Search results for " signaling."

showing 10 items of 1032 documents

Biphasic Erk1/2 activation sequentially involving Gs and Gi signaling is required in beta3-adrenergic receptor-induced primary smooth muscle cell pro…

2013

Abstract The beta3 adrenergic receptor (B3-AR) reportedly induces cell proliferation, but the signaling pathways that were proposed, involving either Gs or Gi coupling, remain controversial. To further investigate the role of G protein coupling in B3-AR induced proliferation, we stimulated primary human myometrial smooth muscle cells with SAR150640 (B3-AR agonist) in the absence or presence of variable G-protein inhibitors. Specific B3-AR stimulation led to an Erk1/2 induced proliferation. We observed that the proliferative effects of B3-AR require two Erk1/2 activation peaks (the first after 3 min, the second at 8 h). Erk1/2 activation at 3 min was mimicked by forskolin (adenylyl-cyclase a…

Beta-3 adrenergic receptorGs alpha subunitMAP Kinase Signaling SystemMyocytes Smooth MuscleProliferationG protein coupled receptorBiologyGTP-Binding Protein alpha Subunits Gi-GoPertussis toxinchemistry.chemical_compoundErk1/2Protein kinasesCyclinsReceptors Adrenergic betaGTP-Binding Protein alpha Subunits GsHumansMolecular BiologyPI3K/AKT/mTOR pathwayCells CulturedG protein-coupled receptorCell ProliferationForskolinColforsinBeta-3 adrenergic receptorCell BiologyCell biologychemistryGene Expression RegulationPertussis ToxinMyometriumFemaleSignal transductionProto-oncogene tyrosine-protein kinase SrcBiochimica et Biophysica Acta (BBA) - Molecular Cell Research
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Wnt activity defines colon cancer stem cells and is regulated by the microenvironment.

2010

Despite the presence of mutations in APC or beta-catenin, which are believed to activate the Wnt signalling cascade constitutively, most colorectal cancers show cellular heterogeneity when beta-catenin localization is analysed, indicating a more complex regulation of Wnt signalling. We explored this heterogeneity with a Wnt reporter construct and observed that high Wnt activity functionally designates the colon cancer stem cell (CSC) population. In adenocarcinomas, high activity of the Wnt pathway is observed preferentially in tumour cells located close to stromal myofibroblasts, indicating that Wnt activity and cancer stemness may be regulated by extrinsic cues. In agreement with this noti…

Beta-cateninColorectal cancerTransplantation HeterologousMice NudeBiologyMiceCancer stem cellParacrine CommunicationmedicineAnimalsHumansAPC microenvironmentbeta CateninHepatocyte Growth FactorWnt signaling pathwayLRP6LRP5Cell BiologyNeoplasms ExperimentalFibroblastsmedicine.diseaseCoculture TechniquesCell biologyNeoplasm ProteinsWnt ProteinsColonic Neoplasmsbiology.proteinNeoplastic Stem CellsHepatocyte growth factorStem cellmedicine.drugSignal Transduction
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Convergence of Wnt signaling on the HNF4alpha-driven transcription in controlling liver zonation.

2009

Background & Aims: In each hepatocyte, the specific repertoire of gene expression is influenced by its exact location along the portocentrovenular axis of the hepatic lobule and provides a reason for the liver functions compartmentalization defined "metabolic zonation." So far, few molecular players controlling genetic programs of periportal (PP) and perivenular (PV) hepatocytes have been identified; the elucidation of zonation mechanisms remains a challenge for experimental hepatology. Recently, a key role in induction and maintenance of the hepatocyte heterogeneity has been ascribed to Wnt/β-catenin pathway. We sought to clarify how this wide-ranging stimulus integrates with hepatocyte s…

Beta-cateninWnt ProteinCellular differentiationBlotting WesternLiver Stem CellFluorescent Antibody TechniqueMice TransgenicBiologyTransfectionSensitivity and SpecificityAnimals; Blotting Western; Cell Differentiation; Cell Proliferation; Cells Cultured; Fluorescent Antibody Technique; Hepatocyte Nuclear Factor 4; Hepatocytes; Humans; Immunoprecipitation; Mice; Mice Knockout; Mice Transgenic; Reverse Transcriptase Polymerase Chain Reaction; Sensitivity and Specificity; Signal Transduction; Transfection; Wnt Proteins; beta Catenin; GastroenterologyMiceliver zonation; wnt signalling; beta catenin; hnf4Gene expressionmedicineAnimalsHumansImmunoprecipitationHepatocyteCells Culturedbeta CateninCell ProliferationMice KnockoutHepatologyAnimalReverse Transcriptase Polymerase Chain ReactionGastroenterologyWnt signaling pathwayCell DifferentiationMolecular biologyWnt Proteinsmedicine.anatomical_structureHepatocyte nuclear factor 4Hepatocyte Nuclear Factor 4Hepatocytebiology.proteinHepatocytesChromatin immunoprecipitationHumanSignal TransductionGastroenterology
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β-Catenin Activation Regulates Tissue Growth Non–Cell Autonomously in the Hair Stem Cell Niche

