Search results for " Cellular"

showing 10 items of 384 documents

Research of a Cellular Automaton Simulating Logic Gates by Evolutionary Algorithms

2003

This paper presents a method of using genetic programming to seek new cellular automata that perform computational tasks. Two genetic algorithms are used : the first one discovers a rule supporting gliders and the second one modifies this rule in such a way that some components appear allowing it to simulate logic gates. The results show that the genetic programming is a promising tool for the search of cellular automata with specific behaviors, and thus can prove to be decisive for discovering new automata supporting universal computation.

Fitness functionTheoretical computer scienceComputer sciencebusiness.industryComputationEvolutionary algorithmGenetic programmingCellular automatonAutomatonMobile automatonGenetic algorithmGenetic representationArtificial intelligencebusinessAsynchronous cellular automaton
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Advanced fluorescence microscopy for in vivo imaging of neuronal activity

2019

Brain function emerges from the coordinated activity, over time, of large neuronal populations placed in different brain regions. Understanding the relationships of these specific areas and disentangling the contributions of individual neurons to overall function remain central goals for neuroscience. In this scenario, fluorescence microscopy has been proved as the tool of choice for in vivo recording of brain activity. Optical advances combined with genetically encoded indicators allow a large flexibility in terms of spatiotemporal resolution and field of view while keeping invasiveness in living animals to a minimum. Here we describe the latest advancements in the field of linear and nonl…

Flexibility (engineering)0303 health sciencesBrain activity and meditationComputer science01 natural sciencesAtomic and Molecular Physics and OpticsElectronic Optical and Magnetic Materials010309 optics03 medical and health scienceslight-sheet microscopy; field-of-view; cellular-resolution; adaptive optics; multiphoton microscopy; GRID CELLS; HIGH-SPEED; LONG-TERM; 2-PHOTON; DEEPLight sheet fluorescence microscopy0103 physical sciencesFluorescence microscopePremovement neuronal activityIn vivo microscopyOptics In vivo imaging MicroscopyNeurosciencePreclinical imagingBrain function030304 developmental biologyOptica
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Maximizing network capacity in an heterogeneous macro-micro cellular scenario

2011

The problem of resource allocation in cellular networks has been traditionally faced at two different levels: at the network level, in terms of frequency planning and reuse pattern design, and at the cell level, in terms of cell capacity optimizations based on channel-dependent scheduling, link adaptation, power control, and so on. While this second aspect has been deeply investigated in literature, the first aspect has been mainly faced with static or semi-dynamic reuse utilization solutions. In this paper we deal with the problem of multi-cellular resource allocation in heterogeneous OFDMA environments with reuse factor equal to 1, where base stations with different power constraints coex…

Frequency-division multiple accessChannel allocation schemesSettore ING-INF/03 - TelecomunicazioniComputer sciencebusiness.industryOrthogonal frequency-division multiplexingLink adaptationRadio Resource Management Cellular networks OFDMAScheduling (computing)Network planning and designBase stationMobile stationCellular networkResource allocationResource managementbusinessComputer networkPower control2011 IEEE Symposium on Computers and Communications (ISCC)
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The Cell Cycle-Specific Growth-Inhibitory Factor Produced by Actinobacillus actinomycetemcomitans Is a Cytolethal Distending Toxin

1998

ABSTRACT Actinobacillus actinomycetemcomitans has been shown to produce a soluble cytotoxic factor(s) distinct from leukotoxin. We have identified in A. actinomycetemcomitans Y4 a cluster of genes encoding a cytolethal distending toxin (CDT). This new member of the CDT family is similar to the CDT produced by Haemophilus ducreyi . The CDT from A. actinomycetemcomitans was produced in Escherichia coli and was able to induce cell distension, growth arrest in G 2 /M phase, nucleus swelling, and chromatin fragmentation in HeLa cells. The three proteins, CDTA, -B and -C, encoded by the cdt locus were all required for toxin activity. Antiserum raised against recombinant CDTC completely inhibited …

G2 PhaseCytolethal distending toxin[SDV]Life Sciences [q-bio]Bacterial ToxinsMolecular Sequence DataRestriction MappingImmunologyMitosismedicine.disease_causeAggregatibacter actinomycetemcomitansMicrobiologyVirulence factorMicrobiologyEscherichia colimedicineHumansAmino Acid SequenceCloning MolecularEscherichia coliBase SequencebiologyToxinACTIVITEAggregatibacter actinomycetemcomitansGENETIQUECell cyclebiology.organism_classificationGrowth InhibitorsRecombinant Proteins[SDV] Life Sciences [q-bio]Infectious DiseasesGenes BacterialMultigene FamilyActinobacillusMolecular and Cellular PathogenesisParasitologyHaemophilus ducreyiHeLa CellsInfection and Immunity
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Galectin-3 Impairment of MYCN-Dependent Apoptosis-Sensitive Phenotype Is Antagonized by Nutlin-3 in Neuroblastoma Cells

