotic tissue formed in response to prolonged CCl(4) exposure (5 weeks) and 60% of them died before the end of the treatment. By contrast, wild-type mice were able to recover after the toxic …

AdultPathologymedicine.medical_specialtyNecrosisGenotypeTissue transglutaminaseHepatitis C virusCCL4medicine.disease_causeGene Expression Regulation EnzymologicPathology and Forensic MedicineExtracellular matrixMiceNecrosisGTP-Binding ProteinsmedicineAnimalsHumansProtein Glutamine gamma Glutamyltransferase 2Mice KnockoutHepatitisLiver injuryTransglutaminasesbiologyCarbon Tetrachloride PoisoningHepatitis C ChronicMiddle Agedmedicine.diseaseMice Inbred C57BLLiverKnockout mousebiology.proteinmedicine.symptomRegular ArticlesThe American Journal of Pathology
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A quantitative study of the pancuronium antagonism at the motor endplate in human organophosphorus intoxication

1995

Nine patients with organophosphorus (OP) intoxication developing neuromuscular transmission defects were given pancuronium 1, 2, or 4 mg intravenously (IV). Thirteen patient controls with hypoxic encephalopathy received similar dosages. The responses were monitored electrophysiologically using single and repetitive nerve stimulation (20 and 50 Hz). In OP patients, pancuronium did not alter the amplitude of the single CMAP, whereas its repetitive discharges were reduced. Severe neuromuscular blocks were reversed only partially by pancuronium 4 mg. In less severe blocks, 1 and 2 mg resulted in marked improvement. In the patient controls, pancuronium 4 mg induced a severe neuromuscular block b…

AdultTime FactorsPhysiologymedicine.medical_treatmentNeuromuscular transmissionAction PotentialsElectromyographyMotor EndplateSynaptic TransmissionNeuromuscular junctionCellular and Molecular Neurosciencechemistry.chemical_compoundOrganophosphate PoisoningPhysiology (medical)medicineHumansPancuroniumRepetitive nerve stimulationAntidoteNeuromuscular BlockadeMovement DisordersDose-Response Relationship Drugmedicine.diagnostic_testbusiness.industryNeuromuscular DiseasesAcetylcholinesteraseElectric Stimulationmedicine.anatomical_structurechemistryAnesthesiaInjections IntravenousToxicityAcetylcholinesteraseNeurology (clinical)businessMuscle & Nerve
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Massive triiodothyronine intoxication: Efficacy of hemoperfusion?

2002

A case of massive accidental triiodothyronine intoxication (1000-fold the usual therapeutic dose, for 8 days) is reported with important disturbances of cardiovascular and central nervous systems that required intensive care support. Serum free triiodothyronine levels were 4789 pmol L-1 on admittance (normal values, 3.5-6.5 pmol L-1). In the absence of a specific treatment, hemoperfusions were performed but failed to accelerate significantly the decay of blood levels of free triiodothyronine (apparent half-life 25.9 hours; 95% confidence interval: 19.8-37.4 hours). The patient, a young woman, made a satisfactory recovery, in spite of important clinical complications.

Adultmedicine.medical_specialtyCritical CareEndocrinology Diabetes and Metabolismmedicine.medical_treatmentNormal valuesEndocrinologyTherapeutic indexCentral Nervous System DiseasesSerum freeInternal medicineIntensive caremedicineHumansTriiodothyroninebusiness.industryPoisoningHemoperfusionConfidence intervalHemoperfusionEndocrinologyCardiovascular DiseasesFree triiodothyronineAnesthesiaTriiodothyronineFemalebusiness
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WIN55,212-2, a cannabinoid receptor agonist, protects against nigrostriatal cell loss in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine mouse model…

2009

Parkinson's disease (PD) is characterized by the progressive loss of nigrostriatal dopamine neurons leading to motor disturbances and cognitive impairment. Current pharmacotherapies relieve PD symptoms temporarily but fail to prevent or slow down the disease progression. In this study, we investigated the molecular mechanisms by which the non-selective cannabinoid receptor agonist WIN55,212-2 (WIN) protects mouse nigrostriatal neurons from 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-induced neurotoxicity and neuroinflammation. Stereological analyses showed that chronic treatment with WIN (4 mg/kg, intraperitoneal), initiated 24 h after MPTP administration, protected against MPTP-ind…

