Search results for "1311"

showing 10 items of 39 documents

Interest of genotyping and phenotyping of drug-metabolizing enzymes for the interpretation of biological monitoring of exposure to styrene

2002

In the field of occupational and/or environmental toxicology, the measurement of specific metabolites in urine may serve to assess exposure to the parent compounds (biological monitoring of exposure). Styrene is one of the chemicals for which biological monitoring programs have been validated and implemented in environmental and occupational medicine. However, inter-individual differences in the urinary excretion exist both for the main end-products (mandelic acid and phenylglyoxylic acid) and for its specific mercapturic acids (phenylhydroxyethylmercapturic acids, PHEMA). This limits to a certain extent the use of these metabolites for an accurate assessment of styrene exposure. In a group…

AdultMalePhenylglyoxylic acidGenotypeMetaboliteUrinary systemPopulation10050 Institute of Pharmacology and Toxicology610 Medicine & healthUrinePharmacologyBiologyPolymerase Chain Reaction3000 General Pharmacology Toxicology and PharmaceuticsExcretionchemistry.chemical_compound1311 GeneticsGeneticsHumansLymphocytesGeneral Pharmacology Toxicology and PharmaceuticseducationGenotypingStyreneGlutathione TransferaseEpoxide Hydrolaseseducation.field_of_studyPolymorphism GeneticGlyoxylatesCytochrome P-450 CYP2E1Environmental ExposureCYP2E1AcetylcysteineIsoenzymesPhenotypeGlutathione S-Transferase piBiochemistrychemistry570 Life sciences; biologyMandelic AcidsBiomarkersPolymorphism Restriction Fragment LengthEnvironmental Monitoring
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Variable pulmonary manifestations in Chitayat syndrome: Six additional affected individuals

2020

Hand hyperphalangism leading to shortened index fingers with ulnar deviation, hallux valgus, mild facial dysmorphism and respiratory compromise requiring assisted ventilation are the key features of Chitayat syndrome. This condition results from the recurrent heterozygous missense variant NM_006494.2:c.266A>G; p.(Tyr89Cys) in ERF on chromosome 19q13.2, encoding the ETS2 repressor factor (ERF) protein. The pathomechanism of Chitayat syndrome is unknown. To date, seven individuals with Chitayat syndrome and the recurrent pathogenic ERF variant have been reported in the literature. Here, we describe six additional individuals, among them only one presenting with a history of assisted ventil…

Adultbronchomalacia2716 Genetics (clinical)hyperphalangismPediatricsmedicine.medical_specialtyAdolescent10039 Institute of Medical Genetics610610 Medicine & healthChitayat syndromeFingersYoung Adult03 medical and health sciences1311 Geneticsrespiratory distressExome SequencingGeneticsmedicineHumansMissense mutationGenetic Predisposition to DiseaseHallux ValgusRespiratory systemChildGenetics (clinical)030304 developmental biologyCHITAYAT SYNDROME0303 health sciencesPierre Robin SyndromebiologyRespiratory distressbusiness.industry030305 genetics & heredityFaciesmedicine.diseasebiology.organism_classificationPhenotype3. Good healthRepressor ProteinsValgusERFChild Preschoolulnar deviation570 Life sciences; biologyFemaleUlnar deviationBronchomalaciabusinessAmerican Journal of Medical Genetics Part A
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Recommendations on the diagnosis and management of Niemann-Pick disease type C

2009

Niemann-Pick disease type C (NP-C) is a lysosomal storage disease in which impaired intracellular lipid trafficking leads to excess storage of cholesterol and glycosphingolipids in the brain and other tissues. it is characterized clinically by a variety of progressive, disabling neurological symptoms including clumsiness, limb and gait ataxia, dysarthria, dysphagia and cognitive deterioration (dementia). Until recently, there has been no disease-modifying therapy available for NP-C, with treatment limited to supportive measures. In most countries, NP-C is managed through specialist centers, with non-specialist support provided locally. However, effective patient Support is hampered by the a…

