Search results for "substitut"

showing 10 items of 1337 documents

The type of GnRH analogue used during controlled ovarian stimulation influences early embryo developmental kinetics: a time-lapse study

2013

OBJECTIVE: To explore if the GnRH analogue used for controlled ovarian stimulation (COS) and the ovulation triggering factor (GnRH agonist + hCG triggering versus GnRH antagonist + GnRH agonist triggering) affect embryo development and kinetics.STUDY DESIGN: In a retrospective cohort study in the Instituto Valenciano de Infertilidad (IVI) Alicante and the Instituto Universitario-IVI Valencia, Spain, 2817 embryos deriving from 400 couples undergoing oocyte donation were analysed. After controlled ovarian stimulation and IVF/intracytoplamic sperm injection, the timing of embryonic cleavages was assessed by a video time-lapse system. The results were analysed using Student's t test for compari…

Pregnancy RateZygoteGonadotropin-Releasing Hormone/adverse effectsmedicine.medical_treatmentGonadotropin-releasing hormoneChorionic GonadotropinGonadotropin-Releasing HormoneEmbryo Culture TechniquesCohort StudiesTime-lapsePregnancyGnRH analogueFertility Agents Female/adverse effectsmedia_commonOocyte DonationObstetrics and GynecologyEmbryoEmbryo transferembryonic structuresSpain/epidemiologyEctogenesis/drug effectsFemaleInfertility Femalehormones hormone substitutes and hormone antagonistsEmbryo qualityAdultAgonistInfertility Female/therapyendocrine systemmedicine.medical_specialtymedicine.drug_classCleavage Stage Ovummedia_common.quotation_subjectFertilization in VitroBiologyTime-Lapse ImagingOvulation InductionInternal medicinemedicineHumansOvulationRetrospective StudiesZygote/drug effectsFertility Agents FemaleOvulation Induction/methodsEmbryo TransferEmbryo developmental kineticsKineticsPregnancy rateEndocrinologyReproductive MedicineSpainEctogenesisChorionic Gonadotropin/pharmacologyOvulation inductionCleavage Stage Ovum/drug effectsEuropean Journal of Obstetrics & Gynecology and Reproductive Biology
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Italian familial defective apolipoprotein B patients share a unique haplotype with other Caucasian patients.

2001

Familial defective apolipoprotein (apo) B-100 together with familial hypercholesterolemia are the two common genetic conditions that cause hypercholesterolemia. Familial defective apolipoprotein B-100 is due to mutations around codon 3500 of the apo B gene. The most-characterized mutation is a G>A transition at nucleotide 10,708 that results in the substitution of arginine by glutamine at codon 3500 (Apo B Arg3500Gln). Two other mutations are caused by a C>T transition, one at nucleotide 10,800 (Apo B Arg3531Cys) and the other at nucleotide 10,707 (apo B Arg3500Trp). In the present study we describe three new Italian cases of familial defective apolipoprotein B-100 (Apo B Arg3500Gln), one f…

ProbandChinaSettore MED/09 - Medicina InternaApolipoprotein BGlutamineEuropean Continental Ancestry GroupHypercholesterolemiaFamilial hypercholesterolemiamedicine.disease_causeArgininePolymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologyWhite PeopleHaplotypemedicineHumansCysteineAlleleCodonGeneApolipoproteins BGeneticsMutationbiologyTransition (genetics)HaplotypeGeneral Medicinemedicine.diseaseEuropeSettore MED/03 - Genetica MedicaAmino Acid SubstitutionHaplotypesItalyApolipoprotein B-100Mutationbiology.proteinlipids (amino acids peptides and proteins)HumanClinical and experimental medicine
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Additive effect of mutations in LDLR and PCSK9 genes on the phenotype of familial hypercholesterolemia.