2012

MYCN amplification occurs in about 20-25% of human neuroblastomas and characterizes the majority of the high-risk cases, which display less than 50% prolonged survival rate despite intense multimodal treatment. Somehow paradoxically, MYCN also sensitizes neuroblastoma cells to apoptosis, understanding the molecular mechanisms of which might be relevant for the therapy of MYCN amplified neuroblastoma. We recently reported that the apoptosis-sensitive phenotype induced by MYCN is linked to stabilization of p53 and its proapoptotic kinase HIPK2. In MYCN primed neuroblastoma cells, further activation of both HIPK2 and p53 by Nutlin-3 leads to massive apoptosis in vitro and to tumor shrinkage an…

Galectin 3Cancer TreatmentGene Dosagelcsh:MedicineApoptosisProtein-Serine-Threonine KinaseBiochemistryPiperazineschemistry.chemical_compoundNeuroblastoma0302 clinical medicineMolecular Cell BiologyBasic Cancer ResearchSignaling in Cellular Processeslcsh:ScienceEnergy-Producing OrganellesApoptotic SignalingNuclear ProteinOncogene Proteins0303 health sciencesN-Myc Proto-Oncogene ProteinMultidisciplinaryCell DeathImidazolesOncogene ProteinNuclear ProteinsTransfectionNutlin3. Good healthGene Expression Regulation NeoplasticProtein TransportCell killingPhenotypeOncologyGalectin-3030220 oncology & carcinogenesisGene Knockdown TechniquesMedicineResearch ArticleSignal TransductionHumanBiologyBioenergeticsProtein Serine-Threonine KinasesN-Myc Proto-Oncogene ProteinModels Biological03 medical and health sciencesNeuroblastomaCell Line TumormedicineHumansBiologyImidazolePiperazineneoplasms030304 developmental biologylcsh:RGene AmplificationChemotherapy and Drug Treatmentmedicine.diseasechemistryCell cultureApoptosisPediatric OncologyCytoprotectionGene Knockdown TechniqueCancer researchlcsh:QTumor Suppressor Protein p53Carrier ProteinsCarrier ProteinDNA Damage
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Mathematical logic and quantum finite state automata

2009

AbstractThis paper is a review of the connection between formulas of logic and quantum finite-state automata in respect to the language recognition and acceptance probability of quantum finite-state automata. As is well known, logic has had a great impact on classical computation, it is promising to study the relation between quantum finite-state automata and mathematical logic. After a brief introduction to the connection between classical computation and logic, the required background of the logic and quantum finite-state automata is provided and the results of the connection between quantum finite-state automata and logic are presented.

General Computer ScienceMeasure-many quantum finite-state automataComputational logicMultimodal logicQuantum dot cellular automatonIntermediate logicMeasure-once quantum finite-state automataNonlinear Sciences::Cellular Automata and Lattice GasesTheoretical Computer ScienceAlgebraTheoryofComputation_MATHEMATICALLOGICANDFORMALLANGUAGESModular logicComputerSystemsOrganization_MISCELLANEOUSComputer Science::Logic in Computer ScienceQuantum finite automataDynamic logic (modal logic)Automata theoryQuantum finite-state automataFirst-order logicAlgorithmComputer Science::Formal Languages and Automata TheoryMathematicsQuantum cellular automatonComputer Science(all)Theoretical Computer Science
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Molecular biomarkers in glaucoma

2013

The seventh annual ARVO/Pfizer Ophthalmic Research Institute conference was held Friday and Saturday, April 29 and 30, 2011, at the Fort Lauderdale Hyatt Regency Pier 66, Fort Lauderdale, Florida. The conference, funded by The ARVO Foundation for Eye Research through a grant from Pfizer Ophthalmics, provided an opportunity to gather experts from within and outside ophthalmology to determine the state of knowledge pertaining to molecular biomarkers associated with glaucoma, as well as the methods to identify and validate them to predict (a) those who would be susceptible to development of glaucoma; (b) markers that will enable prediction of glaucoma progression; and (c) markers that will pre…

Genetic MarkersProteomicsmedicine.medical_specialtygenetic structuresGlaucomaHuman healthCellular and Molecular NeuroscienceOphthalmologyMedicineHumansBiomarkers; Genetic Markers; Genomics; Glaucoma; Humans; Metabolome; Proteomics; Ophthalmology; Sensory Systems; Cellular and Molecular Neuroscience; Medicine (all)Biomarker discoveryBlindnessbusiness.industryMedicine (all)GlaucomaArticlesGenomicsmedicine.diseaseMolecular biomarkerseye diseasesBench to bedsideSensory SystemsOphthalmologyMetabolomeOptometrysense organsbusinessBiomarkers
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Conduct disorder and ADHD: evaluation of conduct problems as a categorical and quantitative trait in the international multicentre ADHD genetics stud…

