Search results for "57"

showing 10 items of 4186 documents

Differentially Expressed tRNA-Derived Small RNAs Co-Sediment Primarily with Non-Polysomal Fractions in Drosophila

2017

Recent studies point to the existence of poorly characterized small regulatory RNAs generated from mRNAs, rRNAs and tRNAs. To explore the subcellular location of tRNA-derived small RNAs, 0–1 and 7–8 h Drosophila embryos were fractionated on sucrose density gradients. Analysis of 12,553,921 deep-sequencing reads from unfractionated and fractionated Drosophila embryos has revealed that tRFs, which are detected mainly from the 5’ends of tRNAs, co-sediment with the non-polysomal fractions. Interestingly, the expression levels of a subset of tRFs change temporally following thematernal-to-zygotic transition in embryos. We detected non-polysomal association of tRFs in S2 cells as well. Differenti…

0301 basic medicineanimal structureslcsh:QH426-470TRNAArticle03 medical and health sciencesExpression patternddc:570PolysomeGeneticstRFDrosophila (subgenus)tRNAGenetics (clinical)biologyTransition (genetics)PolysomeSchneider 2 cellsEmbryobiology.organism_classificationtRF; tRNA; polysome; <i>Drosophila</i>Molecular biologyCell biologylcsh:Genetics030104 developmental biologyTransfer RNADrosophilapolysomeTRFGenes
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Autophagy Stimulation as a Potential Strategy Against Intestinal Fibrosis

2019

We recently observed reduced autophagy in Crohn’s disease patients and an anti-inflammatory effect of autophagy stimulation in murine colitis, but both anti- and pro-fibrotic effects are associated with autophagy stimulation in different tissues, and fibrosis is a frequent complication of Crohn’s disease. Thus, we analyzed the effects of pharmacological modulation of autophagy in a murine model of intestinal fibrosis and detected that autophagy inhibition aggravates, while autophagy stimulation prevents, fibrosis. These effects are associated with changes in inflammation and in collagen degradation in primary fibroblasts. Thus, pharmacological stimulation of autophagy may be useful against …

0301 basic medicineautophagyStimulationInflammationDiseaseIntestinal fibrosis03 medical and health sciencesMice0302 clinical medicineCrohn DiseaseFibrosismedicineintestinal fibrosisMurine colitisAnimalslcsh:QH301-705.5Sirolimusbusiness.industryBrief ReportAutophagyGeneral MedicineFibroblastsmedicine.diseaseFibrosisIntestinesMice Inbred C57BLDisease Models Animal030104 developmental biologylcsh:Biology (General)inflammationCancer research030211 gastroenterology & hepatologyCollagenmedicine.symptomComplicationbusinessImmunosuppressive AgentsCells
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MicroRNAs Dysregulation and Metabolism in Multiple System Atrophy.

2019

Multiple system atrophy (MSA) is an adult onset, fatal disease, characterized by an accumulation of alpha-synuclein (α-syn) in oligodendroglial cells. MicroRNAs (miRNAs) are small non-coding RNAs involved in post-translational regulation and several biological processes. Disruption of miRNA-related pathways in the central nervous system (CNS) plays an important role in the pathogenesis of neurodegenerative diseases, including MSA. While the exact mechanisms underlying miRNAs in the pathogenesis of MSA remain unclear, it is known that miRNAs can repress the translation of messenger RNAs (mRNAs) that regulate the following pathogenesis associated with MSA: autophagy, neuroinflammation, α-syn …

0301 basic medicineautophagyalpha-synucleinCentral nervous systemmultiple system atrophyReviewBiologylcsh:RC321-571neuroinflammationPathogenesis03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAtrophystomatognathic systemmicroRNAmental disordersmedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNeuroinflammationAlpha-synucleinmicroRNAGeneral NeuroscienceAutophagyTranslation (biology)medicine.diseaseCell biologynervous system diseases030104 developmental biologymedicine.anatomical_structurechemistrynervous system030217 neurology & neurosurgeryNeuroscienceFrontiers in neuroscience
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Age‐related ultrastructural changes of the basement membrane in the mouse blood‐brain barrier

2018

Abstract The blood‐brain barrier (BBB) is essential for a functional neurovascular unit. Most studies focused on the cells forming the BBB, but very few studied the basement membrane (BM) of brain capillaries in ageing. We used transmission electron microscopy and electron tomography to investigate the BM of the BBB in ageing C57BL/6J mice. The thickness of the BM of the BBB from 24‐month‐old mice was double as compared with that of 6‐month‐old mice (107 nm vs 56 nm). The aged BBB showed lipid droplets gathering within the BM which further increased its thickness (up to 572 nm) and altered its structure. The lipids appeared to accumulate toward the glial side of the BM. Electron tomography …

0301 basic medicineblood‐brain barrierAgingElectron Microscope TomographyMyocytes Smooth Muscleelectron tomographyBlood–brain barrierMuscle Smooth Vascular03 medical and health sciencesMice0302 clinical medicineMicroscopy Electron TransmissionLipid dropletmedicineAnimalsBasement membraneChemistryNeurodegenerationBrainLipid metabolismBiological TransportCell BiologyOriginal ArticlesLipid Dropletsmedicine.diseaseLipid Metabolismbasement membraneCell biologyCapillariesMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureElectron tomographyAgeingageingBlood-Brain Barrier030220 oncology & carcinogenesisAstrocytesUltrastructureMolecular MedicineOriginal ArticleNeuroglia
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A Basic Architecture of an Autonomous Adaptive System With Conscious-Like Function for a Humanoid Robot.