Agonistmedicine.drug_classbusiness.industryPars compactaGeneral NeuroscienceMPTPCannabinoid Receptor AgonistsSubstantia nigraPharmacologynervous system diseaseschemistry.chemical_compoundnervous systemchemistryDopaminemedicineCannabinoid receptor type 2MPTP Poisoninglipids (amino acids peptides and proteins)businessmedicine.drugEuropean Journal of Neuroscience
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Effects of environmental and occupational lead toxicity and its association with iron metabolism

2021

Abstract Background Discrepancies are present in the findings from clinical trials evaluating a physiological role of iron status in the lead-exposed population. Objective The purpose of this article was to summarize the current understanding of cellular mechanisms of lead toxicity and present a comprehensive review of existing clinical trials related to associations of lead poisoning and iron status. Although an association of iron metabolism pathways that are affected by lead intoxication has been studied, there are still aspects that remain to be elucidated. The existence of additional Pb uptake pathways besides DMT1 transporter-mediated is postulated to non-specifically regulate lead ab…

AnemiaIronPopulationToxicologyLead poisoningOccupational ExposureEnvironmental healthStatistical significancemedicineHumansDrug InteractionsLead (electronics)educationPharmacologyeducation.field_of_studymedicine.diagnostic_testbusiness.industrylead poisoningEnvironmental Exposurelead toxicityIron metabolismmedicine.diseaseClinical trialLeadToxicityEnvironmental PollutantsBlood lead levelbusinessToxicology and Applied Pharmacology
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Therapeutic properties of haemodialysis and blood exchange transfusion in organophosphate poisoning

1976

Human blood was contaminated with nitrostigmine, dimethoate and demeton-S-methyl sulfoxide. It was then dialysed, concentrations of organophosphates were determined and dialysance values calculated. The influence of blood exchange transfusion on poison elimination as well as on the cholinesterase activity of blood, brain and muscle was studied in rats poisoned with nitrostigmine. Haemodialysis was found to be quite an effective method for eliminating demeton-S-methyl sulfoxide and dimethoate, dialysance values of 52.98 ml/min and 59.07 ml/min being found for demeton-S-methyl sulfoxide and dimethoate respectively. Nitrostigmine could not be removed by haemodialysis. These findings suggest th…

Blood transfusionmedicine.medical_treatmentCritical Care and Intensive Care MedicineOrganophosphate poisoning03 medical and health scienceschemistry.chemical_compoundOrganophosphate PoisoningOrganophosphorus Compounds0302 clinical medicineRenal DialysisAnimalsMedicineBlood Transfusion030212 general & internal medicineCholinesteraseParathionbiologyHuman bloodbusiness.industryPoisoningBlood exchange transfusionGeneral Medicinemedicine.diseaseAcetylcholinesteraseRats3. Good healthParathionchemistrySulfoxidesAnesthesiaAcetylcholinesterasebiology.proteinbusinessDimethoate030217 neurology & neurosurgeryEuropean Journal of Intensive Care Medicine
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Optimization of a GF-AAS method for lead testing in blood and urine: A useful tool in acute abdominal pain management in emergency.

2021

Suspicion of lead poisoning is confirmed by its concentration in blood and protoporphyrin red blood cells. At low concentrations, lead influences the synthesis of the heme in the sense of lowering it. Acute and chronic lead intoxication is extremely polymorphic in regards to its clinical manifestations, with digestive, hematological, cardiovascular, renal hepatic and neurological features. The aim of the study was to evaluate the presence of lead in human whole blood and urine harvested before and during chelation treatment in the case of lead poisoning. An atomic absorption spectroscopic method for the analysis of lead was developed using graphite furnace atomic absorption spectrophotomete…

Cancer Researchmedicine.medical_specialtybusiness.industryGeneral MedicineUrineArticlesmedicine.diseaseGastroenterologyLead poisoninglaw.inventionchemistry.chemical_compoundImmunology and Microbiology (miscellaneous)chemistrylawInternal medicineMedicineProtoporphyrinChelation therapybusinessGraphite furnace atomic absorptionLead (electronics)Atomic absorption spectroscopyWhole bloodExperimental and therapeutic medicine
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Effects of a phycotoxin, okadaic acid, on oyster heart cell survival