2006

Patients homozygous or Compound heterozygous for LDLR mutations or double heterozygous for LDLR and apo B R3500Q mutation have higher LDL-C levels. more extensive xanthomatosis and more severe premature coronary disease (pCAD) than simple heterozygotes for mutations in either these genes or for missense mutations in PCSK9 gene. It is not known whether combined mutations in LDLR and PKCS9 are associated with such a severe phenotype. We sequenced Apo B and PCSK9 genes in two patients with the clinical diagnosis of homozygous FH who were heterozygous for LDLR gene mutations. Proband Z.P. (LDL-C 13.39 mmol/L and pCAD) was heterozygous for an LDLR mutation (p.E228K) inherited from her father (LD…

ProbandLDLR geneAdultMaleSettore MED/09 - Medicina InternaApolipoprotein BFamilial hypercholesterolemia (FH); Autosomal dominant hypercholesterolemia 3 (ADH3); LDLR gene; PCSK9 gene; Premature coronary artery diseasePremature coronary artery diseaseLDLR PCSK9Mutation MissenseFamilial hypercholesterolemiaCompound heterozygositymedicine.disease_causeHyperlipoproteinemia Type IIFamilial hypercholesterolemia (FH) Autosomal dominant hypercholesterolemia 3 (ADH3) LDLR gene PCSK9 gene Premature coronary artery diseaseFamilial hypercholesterolemia (FH)medicineMissense mutationHumansCells CulturedGeneticsMutationbiologybusiness.industrySerine EndopeptidasesHeterozygote advantageMiddle Agedmedicine.diseaseAutosomal dominant hypercholesterolemia 3 (ADH3)PedigreePhenotypeSettore MED/03 - Genetica MedicaAmino Acid SubstitutionReceptors LDLPCSK9 geneLDL receptorbiology.proteinlipids (amino acids peptides and proteins)FemaleProprotein ConvertasesProprotein Convertase 9Cardiology and Cardiovascular MedicinebusinessAtherosclerosis
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Widespread selection for high and low secondary structure in coding sequences across all domains of life

2019

AbstractCodon composition, GC-content and local RNA secondary structures can have a profound effect on gene expression and mutations affecting these parameters, even though they do not alter the protein sequence, are not neutral in terms of selection. Although evidence exists that in some cases selection favors more stable RNA secondary structures, we currently lack a concrete idea of how many genes are affected within a species, and if this is a universal phenomenon in nature.We searched for signs of structural selection in a global manner, analyzing a set of one million coding sequences from 73 species representing all domains of life, as well as viruses, by means of our newly developed s…

Protein sequencingEvolutionary biologyThree-domain systemRNABiologySynonymous substitutionProtein secondary structureGeneSelection (genetic algorithm)Nucleic acid secondary structure
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Human apolipoprotein A-I natural variants: molecular mechanisms underlying amyloidogenic propensity

2012

Human apolipoprotein A-I (apoA-I)-derived amyloidosis can present with either wild-type (Wt) protein deposits in atherosclerotic plaques or as a hereditary form in which apoA-I variants deposit causing multiple organ failure. More than 15 single amino acid replacement amyloidogenic apoA-I variants have been described, but the molecular mechanisms involved in amyloid-associated pathology remain largely unknown. Here, we have investigated by fluorescence and biochemical approaches the stabilities and propensities to aggregate of two disease-associated apoA-I variants, apoA-IGly26Arg, associated with polyneuropathy and kidney dysfunction, and apoA-ILys107-0, implicated in amyloidosis in severe…

ProteomicsProtein Foldinglcsh:MedicineProtein aggregationpolymyxinsBiochemistryProtein Structure SecondaryMiceProtein structureneutrophilsMolecular Cell Biologypolycyclic compoundslcsh:ScienceCellular Stress ResponsesMultidisciplinaryProtein StabilityAmyloidosisCiencias QuímicasfluorescenseCell biologymacrophagesBiochemistryToxicityMedicineProtein foldinglipids (amino acids peptides and proteins)medicine.symptomPolyneuropathyResearch ArticleProtein StructureMedicinaLipoproteinsImmunologyBiophysicsInflammationAmyloidogenic ProteinsBiologyProtein ChemistryMicrobiologyCell Lineprotein aggregationmacrophage activationmedicineAnimalsHumansoligomersProtein InteractionsBiologyInflammationamyloidosisApolipoprotein A-IMacrophageslcsh:RImmunityProteinsnutritional and metabolic diseasesmedicine.diseaseApolipoproteinsAmino Acid SubstitutionCell cultureinflammationCiencias Médicaslcsh:QClinical ImmunologyMutant ProteinspolyneuropathyProtein Multimerization
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Regioselective C–H amination of free base porphyrins via electrogenerated pyridinium-porphyrins and stabilization of easily oxidized amino-porphyrins…