2014

Coordinated Hair Growth Wnt/β-catenin signaling is a key pathway that plays a conserved role in regulating stem cell function during adult tissue regeneration. Using time-lapse imaging of live mice, Deschene et al. (p. 1353 ) show that genetic activation of β-catenin within hair follicle stem cells generates axes of hair growth by coordinated cell divisions and cell movements, even when the normal niches—the dermal papillae—are laser-ablated. Activated β-catenin enhances Wnt ligand secretion, and these ligands can then activate Wnt signaling in adjacent cells that do not have activated β-catenin, indicating how activated stem cells could influence neighboring cells during normal growth and …

Beta-cateninWnt ProteinCellular differentiationLigandBiologyLigandsModels BiologicalArticleMiceStem CellmedicineAnimalsStem Cell NicheAnimals; Cell Differentiation; Cell Division; Hair; Hair Follicle; Ligands; Mice; Models Biological; Mutation; Stem Cell Niche; Stem Cells; Tamoxifen; Up-Regulation; Wnt Proteins; beta Catenin; Wnt Signaling Pathway; Medicine (all); MultidisciplinaryWnt Signaling Pathwaybeta CateninMultidisciplinaryintegumentary systemAnimalStem CellsMedicine (all)Regeneration (biology)Mesenchymal stem cellWnt signaling pathwayCell DifferentiationHair follicleUp-RegulationCell biologyWnt ProteinsTamoxifenmedicine.anatomical_structureCateninMutationbiology.proteinStem cellHair FollicleCell DivisionHairScience
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De novo design of protein kinase inhibitors by in silico identification of hinge region-binding fragments.

2013

Protein kinases constitute an attractive family of enzyme targets with high relevance to cell and disease biology. Small molecule inhibitors are powerful tools to dissect and elucidate the function of kinases in chemical biology research and to serve as potential starting points for drug discovery. However, the discovery and development of novel inhibitors remains challenging. Here, we describe a structure-based de novo design approach that generates novel, hinge-binding fragments that are synthetically feasible and can be elaborated to small molecule libraries. Starting from commercially available compounds, core fragments were extracted, filtered for pharmacophoric properties compatible w…

Binding SitesMolecular StructureProtein ConformationIntracellular Signaling Peptides and ProteinsArticlesProtein Serine-Threonine KinasesCrystallography X-RayMAP Kinase Kinase KinasesImmediate-Early ProteinsCSK Tyrosine-Protein KinaseMolecular Docking SimulationSmall Molecule Librariessrc-Family KinasesDrug DesignComputer SimulationProtein Kinase InhibitorsACS chemical biology
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Recent advances in smart biotechnology: Hydrogels and nanocarriers for tailored bioactive molecules depot

2017

Over the past ten years, the global biopharmaceutical market has remarkably grown, with ten over the top twenty worldwide high performance medical treatment sales being biologics. Thus, biotech R&D (research and development) sector is becoming a key leading branch, with expanding revenues. Biotechnology offers considerable advantages compared to traditional therapeutic approaches, such as reducing side effects, specific treatments, higher patient compliance and therefore more effective treatments leading to lower healthcare costs. Within this sector, smart nanotechnology and colloidal self-assembling systems represent pivotal tools able to modulate the delivery of therapeutics. A comprehens…

Bioactive molecules02 engineering and technologyHepatocellular-carcinoma cells01 natural sciencesMiceColloid and Surface ChemistryDrug Delivery SystemsCarbon nano materialNanotechnologyMolecular Targeted TherapyTransgenesRNA Small InterferingPatient complianceTransfer radical polymerizationMicro/nanocarrierMedical treatmentMicro/nanocarriersBioactive molecule deliveryHydrogelsSurfaces and Interfaces021001 nanoscience & nanotechnologyLiposomeBiopharmaceuticalOral deliverySelf-healing hydrogelsIntercellular Signaling Peptides and Proteins0210 nano-technologyAssembling peptide hydrogelsSurfaces and InterfaceNucleic-acid deliveryPlasmidsDiagnostic ImagingSolid lipid nanoparticlesNanotechnology010402 general chemistryAntibodiesSmall Molecule LibrariesCarbon nano-onionsIn-vivoAnimalsHumansPhysical and Theoretical Chemistrybusiness.industryDrug-delivery0104 chemical sciencesBiotechnologyHydrogelSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoMolecular ProbesBioactive molecule delivery; Carbon nano materials; Hydrogels; Liposomes; Micro/nanocarriers; Surfaces and Interfaces; Physical and Theoretical Chemistry; Colloid and Surface ChemistryLiposomesNonviral gene deliveryCarbon nano materialsNanoparticlesBusinessNanocarriers
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PathVisio Analysis: An Application Targeting the miRNA Network Associated with the p53 Signaling Pathway in Osteosarcoma

