Search results for "polymorph"

showing 10 items of 2115 documents

Disruption of the ASTN2 / TRIM32 locus at 9q33.1 is a risk factor in males for Autism Spectrum Disorders, ADHD and other neurodevelopmental phenotypes

2014

Rare copy number variants (CNVs) disrupting ASTN2 or both ASTN2 and TRIM32 have been reported at 9q33.1 by genome-wide studies in a few individuals with neurodevelopmental disorders (NDDs). The vertebrate-specific astrotactins, ASTN2 and its paralog ASTN1, have key roles in glial-guided neuronal migration during brain development. To determine the prevalence of astrotactin mutations and delineate their associated phenotypic spectrum, we screened ASTN2/TRIM32 and ASTN1 (1q25.2) for exonic CNVs in clinical microarray data from 89 985 individuals across 10 sites, including 64 114 NDD subjects. In this clinical dataset, we identified 46 deletions and 12 duplications affecting ASTN2. Deletions o…

MaleReceptors Cell Surface/geneticsAutismChild Development Disorders Pervasive/geneticsGene ExpressionGenome-wide association studyMedical and Health SciencesTripartite Motif ProteinsRisk FactorsReceptors2.1 Biological and endogenous factorsProtein IsoformsNerve Tissue Proteins/geneticsCopy-number variationAetiologyChildGenetics (clinical)Sequence DeletionPediatricGenetics & HeredityGeneticseducation.field_of_studySingle NucleotideArticlesGeneral MedicineExonsBiological SciencesMental HealthPhenotypeAutism spectrum disorderOrgan SpecificityCerebellar cortexChild PreschoolCell SurfaceSpeech delayFemalemedicine.symptomTranscription Initiation SiteAttention Deficit Disorder with Hyperactivity/geneticsChromosomes Human Pair 9HumanPair 9AdultPediatric Research InitiativeChild Development DisordersAdolescentDNA Copy Number VariationsIntellectual and Developmental Disabilities (IDD)Ubiquitin-Protein LigasesPopulationTranscription Factors/geneticsNerve Tissue ProteinsReceptors Cell SurfaceBiologyPolymorphism Single NucleotideChromosomesYoung AdultClinical ResearchProtein Isoforms/geneticsBehavioral and Social ScienceGeneticsmedicineAttention deficit hyperactivity disorderHumansGenetic Predisposition to DiseasePolymorphismPreschooleducationMolecular BiologyGenetic Association StudiesPervasiveGlycoproteinsHuman GenomeNeurosciencesInfant NewbornGlycoproteins/geneticsInfantNewbornmedicine.diseaseBrain DisordersAttention Deficit Disorder with HyperactivityChild Development Disorders PervasiveCase-Control StudiesAutismTranscription Factors
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Genetic variants associated with human eye size are distinct from those conferring susceptibility to myopia

2021

Purpose: Emmetropization requires coordinated scaling of the major ocular components, corneal curvature and axial length. This coordination is achieved in part through a shared set of genetic variants that regulate eye size. Poorly coordinated scaling of corneal curvature and axial length results in refractive error. We tested the hypothesis that genetic variants regulating eye size in emmetropic eyes are distinct from those conferring susceptibility to refractive error.Methods: A genome-wide association study (GWAS) for corneal curvature in 22,180 adult emmetropic individuals was performed as a proxy for a GWAS for eye size. A polygenic score created using lead GWAS variants was tested for…

