Search results for " patologia"

showing 10 items of 779 documents

miR-126-3p and miR-21-5p as Hallmarks of Bio-Positive Ageing; Correlation Analysis and Machine Learning Prediction in Young to Ultra-Centenarian Sici…

2022

Human ageing can be characterized by a profile of circulating microRNAs (miRNAs), which are potentially predictors of biological age. They can be used as a biomarker of risk for age-related inflammatory outcomes, and senescent endothelial cells (ECs) have emerged as a possible source of circulating miRNAs. In this paper, a panel of four circulating miRNAs including miR-146a-5p, miR-126-3p, miR-21-5p, and miR-181a-5p, involved in several pathways related to inflammation, and ECs senescence that seem to be characteristic of the healthy ageing phenotype. The circulating levels of these miRNAs were determined in 78 healthy subjects aged between 22 to 111 years. Contextually, extracellular miR-1…

Aged 80 and overSettore MED/04 - Patologia Generaleageing; inflamm-ageing; endothelial senescence; longevity; miRNAsagingEndothelial Cellsinflamm-ageingGeneral Medicineinflamm-agingMachine LearningMicroRNAslongevityageingendothelial senescenceCentenariansmiRNAsHumansCirculating MicroRNABiomarkersCells; Volume 11; Issue 9; Pages: 1505
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Centenarians as a model to discover genetic and epigenetic signatures of healthy ageing.

2018

Abstract Centenarians are a model of successful ageing. The data favours the theory that, in order to live to 100, it is mandatory to inherit the right genetic variants from parents or acquire epigenetic variants through the environment. Therefore, the study of epigenetic signatures of healthy ageing is becoming an important aspect to identify the role of chromatin modification in ageing and understand how manage this fine-tuning system. So, according to the concept of developmental plasticity, establishment of a longevity phenotype requires a combination of stochastic and non-stochastic events that modulate the genetic substrate and leads to a different outcome. It can be concluded that ce…

Ageing; Diet; Epigenetics; Genetics; Longevity; Stochasticity; Aging; Developmental Biology0301 basic medicineMaleAgingmedia_common.quotation_subjectLongevityBiologyModels BiologicalEpigenesis Genetic03 medical and health sciencesGeneticGeneticsHumansEpigeneticsmedia_commonStochasticitySettore MED/04 - Patologia GeneraleGeneticsAged 80 and overGenetic variantsLongevityEpigeneticDietAgeing030104 developmental biologyAgeingEvolutionary biologyDevelopmental plasticityChromatin modificationSuccessful ageingEpigeneticsFemaleHealthy ageingDevelopmental BiologyMechanisms of ageing and development
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Senescence Markers

2012

Background: Evolutionary theory and empirical evidence suggest that aging is a process of gradual accumulation of damage in cells and tissues of the body. The progressive loss of ability to interact effectively with environmental stimuli is accompanied by progressive modification and adaptations that are influenced by lifestyle and genetic background of the individual. These affect the ability to age successfully, defined both as longevity and/or escaping the major age-related diseases. Some of the most important characteristics of adaptive immunity in aging are compatible with this assumption. Actually, the antigenic load results in the progressive generation of a chronic low grade inflamm…

Aging genetic markersSettore MED/05 - Patologia Clinica
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B Cells Compartment in Centenarian Offspring and Old People

2010

Immunosenescence is considered a major contributory factor to the increased frequency of morbidity and mortality among elderly. On the other hand centenarians are considered the best example of successful ageing. To gain insight into mechanisms of immunosenescence and its clinical relevance, a possible model is represented by centenarians and/or their offspring. Nowadays centenarians are not more a curiosity, but in Europe are 1/8000 inhabitants and it has been demonstrated that the centenarian offspring, who are typically in their 70s and 80s, have a survival advantage when compared with age-matched controls whose parents died at an average life expectancy. Then again, studies on immunosen…

AgingB lymphocyte centenarian immunosenescence longevityOffspringT cellLongevityNaive B cellB-Lymphocyte SubsetsImmunoglobulin DDrug DiscoverymedicineHumansB cellAgedAged 80 and overSettore MED/04 - Patologia GeneralePharmacologybiologybusiness.industryImmunosenescenceMiddle AgedCell Compartmentationmedicine.anatomical_structureAgeingImmunologybiology.proteinAdult ChildrenCentenarianbusinessCurrent Pharmaceutical Design
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NF-κB pathway activators as potential ageing biomarkers: targets for new therapeutic strategies

