Search results for "rats"

showing 10 items of 3537 documents

Inward currents in primary nociceptive neurons of the rat and pain sensations in humans elicited by infrared diode laser pulses

2002

Radiant heat is often used to study nociception in vivo. We now used infrared radiation generated by a diode laser stimulator (wavelength 980 nm) to investigate transduction mechanisms for noxious heat stimuli in acutely dissociated dorsal root ganglion (DRG) neurons of rats in vitro. The laser stimulator offered the unique opportunity to test whether the same stimuli also elicit pain sensations in humans. A specific heat-induced current (I(heat)) was elicited in six of 13 small DRG neurons (diameteror =30 microm) tested in the whole-cell configuration of the patch-clamp mode. Current responses in the seven heat-insensitive neurons were within the range explainable by the temperature depend…

AdultMaleHot TemperaturePatch-Clamp TechniquesPainSensory systemIn Vitro TechniquesMembrane PotentialsRats Sprague-DawleyDorsal root ganglionEvoked Potentials SomatosensoryGanglia SpinalNoxious stimulusmedicinePsychophysicsAnimalsHumansPatch clampNeurons AfferentSkinChemistryLasersNociceptorsMiddle AgedSensory neuronRatsElectrophysiologyKineticsAnesthesiology and Pain MedicineNociceptionmedicine.anatomical_structureNeurologyFemaleNeurology (clinical)Transduction (physiology)Neuroscience
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Spinal Endocannabinoids and CB 1 Receptors Mediate C-Fiber–Induced Heterosynaptic Pain Sensitization

2009

Plastic Pain Perception Drugs and endocannabinoids acting on cannabinoid (CB) receptors have potential in the treatment of certain types of pain. In the spinal cord they are believed to suppress nociception, the perception of pain and noxious stimuli. Pernia-Andrade et al. (p. 760 ) now find that endocannabinoids, which are released in spinal cord by noxious stimulation, may promote rather than inhibit nociception by acting on CB1 receptors. Endocannabinoids not only depress transmission at excitatory synapses in the spinal cord, but also block the release of inhibitory neurotransmitters, thereby facilitating nociception.

AdultMaleInterneuronPainMice TransgenicNeurotransmissionInhibitory postsynaptic potentialSynaptic TransmissionArticleRats Sprague-DawleyMiceYoung AdultPiperidinesReceptor Cannabinoid CB1InterneuronsCannabinoid Receptor ModulatorsmedicineAnimalsHumansPosterior Horn CellNerve Fibers UnmyelinatedMultidisciplinaryExcitatory Postsynaptic PotentialsNeural InhibitionAnatomySpinal cordElectric StimulationRatsMice Inbred C57BLPosterior Horn Cellsmedicine.anatomical_structureNociceptionInhibitory Postsynaptic PotentialsSpinal Cordnervous systemHyperalgesiaHyperalgesiaNeuropathic painPyrazolesFemaleRimonabantmedicine.symptomNeurosciencepsychological phenomena and processesEndocannabinoidsScience
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Increased Neural Activity in Mesostriatal Regions after Prefrontal Transcranial Direct Current Stimulation and L-DOPA Administration

2019

Dopamine dysfunction is associated with a wide range of neuropsychiatric disorders commonly treated pharmacologically or invasively. Recent studies provide evidence for a nonpharmacological and noninvasive alternative that allows similar manipulation of the dopaminergic system: transcranial direct current stimulation (tDCS). In rodents, tDCS has been shown to increase neural activity in subcortical parts of the dopaminergic system, and recent studies in humans provide evidence that tDCS over prefrontal regions induces striatal dopamine release and affects reward-related behavior. Based on these findings, we used fMRI in healthy human participants and measured the fractional amplitude of low…

AdultMaleLevodopamedicine.medical_treatmentDopaminePrefrontal CortexTranscranial Direct Current StimulationLevodopa03 medical and health sciencesNeural activitySpatial similarityYoung Adult0302 clinical medicineDopamineMedicineAnimalsHumansSingle-Blind MethodResearch Articles030304 developmental biologyNeurons0303 health sciencesBrain MappingResting state fMRITranscranial direct-current stimulationbusiness.industryReceptors Dopamine D2General NeuroscienceReceptors Dopamine D1DopaminergicMagnetic Resonance ImagingCorpus StriatumDopamine receptorRats Inbred LewFemalebusinessNeuroscience030217 neurology & neurosurgerymedicine.drug
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Thoracoscopic sympathectomy at the T2 or T3 level facilitates bradykinin-induced protein extravasation in human forearm skin.