2020

Four free base aminoporphyrins were synthesized in two steps via regioselective anodic nucleophilic substitution with pyridine followed by ring opening of the electrogenerated pyridinium with piperidine. The X-ray crystallographic structure of the unstable 2-aminotetraphenylporphyrin was solved. Protonation of this latter compound leads to the stable diiminium porphyrin salt.

Protonation010402 general chemistry01 natural sciencesCatalysischemistry.chemical_compound[CHIM.ANAL]Chemical Sciences/Analytical chemistryPyridinePolymer chemistryMaterials ChemistryNucleophilic substitution[CHIM.COOR]Chemical Sciences/Coordination chemistryAmination[CHIM.ORGA]Chemical Sciences/Organic chemistry010405 organic chemistryMetals and AlloysFree base[CHIM.MATE]Chemical Sciences/Material chemistryGeneral ChemistryPorphyrin0104 chemical sciencesSurfaces Coatings and FilmsElectronic Optical and Magnetic MaterialschemistryCeramics and CompositesPyridiniumPiperidineChemical Communications
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DOES LEPTIN PLAY A CYTOKINE-LIKE ROLE WITHIN THE AIRWAYS OF COPD PATIENTS?

2005

The leptin-leptin receptor system might be up-regulated in the airways of chronic obstructive pulmonary disease (COPD). In bronchial biopsies obtained from normal subjects and smokers, with and without COPD, the present study examined leptin and leptin-receptor expression and their co-localisation in airway and inflammatory cells. Combining immunohistochemistry with terminal deoxynucleotidyl transferase dUTP nick end-labelling techniques, apoptosis in airway and inflammatory cells and in leptin and leptin-receptor expressing cells was investigated. In the epithelial cells both leptin and leptin-receptor expression was higher in normal subjects than in smokers and COPD subjects. By contrast,…

Pulmonary and Respiratory MedicineAdultLeptinMalemedicine.medical_specialtyCopd patientsmedicine.medical_treatmentT-LymphocytesApoptosisBronchiReceptors Cell SurfaceRespiratory MucosaPulmonary Disease Chronic ObstructiveInternal medicineBiopsyMedicineHumansAgedCOPDmedicine.diagnostic_testbusiness.industryLeptindigestive oral and skin physiologyRespiratory diseaseSmokingrespiratory systemMiddle Agedmedicine.diseaseObstructive lung diseaserespiratory tract diseasesRespiratory Function TestsEndocrinologyCytokineTerminal deoxynucleotidyl transferaseCase-Control StudiesImmunologyPhysical therapyReceptors LeptinFemalebusinesshormones hormone substitutes and hormone antagonistsCD8
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Fluticasone/formoterol combination therapy versus budesonide/formoterol for the treatment of asthma: a randomized, controlled, non-inferiority trial …

2012

The inhaled corticosteroid fluticasone propionate (fluticasone) and the long-acting β₂ agonist formoterol fumarate (formoterol) have been combined in a single aerosol inhaler fluticasone/formoterol (flutiform(®)). This study compared the efficacy and safety of fluticasone/formoterol with the combination product budesonide/formoterol (Symbicort(®) Turbohaler(®)).A randomized, double-blind, double-dummy, multicenter, Phase 3 study comprising a 7- (± 3) day screening, 2-4-week run-in, and 12-week treatment periods. Patients aged ≥ 12 years with moderate to severe persistent asthma for ≥ 6 months before screening and forced expiratory volume in one second (FEV₁) 50-80% predicted and ≥ 15% rever…

