Search results for "030215 immunology"

showing 10 items of 670 documents

T cells mediate autoantibody-induced cutaneous inflammation and blistering in epidermolysis bullosa acquisita

2016

AbstractT cells are key players in autoimmune diseases by supporting the production of autoantibodies. However, their contribution to the effector phase of antibody-mediated autoimmune dermatoses, i.e., tissue injury and inflammation of the skin, has not been investigated. In this paper, we demonstrate that T cells amplify the development of autoantibody-induced tissue injury in a prototypical, organ-specific autoimmune disease, namely epidermolysis bullosa acquisita (EBA) – characterized and caused by autoantibodies targeting type VII collagen. Specifically, we show that immune complex (IC)-induced inflammation depends on the presence of T cells – a process facilitated by T cell receptor (…

0301 basic medicineEpidermolysis bullosa acquisitamedicine.medical_specialtyCollagen Type VIINeutrophilsT-LymphocytesGene ExpressionMice NudeInflammationAntigen-Antibody ComplexCell CommunicationEpidermolysis Bullosa AcquisitaArticleMice03 medical and health sciencesCricetulus0302 clinical medicinemedicineAnimalsHumansAutoantibodiesSkinAutoimmune diseaseMice Inbred BALB CMultidisciplinarybusiness.industryT-cell receptorAutoantibodyAntibodies MonoclonalReceptors Antigen T-Cell gamma-deltamedicine.diseaseNatural killer T cellDermatologyImmune complexMice Inbred C57BLDisease Models Animal030104 developmental biologyLymphatic systemImmunoglobulin GImmunologyNatural Killer T-CellsLymph NodesRabbitsmedicine.symptombusinessSpleenSignal Transduction030215 immunologyScientific Reports
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Microevolutionary response of a gut nematode to intestinal inflammation.

2017

7 pages; International audience; Parasitic helminths interfere with the immune response of their hosts to establish long-lasting, chronic infections. While favorable to the parasite, the capacity to dampen the immune response can also provide a benefit to the host in terms of reduced risk of immune disorders and immunopathology. The immunomodulatory role of nematodes has been exploited in clinical trials to treat a number of inflammatory and immune diseases. However, how parasites adapt to an inflammatory environment remains a poorly explored question. Here, we conducted a serial passage experiment where the gut nematode Heligmosomoides polygyrus was maintained for nine generations in mice …

0301 basic medicineGastrointestinal DiseasesInflammationHost-Parasite InteractionsRodent Diseases03 medical and health sciencesMice0302 clinical medicineImmune systemSerial passageImmunopathology[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/Symbiosismedicine[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyAnimalsAdaptationStrongylida InfectionsInflammationExperimental evolutionNematospiroides dubiusbiologyHost (biology)Life history traitsbiology.organism_classificationColitisBiological Evolution3. Good health030104 developmental biologyInfectious DiseasesNematodeExperimental evolutionImmunologybacteria[SDV.IMM]Life Sciences [q-bio]/ImmunologyParasitologyHeligmosomoides polygyrusmedicine.symptomSerial passage030215 immunology[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/Symbiosis
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Gradual development of psoriatic skin lesions by constitutive low-level expression of IL-17A

2016

Psoriasis is a common chronic inflammatory skin disease restricted to humans. The understanding of its pathogenesis has long been hampered by the lack of suitable chronic mouse models. The cytokine IL-17A has emerged as a key player in epithelial immune responses and the defense against extracellular pathogens. Moreover, enhanced expression of IL-17A can turn pathologic and is closely associated with psoriasis. In this study, we generated a novel transgenic mouse model that recapitulates many characteristics of psoriasis. DC-1L-17A(ind) mice with constitutive low-level expression of IL-17A by CD11c(+) cells gradually develop skin lesions during adult life. The lesions preferentially occur a…

0301 basic medicineGenetically modified mousemedicine.medical_treatmentImmunologyCD11cInflammationAnimals Genetically ModifiedPathogenesisMice03 medical and health sciences0302 clinical medicineImmune systemSDG 3 - Good Health and Well-beingPsoriasismedicineAnimalsHumansPsoriasisCells CulturedSkinbusiness.industryInterleukin-17Dendritic Cellsmedicine.diseaseCD11c AntigenDisease Models Animal030104 developmental biologyCytokineImmunologyCytokinesInterleukin 17medicine.symptombusiness030215 immunologyCellular Immunology
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Specificity of human natural antibodies referred to as anti-Tn

