Search results for "sarcoidosis"

showing 10 items of 36 documents

Lung Compartmentalization of Increased TNF Releasing Ability by Mononuclear Phagocytes in Pulmonary Sarcoidosis

1989

The TNF is a monokine with cytotoxic and tumor-necrosing activities; in addition, TNF may play a role in inflammatory processes. The present study evaluates spontaneous and LPS-mediated release of TNF by AMs and autologous peripheral BMs of normal subjects and patients with pulmonary sarcoidosis. A recently developed cytotoxicity assay, specific for detection of TNF activity, was applied. This study demonstrates that (1) unstimulated mononuclear phagocytes released low levels of TNF with no differences between groups; (2) when effector cells were stimulated with LPS, AMs from patients with active pulmonary sarcoidosis released more TNF than AMs recovered from normal subjects and from patien…

AdultLung DiseasesMalePulmonary and Respiratory MedicineSarcoidosisCritical Care and Intensive Care MedicinePathogenesisHumansMedicineMacrophageLungLungTumor Necrosis Factor-alphabusiness.industryMacrophagesRespiratory diseaseMononuclear phagocyte systemCytotoxicity Tests Immunologicmedicine.diseasePulmonary AlveoliMonokinemedicine.anatomical_structureImmunologyLeukocytes MononuclearFemaleTumor necrosis factor alphaSarcoidosisCardiology and Cardiovascular MedicinebusinessBronchoalveolar Lavage FluidChest
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Correlation of Clinical and Immunologic Parameters of the Inflammatory Activity of Pulmonary Sarcoidosis

1991

The evaluation of activation markers such as T4/T8 ratio and HLA-DR expression of lymphocytes of bronchoalveolar lavage (L-BAL) is an important clinical approach for the staging of sarcoidosis. However, it is not known to what extent this is paralleled by an exaggerated lymphocyte function. We investigated the dependence of L-BAL activation markers on the production of interleukin-2 (IL-2) by L-BAL and on the soluble IL-2 receptor serum level (sIL-2R) in 116 patients with sarcoidosis. In none of the combinations tested was a correlation between the two groups of parameters found; r less than 0.5, upper 90% confidence limit of r less than 0.8. Interestingly, IL-2 production is independent of…

AdultLung DiseasesMalePulmonary and Respiratory MedicineSystemic diseaseSarcoidosisT-LymphocytesLymphocyteCD4-CD8 RatioCD4-CD8 RatioEnzyme-Linked Immunosorbent AssayInflammationLymphocyte ActivationHumansMedicineReceptormedicine.diagnostic_testbusiness.industryRespiratory diseaseReceptors Interleukin-2HLA-DR Antigensrespiratory systemmedicine.diseaserespiratory tract diseasesBronchoalveolar lavagemedicine.anatomical_structureImmunologyInterleukin-2FemaleSarcoidosismedicine.symptombusinessBronchoalveolar Lavage FluidAmerican Review of Respiratory Disease
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Increased interleukin 6 production by bronchoalveolar lavage cells in patients with active sarcoidosis

1993

Alveolitis of sarcoidosis is characterized by activated alveolar macrophages (AMs) and T cells. The mediators interleukin-1 (IL-1) and interleukin 6 (IL-6) released by AMs represent essential factors for the progression of the T cells in the cell cycle. The role of IL-1 in pulmonary sarcoidosis has previously been studied; however, the relevance of other mediators (i.e. IL-6) has not yet been evaluated. We measured the spontaneous and lipopolysaccharide (LPS)-induced release of IL-6 and tumor necrosis factor alpha (TNF alpha) by bronchoalveolar lavage cells (BAL) and peripheral blood mononuclear cells (PBMNC) in 6 control subjects (group A) and in 15 patients with sarcoidosis, 10 with activ…

