Search results for "Assay"

showing 10 items of 2241 documents

New benzothieno[3,2-d]-1,2,3-triazines with antiproliferative activity: synthesis, spectroscopic studies, and biological activity.

2014

New benzothieno[3,2-d]-1,2,3-triazines, together with precursors triazenylbenzo[b]thiophenes, were designed, synthesized and screened as anticancer agents. The structural features of these compounds prompted us to investigate their DNA binding capability through UV–vis absorption titrations, circular dichroism, and viscometry, pointing out the occurrence of groove-binding. The derivative 3-(4-methoxy-phenyl)benzothieno[3,2-d]-1,2,3-triazin-4(3H)-one showed the highest antiproliferative effect against HeLa cells and was also tested in cell cycle perturbation experiments. The obtained results assessed for the first time the anticancer activity of benzothieno[3,2-d]-1,2,3-triazine nucleus, and…

Circular dichroismStereochemistryClinical BiochemistryPharmaceutical ScienceAntineoplastic AgentsThiophenesBiochemistryHeLachemistry.chemical_compoundStructure-Activity RelationshipSettore BIO/10 - BiochimicaDrug DiscoveryStructure–activity relationshipMoleculeHumansMolecular BiologyCell ProliferationbiologyDose-Response Relationship DrugMolecular StructureChemistryCell growthTriazinesViscosityCircular DichroismOrganic ChemistryCell CycleBiological activityCell cyclebiology.organism_classificationSettore CHIM/08 - Chimica FarmaceuticaCombinatorial chemistrySettore CHIM/03 - Chimica Generale E InorganicaMolecular MedicineBenzothienotriazines Antiproliferative activity Spectroscopic studies Cell-cycle analysis VLAKSpectrophotometry UltravioletDrug Screening Assays AntitumorDNAHeLa CellsBioorganicmedicinal chemistry letters
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Tumor cells can escape DNA-damaging cisplatin through DNA endoreduplication and reversible polyploidy

2008

Cancer chemotherapy can induce tumor regression followed, in many cases, by relapse in the long-term. Thus this study was performed to assess the determinants of such phenomenon using an in vivo cancer model and in vitro approaches. When animals bearing an established tumor are treated by cisplatin, the tumor initially undergoes a dramatic shrinkage and is characterized by giant tumor cells that do not proliferate but maintain DNA synthesis. After several weeks of latency, the tumor resumes its progression and consists of small proliferating cells. Similarly, when tumor cells are exposed in vitro to pharmacological concentrations of cisplatin, mitotic activity stops initially but cells main…

CisplatinCell BiologyGeneral MedicineBiologyMolecular biologyDNA endoreduplicationGiant cellCancer researchmedicineCytotoxic T cellEndoreduplicationClonogenic assayMitosisMitotic catastrophemedicine.drugCell Biology International
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Genotoxicity of citrus wastewater in prokaryotic and eukaryotic cells and efficiency of heterogeneous photocatalysis by TiO(2).

2012

Abstract The presence of (±)α-pinene, (+)β-pinene, (+)3-carene, and R-(+)limonene terpenes in wastewater of a citrus transformation factory was detected and analyzed, in a previous study, by using Solid Phase Micro-extraction (SPME) followed by GC analyses. Purpose of that research was to compare the genotoxic responses of mixtures of terpenes with the genotoxicity of the individual compounds, and the biological effects of actual wastewater. Genotoxicity was evaluated in the Salmonella reversion assay (Ames test) and in V79 cells by Comet assay. Ames tests indicated that the four single terpenes did not induce an increase of revertants frequency. On the contrary, the mixtures of terpenes ca…

CitrusChromatography GasDNA damageBiophysicsPHOTOCATALYSIS TiO2 GENOTOXICITYmedicine.disease_causeWaste Disposal FluidCatalysisAmes testCell LineTerpenechemistry.chemical_compoundBridged Bicyclo CompoundsCricetulusCricetinaeCyclohexenesmedicineAnimalsRadiology Nuclear Medicine and imagingSolid Phase MicroextractionBicyclic MonoterpenesTitaniumLimoneneRadiationChromatographyPhotolysisRadiological and Ultrasound TechnologyMutagenicity TestsTerpenesComet assayTransformation (genetics)chemistryWastewaterEnvironmental chemistryMonoterpenesSettore CHIM/07 - Fondamenti Chimici Delle TecnologieComet AssayGenotoxicityLimoneneWater Pollutants ChemicalDNA DamageJournal of photochemistry and photobiology. B, Biology
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THE RECOVERY-ELISA—A NOVEL ASSAY TECHNIQUE TO MONITOR THERAPY WITH HUMANIZED ANTIBODIES: THE EXAMPLE OF OMALIZUMAB