Adultmedicine.medical_specialtyNeurology1303 BiochemistryAdolescentEndocrinology Diabetes and Metabolism610 Medicine & healthDiseaseBiochemistry03 medical and health sciencesDysarthriaYoung Adult0302 clinical medicineEndocrinology1311 GeneticsGeneticsLysosomal storage diseasemedicine1312 Molecular BiologyDementiaHumansMass ScreeningIntensive care medicineChildMolecular BiologyMass screening030304 developmental biology0303 health sciencesNiemann–Pick disease type Cbusiness.industryInfant NewbornInfantNiemann-Pick Disease Type CMiddle Agedmedicine.disease3. Good health1310 Endocrinology2712 Endocrinology Diabetes and Metabolism10036 Medical ClinicChild PreschoolPhysical therapyGait Ataxiamedicine.symptombusiness030217 neurology & neurosurgery
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Genetic analysis of dyslexia candidate genes in the European cross-linguistic NeuroDys cohort

2013

The work conducted at the WTCHG was supported by Wellcome Trust grants [076566/Z/05/Z] and [075491/Z/04]; the work in Zurich partly by an SNSF grant [32-108130]. We also thank MAF (Mutation Analysis core Facility) at the Karolinska Institute, Novum, Huddinge. The French part of the project was funded by Agence Nationale de la Recherche (ANR-06-NEURO-019-01 GENEDYS) and Ville de Paris. S Paracchini is a Royal Society University Research Fellow. D Czamara was supported by the Deutsche Forschungsgemeinschaft (German Research Foundation) within the framework of the Munich Cluster for Systems Neurology (EXC 1010 SyNergy). Dyslexia is one of the most common childhood disorders with a prevalence o…

Candidate geneDyslexia10064 Neuroscience Center Zurich10. No inequalityGenetics (clinical)ta515Geneticseducation.field_of_study10093 Institute of PsychologyR10058 Department of Child and Adolescent Psychiatry3. Good healthAssociation studyPhenotype10076 Center for Integrative Human PhysiologyWord-reading[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Reading disability2716 Genetics (clinical)GenotypePopulationLocus (genetics)610 Medicine & healthSpellingQH426 GeneticsBDYBiologyR Medicineta3111Polymorphism Single NucleotideArticleCandidate genesQuantitative Trait HeritableMeta-Analysis as Topic1311 GeneticsDCDC2mental disordersGeneticsmedicineHumanseducationQH426Genetic Association StudiesGenetic associationHaplotypeDyslexiamedicine.diseaseHaplotypesGenetic LociCase-Control Studies570 Life sciences; biology150 PsychologyGenome-Wide Association Study
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Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci

2010

We undertook a meta-analysis of six Crohn's disease genome-wide association studies (GWAS) comprising 6,333 affected individuals (cases) and 15,056 controls and followed up the top association signals in 15,694 cases, 14,026 controls and 414 parent-offspring trios. We identified 30 new susceptibility loci meeting genome-wide significance (P < 5 x 10(-8)). A series of in silico analyses highlighted particular genes within these loci and, together with manual curation, implicated functionally interesting candidate genes including SMAD3, ERAP2, IL10, IL2RA, TYK2, FUT2, DNMT3A, DENND1B, BACH2 and TAGAP. Combined with previously confirmed loci, these results identify 71 distinct loci with gen…