2021

MicroRNAs (miRNAs) are small single-stranded, non-coding RNA molecules involved in the pathogenesis and progression of cancer, including osteosarcoma. We aimed to clarify the pathways involving miRNAs using new bioinformatics tools. We applied WikiPathways and PathVisio, two open-source platforms, to analyze miRNAs in osteosarcoma using miRTar and ONCO.IO as integration tools. We found 1298 records of osteosarcoma papers associated with the word "miRNA". In osteosarcoma patients with good response to chemotherapy, miR-92a, miR- 99b, miR-193a-5p, and miR-422a expression is increased, while miR-132 is decreased. All identified miRNAs seem to be centered on the TP53 network. This is the first …

Bioinformatics Bone tumor Cancer CarcinogenesisMiRNA Oncology Osteosarcoma p53P53 Signaling PathwaymicroRNACancer researchmedicineOsteosarcomaGeneral MedicineBiologymedicine.disease
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The small heat-shock protein α B-crystallin is essential for the nuclear localization of Smad4: impact on pulmonary fibrosis

2014

Idiopathic pulmonary fibrosis (IPF) is a devastating disease characterized by the proliferation of myofibroblasts and the accumulation of extracellular matrix (ECM) in the lungs. TGF-β1 is the major profibrotic cytokine involved in IPF and is responsible for myofibroblast proliferation and differentiation and ECM synthesis. αB-crystallin is constitutively expressed in the lungs and is inducible by stress, acts as a chaperone and is known to play a role in cell cytoskeleton architecture homeostasis. The role of αB-crystallin in fibrogenesis remains unknown. The principal signalling pathway involved in this process is the Smad-dependent pathway. We demonstrate here that αB-crystallin is stron…

Biologymedicine.diseaseHedgehog signaling pathwayPathology and Forensic MedicineCell biologyExtracellular matrixIdiopathic pulmonary fibrosisFibrosisHeat shock proteinPulmonary fibrosisImmunologymedicinesense organsNuclear export signalMyofibroblastThe Journal of Pathology
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Gene therapy with growth factors for periodontal tissue engineering : a review

2011

The treatment of oral and periodontal diseases and associated anomalies accounts for a significant proportion of the healthcare burden, with the manifestations of these conditions being functionally and psychologically debilitating. A challenge faced by periodontal therapy is the predictable regeneration of periodontal tissues lost as a consequence of disease. Growth factors are critical to the development, maturation, maintenance and repair of oral tissues as they establish an extra-cellular environment that is conducive to cell and tissue growth. Tissue engineering principles aim to exploit these properties in the development of biomimetic materials that can provide an appropriate microen…

Biomimetic materialsPeriodontal tissuebusiness.industryGrowth factormedicine.medical_treatmentGenetic enhancementRegeneration (biology)Review-ArticleDentistryContext (language use)Genetic TherapyDiseaseBioinformatics:CIENCIAS MÉDICAS [UNESCO]OtorhinolaryngologyTissue engineeringUNESCO::CIENCIAS MÉDICASBiomaterials and Bioengineering in DentistryGuided Tissue Regeneration PeriodontalmedicineHumansIntercellular Signaling Peptides and ProteinsSurgerybusinessGeneral Dentistry
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Role of calcium in E-selectin induced phenotype of T84 colon carcinoma cells

2003

The adhesion of cancer cells to the endothelium during the metastatic process involves the interaction of specific cell-cell adhesion receptors on the cell surface. E-selectin on endothelial cells and sialyl Lewis X carbohydrate component on tumor cells are mainly implicated in the adhesion of colon carcinoma cells to the endothelium of target organ. In this paper we show that binding of E-selectin to T84 colon tumor cells causes approximately a twofold increase in intracellular calcium concentration. In particular, using two inhibitors of receptor operated calcium channels, CAI and SK&F 96365, we present evidences that the augmentation in cytoplasmic calcium originates from ionic influx fr…

BiophysicsAntineoplastic AgentsCD38BiochemistryCalcium in biologyCell MovementE-selectinTumor Cells CulturedHumansCalcium SignalingPhosphorylationCell adhesionMolecular BiologyCalcium signalingbiologyImidazolesCell BiologyTriazolesCalcium Channel BlockersRecombinant ProteinsCell biologyPhenotypeColonic NeoplasmsCancer cellbiology.proteinTyrosineCalciumNeural cell adhesion moleculeSignal transductionE-SelectinBiochemical and Biophysical Research Communications
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