2013

The therapeutic use of antibodies has grown exponentially, providing new treatment options for various diseases. Monitoring treatment with therapeutic antibodies is a particular challenge because often the target antigen is no longer measurable or the results are unreliable. To overcome this problem, a recovery-ELISA was developed to quantify therapeutic antibody and antigen by a modification of the traditional sandwich immunoassay using omalizumab as an example. Standard serum samples were spiked with IgE and omalizumab in a certain concentration range to create standard curves. After incubation and washout procedures, the reaction was stopped and the plate read with bichromatic absorbance…

Clinical BiochemistryImmunologyEnzyme-Linked Immunosorbent AssayOmalizumabOmalizumabPharmacologyAntibodies Monoclonal HumanizedImmunoglobulin EAntigenmedicineHumansImmunology and Allergybiologybusiness.industryAntibodies MonoclonalImmunoglobulin ESerum samplesAssay techniqueAntibodies Anti-IdiotypicStandard curveMedical Laboratory TechnologyTherapeutic antibodyImmunologybiology.proteinDrug MonitoringAntibodybusinessmedicine.drugJournal of Immunoassay and Immunochemistry
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SMART: Unique splitting-while-merging framework for gene clustering

2014

© 2014 Fa et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Successful clustering algorithms are highly dependent on parameter settings. The clustering performance degrades significantly unless parameters are properly set, and yet, it is difficult to set these parameters a priori. To address this issue, in this paper, we propose a unique splitting-while-merging clustering framework, named "splitting merging awareness tactics" (SMART), which does not require any a priori knowledge of either the number …

Clustering algorithmsMicroarrayslcsh:MedicineGene ExpressionBioinformaticscomputer.software_genreCell SignalingData MiningCluster Analysislcsh:ScienceFinite mixture modelOligonucleotide Array Sequence AnalysisPhysicsMultidisciplinarySMART frameworkConstrained clusteringCompetitive learning modelBioassays and Physiological AnalysisMultigene FamilyCanopy clustering algorithmEngineering and TechnologyData miningInformation TechnologyGenomic Signal ProcessingAlgorithmsResearch ArticleSignal TransductionComputer and Information SciencesFuzzy clusteringCorrelation clusteringResearch and Analysis MethodsClusteringMolecular GeneticsCURE data clustering algorithmGeneticsGene RegulationCluster analysista113Gene Expression Profilinglcsh:RBiology and Life SciencesComputational BiologyCell BiologyDetermining the number of clusters in a data setComputingMethodologies_PATTERNRECOGNITIONSplitting-merging awareness tactics (SMART)Signal ProcessingAffinity propagationlcsh:QGene expressionClustering frameworkcomputer
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Precise mapping of the Goodpasture epitope(s) using phage display, site-directed mutagenesis, and surface plasmon resonance.

2013

Goodpasture disease is an autoimmune disorder mediated by circulating autoantibodies against the noncollagenous-1 (NC1) domain of the alpha 3 chain of type IV collagen (alpha 3(IV)NC1). The structure of Goodpasture epitope(s) has been previously mapped into two main binding regions (E-A and E-B) of the alpha 3(IV)NC1 domain using a residue mutation approach on the highly related alpha 1(IV)NC1 domain. Here we combined phage display and surface plasmon resonance technology to more precisely localize the pathogenic binding sites. Peptides mimicking the Goodpasture epitope(s) were used to identify residues involved in autoantibody binding and found involvement of eight residues previously unre…

Collagen Type IVMalePhage displayautoantibodiesMutantMutagenesis (molecular biology technique)Enzyme-Linked Immunosorbent Assaycollagen type IVAutoantigensEpitopeType IV collagenHumansBinding siteSite-directed mutagenesisAutoantibodiesepitopeChemistryAutoantibodyGoodpasture diseaseMiddle AgedSurface Plasmon ResonanceMolecular biologyNephrologyMutagenesis Site-DirectedBinding Sites Antibodyphage displayCell Surface Display Techniquessurface plasmon resonanceEpitope MappingKidney international
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Full‐thickness tissue engineered oral mucosa for genitourinary reconstruction: A comparison of different collagen‐based biodegradable membranes

2020

Tissue engineering is a method of growing importance regarding clinical application in the genitourinary region. One of the key factors in successfully development of an artificially tissue engineered mucosa equivalent (TEOM) is the optimal choice of the scaffold. Collagen scaffolds are regarded as gold standard in dermal tissue reconstruction. Four distinct collagen scaffolds were evaluated for the ability to support the development of an organotypical tissue architecture. TEOMs were established by seeding cocultures of primary oral epithelial cells and fibroblasts on four distinct collagen membranes. Cell viability was assessed by MTT-assay. The 3D architecture and functionality of the ti…

