Search results for "prostaglandin"

showing 10 items of 232 documents

The role of extracellular calcium in bone metastasis

2016

AbstractThis review summarizes the role of extracellular calcium, as found present in the bone tissue, in the process of bone metastasis.

0301 basic medicinelcsh:Diseases of the musculoskeletal systemIGF insulin-like growth factorPGE-2 prostaglandin E-2Bone tissueFibroblast growth factorM-CSF macrophage colony-stimulating factorPDGF platelet-derived growth factorBone remodelingSK3 small conductance calcium-activated potassium channel 30302 clinical medicineERK extracellular signal-regulated kinaseTGFβ transforming growth factor betaBMP's bone morphogenetic proteinsbiologyAKT AKT8 virus oncogene cellular homologBone metastasislcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogenshumanitiescAMP cyclic adenosine monophosphatemedicine.anatomical_structureOncologyRANKL030220 oncology & carcinogenesisIon channelsCaSR calcium-sensing receptorPTHrP parathyroid hormone-related proteinPlatelet-derived growth factor receptorResearch PaperTRP transient receptor potentialmedicine.medical_specialtychemistry.chemical_elementCalciumRANKL receptor activator of NF-κB ligandlcsh:RC254-28203 medical and health sciencesPLC phospholipase CInternal medicinemedicineExtracellularCaSRET-1 endothelin-1PTEN phosphatase and tensin homolog deleted on chromosome 10business.industryBone metastasismedicine.diseaseFGF fibroblast growth factor030104 developmental biologyEndocrinologyPSA prostate specific antigenchemistryCOPD chronic obstructive pulmonary diseasebiology.proteinCancer researchJNK jun N-terminal kinasePKA protein kinase ARANK receptor activator of NF-κBCalciumlcsh:RC925-935businessMAPK mitogen-activated protein kinaseJournal of Bone Oncology
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Participation of Heme Oxygenase-1 in a Model of Acute Inflammation

2003

In this study, the role of heme oxygenase-1 (HO-1) in the inflammatory response elicited by zymosan in the mouse air pouch model has been examined. This response is characterized by a time-dependent increase in HO-1 expression in the leukocytes migrating into the exudates. At 24–48 h maximal HO-1 expression was accompanied by reduced cyclooxygenase-2 (COX-2) and nitric oxide synthase-2 (NOS-2) expression as well as low levels of inflammatory mediators. Hemin administration into the air pouch caused an elevation of HO-1 protein and bilirubin levels induced by zymosan with inhibition of COX-2 expression. In mouse peritoneal macrophages from hemin-injected mice, we also observed an increased …

0301 basic medicinemedicine.medical_specialtyNitric Oxide Synthase Type IIInflammationGeneral Biochemistry Genetics and Molecular BiologyNitric oxideMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicineAnimalsProstaglandin E2HemeInflammationbiologyZymosanZymosanMembrane ProteinsIsoenzymesHeme oxygenaseDisease Models Animal030104 developmental biologyEndocrinologychemistryCyclooxygenase 2Prostaglandin-Endoperoxide Synthases030220 oncology & carcinogenesisHeme Oxygenase (Decyclizing)ImmunologyMacrophages Peritonealbiology.proteinHeminFemaleCyclooxygenaseNitric Oxide Synthasemedicine.symptomHeme Oxygenase-1medicine.drugHeminExperimental Biology and Medicine
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Role of Redox Signaling and Inflammation in Skeletal Muscle Adaptations to Training

2016

The inflammatory response to exercise-induced muscle damage has been extensively described. Exercise has important modulatory effects on immune function. These effects are mediated by diverse factors including pro-inflammatory cytokines, classical stress hormones, and hemodynamic effects leading to cell redistribution. As has been reported regarding oxidative stress, inflammation can have both detrimental and beneficial effects in skeletal muscle. In this review we will address the role of inflammation on protein metabolism in skeletal muscle. Specifically, we will review studies showing that treatment with cyclooxygenase-inhibiting drugs modulate the protein synthesis response to one bout …

0301 basic medicinemedicine.medical_specialtyprotein synthesisPhysiologyClinical BiochemistryInflammationReviewBiologyBioinformaticsmedicine.disease_causeBiochemistryProinflammatory cytokineMuscle hypertrophyprostaglandins03 medical and health sciences0302 clinical medicineImmune systemInternal medicinemedicineoxidative stressMolecular Biologychemistry.chemical_classificationReactive oxygen specieslcsh:RM1-950Skeletal muscle030229 sport sciencesCell Biology030104 developmental biologyEndocrinologymedicine.anatomical_structurelcsh:Therapeutics. Pharmacologychemistryanti-inflammatoriesmedicine.symptomhypertrophyOxidative stressHormoneAntioxidants
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Tumor-derived immuno-modulators induce overlapping pro-tolerogenic gene expression signatures in human dendritic cells.

