Search results for " Genetics"

showing 10 items of 4169 documents

Extrapolation of the Species Accumulation Curve for Incomplete Species Samplings: A New Nonparametric Approach to Estimate the Degree of Sample Compl…

2015

9 pages; International audience; Incomplete species samplings are deemed to remain the common practice in those groups of animals, such as small or micro- invertebrates, with numerous species that often are more or less difficult to detect in the field. Thus, extrapolating the Species Accumulation Curve as far as possible beyond the actual sample size may thus serve as a useful (although imperfect) surrogate to the desired, but practically inaccessible, complete samplings. In this context, several kinds of theoretical or empirical models for the Species Accumulation Curve and also a lot of estimators of the asymptotic limit of the Curve (i.e. total species richness) have been proposed. The …

[STAT.AP]Statistics [stat]/Applications [stat.AP][ SDE.BE ] Environmental Sciences/Biodiversity and Ecologyspecies accumulation curveSpecies discovery curve[ STAT.AP ] Statistics [stat]/Applications [stat.AP]ExtrapolationNonparametric statisticsExtrapolationChao estimatorSampling (statistics)Sample (statistics)General Biochemistry Genetics and Molecular Biologystop samplingjackknifeStatisticsnon-parametricPoint estimationSpecies richness[SDE.BE]Environmental Sciences/Biodiversity and Ecologypoint estimationspecies richnessGeneral Agricultural and Biological SciencesJackknife resamplingsample completenessMathematicsAnnual Research & Review in Biology
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The selective advantage of cystic fibrosis heterozygotes tested by aDNA analysis: A preliminary investigation

2000

Recently a heterozygote advantage was suggested to explain the high incidence (1:25 carrier individuals in Europeans) of the cystic fibrosis gene. This selective advantage was speculated to be due to a high resistance to chloride-secreting diarrhea, including cholera. Up to now the major efforts to test directly this hypothesis have been limited to animal models.

aDNAPathologymedicine.medical_specialtyCystic fibrosis genecystic fibrosis aDNA ancient DNAmedicine.disease_causeCystic fibrosisNOcystic fibrosis03 medical and health sciencesSelective advantagemedicineancient DNA030304 developmental biology0303 health sciencesbiology030305 genetics & heredityCholera toxinHeterozygote advantagemedicine.diseaseCholeraCystic fibrosis transmembrane conductance regulator3. Good healthDiarrheaAnthropologyImmunologybiology.proteinmedicine.symptom
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Role of Chemerin in Cardiovascular Diseases.

2022

(1) Background: Obesity is closely connected to the pathophysiology of cardiovascular diseases (CVDs). Excess fat accumulation is associated with metabolic malfunctions that disrupt cardiovascular homeostasis by activating inflammatory processes that recruit immune cells to the site of injury and reduce nitric oxide levels, resulting in increased blood pressure, endothelial cell migration, proliferation, and apoptosis. Adipose tissue produces adipokines, such as chemerin, that may alter immune responses, lipid metabolism, vascular homeostasis, and angiogenesis. (2) Methods: We performed PubMed and MEDLINE searches for articles with English abstracts published between 1997 (when the first re…

adipokinechemerin receptorsMedicine (miscellaneous)610 Medicine & health2701 Medicine (miscellaneous)endothelial dysfunctionGeneral Biochemistry Genetics and Molecular Biologyadipokine cardiovascular disease chemerin chemerin receptors chemerin-targeting therapeutic agents endothelial dysfunction inflammation1300 General Biochemistry Genetics and Molecular Biologycardiovascular diseaseinflammation10209 Clinic for Cardiologychemerin-targeting therapeutic agentschemerinBiomedicines
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NAD+ repletion with niacin counteracts cancer cachexia

