Search results for "wistar"

showing 10 items of 1094 documents

Evidence of a flip-flop phenomenon in acamprosate pharmacokinetics: an in vivo study in rats.

2006

The pharmacokinetics of acamprosate were examined in the rat after oral and intravenous administration in order to detect the possible presence of a flip-flop phenomenon. Rats received 9.3 or 73.3 mg/kg of the drug as an intravenous bolus. The same doses were orally administered via gastric intubation. Plasma samples were taken from the jugular vein for determination of acamprosate concentration by liquid scintillation counting. The drug content was also quantified in urine and faeces. The acamprosate bioavailability was close to 20%, the amount recovered in the faeces being around 80% of the administered dose. The terminal slope of the oral plasma curve was significantly lower than that ob…

PharmacologyMaleDose-Response Relationship DrugChemistryTaurineAcamprosateDrug Administration RoutesPharmaceutical ScienceGeneral MedicineAbsorption (skin)UrinePharmacologyBioavailabilityRatsDose–response relationshipAcamprosatePharmacokineticsIn vivoOral administrationmedicineAnimalsPharmacology (medical)Rats Wistarmedicine.drugAlcohol DeterrentsBiopharmaceuticsdrug disposition
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Pharmacological studies of 1-(p-chlorophenyl)propanol and 2-(1-hydroxy-3-butenyl)phenol: Two new non-narcotic analgesics designed by molecular connec…

1997

Abstract Molecular topology has been applied to the design of new analgesic drugs. Linear discriminant analysis and connectivity functions were used to design two potentially suitable drugs which were synthesized and tested for analgesic properties by the acetic acid-induced abdominal constriction test in mice and the tail-flick test in rats. In mice, the compound 1-(p-chlorophenyl)propanol showed higher analgesic activity, both intraperitoneally and orally, than acetylsalicylic acid. 2-(1-Hydroxy-3-butenyl)phenol exhibited a lesser protective effect (70% of that shown by acetylsalicylic acid). In rats, acetylsalicylic acid gave the greatest protection against pain when administered intrape…

PharmacologyStereochemistryPropanolsButanolsAnalgesicPharmaceutical ScienceBiological activity1-PropanolPharmacologyAnalgesics Non-NarcoticRatsNon narcoticPropanolchemistry.chemical_compoundMiceNociceptionchemistryPhenolsIn vivoOral administrationDrug DesignPhenolAnimalsFemaleRats Wistar
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Prevention of the acute neurotoxic effects of phenytoin on rat peripheral nerve by H7, an inhibitor of protein kinase C.

1992

Abstract The neurotoxic effects of a single dose of phenytoin (150 mg/kg body weight) alone or 30 min after H7 (a protein kinase C inhibitor) injection (20 mg/kg body weight) were investigated in terms of peripheral neuromuscular function and Na + ,K + -ATPase activity of the sciatic nerve. This intraperitoneal injection of phenytoin induced complete blockade of muscle action potentials in the dorsal segmental muscles of the rat tail evoked by electric stimulation of the caudal nerve and a 40% decrease in the Na + ,K + -ATPase activity of the rat sciatic nerve when compared with control values, measured as the difference between total and ouabain-insensitive ATPase activity. Prior administr…

PhenytoinMalemedicine.medical_treatmentIntraperitoneal injectionPharmacologyToxicologyNeuromuscular junctionPiperazines1-(5-Isoquinolinesulfonyl)-2-MethylpiperazinemedicineAnimalsPeripheral NervesNa+/K+-ATPaseRats WistarProtein kinase CProtein Kinase CbiologyChemistryIsoquinolinesSciatic NerveElectric StimulationRatsElectrophysiologymedicine.anatomical_structureAnticonvulsantEnzyme inhibitorAnesthesiaPhenytoinbiology.proteinSciatic nerveSodium-Potassium-Exchanging ATPaseInjections Intraperitonealmedicine.drugToxicology
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Modulation of neuronal phospholipase D activity under depolarizing conditions

1999

Neuronal phospholipase D (PLD) activity was hypothesized to be involved in vesicle trafficking and endocytosis and, possibly, transmitter release. We here report that prolonged depolarization of rat hippocampal slices by potassium chloride (KCl) or 4-aminopyridine inhibited PLD activity. Similarly, PLD activity in rat cortical synaptosomes was significantly inhibited by depolarizing agents including veratridine and ouabain. Inhibition of calcium/calmodulin kinase II (CaMKII) which positively modulates synaptosomal PLD activity [Sarri et al. (1998) FEBS Lett. 440, 287-290] by KN-62 caused a further reduction of PLD activity in depolarized synaptosomes. Depolarization-induced inhibition of PL…

