Search results for "proliferation"

showing 10 items of 1193 documents

Genetic-dependency of peroxisomal cell functions - emerging aspects

2003

This paper reviews aspects concerning the genetic regulation of the expression of the well studied peroxisomal genes including those of fatty acid beta-oxidation enzymes; acyl-CoA oxidase, multifunctional enzyme and thiolase from different tissues and species. An important statement is PPARalpha, which is now long known to be in rodents the key nuclear receptor orchestrating liver peroxisome proliferation and enhanced peroxisomal beta-oxidation, does not appear to control so strongly in man the expression of genes involved in peroxisomal fatty acid beta-oxidation related enzymes. In this respect, the present review strengthens among others the emerging concept that, in the humans, the main …

chemistry.chemical_classificationThiolaseFatty AcidsAdaptation BiologicalReceptors Cytoplasmic and NuclearPeroxisome ProliferationPeroxisome proliferator-activated receptorReviewCell BiologyPeroxisomeBiologyLipid MetabolismchemistryNuclear receptorBiochemistryPeroxisomesAnimalsHumansMolecular MedicineGeneFunction (biology)BiogenesisSignal TransductionTranscription FactorsJournal of Cellular and Molecular Medicine
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New Acylated Triterpene Saponins fromPolygala arenaria

2003

Eight new acylated triterpene saponins 1–8 were isolated from the roots of Polygala arenaria as four inseparable (E)/(Z) mixtures of the 4-methoxycinnamoyl and 3,4-dimethoxycinnamoyl derivatives by repeated MPLC over silica gel. Their structures were established mainly by 600-MHz 2D-NMR techniques (1H,1H-COSY, TOCSY, NOESY, HSQC, HMBC) as 3-O-(β-D-glucopyranosyl)presenegenin 28-(O-β-D-galactopyranosyl-(14)-O-[β-D-glucopyranosyl-(13)]-O-β-D-xylopyranosyl-(14)-O-α-L-rhamnopyranosyl-(12)-{4-O-[(E)-4-methoxycinnamoyl]}-β-D-fucopyranosyl) ester and its (Z)-isomer (1/2), 3-O-(β-D-glucopyranosyl)presenegenin 28-(O-β-D-galactopyranosyl-(14)-O-[β-D-glucopyranosyl-(13)]-O-β-D-xylopyranosyl-(14)-O-α-L…

chemistry.chemical_classificationbiologyStereochemistryOrganic ChemistryTenuifolinbiology.organism_classificationBiochemistryPresenegeninCatalysisPolygalaInorganic ChemistryTriterpenechemistryDrug DiscoveryMoietyPhysical and Theoretical ChemistryTwo-dimensional nuclear magnetic resonance spectroscopyLymphocyte Proliferation AssayHelvetica Chimica Acta
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Placental Chorangiocarcinoma a Specific Histological Pattern of Uncertain Incidence and Clinical Impact: Systematic Review of the Literature

2023

Chorangiocarcinoma is a very rare and misdiagnosed placental neoplasm. The unique morphologic features of the lesion distinguish it from other trophoblastic tumors and vascular abnormalities. We present a systematic review of the literature to provide clarity on chorangiocarcinoma entity and biology. A literature search was carried out in December 2022 using the keywords “Placental chorangiocarcinoma”, “Chorangioma”, “Placenta”, and “Throphoblast proliferation”. Articles published from 1988 to 2022 were obtained from Scopus, Google Scholar, and PUBMED. In our review, we examined maternal age, gestational age at the time of delivery, parity, type of pregnancy, placental weight, ultrasound fe…

chorangiocarcinoma; chorangioma; placenta; throphoblast proliferation.placentaGeneral Medicinechorangiocarcinomathrophoblast proliferation.Settore MED/40 - Ginecologia E OstetriciachorangiomaJournal of Clinical Medicine
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A role for peroxisome proliferator-activated receptor gamma in resveratrol-induced colon cancer cell apoptosis.

2014

Scope Resveratrol may function as a chemopreventive agent. A recent clinical study demonstrates a reduction in tumor cell proliferation in colorectal patients receiving repeated oral ingestion of resveratrol. However, gaps remain in our knowledge of the molecular mechanisms by which resveratrol exerts its chemopreventive effect. We have previously demonstrated that resveratrol induces apoptosis in colon cancer cells and that resveratrol can sensitize chemoresistant colon cancer cells to various drugs. Based on its ability to activate peroxisome proliferator-activated receptor gamma (PPARγ) in colon cancer cells, we sought to determine the implication of this nuclear transcription factor in …

