Search results for "34"

showing 10 items of 1830 documents

Research of cardiomyocyte precursors in adult rat heart

2006

Recent reports supported the existence of stem cells in adult hearts. However, phenotype and localization of these cells have not been completely described and it is unknown if cardiac regenerative potential differs from one subject to another. The aims of our work were to identify different populations of cardiac stem cells by the analysis of specific markers and to evaluate the expression variability of these markers in 12 adult rat hearts. The expression of CD9, taube nuss and nanog suggests the presence of stem cells from the earliest stages of embryogenesis in adult myocardium. Their different expression could be associated to the degree of stem cell differentiation. CD34 and c-Kit ant…

MalePathologymedicine.medical_specialtyCellular differentiationAntigens CD34Nerve Tissue ProteinsBiologyNestinStem cells heart expression rat.Intermediate Filament ProteinsmedicineAnimalsCell LineageMyocytes CardiacAntigensRats WistarStem cell transplantation for articular cartilage repairInduced stem cellsMyocardiumStem CellsEndothelial CellsCell DifferentiationAmniotic stem cellsCell BiologyGeneral MedicineGATA4 Transcription FactorRatsEndothelial stem cellProto-Oncogene Proteins c-kitAmniotic epithelial cellsStem cellDevelopmental BiologyAdult stem cellTissue and Cell
researchProduct

The correlation between the severity of radiotherapy-induced glossitis and endothelial cell injury in local tissues in a rat model.

2010

Objectives: To explore the correlation between the severity of radiotherapy-induced glossitis (RTG) and endothelial cell injury in local tissues in a rat model. Study Design: The RTG animal model was designed and used by our team. The Oral mucositis index(OMI) was documented daily. Immunohistochemistry (IHC) Staining of CD34 was utilized to identify endothelial cells in the RTG tissues. Apoptosis of endothelial cells in local lesions due to RTG was detected by the TUNEL assay. The dynamic relationship between the OMI and apoptotic endothelial cells was statistically analyzed by time. Results and Conclusions: The injury and apoptosis of endothelial cells were observed 3 day post-irradiation.…

MalePathologymedicine.medical_specialtyGlossitisCD34Severity of Illness IndexRats Sprague-DawleyGlossitisMucositisMedicineAnimalsRadiation InjuriesGeneral DentistryTUNEL assaybusiness.industryEndothelial Cellsmedicine.disease:CIENCIAS MÉDICAS [UNESCO]StainingRatsEndothelial stem cellDisease Models AnimalOtorhinolaryngologyApoptosisUNESCO::CIENCIAS MÉDICASImmunohistochemistrySurgerybusinessMedicina oral, patologia oral y cirugia bucal
researchProduct

Diagnostic value of CD34 immunostaining in desmoplastic trichilemmoma.

1998

Desmoplastic trichilemmoma (DT) is a variant of trichilemmoma, characterized by a central prominent desmoplastic component which may simulate invasive carcinoma. We have studied the morphologic and immunohistochemical features of seven cases of DT. Immunohistochemistry was performed on paraffin sections using monoclonal antibodies to CD34 (QBEND/10), vimentin and GCDFP-15. CD34 was also tested in seven cases of basal cell carcinoma (BCC), three with outer root sheath differentiation and four with morphea-form features, and five squamous cell carcinomas. Histologically, features of conventional trichilemmoma were seen at the periphery of the seven lesions. In contrast, at the center, the epi…

MalePathologymedicine.medical_specialtyHistologySkin NeoplasmsCD34VimentinAntigens CD34DermatologyBiologyOuter root sheathPathology and Forensic MedicineDiagnosis DifferentialPredictive Value of TestsmedicineCarcinomaBiomarkers TumorHumansVimentinBasal cell carcinomaApolipoproteins DAgedGlycoproteinsNeoplasms Basal CellSkinTrichilemmomaHistocytochemistryMembrane Transport ProteinsMiddle Agedmedicine.diseaseImmunohistochemistryApolipoproteinsCarcinoma Basal Cellbiology.proteinCarcinoma Squamous CellImmunohistochemistryFemaleCarrier ProteinsImmunostainingJournal of cutaneous pathology
researchProduct

Tissue evacuated during joint replacement procedure as a source of mononuclear cells.

