Search results for "thyroiditi"

showing 10 items of 76 documents

Distinct subsets of intrathyroidal regulatory CD4+CD25+ and CD8+CD25+ T cells in Hashimoto's thyroiditis: Potential role in the neoplastic cell growt…

2005

Tregs T regulatory cells hashimoto's thyroiditisSettore MED/13 - Endocrinologia
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Type 1 Diabetes and Autoimmune Thyroid Disease—The Genetic Link

2021

Type 1 diabetes (T1D) and autoimmune thyroid disease (AITD) are the most frequent chronic autoimmune diseases worldwide. Several autoimmune endocrine and non-endocrine disorders tend to occur together. T1D and AITD often cluster in individuals and families, seen in the formation of autoimmune polyendocrinopathy (AP). The close relationship between these two diseases is largely explained by sharing a common genetic background. The HLA antigens DQ2 (DQA1*0501-DQB1*0201) and DQ8 (DQA1*0301-DQB1*0302), tightly linked with DR3 and DR4, are the major common genetic predisposition. Moreover, functional single nucleotide polymorphisms (or rare variants) of various genes, such as the cytotoxic T-lym…

autoimmune polyendocrinopathyendocrine system diseasestype 1 diabetesEndocrinology Diabetes and MetabolismSingle-nucleotide polymorphismGenome-wide association studyCLEC16AHuman leukocyte antigenReviewBiologyPolymorphism Single Nucleotidelcsh:Diseases of the endocrine glands. Clinical endocrinologyPTPN22single nucleotide polymorphismsEndocrinologyimmune system diseasesGenetic predispositionHumansGenetic Predisposition to Diseasesusceptibility genesHLA antigensgenetic linkGeneticslcsh:RC648-665Thyroiditis AutoimmuneFOXP3nutritional and metabolic diseasesAutoimmune polyendocrinopathyDiabetes Mellitus Type 1autoimmune thyroid diseaseFrontiers in Endocrinology
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Other Types of Chaperonopathies

2013

A mechanism causing a chaperonopathy that is introduced in this chapter consists of the absence of a chaperone from the place where it is needed (i.e., chaperonopathies by misplacement). Also in this chapter are discussed the unfolded-protein response (UPR), chaperone-mediated autophagy (CMA), and illustrative examples of chaperonopathies by mistake, or collaborationism. In these conditions, one or more chaperones, apparently normal in structure, perform functions that favor disease rather than the contrary, hence the name of chaperonopathy by mistake or collaborationism (a molecule that ought to protect the cell and the organism promotes pathogenesis instead). Many examples of chaperonopat…

biologybusiness.industryAutophagyMistakeDiseasemedicine.diseasemedicine.disease_causeBioinformaticsMyasthenia gravisThyroiditisAutoimmunityPathogenesisChaperone (protein)biology.proteinMedicinebusiness
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Selenium: A Cure for Autoimmune Thyroiditis.

2021

Autoimmune thyroiditis (AT) is a disease whose incidence has increased dramatically over the past few decades. In iodine sufficient areas, this pathological condition affects up to 5% of the general population. The higher incidence of AT observed in areas of the world with low-selenium (Se) soils apperas to be the consequence of a reduced activity of the intracellular Sedependent glutathione peroxidase activity. Se is involved, through selenoproteins, in the regulation of intracellular antioxidant, redox and anti-inflammatory processes. As a component of selenoproteins, Se exerts various structural and enzymatic functions. A low Se/high fat diet can cause selenoprotein changes and promote t…

business.industryEndocrinology Diabetes and MetabolismThyroiditis Autoimmunechemistry.chemical_elementHashimoto Diseasemedicine.diseaseIodide PeroxidaseAutoimmune thyroiditisSeleniumchemistryImmunologyImmunology and AllergyMedicineHumansSettore MED/49 - Scienze Tecniche Dietetiche ApplicatebusinessSelenium autoimmune thyroiditis redox balanceSeleniumEndocrine, metabolicimmune disorders drug targets
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Response to 'TH1 and TH2 cytokine control of thyrocyte survival in thyroid autoimmunity'

2001

Mirakian et al. question our recent results, which suggest that thyrocyte survival during thyroid autoimmunity depends on differential effects of TH1 and TH2 cytokines1. Thyrocyte destruction in autoimmune thyroiditis is a slow process that lasts several years. We hypothesized that in thyroid autoimmunity the balance between life and death in thyrocytes depends on the predominance over the time of TH2 and TH1 cytokines, whose action is not restricted to immune cells but involves direct modulation of key molecules responsible for survival or death of target cells1.

business.industrymedicine.medical_treatmentImmunologymedicine.diseaseDifferential effectsAutoimmune thyroiditisCytokineImmune systemImmunologyThyroid autoimmunitymedicineImmunology and AllergyTh1 cytokinesbusinessNature Immunology
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Neck liymph nodes in chronic autoimmune thyroiditis: the sonographic pattern.

