Search results for "xanthine"

showing 10 items of 139 documents

Detection of Hypoxanthine from Inosine and Unusual Hydrolysis of Immunosuppressive Drug Azathioprine through the Formation of a Diruthenium(III) Syst…

2021

Hypoxanthine (hpx) is an important molecule for both biochemistry research and biomedical applications. It is involved in several biological processes associated to energy and purine metabolism and has been proposed as a biomarker for a variety of disease states. Consequently, the discovery and development of systems suitable for the detection of hypoxanthine is pretty appealing in this research field. Thus, we have obtained a stable diruthenium (III) compound in its dehydrated and hydrated forms with formula [{Ru(&micro

Models MolecularPurinelcsh:BiotechnologyClinical BiochemistryMolecular Conformationchemistry.chemical_elementinosine02 engineering and technology010402 general chemistry01 natural sciencesArticleHydrolysischemistry.chemical_compoundX-Ray DiffractionLimit of Detectionlcsh:TP248.13-248.65medicineMolecule6-mercaptopurinerutheniumInosinePurine metabolismHypoxanthineazathioprineHydrolysisGeneral Medicine021001 nanoscience & nanotechnologyCombinatorial chemistry0104 chemical sciencesRutheniumchemistryPurineshypoxanthineMicroscopy Electron ScanningbiomarkerCyclic voltammetry0210 nano-technologyImmunosuppressive Agentsmedicine.drugBiosensors
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Preparation of two sets of 5,6,7-trioxygenated dihydroflavonol derivatives as free radical scavengers and neuronal cell protectors to oxidative damag…

2009

An unusual class of 5,6,7-trioxygenated dihydroflavonols (3a-e and 4a-j) were designed and prepared. Their antioxidative properties were assessed by examining their capacities in several in vitro models, including superoxide anion and 1,1-diphenyl-2-picrylhydrazyl (DPPH) radical scavenging, rat liver homogenate lipid peroxidation inhibition, PC12 cells protection from oxidative damage, and xanthine oxidase inhibition. These dihydroflavonols displayed positive quenching abilities towards O(2)(-) and DPPH free radicals, in which the majority exhibited superior antioxidant properties to Vitamin C. cis-Configurated compound (+/-)-3e demonstrated remarkable inhibition to LPO with an IC(50) value…

Models MolecularXanthine OxidaseAntioxidantFlavonolsmedicine.drug_classDPPHmedicine.medical_treatmentClinical BiochemistryMolecular ConformationPharmaceutical Sciencemedicine.disease_causeBiochemistryPC12 CellsAntioxidantsLipid peroxidationRats Sprague-Dawleychemistry.chemical_compoundStructure-Activity RelationshipDrug DiscoverymedicineAnimalsXanthine oxidaseMolecular BiologyXanthine oxidase inhibitorNeuronsSuperoxideOrganic ChemistryFree Radical ScavengersFree radical scavengerRatschemistryBiochemistryMolecular MedicineLipid PeroxidationReactive Oxygen SpeciesOxidative stressBioorganicmedicinal chemistry
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l-Carnitine l-tartrate supplementation favorably affects biochemical markers of recovery from physical exertion in middle-aged men and women.

2009

The purpose of this study was to examine the effects of Carnipure tartrate (Lonza, Allendale, NJ) supplementation (total dose of 2 g/d of l-carnitine) on markers of performance and recovery from physical exertion in middle-aged men and women. Normally active and healthy men (n = 9, 45.4 +/- 5.3 years old) and women (n = 9, 51.9 +/- 5.0 years old) volunteered to participate in the investigation. Double-blind, placebo, balanced treatment presentation and crossover design were used with 3 weeks and 3 days of supplementation followed by a 1-week washout period before the other counterbalanced treatment was initiated. After 3 weeks of each supplementation protocol, each participant then performe…

Muscle tissueAdultMalemedicine.medical_specialtyXanthine OxidaseFree RadicalsEndocrinology Diabetes and MetabolismPhysical ExertionPlaceboEndocrinologyDouble-Blind MethodInternal medicineCarnitinemedicineHumansCarnitineExertionLactic AcidLeg pressTartratesCross-Over Studiesbiologybusiness.industryMyoglobinMiddle AgedCrossover studyMiddle agemedicine.anatomical_structureEndocrinologyPurinesDietary Supplementsbiology.proteinCreatine kinaseFemalebusinessBiomarkersmedicine.drugMetabolism: clinical and experimental
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Redox signaling in acute pancreatitis

2015

Acute pancreatitis is an inflammatory process of the pancreatic gland that eventually may lead to a severe systemic inflammatory response. A key event in pancreatic damage is the intracellular activation of NF-κB and zymogens, involving also calcium, cathepsins, pH disorders, autophagy, and cell death, particularly necrosis. This review focuses on the new role of redox signaling in acute pancreatitis. Oxidative stress and redox status are involved in the onset of acute pancreatitis and also in the development of the systemic inflammatory response, being glutathione depletion, xanthine oxidase activation, and thiol oxidation in proteins critical features of the disease in the pancreas. On th…