endocrine system diseasesColorectal cancerPeroxisome proliferator-activated receptorApoptosisPharmacologyResveratrolresveratrolMESH: ThiazolidinedionesPPAR[ SDV.CAN ] Life Sciences [q-bio]/Cancerchemistry.chemical_compound0302 clinical medicineMESH: StilbenesStilbenesMESH : Cell Proliferation[ SHS.INFO ] Humanities and Social Sciences/Library and information sciencesAnilidesskin and connective tissue diseaseschemistry.chemical_classification0303 health sciencesfood and beveragesCell cycle3. Good healthMESH : ThiazolidinedionesMESH : Colonic Neoplasmscolon cancer030220 oncology & carcinogenesisColonic NeoplasmsS Phase Cell Cycle CheckpointsRosiglitazonehormones hormone substitutes and hormone antagonistsBiotechnologymedicine.drugMESH : PPAR gammaMESH: Cell Line Tumor[SHS.INFO]Humanities and Social Sciences/Library and information sciences[SDV.CAN]Life Sciences [q-bio]/Cancer[SDV.BC]Life Sciences [q-bio]/Cellular BiologyBiologyMESH: Anilides[SHS.INFO] Humanities and Social Sciences/Library and information sciencesMESH : AnilidesMESH : StilbenesRosiglitazone03 medical and health sciences[SDV.CAN] Life Sciences [q-bio]/CancerCell Line TumorMESH: Cell Proliferationmedicine[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyHumans[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biology[SDV.BC] Life Sciences [q-bio]/Cellular BiologyMESH : S Phase Cell Cycle Checkpoints[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular Biologypolyphenols030304 developmental biologyCell ProliferationMESH: Colonic NeoplasmsMESH: HumansCell growthMESH : Cell Line Tumor[ SDV.BC ] Life Sciences [q-bio]/Cellular Biologyorganic chemicalsMESH: ApoptosisMESH : Humansmedicine.diseasePPAR gammaMESH: S Phase Cell Cycle CheckpointschemistryMESH: PPAR gammaApoptosisCancer cellThiazolidinedionesMESH : ApoptosisFood Science
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RASSF1A inhibits estrogen receptor alpha expression and estrogen-independent signalling: implications for breast cancer development

2012

The Ras association domain family 1 isoform A (RASSF1A) is a tumor suppressor whose inactivation is implicated in the development of many human cancers, including breast carcinomas. Little is known about the tumor-suppressive function of RASSF1A in breast tissue and whether its inactivation is mechanistically involved in the initiation and progression of breast tumors. Here, we show that RASSF1A inhibits breast cancer growth in vivo, and suppresses estrogen receptor (ERα) expression and function. Reconstitution of RASSF1A in MCF7 cells led to decreased ERα levels and reduced sensitivity to estrogen (E2). Concomitantly, we observed decreased expression of Id1 as well as the E2-responsive gen…

endocrine systemCancer Researchmedicine.medical_specialtyCell SurvivalGene ExpressionEstrogen receptorApoptosisBreast NeoplasmsCell Cycle ProteinsMice SCIDBiologyMiceBreast cancerDownregulation and upregulationMice Inbred NODInternal medicineGeneticsmedicineAnimalsHumansFulvestrantMolecular BiologyCellular SenescenceCell ProliferationRegulation of gene expressionEstradiolFulvestrantTumor Suppressor ProteinsEstrogen AntagonistsEstrogen Receptor alphaCancerEstrogensCell Cycle Checkpointsmedicine.diseaseGene Expression Regulation NeoplasticEndocrinologyProteolysisMCF-7 CellsCancer researchFemaleEctopic expressionEstrogen receptor alphaNeoplasm TransplantationSignal Transductionmedicine.drugOncogene
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Cilium induction triggers differentiation of glioma stem cells.

2020

Glioblastoma multiforme (GBM) possesses glioma stem cells (GSCs) that promote self-renewal, tumor propagation, and relapse. Understanding the mechanisms of GSCs self-renewal can offer targeted therapeutic interventions. However, insufficient knowledge of GSCs' fundamental biology is a significant bottleneck hindering these efforts. Here, we show that patient-derived GSCs recruit elevated levels of proteins that ensure the temporal cilium disassembly, leading to suppressed ciliogenesis. Depleting the cilia disassembly complex components is sufficient to induce ciliogenesis in a subset of GSCs via relocating platelet-derived growth factor receptor-alpha (PDGFR-α) to a newly induced cilium. Im…

endocrine systemmedicine.medical_treatmentBiologyGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesMice0302 clinical medicineGentamicin protection assayGliomaCiliogenesisCell Line TumormedicineAnimalsHumansCell Self Renewal030304 developmental biologyCell Proliferation0303 health sciencesBrain NeoplasmsCiliumGrowth factorfungiBrainCell DifferentiationGliomaCilium disassemblyCell cyclemedicine.diseaseCell biology030220 oncology & carcinogenesisNeoplastic Stem CellsStem cellNeoplasm Recurrence LocalGlioblastomaCell reports
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Inhibition of KIF20A by BKS0349 reduces endometriotic lesions in a xenograft mouse model

