Search results for "synovia"

showing 10 items of 116 documents

Oxidative stress in osteoarticular diseases

2016

chemistry.chemical_classificationReactive oxygen speciesbusiness.industryOsteoporosisSynovial hyperplasiaOsteoarthritismedicine.diseasemedicine.disease_causechemistry.chemical_compoundImmune systemchemistryRheumatoid arthritisImmunologyMedicinebusinessReactive nitrogen speciesOxidative stress
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Effects of dexamethasone on human synovial fibroblast-like cells, from osteoarthritic joints, in culture.

1990

The effect of Dexamethasone (DEX) on cell division and macromolecular synthesis was investigated in a line (McCoy cells, A 9) of synovial fibroblast-like cells derived from human osteoarthritic joints. DEX markedly reduced the proliferation of McCoy cells in a time and dose-dependent manner. The maximal inhibition (45%) was found at 500 nM DEX 24 h after incubation and was accompanied by the appearance of giant macrophage-like cells. After DEX treatment cells showed increased content of DNA, proteins and RNA together with the reduction of [3H]-thymidine incorporation into the TCA-precipitable fraction.

endocrine systemmedicine.medical_specialtyTime FactorsCell divisionHydrocortisoneSomatic cellCell SurvivalCell CountBiologyGeneral Biochemistry Genetics and Molecular BiologyDexamethasoneCell Linechemistry.chemical_compoundInternal medicineOsteoarthritisSynovial Fluidpolycyclic compoundsmedicineSynovial fluidHumansGeneral Pharmacology Toxicology and PharmaceuticsFibroblastDexamethasoneCell growthGeneral MedicineDNAFibroblastsMolecular biologycultureEndocrinologymedicine.anatomical_structurechemistryCell cultureRNAThymidinehormones hormone substitutes and hormone antagonistsCell Divisionmedicine.drugLife sciences
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Synovial giant cells in rheumatoid arthritis: Expression of cystatin C, but not of cathepsin B

2000

This study was designed to investigate the expression of the matrix degrading proteinase cathepsin B and its endogenous inhibitor cystatin C in rheumatoid arthritis (RA) with special regard to multinucleated synovial giant cells (SGC). We applied an immunohistochemical double-labeling technique. SGC strongly expressed cystatin C and CD68, but were negative for cathepsin B. This staining pattern occurred in osteoclasts as well. Our findings support the idea that in RA matrix destruction by cathepsin B is not mediated by SGC or osteoclasts, but by mononuclear synoviocytes.

inorganic chemicalsPathologymedicine.medical_specialtyArthritisCysteine Proteinase InhibitorsToxicologyGiant CellsCathepsin BCathepsin BPathology and Forensic MedicineArthritis RheumatoidOsteoclastCathepsin L1Synovial FluidmedicineHumansCystatin CCathepsinHyperplasiabiologyCell BiologyGeneral Medicinemedicine.diseaseCystatinsImmunohistochemistryMolecular biologymedicine.anatomical_structureCystatin Ccardiovascular systembiology.proteinCystatinSynovial membraneExperimental and Toxicologic Pathology
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Mechanobiological computational model for the development and formation of synovial joints

2019

El desarrollo de las articulaciones sinoviales se debe a diferentes factores genéticos, bioquímicos y mecánicos. Comienza en el brote de las extremidades, que tienen una masa ininterrumpida de células mesenquimales dentro de su núcleo, el blastema esquelético. La mayoría de estas células blastemales se diferencian en condrocitos; sin embargo, algunas de estas células permanecen, sin diferenciar, en el sitio de la futura articulación (interzona). La separación de los rudimentos ocurre con el proceso de cavitación dentro de la interzona. Después de la cavitación, se produce la morfogénesis articular y el hueso toma su forma final. Una vez finalizado el período embrionario, la articulación sin…

joint developmentjoin onsetUNESCO::CIENCIAS MÉDICAS ::Patología::Histopatologíacomputational modelsarticular cartilagejoint morphogenesissynovial joints:CIENCIAS MÉDICAS ::Patología::Histopatología [UNESCO]
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Human Memory Th17 Cell Populations Change Into Anti-inflammatory Cells With Regulatory Capacity Upon Exposure to Active Vitamin D

