Search results for "porter"

showing 10 items of 920 documents

Association of Monocarboxylate Transporter-1 (MCT1) A1470T Polymorphism (rs1049434) with Forward Football Player Status.

2018

AbstractThe aim of this study was to investigate the association between the MCT1 (monocarboxylate transporter 1) A1470T polymorphism and positional roles in a large cohort of professional football players from five different countries. We compared genotype distributions of the MCT1 A1470T polymorphism between football players (n=694) and non-athlete controls (n=781) from Italy, Poland, Lithuania, Ukraine and Malta, and we analyzed the MCT1 genotype distributions with respect to the players’ positions in the field (e. g. forwards, midfielders, defenders and goalkeepers). Genomic DNA was extracted from either buccal epithelium or peripheral blood using a standard protocol. In the pooled coho…

MaleMonocarboxylic Acid TransportersGenotypePhysical Therapy Sports Therapy and RehabilitationFootball030204 cardiovascular system & hematology03 medical and health sciences0302 clinical medicineGene FrequencySoccerGenotypeHumansOrthopedics and Sports MedicineGenetic differences lactate monocarboxylate transporter-1 repeated sprint ability soccerAlleleAllele frequencyAllelesGenetic Association StudiesPolymorphism GeneticSymportersbiology030229 sport sciencesPeripheral bloodMonocarboxylate transporter 1CohortStandard protocolbiology.proteinDemography
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Elevated serum triiodothyronine and intellectual and motor disability with paroxysmal dyskinesia caused by a monocarboxylate transporter 8 gene mutat…

2008

Monocarboxylate transporter 8 (MCT8 or SLC16A2) is important for the neuronal uptake of triiodothyronine (T3) in its function as a specific and active transporter of thyroid hormones across the cell membrane, thus being essential for human brain development. We report on a German male with Allan-Herndon-Dudley syndrome presenting with severe intellectual and motor disability, paroxysmal dyskinesia combined with truncal muscular hypotonia, and peripheral muscular hypertonia at his current age of 9 years. Additionally, the patient has a lesion in the left putamen region revealed by magnetic resonance imaging and elevated serum T3 levels. The male appeared to have a hemizygous mutation (R271H)…

MaleMonocarboxylic Acid Transportersmedicine.medical_specialtyDevelopmental DisabilitiesDNA Mutational AnalysisEnzyme-Linked Immunosorbent AssayGene mutationArginineLesionDevelopmental NeuroscienceChoreaInternal medicineIntellectual DisabilitymedicineHumansHistidineChildMonocarboxylate transporterAllan–Herndon–Dudley syndromeTriiodothyroninebiologyMuscular hypotoniaSymportersParoxysmal dyskinesiamedicine.diseaseMagnetic Resonance ImagingEndocrinologyPediatrics Perinatology and Child HealthMutationbiology.proteinHypertoniaTriiodothyronineNeurology (clinical)medicine.symptomDevelopmental medicine and child neurology
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Single cell detection of latent cytomegalovirus reactivation in host tissue

2011

The molecular mechanisms leading to reactivation of latent cytomegalovirus are not well understood. To study reactivation, the few cells in an organ tissue that give rise to reactivated virus need to be identified, ideally at the earliest possible time point in the process. To this end, mouse cytomegalovirus (MCMV) reporter mutants were designed to simultaneously express the red fluorescent protein mCherry and the secreted Gaussia luciferase (Gluc). Whereas Gluc can serve to assess infection at the level of individual mice by measuring luminescence in blood samples or by in vivo imaging, mCherry fluorescence offers the advatage of detection of infection at the single cell level. To visualiz…

MaleMuromegalovirusCytomegalovirusGene Expressionmedicine.disease_causeVirusHerpesviridaeGreen fluorescent proteinMiceGaussiaMuromegalovirusSingle-cell analysisGenes ReporterVirologyVirus latencymedicineAnimalsHumansLuciferasesLungMice Inbred BALB CbiologyHerpesviridae Infectionsbiology.organism_classificationmedicine.diseaseVirologyVirus LatencyDisease Models AnimalLuminescent ProteinsCytomegalovirus InfectionsHost-Pathogen InteractionsFemaleVirus ActivationSingle-Cell AnalysismCherryJournal of General Virology
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Inhibition of different GABA transporter systems is required to attenuate epileptiform activity in the CA3 region of the immature rat hippocampus