Candidate geneGenetic LinkagePROTEINGenome-wide association studyInflammatory bowel diseaseGenomeACTIVATION0302 clinical medicineCrohn DiseaseSEQUENCE VARIANTSGenetics0303 health sciencesGenomeNEDD4 FAMILYCOMMON VARIANTSASSOCIATION3. Good health030220 oncology & carcinogenesis10076 Center for Integrative Human PhysiologyComputational Biology; Crohn Disease; Genetic Linkage; Genetic Loci; Genetic Variation; Genome Human; Humans; Reproducibility of Results; Genetic Predisposition to Disease; Genome-Wide Association Study; Geneticsinflammatory-bowel-disease sequence variants common variants nedd4 family association gene identification receptor protein activationHuman/dk/atira/pure/subjectarea/asjc/1300/1311Locus (genetics)610 Medicine & healthBiology03 medical and health sciences1311 GeneticsGenetic linkagemedicineGeneticsHumansGenetic Predisposition to Disease030304 developmental biologyGenetic associationIDENTIFICATIONRECEPTORComputational BiologyGenetic VariationReproducibility of Resultsmedicine.diseaseGENESettore MED/03 - Genetica Medica10199 Clinic for Clinical Pharmacology and ToxicologyGenetic Loci570 Life sciences; biologyHuman genomegenome-wide scan.meta-analysis.crohn's diseaseGenome-Wide Association StudyINFLAMMATORY-BOWEL-DISEASE
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The Study of Carbamoyl Phosphate Synthetase 1 Deficiency Sheds Light on the Mechanism for Switching On/Off the Urea Cycle

2015

12 páginas, 4 figuras, 2 tablas.

Conformational changeCarbamoyl-Phosphate Synthase I Deficiency DiseaseAllosteric regulationCarbamoyl-Phosphate Synthase (Ammonia)Urea cycle diseases610 Medicine & healthBiologyMolecular Dynamics Simulationurologic and male genital diseases03 medical and health sciences0302 clinical medicineGlutamates1311 GeneticsAmmoniaEnzyme StabilityGeneticsmedicine1312 Molecular BiologyHumansUreaHyperammonemiaSite-directed mutagenesisMolecular Biology030304 developmental biologychemistry.chemical_classification0303 health sciencesSite-directed mutagenesisurogenital systemMutagenesisCarbamoyl phosphate synthetase 1HyperammonemiaCarbamoyl phosphate synthetasemedicine.diseaseAllosteric regulation3. Good healthProtein Structure TertiaryRestrained molecular dynamicsKineticsEnzymeBiochemistrychemistry10036 Medical ClinicEnzymeUrea cycleMutationInborn errors030217 neurology & neurosurgerySignal Transduction
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What is the impact of rerouting a cancer diagnosis from emergency presentation to GP referral on resource use and survival? Evidence from a populatio…

2018

Background Studies on alternative routes to diagnosis stimulated successful policy interventions reducing the number of emergency diagnoses and associated mortality risk. A dearth of evidence on the costs of such interventions might prevent new policies from achieving more ambitious targets. Methods We conducted a retrospective cohort study on the population of colorectal (88,051), breast (90,387), prostate (96,219), and lung (97,696) cancer patients diagnosed after a GP referral or an emergency presentation and reported in the Cancer Registry of England. Resource use and survival were compared 1 year before and 5 years after diagnosis (3 years for lung), including the costs of GP referrals…

Emergency Medical ServicesCancer ResearchSurvival0302 clinical medicineNeoplasmsEmergency medical services1306030212 general & internal medicineMedical diagnosisReferral and Consultationeducation.field_of_study1311Health Care CostsEarly diagnosilcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensPrognosisEarly diagnosisPrimary careRoute to diagnosisRoute to diagnosiOncologyPopulation Surveillance030220 oncology & carcinogenesisHealth ResourcesHospital costResearch Articlemedicine.medical_specialty2730ReferralEarly diagnosis; Emergency; Hospital costs; Primary care; Route to diagnosis; Survival; Oncology; Genetics; Cancer ResearchPopulationlcsh:RC254-28203 medical and health sciencesGeneticGeneral PractitionersGeneticsmedicineHumanseducationLung cancerbusiness.industryCancerRetrospective cohort studymedicine.diseaseCancer registryEmergency medicineEmergencyHospital costsbusinessNeoplasms/diagnosis
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Detection of primary DNA damage: applicability to biomonitoring of genotoxic occupational exposure and in clinical therapy