2013

Chronic inflammation is a major biological mechanism underpinning biological ageing process and age-related diseases. Inflammation is also the key response of host defense against pathogens and tissue injury. Current opinion sustains that during evolution the host defense and ageing process have become linked together. Thus, the large array of defense factors and mechanisms linked to the NF-κB system seem to be involved in ageing process. This concept leads us in proposing inductors of NF-κB signaling pathway as potential ageing biomarkers. On the other hand, ageing biomarkers, represented by biological indicators and selected through apposite criteria, should help to characterize biologica…

AgingBiological ageInflammatory network and its effects in ageingImmunologyLarge arrayReviewWorking hypothesisBiological ageing processchemistry.chemical_compoundBiological ageing process Inflammatory network and its effects in ageing NF-κB signaling pathway as hub of inflammatory ageing network Inflammatory biomarkersSettore MED/05 - Patologia ClinicaMedicineSettore MED/04 - Patologia Generalebusiness.industryMechanism (biology)NF-κBInflammatory biomarkersRisk factor (computing)NF-κB signaling pathway as hub of inflammatory ageing networkInflammatory biomarkersAgeingchemistryAgeingImmunologybusinessNeuroscienceImmunity & Ageing
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Pro-inflammatory genetic background and zinc status in old atherosclerotic subjects.

2008

Inflammation and genetics are prominent mechanisms in the pathogenesis of atherosclerosis (AT) and its complications. In this review we discuss the possible impact on AT development of several genetic determinants involved in inflammation, oxidative stress and cytoprotection (IL-6, TNF-alpha, IL-10, CD14, TLR4, MT, HSP70). Genetic polymorphisms of these genes may affect a differential inflammatory response predisposing to AT. However, allelic polymorphisms of genes which increase the risk of AT frequently occur in the general population but, only adequate gene-environment-polymorphism interactions promote the onset of the disease. Zinc deficiency has been suggested as an environmental risk …

AgingCandidate genePopulationInflammationDiseaseBiologymedicine.disease_causeBiochemistryPathogenesisAgeing atherosclerosis inflammation geneticsmedicineSettore MED/05 - Patologia ClinicaHomeostasisHumansAlleleeducationMolecular BiologyAgedSettore MED/04 - Patologia GeneraleAged 80 and overInflammationeducation.field_of_studymedicine.diseaseAtherosclerosisOxidative StressZincNeurologyImmunologyZinc deficiencyMetallothioneinmedicine.symptomOxidative stressBiotechnologyAgeing research reviews
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CCR5 Receptor: Biologic and Genetic Implications in Age-Related Diseases

2007

The CC chemokine receptor 5 (CCR5) is a member of CC-chemokine receptor family. CCR5 has the characteristic structure of a seven transmembrane G protein-coupled receptor (GPCR), which regulates trafficking and effector functions of memory/effector Th1 cells, macrophages, NK cells, and immature dendritic cells. CCR5 and its ligands are important molecules in viral pathogenesis. CCR5 represents the co-receptor for macrophage (M) and dual (T cell and M)-tropic immunodeficiency viruses. Recent evidence has also demonstrated the role of CCR5 in a variety of human diseases, ranging from infectious and inflammatory diseases to cancer. In this article, we describe the involvement of CCR5 in two age…

AgingChemokineReceptors CCR5Chemokine receptor CCR5virusesT cellViral pathogenesisDiseaseLigandsModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyHistory and Philosophy of Sciencecardiovascular diseaseAlzheimer DiseasemedicineHumansMacrophageSettore MED/04 - Patologia GeneraleInflammationGenomebiologyEffectorMacrophagesGeneral Neurosciencevirus diseasesDendritic CellsAtherosclerosisKiller Cells Naturalmedicine.anatomical_structureCardiovascular DiseasesImmunologybiology.proteinMicrogliaCC chemokine receptorsAlzheimer’s diseaseCCR5Gene DeletionAnnals of the New York Academy of Sciences
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Role of Cachexia and Fragility in the Patient Candidate for Cardiac Surgery