2014

GABA transporters (GATs) are an essential element of the GABAergic system, which regulate excitability in the central nervous system and are thus used as targets for anticonvulsive therapy. However, in the immature nervous system the functions of the GABAergic system and the expression profile of GATs are distinct from the adult situation, obscuring to predict how different GAT isoforms influence epileptiform activity. Therefore we analyzed the effects of subtype specific GAT inhibitors on repetitive epileptiform discharges using field potential and whole-cell patch-clamp recordings in the CA3 region of hippocampal slices of immature (postnatal days 4-7) rats. These experiments revealed tha…

MaleNervous systemGABA Plasma Membrane Transport Proteinsgenetic structuresTiagabineCentral nervous systemAction PotentialsHippocampusHippocampal formationPharmacologyGABA AntagonistsOrgan Culture TechniquesSeizuresmedicineAnimalsGABA transporter4-AminopyridineRats WistarbiologyChemistryNeural InhibitionTransporterCA3 Region Hippocampaleye diseasesRatsmedicine.anatomical_structureAnimals Newbornnervous systemNeurologybiology.proteinGABAergicGABA Uptake InhibitorsNeurology (clinical)medicine.drugEpilepsy Research
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The dopamine uptake inhibitor 3 alpha-[bis(4'-fluorophenyl)metoxy]-tropane reduces cocaine-induced early-gene expression, locomotor activity, and con…

2009

Benztropine (BZT) analogs, a family of high-affinity dopamine transporter ligands, are molecules that exhibit pharmacological and behavioral characteristics predictive of significant therapeutic potential in cocaine addiction. Here, we examined in mice the effects of 3 alpha-[bis(4'-fluorophenyl)metoxy]-tropane (AHN-1055) on motor activity, conditioned place preference (CPP) and c-Fos expression in the striatum. AHN-1055 produced mild attenuation of spontaneous locomotor activity at a low dose (1 mg/kg) and weak stimulation at a higher dose (10 mg/kg). In parallel, the BZT analog significantly increased c-Fos expression in the dorsolateral caudoputamen at the high dose, whereas producing ma…

MaleNomifensineConditioning ClassicalAHN-1055cocaineGene ExpressionStimulationStriatumBZT derivativeNucleus accumbensPharmacologyplace preferenceMotor ActivityCocaine-Related DisordersMiceCocaineDopamine Uptake InhibitorsRewardDopaminemedicineAnimalsDopamine transporterPharmacologyBenztropinec-FosbiologyDose-Response Relationship DrugChemistryVentral striatumBrainConditioned place preferencePsychiatry and Mental healthNomifensinemedicine.anatomical_structureSpace Perceptionbiology.proteinStereotyped Behaviorlocomotor activityNeuroscienceProto-Oncogene Proteins c-fosmedicine.drugNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
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Modeling interactions between Human Equilibrative Nucleoside Transporter-1 and other factors involved in the response to gemcitabine treatment to pre…

2014

Background Pancreatic ductal adenocarcinoma (PDAC) is an extremely aggressive malignancy, characterized by largely unsatisfactory responses to the currently available therapeutic strategies. In this study we evaluated the expression of genes involved in gemcitabine uptake in a selected cohort of patients with PDAC, with well-defined clinical-pathological features. Methods mRNA levels of hENT1, CHOP, MRP1 and DCK were evaluated by means of qRT-PCR in matched pairs of tumor and adjacent normal tissue samples collected from PDAC patients treated with gemcitabine after surgical tumor resection. To detect possible interaction between gene expression levels and to identify subgroups of patients a…