2017

Background Different cell populations from bone marrow were used in various clinical trials for cardiac diseases during last decade. Four clinical studies are ongoing in our institution and enroll patients with cardiac diseases, coronary disease, type 2 diabetes, and osteoarthritis. The density gradient is used to separate bone marrow mononuclear cells. Joint replacement procedures were associated with significant loss of tissue. Usually, excess tissue as bone marrow, peripheral blood and fat are removed to clean operation site. The aim of this study is to prove whether removed tissue during joint replacement procedure can be considered as a significant source of mononuclear cells. Methods …

MalePathologymedicine.medical_specialtyJoint replacementmedicine.medical_treatmentArthroplasty Replacement HipPopulationCD34Antigens CD34OsteoarthritisPunctures030204 cardiovascular system & hematologyPeripheral blood mononuclear cellIliac crestTransplantation AutologousMonocytesOsteoarthritis HipIlium03 medical and health sciences0302 clinical medicinemedicineCentrifugation Density GradientHumansOrthopedics and Sports MedicineBone marrow030212 general & internal medicineExcised tissueeducationArthroplasty Replacement Kneeeducation.field_of_studybusiness.industryCell extractionJoint replacementTissue ProcessingMiddle AgedOsteoarthritis Kneemedicine.diseaseFlow CytometryCell countCell yieldmedicine.anatomical_structureMononuclear cellsCase-Control StudiesTissue and Organ HarvestingSurgeryFemaleBone marrowbusinessOriginal Article • HIP - ARTHROPLASTYEuropean journal of orthopaedic surgerytraumatology : orthopedie traumatologie
researchProduct

Collagen membranes of dermal and pericardial origin-In vivo evolvement of vascularization over time.

2020

Aim of the study was to compare the evolvement of vascularization over time of collagen membranes (CMs) of dermal and pericardial origin in an in vivo animal study. Twenty-eight mice underwent implantation of three commercially available CM derived from porcine dermis (homogenous structure: CM1 (Control 1) and bilayer structure: CM2 [Control 2]), from porcine pericardium (CM3; Test 1) as well as CM3 sprayed with silica-enhanced nanostructured hydroxyapatite (CM4, Test 2). After 3, 6, 9, and 12 days, intravital fluorescence microscopy was conducted for determination of capillary diameter, density, flow, and length. At Day 12, samples were examined immunohistologically for expression of fibro…

MalePathologymedicine.medical_specialtyMaterials scienceAngiogenesisSwine0206 medical engineeringBiomedical EngineeringCD34Neovascularization PhysiologicBiocompatible Materials02 engineering and technologyBiomaterialsMiceIn vivomedicineFluorescence microscopePericardiumAnimalsTissue ScaffoldsCD68BilayerMetals and AlloysMembranes ArtificialFibroblast growth factor receptor 4Dermis021001 nanoscience & nanotechnology020601 biomedical engineeringMice Inbred C57BLmedicine.anatomical_structureCeramics and CompositesCollagen0210 nano-technologyPericardiumJournal of biomedical materials research. Part AREFERENCES
researchProduct

Heterozygous nonsense SCN5A mutation W822X explains a simultaneous sudden infant death syndrome.

2008

The sudden, unexpected, and unexplained death of both members of a set of healthy twins (simultaneous sudden infant death syndrome (SSIDS)) is defined as a case in which both infants meet the definition of sudden infant death syndrome individually. A search of the world medical literature resulted in only 42 reported cases of SSIDS. We report the case of a pair of identical, male, monozygotic twins, 138 days old, who suddenly died, meeting the full criteria of SSIDS and where a genetic screen was performed, resulting in a heterozygous nonsense SCN5A mutation (W822X) in both twins. Immunohistochemistry was performed on cardiac tissue samples utilizing polyclonal antibodies anti-Na+ CP type V…