2016

Immature dendritic cells (iDCs) and tolerogenic DCs are essential for the induction and maintenance of peripheral tolerance. Tumors produce immuno-modulatory factors which imprint a pro-tolerogenic, maturation-resistant state in DCs. Here we asked for common markers of differentially tolerized human monocyte-derived DC populations. For this, PBMC-derived monocytes were differentiated to DCs in the presence of established immuno-modulators as released by tumors (IL-6, IL-10, TGF-β, glucocorticoid [GC], prostaglandin E2 [PGE2]). Most unstimulated pro-tolerogenic DC populations commonly over-expressed some tolerance-associated markers (ILT-4, IL-10, HO-1) as compared with iDCs. These markers m…

0301 basic medicinemedicine.medical_treatmentT cellT-LymphocytesImmunologyStimulationBiologyLymphocyte ActivationDinoprostone03 medical and health sciences0302 clinical medicineDownregulation and upregulationNeoplasmsmedicineImmune ToleranceImmunology and AllergyHumansImmunologic FactorsProstaglandin E2GlucocorticoidsCells CulturedAntigen PresentationPeripheral toleranceCell DifferentiationGeneral MedicineDendritic CellsInterleukin 10030104 developmental biologyCytokinemedicine.anatomical_structureImmunologyB7-1 AntigenCytokinesCD80Heme Oxygenase-1030215 immunologymedicine.drugHuman immunology
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Approche nutritionnelle de la thérapie du syndrome de l'œil sec : prévention par les acides gras polyinsaturés

2008

Dry eye disease affects 15% of the population over the age of 65. It is characterized mainly by inflammation of the ocular surface (tear film, conjunctiva and cornea) which represents a relevant target for therapeutic strategies. The side effects and long-term intolerance of anti-inflammatory drugs give rise to an increasing interest for dietary components exhibiting anti-inflammatory properties, such as polyunsaturated fatty acids (PUFAs). In these studies, we developed a scopolamine-induced dry eye model in the rat that mimics changes occurring in Humans suffering from moderate dry eye. We described the time course of clinical signs, inflammation and mucin production loss. We investigated…

ALIMENTATIONPOLYUNSATURED FATTY ACIDSSYNDROME DE l'OEIL SECSURFACE OCULAIRE[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringOCULAR SURFACEPROSTAGLNADINES[SDV.IDA] Life Sciences [q-bio]/Food engineeringACIDES GRAS POLYINSATURESDIETINFLAMMATIONPROSTAGLANDINS[SDV.IDA]Life Sciences [q-bio]/Food engineering[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringDRY EYE DISEASE
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Gastric adenomas: relationship between clinicopathological findings, Helicobacter pylori infection, APC mutations and COX-2 expression.

2006

Gastric adenomas are rare neoplastic growths characterized by localized polypoid proliferations of dysplastic epithelium that tend to progress to infiltrating adenocarcinoma. Therefore, the identification of molecular markers that could reliably recognize adenomas at risk of progression is advocated in the clinical management. In this study we investigated, in a series of gastric adenoma specimens from an area at high risk of gastric cancer, the relationship between clinicopathological characteristics of adenoma and Helicobacter pylori infection, APC mutational status, and COX-2 and the down-stream enzyme mPGES1 expression. Helicobacter pylori infection, detected in 24%, and 33% by histolog…

AdenomaMaleGenes APCAdenomaAdenocarcinomamedicine.disease_causegastric adenomaHelicobacter InfectionsHelicobacter pyloryStomach NeoplasmsmedicineHumansAPC mutationsAgedProstaglandin-E SynthasesAged 80 and overMutationbiologyHelicobacter pyloribusiness.industryCancerAPC mutations; COX-2; gastric adenoma; Helicobacter pyloriHistologyHematologyHelicobacter pyloriCOX-2Middle Agedmedicine.diseasebiology.organism_classificationdigestive system diseasesEpitheliumIntramolecular Oxidoreductasesmedicine.anatomical_structureOncologyDysplasiacyclooxygenase-2Cyclooxygenase 2Gastric MucosaMutationCancer researchAPC-mutations gastric adenoma Helycobacter pyloriAdenocarcinomaFemalebusiness
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Proeryptotic Activity of 4-Hydroxynonenal: A New Potential Physiopathological Role for Lipid Peroxidation Products

2020

Background: Eryptosis is a physiological, apoptosis-like death of injured erythrocytes crucial to prevent premature haemolysis and the pathological sequalae generated by cell-free haemoglobin. When dysregulated, the process is associated to several inflammatory-based pathologies. 4-Hydroxy-trans-2-nonenal (HNE) is an endogenous signalling molecule at physiological levels and, at higher concentrations, is involved in the pathogenesis of several inflammatory-based diseases. This work evaluated whether HNE could induce eryptosis in human erythrocytes. Methods: Measurements of phosphatidylserine, cell volume, intracellular oxidants, Ca++, glutathione, ICAM-1, and ceramide were assessed by flow …