2021

Frailty is the major expression of accelerated aging and describes a decreased resistance to stressors, and consequently an increased vulnerability to additional diseases in elderly people. The vascular aging related to frail phenotype reflects the high susceptibility for cardiovascular diseases and negative postoperative outcomes after cardiac surgery. Sarcopenia can be considered a biological substrate of physical frailty. Malnutrition and physical inactivity play a key role in the pathogenesis of sarcopenia. We searched on Medline (PubMed) and Scopus for relevant literature published over the last 10 years and analyzed the strong correlation between frailty, sarcopenia and cardiovascular…

AgingFood intakemedicine.medical_specialtyCachexiaMEDLINElcsh:TX341-641Reviewfrailtymalnutrition030204 cardiovascular system & hematologyage related syndromeCachexiasarcopeniaSettore MED/34Eating03 medical and health sciences0302 clinical medicinePreoperative CaremedicineHumansAge related syndrome Frailty Malnutrition SarcopeniaSettore MED/05 - Patologia Clinica030212 general & internal medicineCardiac Surgical ProceduresIntensive care medicineNutrition and Dieteticsbusiness.industryStressorPreoperative ExerciseResistance Trainingmedicine.diseaseCardiac surgeryMalnutritionPhenotypevascular agingCardiovascular DiseasesSarcopeniaBlood VesselsVascular agingSedentary Behaviorbusinesslcsh:Nutrition. Foods and food supplyBiomarkersFood ScienceNutrients
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Taste receptor polymorphisms and longevity: a systematic review and meta-analysis

2020

AbstractBitter taste receptors (TAS2R) are involved in a variety of non-tasting physiological processes, including immune-inflammatory ones. Therefore, their genetic variations might influence various traits. In particular, in different populations of South Italy (Calabria, Cilento, and Sardinia), polymorphisms of TAS2R16 and TAS238 have been analysed in association with longevity with inconsistent results. A meta-analytic approach to quantitatively synthesize the possible effect of the previous variants and, possibly, to reconcile the inconsistencies has been used in the present paper. TAS2R38 variants in the Cilento population were also analysed for their possible association with longevi…

AgingGenotypemedia_common.quotation_subjectPopulationLongevityReviewBiologyPolymorphism Single NucleotideReceptors G-Protein-Coupled03 medical and health sciences0302 clinical medicineTaste receptorGenetic variationGenotypeHumansMeta-analysieducation030304 developmental biologymedia_commonImmune-inflammatory responsesSettore MED/04 - Patologia Generale0303 health scienceseducation.field_of_studyLongevityImmune-inflammatory responses; Longevity; Meta-analysis; Taste receptorsTaste receptorsMeta-analysisTAS2R38Evolutionary biologyTasteMeta-analysisImmune-inflammatory responseGeriatrics and GerontologyBitter taste receptors030217 neurology & neurosurgery
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Immunoregulatory role of Jα281 T cells in aged mice developing lupus-like nephritis

2007

Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by the emergence of autoreactive T cells. Humans and mice with SLE have reduced numbers of CD1d-restricted invariant natural killer T (iNKT) cells, suggesting a key role for these cells in its immunopathogenesis. This subset uses an invariant TCR constituted by Valpha14 Jalpha281 chains paired with some Vbeta domains. The regulatory role for iNKT cells in non-autoimmune mice was suggested by our previous results showing that aged Jalpha281 knockout (KO) mice produce anti-dsDNA. Here we show that old Jalpha281 KO mice have proteinuria and antibodies against dsDNA and cardiolipin. Histological analysis of Jalpha281 KO m…

AgingImmunologyReceptors Antigen T-CellEnzyme-Linked Immunosorbent AssayLymphocyte Activationmedicine.disease_causeAutoimmunity Knockout NKT cellsAutoimmunityMicemedicineAnimalsLupus Erythematosus SystemicImmunology and AllergyAutoantibodiesMice KnockoutSettore MED/04 - Patologia GeneraleB-LymphocytesSystemic lupus erythematosusbiologyT-cell receptorAutoantibodyNatural killer T cellMarginal zonemedicine.diseaseImmunohistochemistryLupus NephritisKiller Cells NaturalImmunologybiology.proteinAntibodyNephritisSpleenEuropean Journal of Immunology
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