2019

Autoimmune diseases are characterized by an aberrantly activated immune system, resulting in tissue damage and functional disability in patients. An important therapeutic goal is to restore the deregulated immunological balance between pro- A nd anti-inflammatory T cells. This imbalance is illustrated by elevated levels and activity of memory Th17 cell populations, such as Th17, Th1/Th17, and Th17.1 cells, in various autoimmune diseases. These cells are characterized by the chemokine receptor CCR6, RORC expression and production of IL-17A, IFNγ, and TNFα. Using rheumatoid arthritis (RA) as a model of autoimmune disease, we here demonstrate that pro-inflammatory memory CCR6+ Th cells can swi…

lcsh:Immunologic diseases. Allergy0301 basic medicineAdultMaleReceptors CCR6rheumatoid arthritisCD3 ComplexCD3CellImmunologyAnti-Inflammatory Agentschemical and pharmacologic phenomenavitamin DC-C chemokine receptor type 6Autoimmune DiseasesArthritis Rheumatoid03 medical and health sciencesChemokine receptor0302 clinical medicineImmune systemsynovial fluidRAR-related orphan receptor gammamedicineImmunology and AllergyHumansCells CulturedOriginal ResearchAutoimmune diseasebiologyChemistryTumor Necrosis Factor-alphaInterleukinsMiddle Agedmedicine.diseaseTreg030104 developmental biologymedicine.anatomical_structureImmunologybiology.proteinTh17 CellsTumor necrosis factor alphaFemaleTh17lcsh:RC581-607Immunologic Memory030215 immunologyFrontiers in Immunology
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Artificial cartilage bio-matrix formed of hyaluronic acid and Mg2+-polyphosphate.

2016

Here we show that inorganic polyphosphate (polyP), a polyanionic metabolic regulator consisting of multiple phosphate residues linked by energy-rich phosphoanhydride bonds, is present in the synovial fluid. In a biomimetic approach, to enhance cartilage synthesis and regeneration, we prepared amorphous polyP microparticles with Mg2+ as counterions. The particles were characterised by X-ray diffraction (XRD), energy-dispersive X-ray (EDX) and Fourier transformed infrared spectroscopic (FTIR) analyses. Similar particles were obtained after addition of Mg2+ ions to a solution containing hyaluronic acid, as a major component of the synovial fluid, and soluble Na-polyP. The viscous paste-like ma…

magnesium polyphosphatelcsh:Diseases of the musculoskeletal systemlcsh:Surgeryregenerative medicine02 engineering and technologyCartilage metabolism01 natural sciencesChondrocyteExtracellular matrixchemistry.chemical_compoundCollagen Type IIIChondrocytesX-Ray DiffractionPolyphosphatesHyaluronic acidSpectroscopy Fourier Transform InfraredSynovial FluidmedicineCell AdhesionSynovial fluidHumansMagnesiumRNA MessengerHyaluronic Acidmicroparticles010405 organic chemistryCartilagePolyphosphateSpectrometry X-Ray EmissionSOX9 Transcription Factorlcsh:RD1-811021001 nanoscience & nanotechnology0104 chemical sciencesExtracellular MatrixUp-Regulationosteoarthritismedicine.anatomical_structureCartilageCollagen Type IIIchemistrytissue engineeringBiophysicsMicroscopy Electron Scanninglcsh:RC925-9350210 nano-technologyBiomedical engineering
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Cartilage Regeneration and Tissue Engineering