Adult0301 basic medicineCeramideErythrocyteslcsh:QR1-502PhosphatidylserinesBiochemistryArticleRBClcsh:Microbiology4-HydroxynonenalLipid peroxidationprostaglandins03 medical and health scienceschemistry.chemical_compound0302 clinical medicineeryptosisCell AdhesionHuman Umbilical Vein Endothelial CellsHumansMolecular BiologyCells CulturedCaspaseAldehydesbiologyGlutathionePhosphatidylserineMiddle AgedIntercellular Adhesion Molecule-1Haemolysislipid peroxidation productsGlutathione4-hydroxynonenalCell biology030104 developmental biologychemistryinflammation030220 oncology & carcinogenesisbiology.proteinCalciumLipid PeroxidationIntracellularBiomolecules
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β-Adrenoceptor stimulation up-regulates phosphodiesterase 4 activity and reduces prostaglandin E 2 -inhibitory effects in human neutrophils

2000

Human neutrophils were treated for 4 h with a combination of salbutamol (1 µM), a β2-adrenoceptor agonist, and rolipram (30 µM), a selective phosphodiesterase 4 inhibitor, to investigate whether this treatment produces up-regulation of phosphodiesterase activity with functional consequences. Anion-exchange chromatography coupled with the use of selective activators and inhibitors demonstrated that a phosphodiesterase activity with characteristics of the isoenzyme type 4 was increased in drug-treated cells. Kinetic analysis showed a ~1.5-fold increase in V max without alteration of K m values. The augmented phosphodiesterase activity in drug-treated cells was abolished by actinomycin D. Cycl…

AdultAgonistmedicine.medical_specialtyNeutrophilsmedicine.drug_classStimulationIn Vitro TechniquesBiologyDinoprostoneNeutrophil Activationchemistry.chemical_compoundPDE4BDownregulation and upregulationSuperoxidesInternal medicineCyclic AMPmedicineHumansAlbuterolRNA MessengerEnzyme InhibitorsProstaglandin E2RolipramPharmacologyReverse Transcriptase Polymerase Chain ReactionSuperoxideZymosanZymosanGeneral MedicineAdrenergic AgonistsCyclic Nucleotide Phosphodiesterases Type 4Up-RegulationEndocrinologychemistry3'5'-Cyclic-AMP PhosphodiesterasesReceptors Adrenergic beta-2Roliprammedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Role of prostaglandin E2 in the invasiveness, growth and protection of cancer cells in malignant pleuritis.

2006

The recurrence of pleural effusions is a common event in a variety of neoplastic diseases. The objective of this study was to identify the mechanisms promoting the homing and growth of cancer cells within the pleural space. A cancer cell line recovered from malignant pleural fluids (lung adenocarcinoma cell line) that constitutively expresses cyclooxygenase 2 (COX-2) and all types of prostaglandin receptors was studied. It was first demonstrated using a matrigel system, that malignant pleural fluids increase the invasiveness of adenocarcinoma cells more than congestive heart failure (CHF) pleural fluids. Moreover, exposure to exudative malignant, but not to CHF pleural fluids, increased the…

AdultCancer ResearchPathologymedicine.medical_specialtyLung NeoplasmsPleural effusionAdenocarcinomaDinoprostoneMetastasisCell Line TumormedicineHumansReceptors Prostaglandin ENeoplasm InvasivenessProstaglandin E2PleurisyAgedCell ProliferationMatrigelCyclooxygenase 2 Inhibitorsbusiness.industryNF-kappa BCancerMiddle Agedmedicine.diseaseNeoplasm ProteinsPleural Effusion MalignantUp-RegulationOncologyPleurisyCyclooxygenase 2TalcCancer cellAdenocarcinomabusinessmedicine.drugEuropean journal of cancer (Oxford, England : 1990)
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Effect of loperamide on jejunal electrolyte and water transport, prostaglandin E 2-induced secretion and intestinal transit time in man

1991

Jejunal perfusion was performed in 12 healthy volunteers to evaluate the dose dependent effects of loperamide on intestinal absorption, stimulated secretion and transit. In 6 volunteers intestinal perfusion of the jejunal segment with isotonic NaCl solution was followed by addition of loperamide in increasing doses (2–8 mg·l−1). The volunteers were pretreated with 1 mg·l−1 prostaglandin E2 (PgE2) in the perfusate before addition of 4 mg·l−1 loperamide. Phenolsulphonphtalein (PSP) boluses (2 ml) were given to measure mean transit time (MTT). Loperamide 2 mg·l−1 converted the minor secretion after perfusion with the standard solution (water −1.45 ml·min−1, Na −0.09 and Cl −0.04 mmol·min−1) to…

AdultDiarrheaMalemedicine.medical_specialtyLoperamideAdolescentAbsorption (skin)LoperamideDinoprostoneIntestinal absorptionJejunumChloridesInternal medicinemedicineHumansPharmacology (medical)Prostaglandin E2Gastrointestinal TransitPharmacologyWater transportDose-Response Relationship DrugChemistrySodiumBiological TransportGeneral MedicineWater-Electrolyte BalanceJejunumEndocrinologymedicine.anatomical_structureIntestinal AbsorptionMechanism of actionmedicine.symptomPerfusionmedicine.drugEuropean Journal of Clinical Pharmacology
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