AdultLung DiseasesMalePulmonary and Respiratory Medicinemedicine.medical_specialtySarcoidosisLipopolysaccharidePeripheral blood mononuclear cellchemistry.chemical_compoundInternal medicineHumansMedicineInterleukin 6AgedLungmedicine.diagnostic_testbiologyInterleukin-6Tumor Necrosis Factor-alphabusiness.industryMiddle AgedCell cyclemedicine.diseaseBronchoalveolar lavageEndocrinologymedicine.anatomical_structurechemistryImmunologybiology.proteinFemaleTumor necrosis factor alphaSarcoidosisbusinessBronchoalveolar Lavage FluidLung
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Spontaneous Monokine Release by Alveolar Macrophages in Chronic Sarcoidosis

1991

In pulmonary sarcoidosis an activation of alveolar T lymphocytes and alveolar macrophages (AM) has been demonstrated. There is evidence that in contrast to acute disease a heightened T-cell response cannot be observed in the chronic phase of sarcoidosis. The role of AM in the inflammatory process of chronic sarcoidosis is not yet intensively evaluated. To address this question we measured the release of tumor necrosis factor alpha (TNFα) and interleukin-1 (IL-1) by AM of 39 patients with chronic sarcoidosis (duration > 4 years; 30 active, 9 inactive diseases) without therapy and correlated the monokine release with parameters of T-cell alveolitis and the course of the disease. The T4/T8 …

AdultLung DiseasesMaleSarcoidosisT-Lymphocytesmedicine.medical_treatmentImmunologyCD4-CD8 Ratio610 MedizinBronchoalveolar Lavage Fluid/immunologyTumor Necrosis Factor-alpha/biosynthesisLymphocyte Activation/immunologyLymphocyte ActivationMacrophages AlveolarmedicineHumansImmunology and AllergyMacrophageAntibodies Monoclonal/immunologyInterleukin-1/biosynthesisddc:610Tumor Necrosis Factor-alphabusiness.industryRespiratory diseaseAntibodies MonoclonalInterleukinGeneral MedicineT-Lymphocytes/immunologymedicine.diseaseSarcoidosis/immunologyMonokineLung Diseases/immunologyCytokinemedicine.anatomical_structureChronic DiseaseImmunologyMacrophages Alveolar/immunologyFemaleTumor necrosis factor alphaSarcoidosisPulmonary alveolusbusinessBronchoalveolar Lavage FluidInterleukin-1International Archives of Allergy and Immunology
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Spontaneous interleukin 2 release of bronchoalveolar lavage cells in sarcoidosis is a codeterminator of prognosis

1996

There is mounting evidence that activated interleukin 2 (IL-2)-releasing lymphocytes play a central role in the immunopathogenesis of sarcoidosis by directing inflammatory reactions and granuloma formation. In the context that a significant proportion of these cells accumulates in the lung and releases mediators, we hypothesized that different immunologically defined stages of sarcoidosis can be identified. A cohort of 89 sarcoidosis patients was allocated to four groups according to the following criteria: stage A, a low number of bronchoalveolar lavage (BAL) lymphocytes (20%) without IL-2 release (1 unit/ml in BAL cell culture supernatant); stage B, BAL lymphocytes20%, with IL-2 release (…

AdultMalePulmonary and Respiratory MedicineInterleukin 2T-LymphocytesContext (language use)InflammationStatistics NonparametricPulmonary function testingSarcoidosis PulmonarymedicineHumansCells CulturedChi-Square DistributionLungmedicine.diagnostic_testbusiness.industryrespiratory systemPrognosismedicine.diseaserespiratory tract diseasesBronchoalveolar lavagemedicine.anatomical_structureImmunologyInterleukin-2FemaleSarcoidosismedicine.symptombusinessBronchoalveolar Lavage FluidProgressive diseaseFollow-Up Studiesmedicine.drugLung
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Sudden death due to cardiac sarcoidosis in a case of suspected homicide

1993

In a case of suspected homicide death due to natural causes — cardiac death (SCD) — was found at autopsy. Despite an extensive replacement of myocardial tissue by sarcoid granulomata there was no history of cardiac dysfunction or preceding symptoms. The transmurally infiltrating granulomata and the concomitant fibrosis were predominantly confluent. They occupied vast areas within the interventricular septum and the adjacent posterior wall of the left ventricle. The only other organs involved were mediastinal lymph nodes, which appeared macroscopically normal.

