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showing 10 items of 10618 documents

Common genes associated with antidepressant response in mouse and man identify key role of glucocorticoid receptor sensitivity.

2017

Response to antidepressant treatment in major depressive disorder (MDD) cannot be predicted currently, leading to uncertainty in medication selection, increasing costs, and prolonged suffering for many patients. Despite tremendous efforts in identifying response-associated genes in large genome-wide association studies, the results have been fairly modest, underlining the need to establish conceptually novel strategies. For the identification of transcriptome signatures that can distinguish between treatment responders and nonresponders, we herein submit a novel animal experimental approach focusing on extreme phenotypes. We utilized the large variance in response to antidepressant treatmen…

0301 basic medicineMicroarraysPhysiologyGene ExpressionBioinformaticsBiochemistryBiomarkers PharmacologicalTranscriptomeMice0302 clinical medicineGlucocorticoid receptorMedicine and Health SciencesBiology (General)DepressionGeneral NeuroscienceBrainDrugsAntidepressantsPhenotypeAntidepressive Agents3. Good healthBody FluidsParoxetineBioassays and Physiological AnalysisBloodMice Inbred DBAMultigene FamilyMajor depressive disorderAntidepressantDNA microarrayAnatomyGeneral Agricultural and Biological SciencesResearch ArticleQH301-705.5Antidepressant drug therapy ; Blood ; Gene regulation ; Biomarkers ; Depression ; Gene expression ; Microarrays ; AntidepressantsBiologyResearch and Analysis MethodsGeneral Biochemistry Genetics and Molecular BiologyBlood Plasma03 medical and health sciencesReceptors GlucocorticoidMental Health and PsychiatrymedicineGeneticsAnimalsHumansGene RegulationPharmacologyDepressive Disorder MajorGeneral Immunology and MicrobiologyMechanism (biology)Mood DisordersGene Expression ProfilingBiology and Life Sciencesmedicine.diseaseGene expression profiling030104 developmental biologyGene Expression RegulationCorticosterone030217 neurology & neurosurgeryBiomarkersPLoS biology
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Origin of modern syphilis and emergence of a pandemic Treponema pallidum cluster

2016

The abrupt onslaught of the syphilis pandemic that started in the late fifteenth century established this devastating infectious disease as one of the most feared in human history1 . Surprisingly, despite the availability of effective antibiotic treatment since the mid-twentieth century, this bacterial infection, which is caused by Treponema pallidum subsp. pallidum (TPA), has been re-emerging globally in the last few decades with an estimated 10.6 million cases in 2008 (ref. 2). Although resistance to penicillin has not yet been identified, an increasing number of strains fail to respond to the secondline antibiotic azithromycin3. Little is known about the genetic patterns in current infec…

0301 basic medicineMicrobiologia340 LawCiencias de la SaludAzithromycinGlobal HealthBacterisApplied Microbiology and Biotechnology2726 Microbiology (medical)1307 Cell BiologyGenotypePandemicPhylogenyMolecular EpidemiologyTreponemaPhylogenetic treebiology2404 Microbiology10177 Dermatology ClinicTREPONEMA PALLIDUM10218 Institute of Legal MedicineAnti-Bacterial Agents3. Good health590 Animals (Zoology)//purl.org/becyt/ford/3 [https]ORIGIN OF SYPHILISMalalties de transmissió sexualDNA BacterialMicrobiology (medical)CIENCIAS MÉDICAS Y DE LA SALUDGenotypeImmunology610 Medicine & healthMicrobiologyEvolution Molecular//purl.org/becyt/ford/3.3 [https]10127 Institute of Evolutionary Biology and Environmental Studies03 medical and health sciences1311 GeneticsPhylogeneticsDrug Resistance BacterialGeneticsmedicine2402 Applied Microbiology and BiotechnologyHumansSyphilisTreponema pallidumPandemics2403 ImmunologyMolecular epidemiologyGenetic VariationSequence Analysis DNACell Biologymedicine.diseasebiology.organism_classificationVirologyEnfermedades Infecciosas030104 developmental biologyInfectious disease (medical specialty)570 Life sciences; biologySyphilisGenome Bacterial
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Corrigendum: Phylogeny of Vibrio vulnificus From the Analysis of the Core-Genome: Implications for Intra-Species Taxonomy

2018

Vibrio vulnificus (Vv) is a multi-host pathogenic species currently subdivided into three biotypes (Bts). The three Bts are human-pathogens, but only Bt2 is also a fish-pathogen, an ability that is conferred by a transferable virulence-plasmid (pVvbt2). Here we present a phylogenomic analysis from the core genome of 80 Vv strains belonging to the three Bts recovered from a wide range of geographical and ecological sources. We have identified five well-supported phylogenetic groups or lineages (L). LI comprises a mixture of clinical and environmental Bt1 strains, most of them involved in human clinical cases related to raw seafood ingestion. LII is linked to the aquaculture industry and incl…