2010

Background.  The endogenous peptide bradykinin (BK) is an inflammatory mediator that induces nociceptor activation and sensitization as well as protein extravasation and vasodilation. Objective.  To test the hypothesis if sympathectomy affects BK-induced inflammation in humans. Methods.  Dermal microdialysis was employed on the volar forearm in 10 patients (21–41 years) with regional hyperhidrosis before and three months after preganglionic endoscopic transthoracic sympathetic clipping (ETSC) at the T2 or T3 level and in 10 healthy volunteers (22–36 years). After 60 minutes perfusion with Ringer's solution microdialysis fibers were perfused with BK 10−7 M and 10−5 M for 30 minutes followed …

AdultMaleMicrodialysismedicine.medical_treatmentMicrodialysisVasodilator AgentsBradykininVasodilationBradykininThoracic Vertebraechemistry.chemical_compoundYoung AdultForearmmedicineAnimalsHumansHyperhidrosisSympathectomySkinNeurogenic inflammationbusiness.industryThoracoscopyGeneral MedicineBlood ProteinsExtravasationRatsForearmAnesthesiology and Pain Medicinemedicine.anatomical_structurechemistrySympathectomyRegional Blood FlowAnesthesiaFemaleNeurology (clinical)businessPerfusionPain medicine (Malden, Mass.)
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Uncoupling protein-2 (UCP2) induces mitochondrial proton leak and increases susceptibility of non-alcoholic steatohepatitis (NASH) liver to ischaemia…

2008

Background: The mechanisms of progression from fatty liver to steatohepatitis and cirrhosis are not well elucidated. Mitochondrial dysfunction represents a key factor in the progression of non-alcoholic steatohepatitis (NASH) as mitochondria are the main cellular site of fatty acid oxidation, ATP synthesis and reactive oxygen species (ROS) production. Aims: (1) To evaluate the role of the uncoupling protein 2 in controlling mitochondrial proton leak and ROS production in NASH rats and humans; and (2) to assess the acute liver damage induced by ischaemia–reperfusion in rats with NASH. Methods: Mitochondria were extracted from the livers of NASH humans and rats fed a methionine and choline de…

AdultMaleMitochondrial ROSmedicine.medical_specialtyMitochondria LiverMitochondrionBiologymedicine.disease_causeIon ChannelsMitochondrial ProteinsAdenosine TriphosphateInternal medicinemedicineAnimalsHumansUncoupling proteinUncoupling Protein 2Rats WistarBeta oxidationAdenosine TriphosphatasesMembrane Potential MitochondrialAldehydesFatty liverGastroenterologyMiddle Agedmedicine.diseaseRatsFatty LiverOxidative StressEndocrinologyMitochondrial respiratory chainLiverBiochemistryReperfusion InjuryAcute DiseaseDisease ProgressionFemaleSteatohepatitisReactive Oxygen SpeciesOxidative stressGut
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Characterization of target antigens from anti-neutrophil cytoplasmic antibodies in autoimmune hepatitis type-I.

1997

The occurrence of anti-neutrophil cytoplasmic antibodies (ANCA) has been described in sera of patients with autoimmune hepatitis (AIH). The significance of this finding remains uncertain and the nature of the target antigen(s) has not yet been defined. We studied 32 sera from patients with AIH type-I and prepared extracts of human neutrophils to identify the target antigen(s). A 43 kDa dominant immunoreactive protein was found and identified as the cytoskeletal component actin. Initial studies to define the antigenic determinants identified three different actin domains.

AdultMaleNeutrophilsClinical BiochemistryBlotting WesternMolecular Sequence DataFluorescent Antibody TechniqueAutoimmune hepatitisBiologyBiochemistryAnalytical ChemistryAntibodies Antineutrophil CytoplasmicAutoimmune DiseasesHepatitisEpitopesAntigenimmune system diseasesmedicineTumor Cells CulturedAnimalsHumansAmino Acid SequenceAntigensCytoskeletonAnti-neutrophil cytoplasmic antibodyAgedAged 80 and overMiddle Agedmedicine.diseaseRatsBlotEpitope mappingCytoplasmImmunologybiology.proteinFemaleAntibodyElectrophoresis
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Human Dental Pulp Stem Cells Improve Left Ventricular Function, Induce Angiogenesis, and Reduce Infarct Size in Rats with Acute Myocardial Infarction

2008

Abstract Human dental pulp contains precursor cells termed dental pulp stem cells (DPSC) that show self-renewal and multilineage differentiation and also secrete multiple proangiogenic and antiapoptotic factors. To examine whether these cells could have therapeutic potential in the repair of myocardial infarction (MI), DPSC were infected with a retrovirus encoding the green fluorescent protein (GFP) and expanded ex vivo. Seven days after induction of myocardial infarction by coronary artery ligation, 1.5 × 106 GFP-DPSC were injected intramyocardially in nude rats. At 4 weeks, cell-treated animals showed an improvement in cardiac function, observed by percentage changes in anterior wall thic…

