Search results for "virulence"

showing 10 items of 457 documents

Sulfhydryl G Proteins and Phospholipase A2-Associated G Proteins Are Involved in Adrenergic Signal Transduction in the Rat Pineal Gland

2001

The rat pineal gland with its circadian noradrenaline-regulated melatonin rhythm is an excellent model for studying adrenergic signal transduction with respect to cAMP and cGMP formation. The stimulatory G(s) proteins play a well-established role in this process. In contrast, the potential roles of the inhibitory G(i) proteins, the functionally unclear other G(o) proteins, and a number of G protein subtypes are not known. The present study examines the effects on beta(1)- and beta(1)-plus-alpha(1)-stimulated cAMP and cGMP formation of a number of G protein modulators in rat pinealocyte suspension cultures. The effects of the nitric oxide donor sodium nitroprusside on cGMP were also examined…

MaleNitroprussideArylamine N-AcetyltransferaseG proteinAdrenergicWasp VenomsPhospholipaseBiologyNitric OxidePertussis toxinBenzylisoquinolinesPineal GlandPhospholipases APinealocyteRats Sprague-DawleyPhenylephrineAlkaloidsEndocrinologyPhospholipase A2GTP-Binding ProteinsCyclic AMPAnimalsp-Methoxy-N-methylphenethylamineVirulence Factors BordetellaCyclic GMPSulfhydryl ReagentsIsoproterenolAdrenergic beta-AgonistsRatsReceptors AdrenergicPhospholipases A2Pertussis ToxinBiochemistryEthylmaleimideMastoparanbiology.proteinIntercellular Signaling Peptides and ProteinsAnimal Science and ZoologySignal transductionPeptidesAdrenergic alpha-AgonistsSignal TransductionGeneral and Comparative Endocrinology
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Correlation of virus replication, cytokine (TNF-? and IL-1) producing cells, neuronal necrosis and inflammation after intranasal infection of mice wi…

1995

The number of TNF-alpha and IL-1 beta producing cells was investigated during the acute replication phase of herpes simplex virus (HSV) in trigeminal ganglia after intranasal infection with strains of different virulence. The highly virulent strain WAL replicated strongly and induced many cytokine producing cells early in the ganglia. The low virulent strain HFEM replicated less, only few cytokine producing cells were detected late. The thymidine-kinase negative (TK-) virus 1301 did not replicate but produced some lymphocytic inflammation. The higher the virulence of strains of HSV-1 or -2 was, the stronger was the extent of histopathological lesions; moreover, a dissociation in time betwee…

MaleTime Factorsmedicine.medical_treatmentVirulenceInflammationBiologyVirus Replicationmedicine.disease_causeHerpesviridaeVirusMiceNecrosisT-Lymphocyte SubsetsVirologymedicineAnimalsSimplexvirusAdministration IntranasalNeuronsMice Inbred BALB CTumor Necrosis Factor-alphaHerpes SimplexGeneral MedicineVirologyCytokineHerpes simplex virusTrigeminal GanglionViral replicationmedicine.symptomCD8Interleukin-1Archives of Virology
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Virulence genotype and nematode-killing properties of extra-intestinal Escherichia coli producing CTX-M beta-lactamases.

2006

8 pages; International audience; This study evaluated the virulence potential of Escherichia coli isolates producing CTX-M beta-lactamases. During a 24-month period, 33 extended-spectrum beta-lactamase (ESBL)-producing E. coli, including 14 CTX-M-producers, were isolated from urinary tract infections at N?s University Hospital, France. The prevalence of 14 major virulence factors (VFs) was investigated by PCR and compared with the prevalence in a group of 99 susceptible E. coli isolates. Ten VFs were less prevalent (p <0.05) in the ESBL isolates than the susceptible E. coli, while iutA and traT were more prevalent in ESBL isolates (p <0.05). Moreover, the CTX-M-producing isolates had signif…

MaleTime Factorsvirulence factorsUrine[ SDV.MP.BAC ] Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriologymedicine.disease_causePolymerase Chain Reactionlaw.inventionlaw[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesGenotypepathogenicityCTX-MPolymerase chain reactionEscherichia coli InfectionsPhylogeny0303 health sciencesbiologyVirulenceGeneral MedicineMiddle AgedEnterobacteriaceae3. Good health[ SDV.MHEP.MI ] Life Sciences [q-bio]/Human health and pathology/Infectious diseasesInfectious DiseasesFemaleMicrobiology (medical)[ SDV.MP.PAR ] Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyGenotypeVirulencebeta-LactamasesMicrobiology03 medical and health sciencesImmunocompromised HostDrug Resistance BacterialmedicineEscherichia coliAnimalsHumans[SDV.MP.PAR]Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyTypingCaenorhabditis elegansEscherichia coli030304 developmental biologyAged030306 microbiologybiochemical phenomena metabolism and nutritionbiology.organism_classificationVirologySurvival Analysis[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyNematodeurinary tract infectionBacteria
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Parasite virulence when the infection reduces the host immune response.