NecrosisGSH reduced glutathioneSTAT3 signal transducer and activator of transcription 3ERK extracellular signal-regulated kinasesClinical BiochemistryCCK cholecystokininTRAFs TNF receptor associated factorsReview ArticleIκB kinasePharmacologymedicine.disease_causeBiochemistrySHP small heterodimer partnerSTIM1 stromal interaction molecule 1chemistry.chemical_compoundHATs histone acetyltransferasesMedicineASK1GCL glutamate cysteine ligaseTNF-α tumor necrosis factor alphaIKK IκB kinaseNOS nitric oxide synthaseAcute inflammationHIF hypoxia inducible factorlcsh:QH301-705.5NF-κB nuclear factor kappa BDAMPs damage-associated molecular pattern moleculeslcsh:R5-920biologyGSSG oxidized glutathioneNF-kappa BNLRs nucleotide-binding oligomerization domain (NOD) like receptorsTRADD tumor necrosis factor receptor type 1-associated DEATH domain proteinTRPC3 transient receptor potential channel 3VEGF vascular endothelial growth factorGlutathioneTNFR tumor necrosis factor receptorHMGB1 high-mobility group Box 1 proteinIP3R inositol 145-trisphosphate receptor type 3VCAM-1 Vascular Cell adhesion protein 1Acute DiseaseJNK c-Jun N-terminal kinaseAcute pancreatitisTLRs toll-like receptorsmedicine.symptomlcsh:Medicine (General)Oxidation-ReductionAP-1 activator protein-1Signal TransductionmRNA messenger ribonucleic acidHMGB1ASC apoptosis-associated speck-like protein containing a carboxy-terminal CARDRNS reactive nitrogen speciesPTPs protein tyrosine phosphatasesROS reactive oxygen speciesNADH nicotinamide adenine dinucleotidepHe extracellular pHFAEE fatty acid ethyl estersAP acute pancreatitisHumansXanthine oxidaseCBP CREB-binding proteinRyR endoplasmic reticulum membrane ryanodine receptorsMDA malondialdehydeNO nitric oxideXO xanthine oxidaseASK1 apoptosis signal-regulating kinase-1business.industryOrganic ChemistryAutophagyNADPH nicotinamide adenine dinucleotide phosphateHDACs histone deacetylasesmedicine.diseaseCARS compensatory anti-inflammatory response syndromeXDH xanthine dehydrogenaseIL interleukinIκB inhibitor of kappa BAcute pancreatitisETC Electron transport chainPancreatitisMKPs MAPK phosphatasesSAP severe acute pancreatitischemistrylcsh:Biology (General)DTT dithiothreitolOxidative stressNAC N-acetyl cysteineImmunologybiology.proteinCalciumLysosomesReactive Oxygen SpeciesbusinessMAPK mitogen-activated protein kinaseOxidative stressERCP endoscopic retrograde cholangiopancreatographyRedox Biology
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Protective Effect of Sildenafil against Estradiol-induced ROS production

2010

Several reports suggest that xanthine dehydrogenase (XDH) and its oxidase form (XO) play an important role in various forms of ischemic and vascular injuries. Recently we have demonstrated that 17β-estradiol (E2) induces a significant decrease of the expression and activity of XDH and its conversion to XO in human mammary epithelial cells. E2 is known to induce upregulation of eNOS gene expression in aortic endothelial cells. In light of the ability of XO-derived O2•¯ to combine with •NO to yield ONOO¯, and considering that ONOO¯ converts XDH to XO, it is important to protect tissues against the XO increased activity and ROS increased production, that would in turn react with •NO to augment…

OestrogenOxidative stressXanthine dehydrogenaseSettore BIO/10 - Biochimicaxanthine oxidase
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Nitric oxide and excitatory postsynaptic currents in immature rat sympathetic preganglionic neurons in vitro.

1997

Neuronal nitric oxide synthase immunoreactivity was localized to sympathetic preganglionic neurons of the intermediolateral cell column and cyclic GMP immunoreactivity to nerve fibers projecting into the intermediolateral cell column of 20-25-day-old rats. Whole-cell patch-clamp recordings were made from sympathetic preganglionic neurons in spinal cord slices of immature rats and the role of nitric oxide and cyclic GMP on excitatory postsynaptic currents was studied. Superfusing the slices with the nitric oxide precursor L-arginine (300 microM) increased the amplitude of evoked excitatory postsynaptic currents as well as the frequency of spontaneous miniature excitatory postsynaptic current…

Patch-Clamp TechniquesPostsynaptic CurrentArginineNitroarginineSynaptic TransmissionNitric oxideRats Sprague-Dawleychemistry.chemical_compoundHemoglobinsNerve Fibers1-Methyl-3-isobutylxanthinemedicineAnimalsPhosphodiesterase inhibitorCyclic GMPEvoked PotentialsNeuronsGanglia SympatheticbiologyGeneral NeuroscienceGlutamate receptorImmunohistochemistryElectric StimulationRatsNitric oxide synthasechemistryBiochemistrySpinal Cordbiology.proteinExcitatory postsynaptic potentialBiophysicsNMDA receptorSodium nitroprussideNitric Oxide Synthasemedicine.drugNeuroscience
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Pentoxifylline and Oxypurinol: Potential Drugs to Prevent the “Cytokine Release (Storm) Syndrome” Caused by SARS-CoV-2?