Pulmonary and Respiratory MedicineBudesonideAdultMaleAdolescentFluticasone propionateYoung AdultDouble-Blind Methodimmune system diseasesForced Expiratory VolumeFormoterol FumarateAdministration InhalationImmunology and AllergyMedicineHumansBudesonideFluticasoneAgedbusiness.industryInhalerDry Powder Inhalersrespiratory systemMiddle AgedAsthmarespiratory tract diseasesBronchodilator AgentsAndrostadienesDrug CombinationsBudesonide/formoterolEthanolaminesAnesthesiaPediatrics Perinatology and Child HealthSalbutamolFluticasoneFormoterol FumarateFemaleFormoterolbusinesshormones hormone substitutes and hormone antagonistscirculatory and respiratory physiologymedicine.drugThe Journal of asthma : official journal of the Association for the Care of Asthma
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The effect of budesonide/formoterol maintenance and reliever therapy on the risk of severe asthma exacerbations following episodes of high reliever u…

2012

Abstract Background Divergent strategies have emerged for the management of severe asthma. One strategy utilises high and fixed doses of maintenance treatment, usually inhaled corticosteroid/long-acting β2-agonist (ICS/LABA), supplemented by a short-acting β2-agonist (SABA) as needed. Alternatively, budesonide/formoterol is used as both maintenance and reliever therapy. The latter is superior to fixed-dose treatment in reducing severe exacerbations while achieving similar or better asthma control in other regards. Exacerbations may be reduced by the use of budesonide/formoterol as reliever medication during periods of unstable asthma. We examined the risk of a severe exacerbation in the per…

Pulmonary and Respiratory MedicineBudesonideExacerbationAsthma in primary careSeverity of Illness Indexlaw.inventionRandomized controlled trialDouble-Blind MethodlawAdrenal Cortex HormonesRisk FactorsFormoterol FumarateAdministration InhalationmedicineBudesonide Formoterol Fumarate Drug CombinationHumansAnti-Asthmatic AgentsBudesonideAsthmalcsh:RC705-779Maintenance dosebusiness.industryResearchlcsh:Diseases of the respiratory systemmedicine.diseaseAsthmaBronchodilator AgentsDrug CombinationsTreatment OutcomeBudesonide/formoterolEthanolaminesAnesthesiaDisease ProgressionFormoterol FumarateDrug Therapy CombinationFormoterolbusinesshormones hormone substitutes and hormone antagonistsmedicine.drugRespiratory research
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Knowledge Gaps and Research Priorities in Immune Checkpoint Inhibitor–related Pneumonitis. An Official American Thoracic Society Research Statement

2019

Rationale: Immune checkpoint inhibitors (ICIs) have revolutionized cancer care but are associated with unique adverse events, including potentially life-threatening pneumonitis. The diagnosis of ICI-pneumonitis is increasing; however, the biological mechanisms, clinical and radiologic features, and the diagnosis and management have not been well defined. Objectives: To summarize evidence, identify knowledge and research gaps, and prioritize topics and propose methods for future research on ICI-pneumonitis. Methods: A multidisciplinary group of international clinical researchers reviewed available data on ICI-pneumonitis to develop and refine research questions pertaining to ICI-pneumonitis.…

Pulmonary and Respiratory MedicineOncologymedicine.medical_specialtyBiomedical ResearchLung Neoplasmsmedicine.medical_treatmentMEDLINENSCLCCritical Care and Intensive Care Medicine03 medical and health sciences0302 clinical medicineRisk FactorsCarcinoma Non-Small-Cell LungInternal medicinepolycyclic compoundsmedicineCarcinomaHumansImmunologic FactorsOrganizational Objectives030212 general & internal medicineAdverse effectLung cancerSocieties MedicalPneumonitisAmerican Thoracic Society Documentsinterstitial lung diseasebusiness.industryInterstitial lung diseaseCancerPneumoniaImmunotherapymedicine.diseaseUnited StatesGenes cdclung cancer030228 respiratory systemResearch Designimmunotherapybusinesshormones hormone substitutes and hormone antagonistsAmerican Journal of Respiratory and Critical Care Medicine
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