2010

Parasite infections often induce a reduction in host immune response either because of a direct manipulation of the immune system by the parasite or because of energy depletion. Although infection-induced immunodepression can favour the establishment of the parasite within the host, a too severe immunodepression may increase the risk of infection with opportunistic pathogens, stopping the period over which the parasite can be transmitted to other hosts. Here, we explore how the risk of contracting opportunistic diseases affects the survival of the amphipod Gammarus pulex infected by the acanthocephalan Pomphorhynchus laevis . Previous work with this system has shown that upon infection, G.…

Male[ SDV.MP.PAR ] Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyVirulence[ SDV.IMM.IA ] Life Sciences [q-bio]/Immunology/Adaptive immunologyGeneral Biochemistry Genetics and Molecular BiologyAcanthocephalaHost-Parasite InteractionsPomphorhynchus laevisImmune systemopportunistic pathogensResearch articlesdisease ecology[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisParasite hostingAnimalsAmphipodaGeneral Environmental Science[ SDE.BE ] Environmental Sciences/Biodiversity and EcologyGeneral Immunology and MicrobiologybiologyHost (biology)Risk of infectionGeneral Medicinebiology.organism_classificationvirulenceGammarus pulexPulexImmunologyPomphorhynchus laevisGeneral Agricultural and Biological SciencesWater Microbiologyimmunodepression
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Occurrence of Edwardsiella tarda in wild European eels Anguilla anguilla from Mediterranean Spain

2007

Pure cultures of Edwarsiella tarda were isolated from body ulcers and internal organs of wild European eels caught in a Mediterranean freshwater coastal lagoon (Albufera Lake, Valencia, Spain) over a 1 yr period. Overall, the E. tarda isolation rate from wild eels was 9%, but this increased to 22.8% in diseased individuals. All 22 E. tarda isolates belonged to the 'wild-type' biogroup of the species and were virulent for eels (lethal dose that kills 50% of exposed individuals [LD50 dose]: 10(4.85) to 10(6.83) CFU ind.(-1)), and therefore represented the aetiological agent of the haemorrhagic disease observed in wild European eels. The E. tarda isolates and E. tarda CECT 894T type strain wer…

Mediterranean climatecongenital hereditary and neonatal diseases and abnormalitiesendocrine systemanimal structuresPopulationZoologyVirulenceMicrobial Sensitivity TestsAquatic ScienceIsolation rateFish DiseasesMediterranean seaAnti-Infective AgentsAnguillidaeAgglutination TestsMediterranean SeaPrevalenceAnimalsskin and connective tissue diseaseseducationEdwardsiella tardaEcology Evolution Behavior and SystematicsAntigens Bacterialeducation.field_of_studyVirulencebiologyEcologyEdwardsiella tardaLethal doseEnterobacteriaceae InfectionsAnguillabiology.organism_classificationPhenotypeSpainDiseases of Aquatic Organisms
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Novel strategies in the war against antibiotic resistance

2021

The global threat of antibiotic resistance is steadily growing. Antibiotic resistancemay involve any class of antibiotic, including second- and third-line agents that have been considered to date the last-resort drugs to counteract common infections. We may lose our capability to keep under control many common bacterial infections [1]. Despite this, in the past decade significant research efforts have been made to develop new antibacterial strategies able to treat multidrug-resistant infections; however, no new therapeutic approach has yet reached the clinic [2,3]. In order to identify new valuable antimicrobial drugs, it is important to consider the main bacterial resistance mechanisms in …

Methicillin-Resistant Staphylococcus aureusPharmacologyVirulencebiologyChemistryEscherichia coli ProteinsProtein Disulfide-IsomerasesBiofilmDrug Resistance MicrobialAnti-Bacterial AgentsMicrobiologyDsbAAntibiotic resistanceBiofilmsDrug DiscoveryEscherichia colibiology.proteinMolecular Medicineantibiotic resistance antivirulence strategy biofilm DsbA MRSA
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Therapeutic Strategies To Counteract Antibiotic Resistance in MRSA Biofilm‐Associated Infections

2021

Methicillin-resistant Staphylococcus aureus (MRSA) has emerged as one of the leading causes of persistent human infections. This pathogen is widespread and is able to colonize asymptomatically about a third of the population, causing moderate to severe infections. It is currently considered the most common cause of nosocomial infections and one of the main causes of death in hospitalized patients. Due to its high morbidity and mortality rate and its ability to resist most antibiotics on the market, it has been termed a “superbug”. Its ability to form biofilms on biotic and abiotic surfaces seems to be the primarily means of MRSA antibiotic resistance and pervasiveness. Importantly, more tha…