2008

Contains fulltext : 71374.pdf (Publisher’s version ) (Closed access) Attention-deficit/hyperactivity disorder (ADHD) is typically characterized by inattention, excessive motor activity, impulsivity, and distractibility. Individuals with ADHD have significant impairment in family and peer relations, academic functioning, and show high co-morbidity with a wide range of psychiatric disorders including oppositional defiant disorder (ODD), conduct disorder (CD), anxiety disorder, depression, substance abuse, and pervasive developmental disorder (PDD). Family studies suggest that ADHD + CD represents a specific subtype of the ADHD disorder with familial risk factors only partly overlapping with t…

Genetics and epigenetic pathways of disease [NCMLS 6]2804 Cellular and Molecular NeuroscienceMedizinPoison controlComorbidityNeuroinformatics [DCN 3]Linkage DisequilibriumCohort Studies2738 Psychiatry and Mental Health0302 clinical medicinePerception and Action [DCN 1]Genetics(clinical)ChildGenetics (clinical)Oligonucleotide Array Sequence Analysis0303 health sciencesAntisocial Personality Disorder10058 Department of Child and Adolescent Psychiatry3. Good healthPedigreeEuropePsychiatry and Mental healthConduct disorderAttention Deficit and Disruptive Behavior DisordersADHD -- conduct disorder -- genome wide association study -- genetic association information networkmedicine.symptomPsychologyFunctional Neurogenomics [DCN 2]Anxiety disorderAlgorithmsClinical psychologyConduct DisorderGenetic Markers2716 Genetics (clinical)Quantitative Trait LociContext (language use)610 Medicine & healthChild Behavior DisordersImpulsivityPolymorphism Single NucleotideMental health [NCEBP 9]behavioral disciplines and activitiesGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceCognitive neurosciences [UMCN 3.2]mental disordersmedicinePervasive developmental disorderHumansddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und Jugendalters030304 developmental biologyPsychiatric Status Rating ScalesGenome HumanAntisocial personality disordermedicine.diseaseComorbidityGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with Hyperactivity030217 neurology & neurosurgeryGenome-Wide Association Study
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Genetic and molecular analysis of six tumor suppressor genes in Drosophila melanogaster

1990

Six Drosophila melanogaster tumor suppressor genes causing malignant or benign tumors in specific cell types are described. The wild-type alleles of these genes are instrumental in the differentiation of particular cell types. In the homozygous state, recessive mutations in the genes interrupt the differentiation of the cells and thus cause their uncontrolled, autonomous, lethal proliferation. The tumors show all major characteristics of malignant and benign neoplastic growth. Genomic sequences of four of the genes have been identified and are currently being characterized. ImagesFIGURE 1.FIGURE 2.FIGURE 2.

GeneticsCell typebiologyHealth Toxicology and MutagenesisRestriction MappingPublic Health Environmental and Occupational HealthNeoplastic growthNeoplasms Experimentalbiology.organism_classificationMolecular analysislaw.inventionMolecular and Cellular Aspects of Transformation and DifferentiationRestriction mapDrosophila melanogasterlawSuppressorAnimalsGenes LethalGenes Tumor SuppressorDrosophila melanogasterAlleleGeneEnvironmental Health Perspectives
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MAD2 depletion triggers premature cellular senescence in human primary fibroblasts by activating a P53 pathway preventing aneuploid cells propagation.

2012

The spindle assembly checkpoint (SAC) is a cellular surveillance mechanism that ensures faithful chromosome segregation during mitosis and its failure can result in aneuploidy. Previously, it was suggested that reduction of the MAD2 gene, encoding a major component of the SAC, induced aneuploidy in human tumor cells. However, tumor cell lines contain multiple mutations that might affect or exacerbate the cellular response to Mad2 depletion. Thus, the scenario resulting by Mad2 depletion in primary human cells could be different and more complex that the one depicted so far. We used primary human fibroblasts (IMR90) and epithelial breast cells (MCF10A) to gain further insight on the effects …

Genome instabilityCyclin-Dependent Kinase Inhibitor p21Cell cycle checkpointMad2PhysiologyClinical BiochemistryMAD2 depletion Aneuploidy Premature cellular senescence TP53Cell Cycle ProteinsBiologyCyclin-dependent kinaseChromosome instabilityChromosomal InstabilityTumor Suppressor Protein p14ARFHumansGene SilencingRNA Small InterferingMitosisCells CulturedCellular SenescenceCell ProliferationCalcium-Binding ProteinsCell BiologyCell Cycle CheckpointsFibroblastsAneuploidybeta-GalactosidaseCell biologyRepressor ProteinsSpindle checkpointSettore BIO/18 - GeneticaGene Expression RegulationMad2 Proteinsbiology.proteinM Phase Cell Cycle CheckpointsTumor Suppressor Protein p53Cell agingSignal Transduction
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