MaleRefractive errorLinkage disequilibriumgenetic structuresLOCIEmmetropiaGenome-wide association studyPRSS56AXIAL LENGTHLinkage Disequilibrium0302 clinical medicineCORNEAL CURVATURECorneaMyopiarefractive errorChild0303 health sciencesHERITABILITYMiddle Agedgenetic correlationAxial Length Eyemedicine.anatomical_structureeye sizeGROWTHFemaleAdultmedicine.medical_specialtyUK BiobankAdolescentBiologyRefraction OcularGenetic correlationPolymorphism Single NucleotideOCULAR COMPONENT DIMENSIONS03 medical and health sciencesYoung AdultOphthalmologymedicineGeneticsHumansGenetic Predisposition to Disease3125 Otorhinolaryngology ophthalmologyGENOME-WIDE ASSOCIATIONMETAANALYSIS030304 developmental biologyAgedmedicine.diseaseeye diseasesGenetic marker030221 ophthalmology & optometryHuman eyesense organsGenome-Wide Association StudyInvestigative Ophthalmology & Visual Science
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A locus on 2p12 containing the co-regulated MRPL19 and C2ORF3 genes is associated to dyslexia.

2007

DYX3, a locus for dyslexia, resides on chromosome 2p11-p15. We have refined its location on 2p12 to a 157 kb region in two rounds of linkage disequilibrium (LD) mapping in a set of Finnish families. The observed association was replicated in an independent set of 251 German families. Two overlapping risk haplotypes spanning 16 kb were identified in both sample sets separately as well as in a joint analysis. In the German sample set, the odds ratio for the most significantly associated haplotype increased with dyslexia severity from 2.2 to 5.2. The risk haplotypes are located in an intergenic region between FLJ13391 and MRPL19/C2ORF3. As no novel genes could be cloned from this region, we hy…

MaleRibosomal ProteinsCandidate geneLinkage disequilibriumHeterozygoteTranscription GeneticLocus (genetics)BiologyPolymorphism Single NucleotideLinkage DisequilibriumDyslexiaEvolution MolecularMitochondrial Proteins03 medical and health sciences0302 clinical medicineIntergenic regionGene mappingDCDC2GermanyGeneticsmedicineAnimalsHumansFamilyMolecular BiologyGenetics (clinical)FinlandPhylogeny030304 developmental biologyGenetics0303 health sciencesHaplotypeDyslexiaBrainChromosome MappingGeneral Medicinemedicine.diseaseRepressor ProteinsPhenotypeHaplotypesChromosomes Human Pair 2Female030217 neurology & neurosurgeryHuman molecular genetics
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XRCC5 as a Risk Gene for Alcohol Dependence : Evidence from a Genome-Wide Gene-Set-Based Analysis and Follow-up Studies in Drosophila and Humans

2015

Genetic factors play as large a role as environmental factors in the etiology of alcohol dependence. Although genome-wide association studies (GWAS) enable systematic searches for loci not hitherto implicated in the etiology of alcohol dependence, many true findings may be missed due to correction for multiple testing. The aim of the present study was to circumvent this limitation by searching for biological system-level differences, and then following up these findings in humans and animals. Gene-set based analysis of GWAS data from 1333 cases and 2168 controls identified 19 significantly associated gene-sets of which five could be replicated in an independent sample. Clustered in these ge…

MaleRiskAdolescentMedizinGenome-wide association studyBiologyPolymorphism Single NucleotideWhite PeopleAnimals Genetically ModifiedRNA interferenceGermanyGenetic variationAnimalsHumansGene silencingGenetic Predisposition to DiseaseKu AutoantigenGeneGenetic associationPharmacologyGeneticsEthanolAlcohol dependenceDNA HelicasesCentral Nervous System DepressantsPhenotypeAlcoholismPsychiatry and Mental healthDrosophila melanogasterFemaleOriginal ArticleFollow-Up StudiesGenome-Wide Association Study
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The relation between obesity, abdominal fat deposit and the angiotensin-converting enzyme gene I/D polymorphism and its association with coronary hea…

2004

To analyse the relation between overweight, obesity and fat distribution with I/D polymorphism of the angiotensin-converting enzyme (ACE) gene and its association with coronary heart disease (CHD). Cross-sectional, case–control study. A total of 185 cases (141 males) who had suffered at least one episode of CHD and 182 controls (127 males). Body mass index, waist circumference, blood pressure, plasma total cholesterol, triglycerides, HDL cholestrol and fasting glucose were measured with standard methods, genotyping the I/D polymorphism of ACE gene. Obesity and abdominal fat deposit are associated with CHD in women, but not independently. We have found an association between obesity and abdo…