2008

Okadaic acid (OA) is a dinoflagellate toxin which accumulates in shellfish producing diarrhetic shellfish poisoning (DSP) in humans. It was found that OA is a highly selective inhibitor of protein phosphatase types 1 (PP1) and 2A (PP2A) which produces a marked increase in phosphorylation of several proteins, including p38 mitogen-activated protein (MAP) kinase. The cytotoxicity attributed to OA and the effects on p38 MAP kinase and calcium current were examined in the oyster Crassostrea gigas in this study. Data showed that p38 MAP kinase is strongly expressed in oyster heart and that OA bioaccumulated in cultured heart cells. Hence the effects of OA was tested in vitro and in vivo on oyste…

ChronotropicOysterbiologyKinaseHealth Toxicology and MutagenesisPhosphataseProtein phosphatase 2Okadaic acidPollutionMolecular biologychemistry.chemical_compoundchemistrybiology.animalEnvironmental ChemistryDiarrhetic shellfish poisoningProtein kinase CToxicological & Environmental Chemistry
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rRNA probing of chromosomal DNA of epidemic and sporadic isolates of Salmonella enterica subsp. Enterica serovar kottbus from Northern and Southern I…

1990

Fifty-two strains of Salmonella enterica subsp. enterica serovar Kottbus, identified at the Centres of Enterobacteriaceae of Northern and Southern Italy, were investigated by molecular genetic methods. Thirteen isolates were recovered during two food-poisoning outbreaks that occurred in May 1987 in Lombardy. The rDNA gene restriction patterns, obtained by probing endonuclease cleaved chromosomal DNA with photobiotin labeled Escherichia coli rRNA, revealed some heterogeneity among strains isolated from Southern Italy, whereas Northern Italy isolates exhibited virtually identical banding patterns.

DNA BacterialSerotypeEpidemiologyPopulationmedicine.disease_causeDisease OutbreaksMicrobiologySalmonellaEscherichia coliHumansMedicineGeneEscherichia colibiologybusiness.industryGenetic VariationNucleic Acid HybridizationOutbreakRNA ProbesRibosomal RNAbiology.organism_classificationDNA FingerprintingEnterobacteriaceaeBlotting SouthernItalyRNA RibosomalSalmonella entericaSalmonella Food PoisoningSalmonella enterica subsp. entericabusinessEuropean Journal of Epidemiology
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Staphylococcal food poisoning case and molecular analysis of toxin genes in Staphylococcus aureus strains isolated from food in Sicily, Italy.

2014

A case of staphylococcal food poisoning was observed in two individuals of the same family after consumption of primosale, a semiripened sheep cheese produced in Sicily. Staphylococcus aureus isolated from the cheese produced enterotoxin C (SEC) and carried both the enterotoxin C (sec) and the toxic shock syndrome toxin (tsst-1) gene. Following this case, an extensive survey was conducted on 971 food samples (raw milk, cheese, meat, and food preparations). S. aureus was detected in 102 of 971 food samples, from all types of food with the exception of ricotta cheese. The tsst-1 gene was present in 42% of the strains, either alone or in combination with other toxin genes. The enterotoxin C ge…

DNA BacterialStaphylococcus aureusMeatBacterial ToxinsEnterotoxinBiologySettore BIO/19 - Microbiologia Generalemedicine.disease_causeApplied Microbiology and BiotechnologyMicrobiologyPolymerase Chain ReactionMicrobiologyEnterotoxinsmedicineAnimalsFood scienceGeneSicilyfood safety.SuperantigensToxinToxic shock syndrome toxinRaw milktoxin geneMolecular analysisStaphylococcal Food PoisoningStaphylococcus aureusConsumer Product Safetytypical dairy productStaphylococcus aureuFood Microbiologyfood poisoningAnimal Science and ZoologyDairy ProductsStaphylococcal Food PoisoningFood ScienceFoodborne pathogens and disease
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