MaleOncologyCHOPEquilibrative nucleoside transporter 1BioinformaticsDeoxycytidineCohort StudiesPancreatic ductal adenocarcinomachemistry.chemical_compoundMedicine(all)Transcription Factor CHOPbiologyDCKGeneral MedicineMiddle AgedSurvival RateDisease ProgressionAdenocarcinomaFemaleMRP1DeoxycytidineMultidrug Resistance-Associated ProteinsCarcinoma Pancreatic Ductalmedicine.drugmedicine.medical_specialtyAntineoplastic AgentsAdenocarcinomaMalignancyhENT1General Biochemistry Genetics and Molecular BiologyEquilibrative Nucleoside Transporter 1Internal medicinemedicineHumansRNA MessengerSurvival rateAgedBiochemistry Genetics and Molecular Biology(all)business.industryResearchRECPAMmedicine.diseaseGemcitabineGemcitabinechemistrybiology.proteinPancreatic ductal adenocarcinoma hENT1 CHOP MRP1 DCK RECPAMbusinessTranscription Factor CHOPCHOP
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Dehydroepiandrosterone up-regulates the Adrenoleukodystrophy-related gene (ABCD2) independently of PPAR alpha in rodents

2007

International audience; X-linked adrenoleukodystrophy (X-ALD) is a neurodegenerative disease caused by mutations in the ABCD1 gene, which encodes a peroxisomal ABC transporter, ALDP, supposed to participate in the transport of very long chain fatty acids (VLCFA). The adrenoleukodystrophyrelated protein (ALDRP), which is encoded by the ABCD2 gene, is the closest homolog of ALDP and is considered as a potential therapeutic target since functional redundancy has been demonstrated between the two proteins. Pharmacological induction of Abcd2 by fibrates through the activation of PPARa has been demonstrated in rodent liver. DHEA, the most abundant steroid in human, is described as a PPARa activat…

MalePEROXISOMEProhormonePeroxisome proliferator-activated receptorATP-binding cassette transporterBiochemistryMice0302 clinical medicineABC TRANSPORTERSPPAR-ALPHAAdrenal GlandsTestisDHEACells Culturedchemistry.chemical_classification0303 health sciencesSex CharacteristicsbiologyBrainGeneral MedicineOrgan SizePeroxisome3. Good healthUp-RegulationLiverAdrenoleukodystrophyFemalemedicine.drugAndrostenediolmedicine.medical_specialtyADRENOLEUKODYSTROPHYATP Binding Cassette Transporter Subfamily D03 medical and health sciencesABCD3Internal medicinemedicineABCD2AnimalsPPAR alpha[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyRats Wistar030304 developmental biologyActivator (genetics)Body Weightnutritional and metabolic diseasesMembrane ProteinsDehydroepiandrosteronemedicine.diseaseRatsMice Inbred C57BLEndocrinologychemistrybiology.proteinHepatocytesATP-Binding Cassette TransportersAcyl-CoA Oxidase030217 neurology & neurosurgery
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Does parental expressed emotion moderate genetic effects in ADHD? An exploration using a genome wide association scan.

2008

Contains fulltext : 70497.pdf (Publisher’s version ) (Closed access) Studies of gene x environment (G x E) interaction in ADHD have previously focused on known risk genes for ADHD and environmentally mediated biological risk. Here we use G x E analysis in the context of a genome-wide association scan to identify novel genes whose effects on ADHD symptoms and comorbid conduct disorder are moderated by high maternal expressed emotion (EE). SNPs (600,000) were genotyped in 958 ADHD proband-parent trios. After applying data cleaning procedures we examined 429,981 autosomal SNPs in 909 family trios. ADHD symptom severity and comorbid conduct disorder was measured using the Parental Account of Ch…