AdultMalemedicine.medical_specialtyPathologySarcoidosisAutopsySudden deathPathology and Forensic MedicineDiagnosis DifferentialHomicideFibrosishemic and lymphatic diseasesInternal medicineHumansMedicineInterventricular septumbusiness.industryMyocardiummedicine.diseaseDeath Sudden Cardiacmedicine.anatomical_structureVentricleConcomitantCardiologyLymphCardiomyopathiesHomicidebusinessEndocardiumInternational Journal of Legal Medicine
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GRANULOMATOUS MASTITIS DURING CHRONIC ANTIDEPRESSANT THERAPY: IS IT POSSIBLE A CONSERVATIVE THERAPEUTIC APPROACH?

2012

Granulomatous mastitis is a rare benign inflammatory disease of the breast with multiple etiologies such as tuberculosis, sarcoidosis, foreign body reaction, and mycotic and parasitic infections. In contrast, idiopathic granulomatous mastitis (IGM) is characterized by the presence of chronic granulomatous lobulitis in the absence of an obvious etiology. Clinically and radiologically it may mimic breast carcinoma and so awareness of surgeons, pathologists, and radiologists is essential to avoid unnecessary mastectomies. Cases of IGM are reported during antidepressant therapy in patients also showing high levels of prolactinemia. In these cases, we believe that surgical excision must be avoid…

Antidepressant therapyCancer Researchmedicine.medical_specialtyTuberculosisbusiness.industryDiseaseGranulomatous mastitismedicine.diseaseBrief CommunicationDermatologySurgeryHyperprolactinemiaTherapeutic approachSettore MED/18 - Chirurgia GeneraleBreast cancerOncologySelective serotonin reuptake inhibitorsmedicineEtiologySarcoidosisantidepressant therapy idiopathic granulomatous mastitis selective serotin reuptake inhibitorsIdiopathic granulomatous mastitisbusinessPathological
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Proteomic analysis of Kveim reagent identifies targets of cellular immunity in sarcoidosis

2017

Background Kveim-reagent (Kv) skin testing was a historical method of diagnosing sarcoidosis. Intradermal injection of treated sarcoidosis spleen tissue resulted in a granuloma response at injection site by 4–6 weeks. Previous work indicates proteins as the possible trigger of this reaction. We aimed to identify Kv-specific proteins and characterise the ex vivo response of Peripheral Blood Mononuclear Cells (PBMCs) from sarcoidosis, tuberculosis and healthy control patients when stimulated with both Kv and selected Kv-specific proteins. Methods Kv extracts were separated by 1D-SDS-PAGE and 2D-DIGE and then underwent mass spectrometric analysis for protein identification. Sarcoidosis and con…