0301 basic medicineMicrobiology (medical)030106 microbiologylcsh:QR1-502VirulenceMicrobiologiaSNPVibrio vulnificusGenomeMicrobiologylcsh:Microbiologymicrobial evolution03 medical and health sciencesPhylogeneticsPhylogenomicspathovarVibrio vulnificusOriginal ResearchGeneticsPhylogenetic treebiologyCorrectionpathogensbiology.organism_classificationbiotypeVibriovirulence plasmid030104 developmental biologycore genomePathovarBacteris patògensFrontiers in Microbiology
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Metabolic complementation in bacterial communities: Necessary conditions and optimality

2016

Bacterial communities may display metabolic complementation, in which different members of the association partially contribute to the same biosynthetic pathway. In this way, the end product of the pathway is synthesized by the community as a whole. However, the emergence and the benefits of such complementation are poorly understood. Herein, we present a simple model to analyze the metabolic interactions among bacteria, including the host in the case of endosymbiotic bacteria. The model considers two cell populations, with both cell types encoding for the same linear biosynthetic pathway. We have found that, for metabolic complementation to emerge as an optimal strategy, both product inhib…

0301 basic medicineMicrobiology (medical)Cell typeSystems biology030106 microbiologyCelllcsh:QR1-502Computational biologyBiologyMicrobiologylcsh:Microbiology03 medical and health sciencesMetabolic complementationmetabolic modelingHypothesis and TheoryBotanymedicineCinara cedricross-feedingEndosymbiotic bacteriaHost (biology)biology.organism_classificationkinetic modelingComplementation030104 developmental biologymedicine.anatomical_structureProduct inhibitionendosymbiotic bacteriaMetabolic ModellingoptimizationBacteria
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First phenotypic and genotypic description of Fasciola hepatica infecting highland cattle in the state of Mexico, Mexico

2018

Abstract Fascioliasis is a plantborne and zoonotic parasitic disease caused by fasciolid liver flukes. Fasciola hepatica is the only fasciolid species described in the Americas. Human fascioliasis endemic areas are mainly located in high altitude areas of the Americas. Given the necessity to characterize F. hepatica populations involved, the phenotypic and genotypic features of fasciolid adults infecting cattle in the highland area of Toluca, State of Mexico, Mexico, were analyzed and compared to fasciolid materials from the Northern Bolivian Altiplano, representing the altiplanic transmission pattern in a hyperendemic scenario. A computer image analysis system (CIAS) was applied on the bas…

0301 basic medicineMicrobiology (medical)FascioliasisGenotypeRange (biology)030231 tropical medicinebiology.animal_breedPopulationCattle DiseasesZoologyDNA RibosomalMicrobiology03 medical and health sciences0302 clinical medicineIntergenic regionHepaticaparasitic diseasesGeneticsAnimalsFasciola hepaticaGeography MedicaleducationMexicoMolecular BiologyRibosomal DNAEcology Evolution Behavior and SystematicsBase Compositioneducation.field_of_studybiologyHighland CattleFasciola hepatica030108 mycology & parasitologyLiver flukebiology.organism_classificationPhenotypeInfectious DiseasesHaplotypesCattleSequence AnalysisInfection, Genetics and Evolution
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Subtle genotypic changes can be observed soon after diagnosis in Mycobacterium tuberculosis infection.

2016

Clonal variants of Mycobacterium tuberculosis (MTB) coexist in specific patients, although the dynamics of their emergence is unknown. We used MIRU-VNTR to detect microevolution leading to variants of MTB in 3 out of 19 patients (15%) soon after diagnosis (61-85 days). Most harbored SNPs and for some of them a potential functional role was suggested. Microevolution in tuberculosis seems to occur sooner and more often than expected and could affect tracking of transmission.

0301 basic medicineMicrobiology (medical)Functional roleAdultMaleTuberculosisGenotyping Techniques030106 microbiologyAdaptation BiologicalSingle-nucleotide polymorphismBiologyMicrobiologyPolymorphism Single NucleotideMycobacterium tuberculosisEvolution Molecular03 medical and health sciencesGenotypemedicineHumansTuberculosisAgedAged 80 and overTransmission (medicine)MicroevolutionGenetic VariationGeneral MedicineMycobacterium tuberculosisMiddle Agedbacterial infections and mycosesmedicine.diseasebiology.organism_classificationVirologyInfectious DiseasesFemaleInternational journal of medical microbiology : IJMM
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Genome-scale analysis of evolutionary rate and selection in a fast-expanding Spanish cluster of HIV-1 subtype F1.