MaleParentsCandidate geneGenetics and epigenetic pathways of disease [NCMLS 6]Medizin2804 Cellular and Molecular NeuroscienceHostilityNeuroinformatics [DCN 3]2738 Psychiatry and Mental Health0302 clinical medicineGene FrequencyPerception and Action [DCN 1]Gene–environment interactionParent-Child RelationsChildGenetics (clinical)NeuregulinsbiologySLC1A1Intracellular Signaling Peptides and Proteins10058 Department of Child and Adolescent PsychiatryPsychiatry and Mental healthExpressed EmotionExcitatory Amino Acid Transporter 3Conduct disorderChild PreschoolFemalemedicine.symptomFunctional Neurogenomics [DCN 2]Clinical psychologyGenetic Markers2716 Genetics (clinical)AdolescentQuantitative Trait Loci610 Medicine & healthSingle-nucleotide polymorphismContext (language use)Mental health [NCEBP 9]Polymorphism Single NucleotideGenomic disorders and inherited multi-system disorders [IGMD 3]Interviews as Topic03 medical and health sciencesCellular and Molecular NeuroscienceCognitive neurosciences [UMCN 3.2]mental disordersmedicineExpressed emotionHumansddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersAllelesProbabilityModels Geneticbusiness.industryGenome Humanmedicine.disease030227 psychiatryGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with Hyperactivitybiology.proteinbusiness030217 neurology & neurosurgeryGenome-Wide Association Study
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Regulation of organic anion transporters in a new rat model of acute and chronic cholangitis resembling human primary sclerosing cholangitis

2002

Primary sclerosing cholangitis (PSC) is a cholestatic liver disease of unknown etiology. Although the primary defect affects cholangiocytes, cholestatic injury of hepatocytes may promote further liver damage. Since down-regulation of hepatocellular organic anion transporters is implicated in the molecular pathogenesis of cholestasis, expression of these transporters was determined in a novel rat model, which closely resembles human PSC.Hepatic protein and mRNA expression of basolateral (Ntcp, Oatp1, 2 and 4) and canalicular (Mrp2, Bsep) organic anion transporters were analyzed 1, 4 and 12 weeks after induction of experimental PSC by 2,4,6-trinitrobenzenesulfonic acid (TNBS).Specific down-re…

MalePathologymedicine.medical_specialtyOrganic Cation Transport ProteinsOrganic anion transporter 1Cholangitis SclerosingGene ExpressionOrganic Anion Transporters Sodium-DependentInflammationOrganic Anion Transporters Sodium-Independentdigestive systemPrimary sclerosing cholangitisSolute Carrier Organic Anion Transporter Family Member 1B3CholestasismedicineAnimalsRNA MessengerChronic CholangitisLiver injurySymportersHepatologybiologybusiness.industryMultidrug resistance-associated protein 2Membrane Transport ProteinsTransportermedicine.diseasedigestive system diseasesRatsDisease Models AnimalRats Inbred LewAcute DiseaseChronic Diseasebiology.proteinATP-Binding Cassette TransportersFemalemedicine.symptomCarrier ProteinsbusinessJournal of Hepatology
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DNA-Ploidy, Morphometric-Stereological and P-Glycoprotein Study of Superficial Bladder Carcinomas

1992

We carried out a DNA-ploidy, morphometric-stereologic and P-glycoprotein study on 40 newly diagnosed superficial bladder cancer patients (G1-G2), correlating the results with histological grade and clinical outcome. Variations in the number of patients who present recurrences, progression or remain tumor-free during the whole follow-up period (at least 5 years) were not significant when related to nuclear size, proliferative diploid index, presence of aneuploidy and expression of P-glycoprotein. It is striking how the majority of disease-free subjects showed a proliferative diploid index higher than 10%. Moreover, 3 of them presented an aneuploid cell population. In our study, only histolog…

MalePathologymedicine.medical_specialtyUrologyCellPopulationAneuploidyNewly diagnosedBiomarkers TumormedicineHumansIn patientATP Binding Cassette Transporter Subfamily B Member 1educationDna ploidyP-glycoproteineducation.field_of_studyMembrane GlycoproteinsPloidiesbiologybusiness.industryDNA Neoplasmmedicine.diseaseNeoplasm Proteinsmedicine.anatomical_structureUrinary Bladder Neoplasmsbiology.proteinFemalePloidybusinessFollow-Up StudiesEuropean Urology
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