2019

AbstractSeveral studies have suggested a possible etiological association between ovarian endometriosis and ovarian cancer. Evidence has shown that KIF20A overexpression might confer a malignant phenotype to ovarian tumors by promoting proliferation and inhibiting apoptosis. However, no data about the role of KIF20A in endometriosis have been described. In this study, the human endometrium (n = 4) was transfected by mCherry adenovirus and intraperitoneally implanted in mice. Subsequently, mice were divided in three groups (n = 8/group) that were treated with Vehicle, BKS0349 (KIF20A-antagonist) or cabergoline (dopamine receptor agonist) for 21 days. mCherry-labeled endometriotic lesions wer…

endometriosisEmbryologyCabergolineEndometriosisEndometriosisKinesinsMice NudeApoptosisBiologyPeritoneal DiseasesEndometriumAndrologyEndometriumMiceGeneticsmedicineAnimalsHumansKIF20AMolecular BiologyCell ProliferationTUNEL assayOptical ImagingapoptosisObstetrics and GynecologyCell BiologyCell cyclemedicine.diseaseDisease Models Animalcell proliferationmedicine.anatomical_structureReproductive MedicineApoptosisOvarian EndometriosisHeterograftsImmunohistochemistrycell cycleFemaleOvarian cancerDevelopmental BiologyMolecular Human Reproduction
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Evaluation of PAI-1 in endometriosis using a homologous immunocompetent mouse model

2018

To analyze the role of PAI-1 (plasminogen activator inhibitor 1) in endometriotic lesion growth, we studied the effect of PAI-1 inhibition by PAI-039 using a homologous mouse model of endometriosis that allows noninvasive monitoring. Endometrial tissue from donor mice was collected, labeled with mCherry adenovirus, and implanted into a subcutaneous pocket on the ventral abdomen of recipient mice. Seven days after transplantation, mice were randomly allocated in two groups and treated once daily for 2 weeks with either vehicle (control group) or PAI-1 inhibitor (PAI-039 group). Endometriotic lesion size generated in recipient mice was monitored by mCherry signal. Animals were euthanized 21 d…

endometriosisPathologymedicine.medical_specialtyAngiogenesismouse modelnoninvasive monitoringEndometriosisEndometriosisPAI-1FibrinLesionNeovascularization03 medical and health scienceschemistry.chemical_compoundEndometriumMiceangiogenesis0302 clinical medicineIn vivoSerpin E2medicineAnimalsCell Proliferation030219 obstetrics & reproductive medicinebiologyIndoleacetic AcidsNeovascularization PathologicCell BiologyGeneral Medicinemedicine.diseaseTransplantationDisease Models AnimalReproductive Medicinechemistry030220 oncology & carcinogenesisPlasminogen activator inhibitor-1biology.proteinFemalefibrinolysismedicine.symptom
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Sildenafil inhibits the proliferation of cultured human endothelial cells.

2013

The proliferation of endothelial cells plays a crucial role in the development of intraplaque angiogenesis (IPA). IPA is a major source of intraplaque hemorrhage and therefore contributes to the destabilization of atherosclerotic plaques. Therefore, the aim of the present study was to examine, whether sildenafil inhibits endothelial cell growth. The proliferation of human endothelial cells derived from umbilical cord veins (HUVEC) was examined on DNA level by measurements of (3H)-thymidine incorporation. Cell viability was analyzed using trypan blue staining. The proliferation of cultured human endothelial cells was significantly decreased by 1 μmol/l (-48.4%) and 10 μmol/l (-89.6%) sildena…

endothelial proliferationsildenafilcardiovascular systematherosclerosisbasic fibroblast growth factorArticleplaque angiogenesisrespiratory tract diseasesInternational journal of biomedical science : IJBS
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Biomimetic strategy towards gelatin coatings on PET. Effect of protocol on coating stability and cell-interactive properties

2019

Gelatin-modified poly(ethylene terephthalate) (PET) surfaces have been previously realized via an intermediate dopamine coating procedure that resulted in surfaces with superior haemocompatibility compared to unfunctionalized PET. The present study addresses the biocompatibility assessment of these coated PET surfaces. In this context, the stability of the gelatin coating upon exposure to physiological conditions and its cell-interactive properties were investigated. The proposed gelatin–dopamine-PET surfaces showed an increased protein coating stability up to 24 days and promoted the attachment and spreading of both endothelial cells (ECs) and smooth muscle cells (SMCs). In parallel, physi…

food.ingredientBiocompatibilityCellBiomedical Engineering02 engineering and technologyengineering.material010402 general chemistry01 natural sciencesGelatinfoodCoatingSmooth muscleBiomimeticsmedicineGeneral Materials ScienceChemistryPolyethylene TerephthalatesGeneral ChemistryGeneral Medicine021001 nanoscience & nanotechnology0104 chemical sciencesPolyestermedicine.anatomical_structureengineeringSurface modificationGelatin0210 nano-technologyVascular graftBiomedical engineeringSURFACE MODIFICATION; CHEMISTRY; POLYESTER; ADHESION; FUNCTIONALIZATION; BIOCOMPATIBILITY; IMMOBILIZATION; PROLIFERATION; COMPATIBILITY; BIOMATERIALS
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