Phosphatidylinositol 45-DiphosphateTime FactorsBiophysicschemistry.chemical_elementCalciumHippocampusBiochemistryOuabainMembrane PotentialsPotassium Chloridechemistry.chemical_compoundStructural BiologyCa2+/calmodulin-dependent protein kinaseSynaptosomeElectrochemistryPhospholipase DGeneticsmedicineAnimalsPhospholipase D activityEnzyme InhibitorsRats WistarMolecular BiologyProtein Kinase CProtein Synthesis InhibitorsSynaptosomePhospholipase DCalcium/calmodulin-dependent protein kinase IINeomycinDepolarizationPhosphatidylinositol-45-bisphosphateCell BiologyRatsCell biologyenzymes and coenzymes (carbohydrates)chemistryCalcium-Calmodulin-Dependent Protein KinasesDepolarizationlipids (amino acids peptides and proteins)VeratridineSynaptosomesmedicine.drugFEBS Letters
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Control of oxidative metabolism in volume-overloaded rat hearts: effects of different lipid substrates.

1994

The relationship between intracellular energy parameters and myocardial O2 consumption (VO2) was studied in control and volume-overloaded hearts perfused with different lipid substrates and over a range of left ventricular work loads. In control hearts, a unique linear relationship between log of cytosolic [ATP]/[ADPf].[Pi] (where [ADPf] is concentration of free ADP) and myocardial VO2 was observed between low and high work loads for both fatty acids studied. In volume-overloaded hearts perfused in the presence of exogenous palmitate, the slope of the relationship between log [ATP]/[ADPf].[Pi] and myocardial VO2 was considerably depressed. It would seem that, under these conditions, much o…

PhosphocreatinePhysiologyRespiratory chainPalmitic AcidCardiomegalyPalmitic AcidsIn Vitro TechniquesVentricular Function LeftPhosphatesAdenosine TriphosphateOxygen ConsumptionPhysiology (medical)RespirationPiAnimalsRespiratory systemRats WistarATP synthasebiologyMyocardiumSubstrate (chemistry)HeartCreatineNADRatsAdenosine DiphosphateCytosolBiochemistrybiology.proteinCaprylatesCardiology and Cardiovascular MedicineEnergy MetabolismIntracellularThe American journal of physiology
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Stimulus-induced gamma activity in the electrocorticogram of freely moving rats: the neuronal signature of novelty detection.

2009

To investigate the cortical activity pattern associated with the exploration and identification of a novel object we recorded the intracranial electrocorticogram (ECoG) in the barrel cortex of freely moving adult rats using wireless technology. We report here that the exploration and detection of a novel object correlate with a transient increase of synchronized oscillatory activity in the 40–47 Hz frequency band. This specific cortical activity pattern occurs 200–300 ms after the first sensory contact with the novel stimulus and decreases in power in the subsequent recording sessions with the same object. During the first explorative session the increase in 40–47 Hz is associated with a si…

PhysicsMaleNeuronsmedicine.diagnostic_testCentral nervous systemSensory systemElectroencephalographySomatosensory CortexBarrel cortexStimulus (physiology)Somatosensory systemNovelty detectionRatsBehavioral NeuroscienceElectrophysiologymedicine.anatomical_structuremedicineExploratory BehaviorAnimalsTelemetryRats WistarElectrocorticographyNeuroscienceBehavioural brain research
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Comparative enhancer effects of Span20 with Tween20 and Azone on the in vitro percutaneous penetration of compounds with different lipophilicities.

2000

Sorbitan monolaurate (Span20) was used in this study to analyze the influence of the polar functional group on the effects that non-ionic surfactants have on skin permeability. Its ethoxylate derivative polysorbate 20 (Tween20) and Azone, both with the same C12 alkyl chain as Span20, were used for comparative purposes. We evaluated the relative potency of the three molecules as enhancers in the permeability of a series of compounds with lipophilicities ranging from log Poct=-0.95 to log Poct=2.33. The influence of the enhancer concentration was also studied. For this purpose the epidermis of Wistar rat was pretreated with ethanolic solutions (1 and 5%, w/v) of each enhancer. Our results ind…

PolysorbateChromatographySkin AbsorptionPharmaceutical ScienceSorbitan monolaurateExcipientPolysorbatesAzepinesAdministration CutaneousDosage formRatschemistry.chemical_compoundSurface-Active AgentschemistryLipophilicitymedicineAnimalsPolysorbate 20Rats WistarEnhancerAzonemedicine.drugHexosesInternational journal of pharmaceutics
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Activation of alpha-1A adrenoceptors mobilizes calcium from the intracellular stores in myocytes from rat portal vein.