AdultMalePathologymedicine.medical_specialtyAdolescentAngiogenesismedicine.medical_treatmentMyocytes Smooth MuscleCell- and Tissue-Based TherapyMyocardial InfarctionNeovascularization PhysiologicBiologystem cell therapyventricular remodelingVentricular Function LeftRats Nudeleft ventricular functionDental pulp stem cellsmedicineAnimalsHumansMyocytes CardiacMyocardial infarctionVentricular remodelingDental PulpCell ProliferationUltrasonographymesenchymal stem cellsStem CellsCardiac muscleCell DifferentiationMesenchymal Stem CellsAmniotic stem cellsCell BiologyStem-cell therapyAnatomymedicine.diseasedental pulp stem cellsRatsRetroviridaemedicine.anatomical_structureMolecular MedicineStem cellRetroviridae InfectionsStem Cell TransplantationDevelopmental BiologyStem Cells
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Reconstruction of critical-size mandibular defects in immunoincompetent rats with human adipose-derived stromal cells

2013

In patients with bony defects, autologous bone grafts are the "gold standard" for reconstruction. In children, autologous bone harvesting is limited but tissue engineering offers an alternative. Next to bone marrow, adipose tissue is a source of mesenchymal stromal cells, and adipose-derived stromal cells (ADSC) can differentiate into osteocytes. The aim of this study was to evaluate the efficacy of bioactive implants (ADSC in fibrin glue) for repair of critical-size mandibular defects in athymic rats. Human adult ADSC embedded in fibrin glue were implanted into a critical-size defect in the rat mandible and their efficacy was compared to those of protected bone healing (pbh), autologous bo…

AdultMalePathologymedicine.medical_specialtyStromal cellAdipose tissueFibrin Tissue AdhesiveBone healingOsteocytesImmunocompromised HostRandom AllocationRats NudeTissue engineeringOsteogenesisImage Processing Computer-AssistedAnimalsHumansMedicineMandibular DiseasesIn patientAutograftsFibrin glueWound HealingBone TransplantationTissue EngineeringTissue Scaffoldsbusiness.industryMesenchymal stem cellMesenchymal Stem CellsX-Ray MicrotomographyAnatomyRatsmedicine.anatomical_structureAdipose TissueOtorhinolaryngologySurgeryBone marrowMandibular ReconstructionOral SurgeryTomography X-Ray ComputedbusinessJournal of Cranio-Maxillofacial Surgery
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Hsp60 response in experimental and human temporal lobe epilepsy

2015

The mitochondrial chaperonin Hsp60 is a ubiquitous molecule with multiple roles, constitutively expressed and inducible by oxidative stress. In the brain, Hsp60 is widely distributed and has been implicated in neurological disorders, including epilepsy. A role for mitochondria and oxidative stress has been proposed in epileptogenesis of temporal lobe epilepsy (TLE). Here, we investigated the involvement of Hsp60 in TLE using animal and human samples. Hsp60 immunoreactivity in the hippocampus, measured by Western blotting and immunohistochemistry, was increased in a rat model of TLE. Hsp60 was also increased in the hippocampal dentate gyrus neurons somata and neuropil and hippocampus proper …

AdultMalePathologymedicine.medical_specialtyanimal structuresHippocampuschemical and pharmacologic phenomenaBiologyHippocampal formationHippocampuscomplex mixturesEpileptogenesisArticleTemporal lobeYoung AdultEpilepsymedicineNeuropilAnimalsHumansTemporal lobe epilepsyMultidisciplinaryHippocampus properDentate gyrusfungiChaperonin 60Middle Agedmedicine.diseaseImmunohistochemistryRatsmedicine.anatomical_structureEpilepsy Temporal Lobenervous systemDentate GyrusFemale
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Cyclooxygenase 2-selective and nonselective nonsteroidal anti-inflammatory drugs induce oxidative stress by up-regulating vascular NADPH oxidases.

2008

Cyclooxygenase 2-selective inhibitors (coxibs) and nonselective nonsteroidal anti-inflammatory drugs (NSAIDs) are associated with an increase in cardiovascular events. The current study was designed to test the effect of coxibs and nonselective NSAIDs on vascular superoxide and nitric oxide (NO) production. mRNA expression of endothelial NO synthase (eNOS) and of the vascular NADPH oxidases was studied in spontaneously hypertensive rats (SHR) and in human endothelial cells. The expression of Nox1, Nox2, Nox4, and p22phox was increased markedly by the nonselective NSAIDs diclofenac or naproxen and moderately by rofecoxib or celecoxib in the aorta and heart of SHR. The up-regulation of NADPH …

AdultMalePharmacologychemistry.chemical_compoundEnosRats Inbred SHRAnimalsHumansPharmacologyNADPH oxidasebiologyCyclooxygenase 2 InhibitorsNitrotyrosineAnti-Inflammatory Agents Non-SteroidalNOX4NADPH Oxidasesbiology.organism_classificationRatsUp-RegulationOxidative StresschemistryCyclooxygenase 2NOX1Apocynincardiovascular systembiology.proteinMolecular MedicineFemaleP22phoxEndothelium VascularPeroxynitriteThe Journal of pharmacology and experimental therapeutics
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