2020

International audience; To understand the role of human natural IgM known as antibodies against the carbohydrate epitope Tn, the antibodies were isolated using GalNAcα−Sepharose affinity chromatography, and their specificity was profiled using microarrays (a glycan array printed with oligosaccharides and bacterial polysaccharides, as well as a glycopeptide array), flow cytometry, and inhibition ELISA. The antibodies bound a restricted number of GalNAcα-terminated oligosaccharides better than the parent monosaccharide, e.g., 6-O-Su-GalNAcα and GalNAcα1−3Galβ1−3(4)GlcNAcβ. The binding with several bacterial polysaccharides that have no structural resemblance to the affinity ligand GalNAcα was…

0301 basic medicineGlycanGlycansImmunologyTn antigenAntibody Affinity[SDV.CAN]Life Sciences [q-bio]/CancerAnti-Glycan antibodiesEpitopeAntigen-Antibody ReactionsEpitopesJurkat Cells03 medical and health sciences0302 clinical medicineAffinity chromatography[SDV.CAN] Life Sciences [q-bio]/CancerAntibody SpecificityNeoplasmsTn antigenHumansAntigens Tumor-Associated CarbohydrateAmino Acid SequenceMolecular BiologyPeptide sequenceCancerbiologyChemistryBacterial polysaccharideImmunity Innate3. Good health030104 developmental biologyCarbohydrate SequenceImmunoglobulin MBiochemistryNatural antibodiesbiology.proteinParatopeAntibody030215 immunology
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Polyglobulie rare par mutation du gène EGLN1 : à propos d’un cas et revue de la littérature

2020

Resume Introduction Le diagnostic etiologique d’une polyglobulie est souvent simple, l’origine etant le plus souvent acquise. Parfois il faut rechercher des formes rares, hereditaires, l’algorithme decisionnel reposant sur le dosage de l’erythropoietine serique et sur la mesure de la pression en oxygene saturant l’hemoglobine a 50 % (P50). Ces pathologies sont en rapport avec des anomalies du recepteur a l’erythropoietine, des hemoglobines hyperaffines ou a des dysfonctionnements de la voie de regulation du facteur HIF (Hypoxia-Inducible Factor) implique dans la sensibilite renale a l’hypoxie. Observation Nous rapportons le cas d’un patient de 56 ans qui presente une polyglobulie inexplique…

0301 basic medicineGynecology03 medical and health sciencesmedicine.medical_specialty030104 developmental biology0302 clinical medicinebusiness.industryGastroenterologyInternal MedicineMedicineHereditary Erythrocytosisbusiness030215 immunologyLa Revue de Médecine Interne
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MPLA-coated hepatitis B virus surface antigen (HBsAg) nanocapsules induce vigorous T cell responses in cord blood derived human T cells.

2016

Chronic hepatitis B virus (HBV) infection is the most prevalent serious liver infection in the world. A frequent route of infection represents mother-to-child transmission. Efficient control of HBV replication depends on antigen-specific cellular immune response mediated by dendritic cells (DCs). Aim of the present study was to evaluate optimized adjuvant combinations, efficiently maturing monocyte-derived neonatal and adult dendritic cells (moDCs). In addition, the potential of polymeric HBsAg-nanocapsules (HBsAg-NCs) was investigated regarding up-take by moDCs and the subsequent induction of specific T cell responses in a human co-culture model. Simultaneous stimulation of moDCs with MPLA…

0301 basic medicineHBsAgHepatitis B virusT cellT-LymphocytesBiomedical EngineeringPharmaceutical ScienceMedicine (miscellaneous)Bioengineeringmedicine.disease_causeVirus03 medical and health sciences0302 clinical medicineImmune systemNanocapsulesmedicineHumansGeneral Materials ScienceHepatitis B VaccinesHepatitis B virusLiver infectionHepatitis B Surface Antigensbusiness.industryDendritic CellsHepatitis Bmedicine.diseaseFetal BloodHepatitis BVirology030104 developmental biologymedicine.anatomical_structureImmunologyAntigens SurfaceMolecular MedicinebusinessCD80030215 immunologyNanomedicine : nanotechnology, biology, and medicine
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Th2 and Th1 Responses: Clear and Hidden Sides of Immunity Against Intestinal Helminths.

2017

Intestinal helminthiases affect millions of people worldwide, mainly in developing regions, where they cause a significant negative impact on human health and socioeconomic growth of affected populations. However, intestinal helminthiases are still among the most neglected tropical diseases. Protective immunity against intestinal helminths is associated with development of type 2 responses. Nevertheless, in some host–intestinal helminth combinations, local Th1 responses are initiated, inducing chronicity. The usage of helminth–mouse models is useful for elucidating the mechanisms behind the initiation of each type of response. Herein, the current knowledge on these topics is reviewed, payin…