1994

International audience; Intracellular free Ca++ concentration ([Ca++]i) was monitored using the fluorescence from the dye fura-2-acetoxymethylester in single myocytes from rat portal vein. In the presence of oxodipine (a L-type Ca++ channel inhibitor), norepinephrine (10 microM) evoked transient increases in [Ca++]i which were related to release of Ca++ from intracellular stores. The alpha-1 adrenoceptors mediating intracellular Ca++ release and inositol phosphate accumulation were identified by using subtype-selective agonists and antagonists. Pretreatment with chloroethylclonidine had little effect on the norepinephrine-induced increase in [Ca++]i and inositol phosphate accumulation. In c…

Portal VeinInositol Phosphates[SDV]Life Sciences [q-bio]Molecular Sequence Data[SDV.BC]Life Sciences [q-bio]/Cellular Biology[SDV.BC.BC]Life Sciences [q-bio]/Cellular Biology/Subcellular Processes [q-bio.SC]In Vitro TechniquesAntibodiesMuscle Smooth VascularRats[SDV] Life Sciences [q-bio]NorepinephrineChloride ChannelsReceptors Adrenergic alpha-1[SDV.BC.BC] Life Sciences [q-bio]/Cellular Biology/Subcellular Processes [q-bio.SC]AnimalsCalciumAmino Acid SequenceRats Wistar[SDV.BC] Life Sciences [q-bio]/Cellular BiologyAdrenergic alpha-AntagonistsCells CulturedSignal Transduction
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Cytoprotective effect of NMDA receptor antagonists on prion protein (PrionSc)-induced toxicity in rat cortical cell cultures

1993

Rat cortical cells were incubated with the Scrapie prion protein, PrionSc. At concentrations of 3 ng/ml of PrionSc and higher, the viability of the cells decreased significantly after a 12-h incubation period. Simultaneously, the degree of DNA fragmentation increased. In control experiments with antibodies against PrionSc, PrionSc lost its deleterious effect on neurons. PrionSc did not affect the viability of astrocytes. Drugs known to block NMDA receptor channels, such as memantine (1-amino-3,5-dimethyl-adamantane) (Mem), its analogue 1-N-methylamino-3,5-dimethyl-adamantane as well as (+)-5-methyl-10,11-dihydro-5H-dibenzo[a,d]cyclohepten-5,10-imine maleate (MK-801) prevented the effect of …

PrPSc ProteinsCell SurvivalPrionsNerve Tissue ProteinsScrapiePharmacologyReceptors N-Methyl-D-AspartateIncubation periodNeuroblastomaTumor Cells CulturedmedicineAnimalsRats WistarCells CulturedCerebral CortexNeuronsPharmacologybiologyMemantineCalcium Channel BlockersIn vitroRatsAstrocytesLiposomesToxicityImmunologybiology.proteinDNA fragmentationNMDA receptorAntibodymedicine.drugEuropean Journal of Pharmacology: Molecular Pharmacology
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Effect of flupirtine on Bcl-2 and glutathione level in neuronal cells treated in vitro with the prion protein fragment (PrP106-126).

1997

Flupirtine, trade name Katadolon, is a centrally acting nonopioid analgesic that has recently been found to display cytoprotective activity in vitro and in vivo on neurons induced to undergo apoptosis. This report shows that the PrP106-126 fragment of the prion protein, which is the likely etiological agent for a series of encephalopathies, is toxic to cortical neurons in vitro. Simultaneously, PrP106-126 influences the molecular GSH content and the bcl-2 expression in neurons. Significant toxicity (32% reduction in cell viability) was observed at a concentration of 50 microM of the peptide after 9 days of incubation, while at higher concentrations toxicity increased to 70%. Neurotoxicity w…

PrionsMolecular Sequence DataAminopyridinesApoptosisPharmacologyBiologychemistry.chemical_compoundDevelopmental NeurosciencemedicineAnimalsAmino Acid SequenceRats WistarCytotoxicityCells CulturedNeuronsNeurotoxicityGlutathioneAnalgesics Non-Narcoticmedicine.diseaseGlutathioneIn vitroPeptide FragmentsGenes bcl-2RatsOxidative StressNeuroprotective AgentsNeurologychemistryGene Expression RegulationProto-Oncogene Proteins c-bcl-2ApoptosisCell cultureImmunologyToxicityFlupirtineOxidation-Reductionmedicine.drugExperimental neurology
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