2023

AbstractCachexia is a debilitating wasting syndrome and highly prevalent comorbidity in cancer patients. It manifests especially with energy and mitochondrial metabolism aberrations that promote tissue wasting. We recently identified nicotinamide adenine dinucleotide (NAD+) loss to associate with muscle mitochondrial dysfunction in cancer hosts. In this study we confirm that depletion of NAD+ and downregulation of Nrk2, an NAD+ biosynthetic enzyme, are common features of severe cachexia in different mouse models. Testing NAD+ repletion therapy in cachectic mice reveals that NAD+ precursor, vitamin B3 niacin, efficiently corrects tissue NAD+ levels, improves mitochondrial metabolism and amel…

aineenvaihduntahäiriötMultidisciplinaryenergy metabolismcancerGeneral Physics and AstronomysyöpätauditGeneral Chemistrymetabolic diseasesaineenvaihduntaGeneral Biochemistry Genetics and Molecular Biology
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Distinctive amino acid residue periodicities in terminal sequences of type III and type I secreted proteins from proteobacteria

2007

AbstractThe Fourier transform (FT) method was applied to specify the distribution of 14 predefined groups of amino acids (64 residues) at both termini of annotated type III and type I secreted proteins from proteobacteria. Type I proteins displayed a higher occurrence of significant periodicities at both C-and N-termini, indicating potent features to discriminate between secretion types, particularly by the use of variables selected from the full periodicity profiles at 19 orders of FT. The Fishers linear discriminant analysis, together with the stepwise selection of variables throughout equal pairs of combinations for all predefined groups of residues, revealed the C-terminal harmonics of …

amino acid periodicityQH301-705.5Computational biologyBiologyBioinformaticsGeneral Biochemistry Genetics and Molecular Biologysymbols.namesakeDiscriminant function analysisprotein secretionBiology (General)chemistry.chemical_classificationGeneral Immunology and MicrobiologyGeneral NeuroscienceStepwise regressiondiscriminant analysisLinear discriminant analysisbiology.organism_classificationAmino acidSecretory proteinFourier transformchemistryTest setsymbolsProteobacteriaGeneral Agricultural and Biological SciencesproteobacteriaOpen Life Sciences
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A two-stage genome-wide association study of sporadic amyotrophic lateral sclerosis

2009

The cause of sporadic amyotrophic lateral sclerosis (ALS) is largely unknown, but genetic factors are thought to play a significant role in determining susceptibility to motor neuron degeneration. To identify genetic variants altering risk of ALS, we undertook a two-stage genome-wide association study (GWAS): we followed our initial GWAS of 545 066 SNPs in 553 individuals with ALS and 2338 controls by testing the 7600 most associated SNPs from the first stage in three independent cohorts consisting of 2160 cases and 3008 controls. None of the SNPs selected for replication exceeded the Bonferroni threshold for significance. The two most significantly associated SNPs, rs2708909 and rs2708851 …

amyotrophic lateral sclerosisLinkage disequilibriumPopulationamyotrophic lateral sclerosis; genetics; GWASingle-nucleotide polymorphismGenome-wide association studyBiologyGWAPolymorphism Single Nucleotide03 medical and health sciences0302 clinical medicineGenotypeGeneticsmedicineHumansPolymorphismAmyotrophic lateral sclerosiseducationMolecular BiologyGenetics (clinical)030304 developmental biologyGenetics0303 health scienceseducation.field_of_studyGenomeSLA wide genome screeningGenome HumanAssociation Studies ArticlesCase-control studySingle NucleotideGeneral MedicineOdds ratiomedicine.diseaseSettore MED/26 - NEUROLOGIAAmyotrophic Lateral Sclerosis; genetics Case-Control Studies Genome; Human Genome-Wide Association Study Humans Polymorphism; Single NucleotideCase-Control Studies030217 neurology & neurosurgeryHumanGenome-Wide Association StudyHuman Molecular Genetics
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NAD+ Precursors and Antioxidants for the Treatment of Amyotrophic Lateral Sclerosis