MalePathologymedicine.medical_specialtyNav1.5 protein functionv1.5 protein functionmedia_common.quotation_subject2734Nonsense mutationNonsenseNa+ channel functionMuscle ProteinsSocio-culturaleBiology+Nav1.5 protein function; Na+ channel function; SCN5A gene mutation; Simultaneous sudden infant death syndrome; W822X mutation; Codon Nonsense; Diseases in Twins; Humans; Infant; Male; Muscle Proteins; NAV1.5 Voltage-Gated Sodium Channel; Sodium Channels; Sudden Infant Death; 2734Sudden deathSodium ChannelsNAV1.5 Voltage-Gated Sodium ChannelPathology and Forensic MedicinePathogenesisSCN5A gene mutationDiseases in TwinsmedicineHumansSimultaneous sudden infant death syndromeSCN5A gene mutationW822X mutationNa+ channel functionNav1.5 protein functionNaSimultaneous sudden infant death syndrome SCN5A gene mutation W822X mutation Na+ channel function Nav1.5 protein function CodonMolecular BiologyCellular localizationmedia_commonSimultaneous sudden infant death syndromeSettore BIO/16 - Anatomia UmanaSimultaneous sudden infant death syndrome SCN5A gene mutation W822X mutation Na+ channel function Nav1.5 protein functionW822X mutationInfantCell BiologyGeneral MedicineSudden infant death syndromeNonsenseTerminal deoxynucleotidyl transferaseCodon NonsenseImmunohistochemistryNa; v; 1.5 protein function; Na; +; channel function; SCN5A gene mutation; Simultaneous sudden infant death syndrome; W822X mutationchannel functionSudden Infant Death
researchProduct

Liver regeneration induced by a designer human IL‐6/ sIL‐6R fusion protein reverses severe hepatocellular injury

2000

The cytokine IL-6 plays a significant role in liver regeneration in conjunction with additional growth factors (HGF, TNF-α, and TGF-α). Many IL-6 effects depend on a naturally occurring soluble IL-6 receptor (sIL-6R). Here, the chimeric protein hyper-IL-6, constructed from the human IL-6 protein fused to a truncated form of its receptor, was found to have superagonistic IL-6 properties, and as such, enhanced liver cell regeneration. Hyper-IL-6 reversed the state of hepatotoxicity and enhanced the survival rates of rats suffering from fulminant hepatic failure after D-galactosamine administration. The hyper-IL-6 protein has a significant potential for use in the treatment of severe human liv…

MalePathologymedicine.medical_specialtyRecombinant Fusion Proteinsmedicine.medical_treatmentApoptosisGalactosamineThioacetamideBiochemistryFulminant hepatic failureGeneticsmedicineAnimalsHumansReceptorInterleukin 6Molecular BiologybiologyInterleukin-6ChemistryLiver cellRegeneration (biology)Receptors InterleukinReceptors Interleukin-6Fusion proteinRats Inbred F344Liver regenerationLiver RegenerationRatsDisease Models AnimalCytokineCancer researchbiology.proteinCell DivisionLiver FailureBiotechnologyThe FASEB Journal
researchProduct

Biphasic dermatofibrosarcoma protuberans with a labyrinthine plexiform high‐grade fibrosarcomatous transformation

2013

Several variants of dermatofibrosarcoma protuberans, a low-grade superficial sarcoma, are well recognized. The most prognostically important is the fibrosarcomatous variant. We report a case of biphasic dermatofibrosarcoma protuberans in which the high-grade component exhibited a previously undescribed plexiform pattern. A clinicopathological study complemented with immunohistochemical, ultrastructural, reverse transcription polymerase chain reaction and fluorescence in situ hybridization analyses of this unique case. Histopathologically, a conventional low-grade dermatofibrosarcoma protuberans was admixed with intratumoral high-grade areas showing a striking labyrinthine plexiform pattern …

MalePathologymedicine.medical_specialtySkin NeoplasmsHistologyOncogene Proteins FusionFibrosarcomaCD34DermatologyBiologyPathology and Forensic MedicinemedicineDermatofibrosarcoma protuberansHumansIn Situ Hybridization FluorescencePDGFBmedicine.diagnostic_testDermatofibrosarcomaMiddle AgedPrognosismedicine.diseaseImmunohistochemistryCell Transformation NeoplasticCOL1A1/PDGFB Fusion GeneImmunohistochemistrySarcomaImmunostainingFluorescence in situ hybridizationJournal of Cutaneous Pathology
researchProduct