MaleRiskmedicine.medical_specialtyWaistGenotypeEndocrinology Diabetes and MetabolismMedicine (miscellaneous)Coronary DiseaseOverweightPeptidyl-Dipeptidase APolymorphism (computer science)Internal medicineAbdomenMedicineHumansObesityAgedNutrition and DieteticsPolymorphism Geneticbiologybusiness.industryCase-control studyAngiotensin-converting enzymeMiddle Agedmedicine.diseaseObesityBlood pressureEndocrinologyCross-Sectional StudiesLogistic ModelsAdipose TissueCase-Control Studiesbiology.proteinBody ConstitutionFemalemedicine.symptombusinessBody mass indexInternational journal of obesity (2005)
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Differences in the Density of Fungiform Papillae and Composition of Saliva in Patients With Taste Disorders Compared to Healthy Controls

2017

International audience; This study investigated the relation of the fungiform taste papillae density and saliva composition with the taste perception of patients suffering from diagnosed taste disorders. For this purpose, 81 patients and 40 healthy subjects were included. Taste was measured by means of regional and whole mouth chemosensory tests, and electrogustometry. Olfaction was assessed using the Sniffin Sticks. Fungiform papillae were quantified using the "Denver Papillae Protocol for Objective Analysis of Fungiform Papillae". In addition, salivary parameters [flow rate, total proteins, catalase, total anti-oxidative capacity (TAC), carbonic anhydrase VI (caVI), and pH] were determine…

MaleSalivaTastefungiforme papillaePhysiology[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutritionsignal-detection measuresDentistryPhysiologyperceptiontasteBehavioral Neuroscience0302 clinical medicineMedicine030223 otorhinolaryngologyLingual papillaAged 80 and overElectrogustometryTaste PerceptionMiddle AgedTaste BudsSensory SystemsTaste disorderchorda tympani nerveFemalemedicine.symptomperformanceoleic-acidAdultgene polymorphismOlfactiontaste disordersbud density03 medical and health sciencesYoung Adultstomatognathic systemPhysiology (medical)HumansAgedsalivabusiness.industryBeck Depression Inventorytongue-regionAgeusiasensitivityCase-Control Studiesbusinessintensity[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgery
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Association between the APOA2 promoter polymorphism and body weight in Mediterranean and Asian populations: replication of a gene–saturated fat inter…

2011

Objective: The APOA2 gene has been associated with obesity and insulin resistance (IR) in animal and human studies with controversial results. We have reported an APOA2–saturated fat interaction determining body mass index (BMI) and obesity in American populations. This work aims to extend our findings to European and Asian populations. Methods: Cross-sectional study in 4602 subjects from two independent populations: a high-cardiovascular risk Mediterranean population (n=907 men and women; aged 67±6 years) and a multiethnic Asian population (n=2506 Chinese, n=605 Malays and n=494 Asian Indians; aged 39±12 years) participating in a Singapore National Health Survey. Anthropometric, clinical, …

MaleSaturated fatsaturated fatGenotypeEndocrinology Diabetes and MetabolismSaturated fatPopulationMedicine (miscellaneous)BiologyPolymorphism Single NucleotideMediterranean BasinArticleWhite PeopleAPOA2Body Mass IndexAsian PeopleGene interactioninsulin resistanceGenotypegene-diet interactionmedicineHumansGenetic Predisposition to DiseaseObesityeducationGeneAllelesAgedGeneticseducation.field_of_studyNutrition and DieteticsBody WeightInsulin resistancemedicine.diseaseDietary FatsObesityCross-Sectional StudiesCardiovascular DiseasesGene–diet interactionFemaleBody mass indexApolipoprotein A-IIInternational Journal of Obesity
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Association analyses identify 31 new risk loci for colorectal cancer susceptibility