2021

Charcot first described amyotrophic lateral sclerosis (ALS) between 1865 and 1874 as a sporadic adult disease resulting from the idiopathic progressive degeneration of the motor neuronal system, resulting in rapid, progressive, and generalized muscle weakness and atrophy. There is no cure for ALS and no proven therapy to prevent it or reverse its course. There are two drugs specifically approved for the treatment of ALS, riluzol and edaravone, and many others have already been tested or are following clinical trials. However, at the present moment, we still cannot glimpse a true breakthrough in the treatment of this devastating disease. Nevertheless, our understanding of the pathophysiology…

amyotrophic lateral sclerosisbusiness.industryQH301-705.5NAD<sup>+</sup>Medicine (miscellaneous)Degeneration (medical)Diseasemedicine.disease_causemedicine.diseaseGeneral Biochemistry Genetics and Molecular BiologyPathophysiologychemistry.chemical_compoundAtrophyantioxidantschemistryEdaravoneMedicinemotor neuronsoxidative stressNAD+ kinaseAmyotrophic lateral sclerosisBiology (General)businessNeuroscienceOxidative stressBiomedicines
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Chemically induced mouse models of intestinal inflammation

2007

Animal models of intestinal inflammation are indispensable for our understanding of the pathogenesis of Crohn disease and ulcerative colitis, the two major forms of inflammatory bowel disease in humans. Here, we provide protocols for establishing murine 2,4,6-trinitro benzene sulfonic acid (TNBS)-, oxazolone- and both acute and chronic dextran sodium sulfate (DSS) colitis, the most widely used chemically induced models of intestinal inflammation. In the former two models, colitis is induced by intrarectal administration of the covalently reactive reagents TNBS/oxazolone, which are believed to induce a T-cell-mediated response against hapten-modified autologous proteins/luminal antigens. In …

animal diseasesdigestive systemInflammatory bowel diseaseGeneral Biochemistry Genetics and Molecular BiologyPathogenesisOxazoloneMicechemistry.chemical_compoundAntigenmedicineAnimalsColitisbiologyDextran SulfateOxazoloneEpithelial CellsColitismedicine.diseaseUlcerative colitisdigestive system diseasesDisease Models AnimalTrinitrobenzenesulfonic AcidchemistryImmunologybiology.proteinAntibodyHaptenNature Protocols
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Prediction model for aneuploidy in early human embryo development revealed by single-cell analysis.

2014

Aneuploidies are prevalent in the human embryo and impair proper development, leading to cell cycle arrest. Recent advances in imaging and molecular and genetic analyses are postulated as promising strategies to unveil the mechanisms involved in aneuploidy generation. Here we combine time-lapse, complete chromosomal assessment and single-cell RT–qPCR to simultaneously obtain information from all cells that compose a human embryo until the approximately eight-cell stage (n=85). Our data indicate that the chromosomal status of aneuploid embryos (n=26), including those that are mosaic (n=3), correlates with significant differences in the duration of the first mitotic phase when compared with e…

animal structuresCellular differentiationGeneral Physics and AstronomyAneuploidyBiologyModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyArticleTranscriptomeSingle-cell analysismedicineHumansGenetic TestingMitosisCell ProliferationGeneticsMultidisciplinaryGene Expression ProfilingGene Expression Regulation DevelopmentalEmbryoCell DifferentiationGeneral Chemistrymedicine.diseaseAneuploidyGene expression profilingembryonic structuresPloidyNature communications
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Social Interactions of Dat-Het Epi-Genotypes Differing for Maternal Origins: The Development of a New Preclinical Model of Socio-Sexual Apathy

2021

Social interaction is essential for life but is impaired in many psychiatric disorders. We presently focus on rats with a truncated allele for dopamine transporter (DAT). Since heterozygous individuals possess only one non-mutant allele, epigenetic interactions may unmask latent genetic predispositions. Homogeneous “maternal” heterozygous offspring (termed MAT-HET) were born from dopamine-transporter knocked-out (DAT-KO) male rats and wild-type (WT) mothers

animal structuresOffspringQH301-705.5socio-sexual rewardMedicine (miscellaneous)PhysiologyHippocampusArticleGeneral Biochemistry Genetics and Molecular Biologysocial behavior03 medical and health sciences0302 clinical medicineGenetic predispositionAlleleBiology (General)Prefrontal cortexdopamine transporterDopamine transporterEstrous cyclebiologyapplied_psychology030227 psychiatrySettore BIO/14 - Farmacologiabiology.proteinparent-of-origin effectAbnormality030217 neurology & neurosurgeryBiomedicines
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