Solitary Fibrous Tumor: Integration of Clinical, Morphologic, Immunohistochemical and Molecular Findings in Risk Stratification and Classification Ma…

2021

Although solitary fibrous tumors (SFTs) have an unpredictable evolution, some specific clinicopathologic factors have been associated with the final outcome. We retrieved clinical, pathological and molecular data of 97 patients with a histological diagnosis of SFT and Signal transducer and activator of transcription 6 (STAT6) positivity. We retrospectively studied the pathological factors predictive of recurrence/metastasis and compared them with the clinical outcome. A wide immunohistochemical study and molecular analysis to detect NAB2/STAT6 gene fusion, tumor protein-53 (TP53) and/or (telomerase reverse transcriptase) TERT promotor mutation were performed. The risk of metastasis was calc…

MalePathologymedicine.medical_specialtySolitary fibrous tumorQH301-705.5CD99CD34APAF-1Risk AssessmentArticleCatalysisMetastasisInorganic ChemistryBiomarkers TumormedicineHumanssolitary fibrous tumorTelomerase reverse transcriptaseBiology (General)risk stratification systemsPhysical and Theoretical ChemistryQD1-999Molecular BiologyPathological<i>p53</i> mutationSpectroscopyRetrospective StudiesSTAT6<i>HTER</i> mutationbusiness.industryOrganic ChemistrySoft tissueGeneral MedicineMiddle AgedPrognosismedicine.diseaseCombined Modality TherapyImmunohistochemistryp53 mutationComputer Science ApplicationsChemistryHTER mutationSolitary Fibrous TumorsImmunohistochemistryFemaleNeoplasm Recurrence LocalbusinessFollow-Up StudiesInternational Journal of Molecular Sciences
researchProduct

Mathematical models for the diffusion magnetic resonance signal abnormality in patients with prion diseases

2014

In clinical practice signal hyperintensity in the cortex and/or in the striatum on magnetic resonance (MR) diffusion-weighted images (DWIs) is a marker of sporadic Creutzfeldt–Jakob Disease (sCJD). MR diagnostic accuracy is greater than 90%, but the biophysical mechanisms underpinning the signal abnormality are unknown. The aim of this prospective study is to combine an advanced DWI protocol with new mathematical models of the microstructural changes occurring in prion disease patients to investigate the cause of MR signal alterations. This underpins the later development of more sensitive and specific image-based biomarkers. DWI data with a wide a range of echo times and diffusion weightin…

MalePathologysCJD sporadic Creutzfeldt–Jakob diseaseROI region of interestPrion diseasePrPSc prion protein scrapieElectroencephalographyFOV field of viewlcsh:RC346-429Prion DiseasesADC apparent diffusion coefficientTI inversion timeRPE rapidly progressive encephalopathyAged 80 and overTE echo timeBrain Mappingmedicine.diagnostic_testBrainRegular ArticleMiddle AgedBIC Bayesian information criterionTR repetition timemedicine.anatomical_structureNeurologylcsh:R858-859.7FemaleMPRAGE magnetization-prepared rapid acquisition gradient-echoAbnormalitySS-SE single shot spin-echoAdultmedicine.medical_specialtyCognitive NeuroscienceCreutzfeldt–Jakob diseaseCNR contrast to noise ratioEPI echo-planar imagingNeuropathologyPrPC prion protein cellularGrey matterSpongiform degenerationlcsh:Computer applications to medicine. Medical informaticsEEG electroencephalogramDiffusion MRINeuroimagingImage Interpretation Computer-AssistedmedicineHumansRadiology Nuclear Medicine and imaginglcsh:Neurology. Diseases of the nervous systemAgedCJD Creutzfeldt–Jakob diseaseGSS Gerstmann–Sträussler–Scheinker syndromebusiness.industryDWI diffusion weighted imagingDiffusion MRI; Biophysical models; Creutzfeldt-Jakob disease; Prion disease; Spongiform degenerationMagnetic resonance imagingModels TheoreticalHyperintensityCreutzfeldt-Jakob diseaseDiffusion Magnetic Resonance ImagingNeurology (clinical)businessBiophysical modelsDiffusion MRINeuroImage: Clinical
researchProduct