2019

Abstract Articular cartilage in synovial joints is a hyaline cartilage highly hydrated with a rigorous order of cells and fibers and a specific content of proteoglycans and glycoproteins. It provides a low-friction surface, participates in the lubrication of the synovial joints, and distributes the forces to the underlying bone. It is an avascular and aneural tissue where small metabolites diffuse to and from cells. Unlike hyaline cartilage in other locations, articular cartilage lacks perichondrium, a layer of fibrous tissue around it that serves as the source of new cartilage cells. Thus, although extracellular matrix undergoes continuous remodeling throughout life, the ability to repair …

medicine.anatomical_structureTissue engineeringHyaline cartilageChemistryRegeneration (biology)CartilagemedicinePerichondriumOsteoarthritisSynovial membraneChondrogenesismedicine.diseaseCell biology
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C1q-bearing immune complexes detected by a monoclonal antibody to human C1q in rheumatoid arthritis sera and synovial fluids

1991

Using a monoclonal antibody directed against the C-chain of human C1q, we detected C1q-bearing immune complexes (IC) in sera and synovial fluids of rheumatoid arthritis (RA) patients. In a sandwich-ELISA, C1q-bearing IC were captured by the solid-phase monoclonal antibody and then detected with peroxidase-labeled F(ab')2-antibodies to either human IgG or IgM. The results of this assay were compared to an ELISA-modification of the C1q-solid-phase binding assay (C1q-SPBA). C1q-bearing IC were detected in 81.1% of RA-sera and the 65.2% of RA-synovial fluids. IgG as well as IgM was present in 72.6% of the sera and 70% of the synovial fluids which were positive in both assays. Most RA sera that …

medicine.drug_classImmunologyEnzyme-Linked Immunosorbent Assaychemical and pharmacologic phenomenaAntigen-Antibody ComplexMonoclonal antibodyComplement Hemolytic Activity AssayArthritis RheumatoidImmunoglobulin Fab FragmentsClassical complement pathwayImmune systemRheumatologyimmune system diseasesOsteoarthritisSynovial FluidmedicineHumansImmunology and AllergySynovial fluidskin and connective tissue diseasesbiologybusiness.industryComplement C1qAntibodies Monoclonalmedicine.diseaseImmune complexImmunoglobulin MImmunoglobulin GRheumatoid arthritisMonoclonalImmunologybiology.proteinAntibodybusinessRheumatology International
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Blocking Jak/STAT signalling using tofacitinib inhibits angiogenesis in experimental arthritis

2021

Abstract Objective During rheumatoid arthritis (RA), the angiogenic processes, occurring with pannus-formation, may be a therapeutic target. JAK/STAT-pathway may play a role and the aim of this work was to investigate the inhibiting role of a JAK-inhibitor, tofacitinib, on the angiogenic mechanisms occurring during RA. Methods After ethical approval, JAK-1, JAK-3, STAT-1, STAT-3 and VEGF expression was evaluated on RA-synovial-tissues. In vitro, endothelial cells (ECs), stimulated with 20 ng/ml of VEGF and/or 1 μM of tofacitinib, were assessed for tube formation, migration and proliferation, by Matrigel, Boyden chamber assay and ki67 gene-expression. In vivo, 32 mice received collagen (coll…

medicine.medical_specialtyAngiogenesisArthritisDiseases of the musculoskeletal systemPharmacologyPyrroleMiceRheumatoid arthritis Angiogenesis TofacitinibPiperidinePiperidinesIn vivoInternal medicineMedicineAnimalsHumansPyrrolesRheumatoid arthritisRheumatoid arthritiTube formationMatrigelEndothelial CellTofacitinibbusiness.industryAnimalSynovial MembraneEndothelial Cellsmedicine.diseaseArthritis ExperimentalRheumatologyAngiogenesiPyrimidinesPyrimidineRC925-935TofacitinibRheumatoid arthritisAngiogenesisbusinessHumanResearch Article
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Mutational analysis of E-cadherin, β-catenin and APC genes in synovial sarcomas

2010

medicine.medical_specialtyMutationPathologyHistologyCadherinCell adhesion moleculeCancerAnatomical pathologyGeneral MedicineBiologymedicine.disease_causemedicine.diseaseSynovial sarcomaPathology and Forensic MedicineCateninmedicineCancer researchGeneHistopathology
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