Methicillin-Resistant Staphylococcus aureusmedicine.medical_specialtymedicine.drug_classMRSA biofilm antibiotic-resistance antivirulence strategy eradicating agentsAntibioticsPopulationbeta-Lactamsmedicine.disease_cause01 natural sciencesBiochemistryHigh morbidityAntibiotic resistanceDrug Resistance BacterialDrug DiscoveryHumansMedicineGeneral Pharmacology Toxicology and PharmaceuticsIntensive care medicineeducationProtein Kinase InhibitorsPathogenOxazolidinonesPharmacologyeducation.field_of_study010405 organic chemistrybusiness.industryMortality rateOrganic ChemistryBiofilmStaphylococcal Infectionsbiochemical phenomena metabolism and nutritionAnti-Bacterial Agents0104 chemical sciences010404 medicinal & biomolecular chemistryStaphylococcus aureusBiofilmsPhenazinesMolecular MedicinebusinessChemMedChem
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Increase in gut microbiota after immune suppression in baculovirus-infected larvae.

2013

Spodoptera exigua microarray was used to determine genes differentially expressed in S. exigua cells challenged with the species-specific baculovirus SeMNPV as well as with a generalist baculovirus, AcMNPV. Microarray results revealed that, in contrast to the host transcriptional shut-off that is expected during baculovirus infection, S. exigua cells showed a balanced number of up- and down-regulated genes during the first 36 hours following the infection. Many immune-related genes, including pattern recognition proteins, genes involved in signalling and immune pathways as well as immune effectors and genes coding for proteins involved in the melanization cascade were found to be down-regul…

MicroarraysApplied MicrobiologyvirusesGut floraTranscriptomesBiology (General)Immune ResponseEffectorViral Immune EvasionMicrobiotaAgricultureGenomicsFunctional GenomicsHost-Pathogen InteractionIntestinesLarvaResearch ArticleQH301-705.5Mechanisms of Resistance and SusceptibilityImmunologyVirulenceBiologySpodopteraSpodopteraImmune SuppressionMicrobiologydigestive systemVirusMicrobiologyMolecular GeneticsImmune systemIntegrated ControlGenome Analysis ToolsVirologyMicrobial ControlExiguaGeneticsImmune ToleranceAnimalsGene RegulationMolecular BiologyGeneBiologyImmunity to InfectionsMicrobial PathogensImmunityComputational BiologyImmune DefenseRC581-607biology.organism_classificationNucleopolyhedrovirusesParasitologyPest ControlImmunologic diseases. AllergyGenome Expression AnalysisPLoS Pathogens
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What Do We Know about Candida auris? State of the Art, Knowledge Gaps, and Future Directions

2021

Candida auris has unprecedently emerged as a multidrug resistant fungal pathogen, considered a serious global threat due to its potential to cause nosocomial outbreaks and deep-seated infections with staggering transmissibility and mortality, that has put health authorities and institutions worldwide in check for more than a decade now. Due to its unique features not observed in other yeasts, it has been categorised as an urgent threat by the Centers for Disease Control and Prevention and other international agencies. Moreover, epidemiological alerts have been released in view of the increase of healthcare-associated C. auris outbreaks in the context of the COVID-19 pandemic. This review su…

Microbiology (medical)<i>Candida auris</i>Coronavirus disease 2019 (COVID-19)diagnosisQH301-705.5pathogenesisOutbreakContext (language use)Fungal pathogenMicrobiologyDisease controlvirulenceGeographyCandida aurisVirologyDevelopment economicsPandemiccandidaemiaepidemiologyBiology (General)Outbreak controlMicroorganisms
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In Situ Characterization of Hfq Bacterial Amyloid: A Fourier-Transform Infrared Spectroscopy Study

2019

Hfq is a bacterial protein that regulates gene expression at the post-transcriptional level in Gram-negative bacteria. We have previously shown that Escherichia coli Hfq protein, and more precisely its C-terminal region (CTR), self-assembles into an amyloid-like structure in vitro. In the present work, we present evidence that Hfq unambiguously forms amyloid structures also in vivo. Taking into account the role of this protein in bacterial adaptation and virulence, our work opens possibilities to target Hfq amyloid self-assembly and cell location, with potential to block bacterial adaptation and treat infections. Fil: Partouche, David. Centre National de la Recherche Scientifique; Francia. …

Microbiology (medical)AmyloidOtras Ingenierías y Tecnologíasprotein fibrillation inhibitionVirulencelcsh:MedicineINGENIERÍAS Y TECNOLOGÍASmedicine.disease_causeArticleHFQHfq03 medical and health sciencesIn vivoGene expressionmedicineImmunology and Allergybacterial amyloidSerum amyloid Aprotein fibrilsMolecular BiologyEscherichia coli030304 developmental biology//purl.org/becyt/ford/2.11 [https]Hfq protein0303 health sciencesGeneral Immunology and Microbiologybiology030306 microbiologyChemistrylcsh:Rfunctional amyloidbiology.organism_classificationCell biologyInfectious Diseases//purl.org/becyt/ford/2 [https]FTIRbiology.proteinBacteriaPathogens
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