2018

In developing a humanoid robot, there are two major objectives. One is developing a physical robot having body, hands, and feet resembling those of human beings and being able to similarly control them. The other is to develop a control system that works similarly to our brain, to feel, think, act, and learn like ours. In this article, an architecture of a control system with a brain-oriented logical structure for the second objective is proposed. The proposed system autonomously adapts to the environment and implements a clearly defined “consciousness” function, through which both habitual behavior and goal-directed behavior are realized. Consciousness is regarded as a function for effecti…

0301 basic medicinebrain-oriented systemComputer sciencelcsh:Mechanical engineering and machinerymedia_common.quotation_subjectlcsh:QA75.5-76.9503 medical and health sciences0302 clinical medicineArtificial IntelligenceAdaptive systemHypothesis and Theorylcsh:TJ1-1570Binding problemAdaptation (computer science)Function (engineering)goal-directed behaviorLibet’s experimentmedia_commonRobotics and AIautonomous adaptationhabitual behaviorArtificial neural networkbusiness.industryComputer Science Applicationsimage processing030104 developmental biologybinding problemRobotlcsh:Electronic computers. Computer scienceArtificial intelligencemodel of consciousnessConsciousnessbusiness030217 neurology & neurosurgeryHumanoid robotFrontiers in robotics and AI
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SANS (USH1G) Molecularly Links the Human Usher Syndrome Protein Network to the Intraflagellar Transport Module by Direct Binding to IFT-B Proteins.

2019

The human Usher syndrome (USH) is a retinal ciliopathy, characterized by profound congenital deafness, variable vestibular dysfunction and pre-pubertal onset of retinitis pigmentosa. In the effected sensory cells, USH protein networks are assumed to function in ciliary transport processes. The USH1G protein SANS is a scaffold of the ciliary/periciliary USH protein network of photoreceptor cells. Moreover, SANS is associated with microtubules, the transport routes for protein delivery toward the cilium. To enlighten the role of SANS in ciliary transport processes, we aimed to identify transport related proteins associated with SANS. The intraflagellar transport (IFT) system is a conserved me…

0301 basic medicineciliary transportIFTPhotoreceptor cell570 Life sciences03 medical and health sciencesCell and Developmental Biology0302 clinical medicineprimary ciliaMicrotubuleIntraflagellar transportRetinitis pigmentosamedicinephotoreceptor celllcsh:QH301-705.5USH interactomeOriginal ResearchChemistryCiliumCell Biologymedicine.diseaseCell biologyCiliopathy030104 developmental biologymedicine.anatomical_structureciliopathylcsh:Biology (General)030220 oncology & carcinogenesisUSH1GAnkyrin repeatsense organsCiliary baseUsher syndrome570 BiowissenschaftenDevelopmental BiologyFrontiers in cell and developmental biology
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Disruption of otoferlin alters the mode of exocytosis at the mouse inner hair cell ribbon synapse

2019

Sound encoding relies on Ca2+-mediated exocytosis at the ribbon synapse between cochlear inner hair cells (IHCs) and type I spiral ganglion neurons (SGNs). Otoferlin, a multi-C-2 domain protein, is proposed to regulate Ca2+-triggered exocytosis at this synapse, but the precise mechanisms of otoferlin function remain to be elucidated. Here, performing whole-cell voltage-clamp recordings of excitatory postsynaptic currents (EPSCs) from SGNs in otoferlin mutant mice, we investigated the impact of Otof disruption at individual synapses with single release event resolution. Otof deletion decreased the spontaneous release rate and abolished the stimulus-secretion coupling. This was evident from f…

0301 basic medicinecochleaRibbon synapsehair cellExocytosislcsh:RC321-571Synapse03 medical and health sciencesCellular and Molecular Neuroscienceotoferlin0302 clinical medicinemedicineOTOFauditoryMolecular Biologylcsh:Neurosciences. Biological psychiatry. NeuropsychiatrySpiral ganglionOriginal Researchribbon synapsecalciumChemistryDepolarizationCell biology030104 developmental biologymedicine.anatomical_structureEPSCExcitatory postsynaptic potentialHair cellspiral ganglion neuron030217 neurology & neurosurgeryNeuroscience
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Human Mesenchymal Stem Cells Prevent Neurological Complications of Radiotherapy