1995

The biological effect of putative genotoxic chemicals in the work place environment was monitored in peripheral mononuclear blood cells of exposed workers. DNA strand breaks, alkali-labile sites of DNA and DNA cross-links were measured using the alkaline filter elution method. A dose dependent increase in DNA damage was found in sterilization workers exposed to ethylene oxide and metal workers with exposure towards N-nitrosodiethanolamine. Two subpopulations with different response to the external exposure were found in nonsmoking sterilization workers. Nurses handling antineo-plastic agents without adequate safety provisions showed a statistically significantly higher rate of DNA strand br…

Ethylene OxideMaleDNA damagemedicine.medical_treatmentNurses10050 Institute of Pharmacology and ToxicologyAntineoplastic Agents610 Medicine & healthPharmacologyDNA Strand Break3000 General Pharmacology Toxicology and PharmaceuticsCell Linechemistry.chemical_compound1311 GeneticsOccupational ExposureBiomonitoringGeneticsmedicineCarcinomaAnimalsHumansDiethylnitrosamineGeneral Pharmacology Toxicology and PharmaceuticsOvarian NeoplasmsChemotherapybusiness.industrySterilizationDNASterilization (microbiology)medicine.diseaseHodgkin DiseasechemistryCarcinogens570 Life sciences; biologyFemaleOccupational exposurebusinessDNADNA DamageEnvironmental Monitoring
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Mutations in SLC20A2 link familial idiopathic basal ganglia calcification with phosphate homeostasis.

2012

Familial idiopathic basal ganglia calcification (IBGC) is a genetic condition with a wide spectrum of neuropsychiatric symptoms, including parkinsonism and dementia. Here, we identified mutations in SLC20A2, encoding the type III sodium-dependent phosphate transporter 2 (PiT2), in IBGC-affected families of varied ancestry, and we observed significantly impaired phosphate transport activity for all assayed PiT2 mutants in Xenopus laevis oocytes. Our results implicate altered phosphate homeostasis in the etiology of IBGC.

Genetic Markersmedicine.medical_specialtyGenetic LinkageMolecular Sequence DataMutation MissenseXenopusBasal ganglia calcification610 Medicine & healthPhosphates10052 Institute of PhysiologyXenopus laevis03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAsian PeopleBasal Ganglia Diseases1311 GeneticsCalcinosisGenetic linkageInternal medicineGeneticsmedicineAnimalsHomeostasisHumansBasal ganglia disease030304 developmental biology0303 health sciencesBase SequencebiologySodium-Phosphate Cotransporter Proteins Type IIIParkinsonismCalcinosisSequence Analysis DNAmedicine.diseasePhosphatebiology.organism_classificationPedigreeEndocrinologychemistry10076 Center for Integrative Human PhysiologyOocytes570 Life sciences; biologyLod Score030217 neurology & neurosurgeryHomeostasisChromosomes Human Pair 8Nature genetics
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Meta-analysis and imputation refines the association of 15q25 with smoking quantity.

2010

Smoking is a leading global cause of disease and mortality(1). We established the Oxford-GlaxoSmithKline study (Ox-GSK) to perform a genome-wide meta-analysis of SNP association with smoking-related behavioral traits. Our final data set included 41,150 individuals drawn from 20 disease, population and control cohorts. Our analysis confirmed an effect on smoking quantity at a locus on 15q25 (P = 9.45 x 10(-19)) that includes CHRNA5, CHRNA3 and CHRNB4, three genes encoding neuronal nicotinic acetylcholine receptor subunits. We used data from the 1000 Genomes project to investigate the region using imputation, which allowed for analysis of virtually all common SNPs in the region and offered a …

Genetics0303 health scienceseducation.field_of_study/dk/atira/pure/subjectarea/asjc/1300/1311PopulationSingle-nucleotide polymorphismGenome-wide association studyLocus (genetics)BiologyArticle3. Good health03 medical and health sciences0302 clinical medicineGenome-Wide Association; Nicotine Dependence; Lung-Cancer; Susceptibility Locus; Risk-Factors; Disease; Genes; SNPS; Colaus StudyGeneticsSNP1000 Genomes ProjectAlleleeducation030217 neurology & neurosurgeryImputation (genetics)genome-wide association study; smoking initiation; smoking quantity030304 developmental biologyNature genetics
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