2018

Abstract This work is aimed at assessing the presence of positive selection and/or shifts of the evolutionary rate in a fast-expanding HIV-1 subtype F1 transmission cluster affecting men who have sex with men in Spain. We applied Bayesian coalescent phylogenetics and selection analyses to 23 full-coding region sequences from patients belonging to that cluster, along with other 19 F1 epidemiologically-unrelated sequences. A shift in the overall evolutionary rate of the virus, explained by positively selected sites in the cluster, was detected. We also found one substitution in Nef (H89F) that was specific to the cluster and experienced positive selection. These results suggest that fast tran…

0301 basic medicineMicrobiology (medical)GenotypeBayesian probabilityGenome scaleEpitopes T-LymphocyteHIV InfectionsGenome ViralBiologyDisease clusterMicrobiologyArticlelaw.inventionMen who have sex with menCoalescent theoryEvolution MolecularSubtype F103 medical and health sciencesSex FactorslawPhylogeneticsDatabases GeneticGeneticsHumansSelection GeneticSelectionMolecular BiologyAntigens ViralEcology Evolution Behavior and SystematicsSelection (genetic algorithm)PhylogenyRecombination GeneticGenomicsMen who have sex with men030104 developmental biologyInfectious DiseasesTransmission (mechanics)Evolutionary biologySpainHIV-1Transmission clusterInfection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases
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Gliding Motility and Expression of Motility-Related Genes in Spreading and Non-spreading Colonies of Flavobacterium columnare

2018

Gliding motility facilitates the movement of bacteria along surfaces in many Bacteroidetes species and results in spreading colonies. The adhesins required for the gliding are secreted through a gliding motility-associated protein secretion system, known as the type IX secretion system (T9SS). The fish pathogen Flavobacterium columnare produces spreading (rhizoid [Rz], soft [S]) and non-spreading (rough [R]) colony types, of which only the spreading Rz type is virulent. In this study, we explored the spreading behavior of these colony types by microscopic imaging and measured the expression of genes associated with gliding motility and T9SS (gldG, gldH, gldL, sprA, sprB, sprE, sprF, sprT, a…

0301 basic medicineMicrobiology (medical)Gliding motility030106 microbiologylcsh:QR1-502MotilityVirulenceFlavobacteriumMicrobiologylcsh:MicrobiologybakteeritMicrobiology03 medical and health sciencesFlavobacterium columnarenutrientscolony typeGene expressionSecretiongeeniekspressiobacteriabiologyta1183RT-qPCRta1182liikebiology.organism_classificationBacterial adhesinFlavobacterium columnarecolony spreadingT9SSgene expressiongliding motilityleviäminenBacteriatype IX secretion systemFrontiers in Microbiology
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Molecular epidemiology and drug-resistance mechanisms in carbapenem-resistant Klebsiella pneumoniae isolated in patients from a tertiary hospital in …

2020

Abstract Objectives The aim of this study has been to characterize carbapenem-resistant Klebsiella pneumoniae isolates and to determine the resistance mechanisms involved, the clonal relationship between strains and clinical and demographical data of the infected patients. Methods Clinical and demographical data from patients were collected and statistically analysed. Antimicrobial susceptibility testing was performed and resistance genes were detected both phenotypically and genotypically. Conjugation assays were performed to show horizontal transferability of resistance genes. Clonal relationship was also studied. Next-generation sequencing (NGS) was performed to obtain information regard…

0301 basic medicineMicrobiology (medical)Klebsiella pneumoniaeTetracycline030106 microbiologyImmunologyVirulenceDrug resistanceResistance mechanismsMicrobiologyMicrobiologyTertiary Care Centers03 medical and health sciences0302 clinical medicinePlasmidGenotypemedicineImmunology and AllergyHumans030212 general & internal medicineMolecular EpidemiologyMolecular epidemiologybiologybiology.organism_classificationCarbapenemasesQR1-502Klebsiella InfectionsKlebsiella pneumoniaeCarbapenemsPharmaceutical PreparationsSpainMultilocus sequence typingmedicine.drugJournal of Global Antimicrobial Resistance
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Highly heterogeneous mutation rates in the hepatitis C virus genome.

2016

Spontaneous mutations are the ultimate source of genetic variation and have a prominent role in evolution. RNA viruses such as hepatitis C virus (HCV) have extremely high mutation rates, but these rates have been inferred from a minute fraction of genome sites, limiting our view of how RNA viruses create diversity. Here, by applying high-fidelity ultradeep sequencing to a modified replicon system, we scored >15,000 spontaneous mutations, encompassing more than 90% of the HCV genome. This revealed >1,000-fold differences in mutability across genome sites, with extreme variations even between adjacent nucleotides. We identify base composition, the presence of high- and low-mutation clusters a…

0301 basic medicineMicrobiology (medical)Mutation rateGenotypeHepatitis C virusImmunologyGenome ViralHepacivirusBiologymedicine.disease_causeVirus ReplicationApplied Microbiology and BiotechnologyMicrobiologyGenome03 medical and health sciencesMutation RateMolecular evolutionGenetic variationGeneticsmedicineHumansTransversionGenetics030102 biochemistry & molecular biologyNucleotidesGenetic VariationHigh-Throughput Nucleotide SequencingCell BiologyResistance mutationHepatitis C030104 developmental biologyViral replicationRNA ViralRepliconNature microbiology
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