2019

Radiotherapy is a highly effective tool for the treatment of brain cancer. However, radiation also causes detrimental effects in the healthy tissue, leading to neurocognitive sequelae that compromise the quality of life of brain cancer patients. Despite the recognition of this serious complication, no satisfactory solutions exist at present. Here we investigated the effects of intranasal administration of human mesenchymal stem cells (hMSCs) as a neuroprotective strategy for cranial radiation in mice. Our results demonstrated that intranasally delivered hMSCs promote radiation-induced brain injury repair, improving neurological function. This intervention confers protection against inflamma…

0301 basic medicinecognitionmedicine.medical_treatmentneurocognitive sequelaeStem cellsBioinformaticsBrain cancer0302 clinical medicineCognitionOriginal ResearchCREBNeuroprotección:Analytical Diagnostic and Therapeutic Techniques and Equipment::Therapeutics::Radiotherapy [Medical Subject Headings]Neurocognitive sequelaeNeuroprotectionneuroprotectionmedicine.symptomStem cellCélulas madreNeoplasias encefálicas:Diseases::Neoplasms::Neoplasms by Site::Nervous System Neoplasms::Central Nervous System Neoplasms::Brain Neoplasms [Medical Subject Headings]Brain tumorInflammationNeuroprotectionlcsh:RC321-57103 medical and health sciencesCellular and Molecular NeuroscienceRadioterapiastem cellsmedicinelcsh:Neurosciences. Biological psychiatry. Neuropsychiatry:Chemicals and Drugs::Enzymes and Coenzymes::Enzymes::Transferases::Acyltransferases::Acetyltransferases::p300-CBP Transcription Factors::CREB-Binding Protein [Medical Subject Headings]radiotherapybrain cancerCogniciónRadiotherapybusiness.industryMesenchymal stem cellmedicine.diseaseequipment and suppliesIntranasal cell deliveryRadiation therapy030104 developmental biology:Anatomy::Cells::Stem Cells [Medical Subject Headings]Nasal administrationbusinessNeurocognitive030217 neurology & neurosurgeryintranasal cell deliveryNeuroscienceFrontiers in Cellular Neuroscience
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A Weaning Reaction to Microbiota Is Required for Resistance to Immunopathologies in the Adult.

2019

International audience; Microbes colonize all body surfaces at birth and participate in the development of the immune system. In newborn mammals, the intestinal microbiota is first shaped by the dietary and immunological components of milk and then changes upon the introduction of solid food during weaning. Here, we explored the reactivity of the mouse intestinal immune system during the first weeks after birth and into adulthood. At weaning, the intestinal microbiota induced a vigorous immune response—a “weaning reaction”—that was programmed in time. Inhibition of the weaning reaction led to pathological imprinting and increased susceptibility to colitis, allergic inflammation, and cancer …

0301 basic medicinecolitis[SDV]Life Sciences [q-bio]short-chain fatty acidsImmunologyRetinoic acidTretinoinWeaningBiologyT-Lymphocytes Regulatoryregulatory T cellsAllergic inflammation03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineImmune systemRAR-related orphan receptor gammamicrobiotamedicineImmunology and AllergyWeaningAnimalsinflammatory pathologyColitisImprinting (psychology)Intestinal Mucosaneonatal periodNuclear Receptor Subfamily 1 Group F Member 3medicine.diseaseFatty Acids Volatile3. Good healthGastrointestinal Microbiome[SDV] Life Sciences [q-bio]Mice Inbred C57BL030104 developmental biologyInfectious DiseaseschemistryAnimals NewbornSolid food030220 oncology & carcinogenesisImmunologymucosal immunityImmunity
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MECP2 impairs neuronal structure by regulating KIBRA

2016

Using a Drosophila model of MECP2 gain-of-function, we identified memory associated KIBRA as a target of MECP2 in regulating dendritic growth. We found that expression of human MECP2 increased kibra expression in Drosophila, and targeted RNAi knockdown of kibra in identified neurons fully rescued dendritic defects as induced by MECP2 gain-of-function. Validation in mouse confirmed that Kibra is similarly regulated by Mecp2 in a mammalian system. We found that Mecp2 gain-of-function in cultured mouse cortical neurons caused dendritic impairments and increased Kibra levels. Accordingly, Mecp2 loss-of-function in vivo led to decreased Kibra levels in hippocampus, cortex, and cerebellum. Togeth…

0301 basic medicinecongenital hereditary and neonatal diseases and abnormalitiesCerebellumMethyl-CpG-Binding Protein 2Dendritic morphologyHippocampusDisease modelsHippocampusArticlelcsh:RC321-571MECP2Mice03 medical and health sciencesMemoryRNA interferencemental disordersmedicineAnimalsHumanslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCerebral CortexNeuronsGene knockdownMECP2 duplication syndromebiologybiology.organism_classificationMECP2nervous system diseasesCortex (botany)Disease Models AnimalDrosophila melanogaster030104 developmental biologymedicine.anatomical_structureNeurologyCerebral cortexDrosophilaDrosophila melanogasterNeuroscienceNeurobiology of Disease
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