2013

chronic autoimmune thyroiditisNeck liymph nodesSettore MED/13 - Endocrinologia
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2021

BACKGROUND Studies investigating a potential association between hypothyroidism and non-alcoholic fatty liver disease (NAFLD) showed conflicting results and large-scale population-based data from Germany on this topic are currently missing. OBJECTIVE It was the aim of this analysis to investigate the impact of thyroid gland disorders on the prevalence of NAFLD in Germany. METHODS In this case-control study, using the German disease Analyzer database (IQVIA), NAFLD patients were matched to patients without NAFLD by age, sex, index year, treating physician, diabetes mellitus type II, and obesity. The main outcome of the study was an association between thyroid gland disorders (hypothyroidism,…

education.field_of_studymedicine.medical_specialtyendocrine system diseasesbusiness.industryPopulationFatty liverThyroidGastroenterologynutritional and metabolic diseasesThyroid Gland Disordermedicine.diseaseLower riskGastroenterologydigestive system diseasesAutoimmune thyroiditisLiver diseasemedicine.anatomical_structureOncologyInternal medicinemedicineRisk factorbusinesseducationUnited European Gastroenterology Journal
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Autoimmune thyroid disease: new models of cell death in autoimmunity

2002

Autoimmunity to thyroid antigens leads to two distinct pathogenic processes with opposing clinical outcomes: hypothyroidism in Hashimoto's thyroiditis and hyperthyroidism in Graves' disease. The high frequency of these diseases and easy accessibility of the thyroid gland has allowed the identification of key pathogenic mechanisms in organ-specific autoimmune diseases. In early investigations, antibody- and T-cell-mediated death mechanisms were proposed as being responsible for autoimmune thyrocyte depletion. Later, studies on apoptosis have provided new insights into autoimmune target destruction, indicating the involvement of death receptors and cytokine-regulated apoptotic pathways in the…

endocrine systemHistoryProgrammed cell deathFas Ligand Proteinendocrine system diseasesImmunologyThyroid GlandApoptosisAutoimmunityDiseasemedicine.disease_causeThyroiditisEducationAutoimmunityPathogenesisAntigenSettore MED/04 - PATOLOGIA GENERALEHumansMedicinefas ReceptorMembrane Glycoproteinsbiologybusiness.industryThyroidThyroiditis Autoimmunemedicine.diseaseGraves DiseaseComputer Science Applicationsmedicine.anatomical_structureModels AnimalImmunologybiology.proteinAntibodybusinessT-Lymphocytes CytotoxicNature Reviews Immunology
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Thyroïdites : où en est-on en 2019 ?

2020

Thyroiditis is a frequent and mostly benign disease that can sometimes disrupt the thyroid balance. Their diagnosis, as well as their aetiology, is a necessary step in the management of the patients. Painful thyroiditis includes acute thyroiditis of infectious origin and subacute thyroiditis. The first one can be treated by antibiotics or antifungals depending on the germ found. The second one will be treated with non-steroidal anti-inflammatory drugs or corticosteroids. In cases of Hashimoto's thyroiditis with overt hypothyroidism, replacement therapy with L-thyroxine will be adapted to the TSH level. As amiodarone treatment provides dysthyroidism, the thyroid status should be monitored re…

endocrine systemPediatricsmedicine.medical_specialtyendocrine system diseasesmedicine.medical_treatment[SDV]Life Sciences [q-bio]030209 endocrinology & metabolismContext (language use)AmiodaroneThyroiditis03 medical and health sciences0302 clinical medicineInternal MedicinemedicineSubacute thyroiditisbusiness.industryThyroid diseaseThyroidGastroenterologyImmunotherapymedicine.disease3. Good health[SDV] Life Sciences [q-bio]medicine.anatomical_structure030220 oncology & carcinogenesisEtiologybusinesshormones hormone substitutes and hormone antagonistsmedicine.drug
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Bioassays for TSH Receptor Antibodies: Quo Vadis?

2015

Autoantibodies (Ab) to the TSH receptor (TSHR) are responsible for many of the clinical manifestations of Graves' disease (GD) and are specific biomarkers of this autoimmune thyroid disorder (AITD) [1,2,3]. These Ab can be measured either via competitive-binding immunoassays or with bioassays [4]. Antibody-binding assays only report the presence or absence of TSHR-Ab and their concentrations, but do not indicate their functional activity. Bioassays, in contrast, indicate whether TSHR-Ab have stimulatory or blocking activity [5]. Historically, bioassays for TSHR-Ab were research tools used to study the pathophysiology of GD. Recently, however, there are increasing data that demonstrate the c…

endocrine systemendocrine system diseasesbiologybusiness.industryEndocrinology Diabetes and MetabolismThyroidAutoantibodymedicine.diseaseeye diseasesThyroiditisThyroid disordermedicine.anatomical_structureEditorialImmunologymedicinebiology.proteinBioassayBiomarker (medicine)AntibodyChronic thyroiditisbusiness
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