2019

Colorectal cancer (CRC) is a leading cause of cancer-related death worldwide, and has a strong heritable basis. We report a genome-wide association analysis of 34,627 CRC cases and 71,379 controls of European ancestry that identifies SNPs at 31 new CRC risk loci. We also identify eight independent risk SNPs at the new and previously reported European CRC loci, and a further nine CRC SNPs at loci previously only identified in Asian populations. We use in situ promoter capture Hi-C (CHi-C), gene expression, and in silico annotation methods to identify likely target genes of CRC SNPs. Whilst these new SNP associations implicate target genes that are enriched for known CRC pathways such as Wnt …

MaleScienceInheritance Patternscancer genetics/Datasets as Topiccolorectal cancerGenome-wide association studiesPolymorphism Single NucleotideArticleWhite PeopleAsian PeopleRisk FactorsCancer genomicsHumansGenetic Predisposition to Diseaselcsh:ScienceCancer geneticsneoplasmscancer genomicsQgenomiikkaMiddle AgedColorectal cancerdigestive system diseasesperäsuolisyöpäsyöpägeenitGenetic LociCase-Control Studiesgenome-wide association studieslcsh:QsyöpätauditFemaleColorectal NeoplasmsGenome-Wide Association StudyNature Communications
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Fetuin-A serum levels are not correlated to kidney function in long-lived subjects

2012

Objectives: Serum Fetuin A has been identified as an inhibitor of ectopic calcification. It is reduced in subjects with chronic kidney disease (CKD) and it has been proposed as a potential link between CKD and the higher prevalence of arterial calcification observed in these patients. During ageing both the stiffening of arterial wall due to calcification and a decline in kidney function are frequent. The aim of the study is to investigate if Fetuin A serum levels are associated with ageing and with AHSG T256S polymorphism. Moreover, we aim at investigate whether serum Fetuin A is correlated to kidney function in this setting of senescence. Design and Methods: 256 health long-lived subjects…

MaleSenescenceAgingmedicine.medical_specialtyFetuin Aalpha-2-HS-GlycoproteinClinical BiochemistryGene ExpressionRenal functionKidneyKidney Function TestsPolymerase Chain ReactionPolymorphism Single NucleotideEctopic calcificationElderlyInternal medicinemedicineHumansCystatin Ckidney functionAged 80 and overbiologybusiness.industryCalcinosisGeneral MedicineSerum Fetuin AKidney diseasemedicine.diseaseFetuinArterial calcificationSettore BIO/12 - Biochimica Clinica E Biologia Molecolare ClinicaAHSG T256S polymorphismEndocrinologyFetuin A Cystatin C Elderly Kidney disease AHSGCystatin CAHSGbiology.proteinKidney Failure ChronicFemalebusinessPolymorphism Restriction Fragment LengthCalcificationKidney disease
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p53 mutations in L3-loop zinc-binding domain, DNA-ploidy, and S phase fraction are independent prognostic indicators in colorectal cancer: A prospect…

2002

p53 gene alterations are among the most common events observed in colorectal cancer,and are accompanied frequently by DNA aneuploidy and high proliferative activity. The prognostic significance of such mutations remains controversial. We prospectively evaluated the prognostic significance of p53 mutations, DNA-ploidy, and S phase fraction (SPF) in a consecutive series of 160 colorectal cancer patients (median follow-up 71 months). Tumor DNA was screened for p53 mutations by PCR/single-strand conformational polymorphism/sequencing. DNA-ploidy and SPF were assessed by DNA flow cytometry. p53 mutations were detected in 68 of 160 (42.5%) cases. In 56% (38 of 68) of these, p53 mutations were fou…

MaleSettore MED/06 - Oncologia Medicaprotein p53S PhaseBiomarkers TumorHumansProspective StudiesCodonAgedPloidiesPolymorphism GeneticDNA NeoplasmExonsDNAMiddle AgedGenes p53PrognosisSurvival AnalysisProtein Structure TertiaryItalyMultivariate AnalysisMutationFemaleCarrier ProteinsColorectal NeoplasmsFollow-Up Studies
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