2020

Background: COVID-19, caused by SARS-CoV-2, is a potentially lethal, rapidly-expanding pandemic and many efforts are being carried out worldwide to understand and control the disease. COVID-19 patients may display a cytokine release syndrome, which causes severe lung inflammation, leading, in many instances, to death. Objective: This paper is intended to explore the possibilities of controlling the COVID-19-associated hyperinflammation by using licensed drugs with anti-inflammatory effects. Hypothesis: We have previously described that pentoxifylline alone, or in combination with oxypurinol, reduces the systemic inflammation caused by experimentally-induced pancreatitis in rats. Pentoxifyl…

Pharmacology0303 health sciencesbusiness.industryInflammationmedicine.diseaseSystemic inflammationPentoxifyllineProinflammatory cytokine03 medical and health scienceschemistry.chemical_compoundCytokine release syndrome0302 clinical medicinechemistry030220 oncology & carcinogenesisDrug DiscoveryImmunologyMedicineAcute pancreatitisPancreatitismedicine.symptomXanthine oxidasebusiness030304 developmental biologymedicine.drugCurrent Pharmaceutical Design
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Effects of caffeine intake on the pharmacokinetics of melatonin, a probe drug for CYP1A2 activity

2003

Aims  The aim of this study was to assess the influence of concomitant caffeine intake on the pharmacokinetics of oral melatonin, a probe drug for CYP1A2 activity. Methods  Twelve healthy subjects, six smokers and six nonsmokers, were given melatonin (6 mg) either alone or in combination with caffeine (3 × 200 mg). Blood samples for the analysis of melatonin or caffeine and paraxanthine were taken from 1 h before until 6 h after intake of melatonin. Subjects were genotyped with respect to the CYP1A2*1F (C734A) polymorphism. Results  When caffeine was coadministered the Cmax and AUC of melatonin were increased on average by 142% (P = 0.001, confidence interval on the difference 44, 80%) and …

PharmacologyChemistryCYP1A2CmaxPharmacologyCrossover studyBioavailabilityMelatoninchemistry.chemical_compoundPharmacokineticsmedicinePharmacology (medical)CaffeineParaxanthinemedicine.drugBritish Journal of Clinical Pharmacology
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Discovery of new antimalarial compounds by use of molecular connectivity techniques.

1999

Abstract Molecular connectivity has been applied to the search for new compounds with antimalarial activity. Linear discriminant analysis and connectivity functions were used to select several potentially suitable drugs which were tested for antimalarial properties by use of an in-vitro micro test which estimates parasite growth by measurement of incorporation of [3H]hypoxanthine. Hexetidine stands out among the compounds selected. Activity assays were performed with Plasmodium falciparum passou and 3CD7 strains, for which the IC50 values (doses resulting in 50% inhibition) were 320 and 400 ng mL−1 respectively. These results are comparable with those obtained for quinine chlorhydrate (IC50…

PharmacologyDrugQuininebiologyStereochemistrymedia_common.quotation_subjectPlasmodium falciparumPharmaceutical SciencePlasmodium falciparumBiological activityHexetidineChloroquine sulphatebiology.organism_classificationchemistry.chemical_compoundAntimalarialsBiochemistrychemistryDrug DesignmedicineAnimalsHumansIC50Hypoxanthinemedicine.drugmedia_commonThe Journal of pharmacy and pharmacology
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Zur Wirkung von Butazolidin im Intermedi�rstoffwechsel

1956

Butazolidin (Phenylbutazone) inhibits the oxidative decarboxylation of pyruvate and α-ketoglutarate in a final concentration of 10 mg-% (3,24 · 10−4 m). Data are presented suggesting that the β-ketothiolase is inhibited. The following enzymes or enzyme systems are not inhibited: The enzymes of the respiratory chain, the enzymes of the citric acid cycle with exception of α-ketoglutaric oxidase, the glycolysis of hexosediphosphate (slight inhibition), acetate thiokinase, sulfanilamid transacetylase, pyruvic decarboxylase from yeast, arginase, xanthine oxidase, and D-amino acid oxidase.

Pharmacologychemistry.chemical_classificationOxidase testRespiratory chainGeneral MedicineCitric acid cyclechemistry.chemical_compoundEnzymechemistryBiochemistryAcetate thiokinaseXanthine oxidaseOxidative decarboxylationPyruvate decarboxylaseNaunyn-Schmiedebergs Archiv f�r Experimentelle Pathologie und Pharmakologie
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