0301 basic medicineHelminthiasisHelminthiasisBiologyHost-Parasite Interactions03 medical and health sciencesHuman health0302 clinical medicineImmune systemTh2 CellsImmunityHelminthsmedicineHelminthsAnimalsHumansDeveloping regionsIntestinal Diseases ParasiticTh1 Cellsmedicine.diseaseImmunity Humoral030104 developmental biologyInfectious DiseasesImmunologyNeglected tropical diseasesParasitologyTh1 response030215 immunologyTrends in parasitology
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The immunoglobulin γ marker 17 allotype and KIR/HLA genes prevent the development of chronic hepatitis B in humans

2020

Hepatitis B virus (HBV) infection causes a self-limiting disease in most individuals. However, < 10% of infected subjects develop a chronic disease. Genetic host variability of polymorphic genes at the interface of innate and acquired immunity, such as killer immunoglobulin-like receptors (KIR), their human leucocyte antigen (HLA) and IgG allotypes (GM), could explain this different clinical picture. We previously showed a protective role of the KIR2DL3 gene for the development of chronic hepatitis B (CHB), and a detrimental role of the KIR ligand groups, HLA-A-Bw4 and HLA-C2. We have expanded the previous analysis genotyping patients for GM23 and GM3/17 allotypes. The comparison of the …

0301 basic medicineHepatitis B virusKIR LigandImmunologyhepatitis B viruHuman leukocyte antigenHLA-C Antigensmedicine.disease_causeRisk Assessment03 medical and health sciences0302 clinical medicineHepatitis B ChronicGene FrequencyImmunoglobulin Gm AllotypesRisk Factorskiller immunoglobulin-like receptorImmunology and AllergyMedicineHumansGenetic Predisposition to DiseaseGenotypingHepatitis B virusSettore MED/04 - Patologia Generalebiologybusiness.industryOriginal ArticlesProtective FactorsAcquired immune systemAllotypeγ marker030104 developmental biologyPhenotypeHLA-B AntigensReceptors KIR2DL3Case-Control StudiesImmunologyHost-Pathogen Interactionsbiology.proteinGene polymorphismAntibodyhepatitis B virus; human leucocyte antigen; killer immunoglobulin-like receptor; ? markerbusiness030215 immunologyhuman leucocyte antigen
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Overview of the interaction of helminth extracellular vesicles with the host and their potential functions and biological applications.

2021

Helminth Extracellular Vesicles (EVs) have emerged as important mediators in host-parasite communications, participating in the parasite survival and its pathogenic effects. In the last decade, a growing amount of information reporting the isolation and characterization of EVs from different helminth species has appeared, but unfortunately, few reports have focused on functional studies of helminth EVs in different cell lines, organoids or animal models. We here review these in vitro and in vivo studies, which clearly demonstrate that helminths secrete EVs, which affect their environment. Helminth EVs are actively internalized by different cell lines, modulating cellular functions important…

0301 basic medicineHost (biology)ImmunologyCellular functionsHelminthiasisComputational biologyBiologyExtracellular vesiclesHost-Parasite Interactions03 medical and health sciencesExtracellular Vesicles030104 developmental biology0302 clinical medicineImmune systemHelminthsparasitic diseasesHelminthsAnimalsHumansSecretionFunctional studiesInformation reportingMolecular Biology030215 immunologyMolecular immunology
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Role of Immunogenetics in the Outcome of HCMV Infection: Implications for Ageing

2019

The outcome of host-virus interactions is determined by a number of factors, some related to the virus, others to the host, such as environmental factors and genetic factors. Therefore, different individuals vary in their relative susceptibility to infections. Human cytomegalovirus (HCMV) is an important pathogen from a clinical point of view, as it causes significant morbidity and mortality in immunosuppressed or immunosenescent individuals, such as the transplanted patients and the elderly, respectively. It is, therefore, important to understand the mechanisms of virus infection control. In this review, we discuss recent advances in the immunobiology of HCMV-host interactions, with partic…

0301 basic medicineHuman cytomegalovirusAgingCellular immunityvirusesCytomegalovirusReviewlcsh:Chemistry0302 clinical medicineHLA AntigensGenotypeMedicineantibodieslcsh:QH301-705.5SpectroscopyimmunosenescenceImmunity CellularbiologyGeneral MedicineImmunosenescenceGMComputer Science ApplicationsKIRHLAantibodieCytomegalovirus InfectionsHost-Pathogen InteractionsAntibodyGenotypeNKCongenital cytomegalovirus infectionHuman leukocyte antigenelderlyCatalysisVirusInorganic Chemistry03 medical and health sciencesImmunogeneticsAnimalsHumansGenetic Predisposition to DiseasePhysical and Theoretical ChemistryMolecular BiologyHCMVSettore MED/04 - Patologia Generalebusiness.industryOrganic Chemistrymedicine.diseaseImmunity Humoral030104 developmental biologylcsh:Biology (General)lcsh:QD1-999Immunologybiology.proteinbusiness030215 immunology
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