Bacterial DiseasesMaleProteomics0301 basic medicineCellular immunityPhysiologylcsh:MedicineVimentinBiochemistry0302 clinical medicineTandem Mass SpectrometryImmune PhysiologyMedicine and Health SciencesMedicinelcsh:ScienceCITRULLINATED VIMENTINInnate Immune SystemImmunity CellularMultidisciplinarybiologyMYCOBACTERIAL CATALASE-PEROXIDASEPEPTIDESMiddle Aged3. Good healthMultidisciplinary SciencesInfectious DiseasesGranulomaCytokinesScience & Technology - Other TopicsElectrophoresis Polyacrylamide GelFemaleResearch ArticleAdultSarcoidosisGeneral Science & TechnologyInflammatory DiseasesSYSTEMIC SARCOIDOSISImmunologyANTIGENTUBERCULOSISPeripheral blood mononuclear cell03 medical and health sciencesImmune systemRheumatologyAntigenMD MultidisciplinaryVimentinHumansSecretionScience & Technologybusiness.industrylcsh:RBiology and Life SciencesProteinsMolecular DevelopmentTropical Diseasesmedicine.diseaseRHEUMATOID-ARTHRITISCytoskeletal Proteins030104 developmental biology030228 respiratory systemImmune SystemImmunologybiology.proteinT-CELLSIndicators and Reagentslcsh:QCytokine secretionBODIESPhysiological ProcessesbusinessSpleenEx vivoDevelopmental BiologyRCRESPONSES
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Reduced T-cell receptor CD3ζ-chain protein and sustained CD3ε expression at the site of mycobacterial infection

2001

Control of mycobacterial infection by the cellular immune system relies both on antigen-presenting cells and on T lymphocytes. The quality of an effective cellular immune response is dependent on functional signal transduction residing in the cytoplasmic tails of the T-cell receptor CD3 components. In order to investigate potential effects of mycobacteria on T-cell receptor signalling, we examined the protein expression of T-cell signal transduction molecules (CD3zeta, ZAP-70, p59fyn, p56lck). In Western blots of peripheral blood mononuclear cells of Mycobacterium tuberculosis infected patients, only the CD3zeta-chain showed a marked reduction in protein expression. To investigate the situa…

CD3 ComplexCD3ImmunologyPalatine TonsilReceptors Antigen T-CellFluorescent Antibody TechniqueImmunofluorescenceProto-Oncogene Proteins c-fynPeripheral blood mononuclear cellImmunoenzyme TechniquesImmune systemSarcoidosis PulmonaryProto-Oncogene ProteinsmedicineImmunology and AllergyHumansReceptorTuberculosis PulmonaryMycobacterium InfectionsGranulomaZAP-70 Protein-Tyrosine Kinasemedicine.diagnostic_testbiologyT-cell receptorMembrane ProteinsOriginal ArticlesProtein-Tyrosine KinasesMolecular biologyLeprosy LepromatousLymphatic systemLymphocyte Specific Protein Tyrosine Kinase p56(lck)Immunologybiology.proteinInterleukin-2Signal transductionSignal Transduction
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Assessment of local cellular immunity in lung cancer by bronchoalveolar lavage.

1990

Small cell lung cancer (SCLC) is the most malignant of the pulmonary neoplasms and is associated with a poor local cellular immune response. 16 patients with non small cell lung cancer (NSCLC) and 11 patients with SCLC underwent bronchoalveolar lavage (BAL) in the lung which harbored the tumor in order to investigate the lymphocyte surface antigens utilizing the immunoperoxidase technique. Analysis of blood lymphocytes was performed in parallel. 8 patients with previous sarcoidosis in complete remission who underwent BAL and 10 normal blood donors served as controls. Among blood lymphocytes the CD3+, CD4+ and CD16+ cell populations were elevated significantly and the T4/T8 ratio was elevate…

CD4-Positive T-LymphocytesCellular immunityPathologymedicine.medical_specialtyLung NeoplasmsLymphocyteT-LymphocytesAdenocarcinomaLeukocyte CountImmune systemAntigens CDCarcinoma Non-Small-Cell LungDrug DiscoveryImmune ToleranceMedicineHumansCarcinoma Small CellLung cancerGenetics (clinical)Lungmedicine.diagnostic_testImmunoperoxidasebusiness.industryReceptors Interleukin-2General Medicinerespiratory systemmedicine.diseaserespiratory tract diseasesBronchoalveolar lavagemedicine.anatomical_structureImmunologyCarcinoma Squamous CellMolecular MedicineSarcoidosisbusinessBronchoalveolar Lavage FluidKlinische Wochenschrift
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