Collagen Type IVScaffoldMaterials scienceSwineBiomedical EngineeringTenascinBiocompatible MaterialsMatrix (biology)Fibroblast migrationBiomaterials03 medical and health sciences0302 clinical medicineTissue engineeringAbsorbable ImplantsMaterials TestingmedicineAnimalsViability assayOral mucosaFibroblastCells CulturedTissue EngineeringTissue ScaffoldsbiologyKeratin-13Mouth MucosaEpithelial CellsMembranes ArtificialTenascin030206 dentistryFibroblastsPlastic Surgery ProceduresCoculture TechniquesUrogenital Surgical ProceduresCell biologymedicine.anatomical_structure030220 oncology & carcinogenesisbiology.proteinJournal of Biomedical Materials Research Part B: Applied Biomaterials
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Regulation of type IV collagen gene expression and degradation in fast and slow muscles during dexamethasone treatment and exercise.

2003

Glucocorticoids have anti-anabolic effects on many tissues and can cause muscle atrophy. However, their effects on type IV collagen gene expression and degradation in skeletal muscle have not been studied previously. Rats were treated daily with dexamethasone or saline. Half the groups of experimental and control animals were also subjected to daily endurance or uphill running exercise to determine the possible preventive effects of exercise. After an experimental period of 3 or 10 days, the extensor digitorum longus, soleus and tibialis anterior muscles were studied. Dexamethasone treatment for 10 days reduced muscle weight and type IV collagen mRNA abundance in all muscles. Gene expressio…

Collagen Type IVmedicine.medical_specialtyPhysiologyClinical BiochemistryAnti-Inflammatory AgentsRadioimmunoassayMatrix metalloproteinaseDexamethasoneRats Sprague-DawleyType IV collagenPhysiology (medical)Internal medicinePhysical Conditioning AnimalGene expressionmedicineAnimalsRNA MessengerReceptorMuscle SkeletalGlucocorticoidsDexamethasoneRegulation of gene expressionTissue Inhibitor of Metalloproteinase-2ChemistrySkeletal muscleBlotting NorthernMuscle atrophyRatsEndocrinologymedicine.anatomical_structureMuscle Fibers Slow-TwitchGene Expression RegulationMuscle Fibers Fast-TwitchMatrix Metalloproteinase 2Femalemedicine.symptommedicine.drugPflugers Archiv : European journal of physiology
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2-Cinnamamido, 2-(3-phenylpropiolamido), and 2-(3-phenylpropanamido)benzamides: synthesis, antiproliferative activity, and mechanism of action

2013

Abstract Several new benzamides 4a–q were synthesized by stirring in pyridine the acid chlorides 3a–q with the appropriate anthranilamide derivatives 2a–g. Some of the synthesized compounds were evaluated for their in vitro antiproliferative activity against a panel of 5 human cell lines (K562 human chronic myelogenous leukemia cells, MCF-7 breast cancer cells, HTC-116 and HT26 colon cancer cells and NCI H460 non-small cell lung cancer cells).

Colorectal cancerAntineoplastic AgentsApoptosisPharmacologyArticleStructure-Activity RelationshipDrug DiscoveryTumor Cells CulturedmedicineHumansStructure–activity relationshipCell ProliferationPharmacologyDose-Response Relationship DrugMolecular StructureChemistryCell growthOrganic ChemistryGeneral Medicinemedicine.disease2-cinnamamidobenzamides 2-(3-phenylpropiolamido)benzamides 2-(3-phenylpropanamido)benzamides antiproliferative activity apoptosisSettore CHIM/08 - Chimica FarmaceuticaMechanism of actionApoptosisBenzamidesMCF-7 CellsNon small cellDrug Screening Assays Antitumormedicine.symptomK562 CellsChronic myelogenous leukemiaK562 cells
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Dynamic regulation of the cancer stem cell compartment by Cripto-1 in colorectal cancer.

2015

Stemness was recently depicted as a dynamic condition in normal and tumor cells. We found that the embryonic protein Cripto-1 (CR1) was expressed by normal stem cells at the bottom of colonic crypts and by cancer stem cells (CSCs) in colorectal tumor tissues. CR1-positive populations isolated from patient-derived tumor spheroids exhibited increased clonogenic capacity and expression of stem-cell-related genes. CR1 expression in tumor spheroids was variable over time, being subject to a complex regulation of the intracellular, surface and secreted protein, which was related to changes of the clonogenic capacity at the population level. CR1 silencing induced CSC growth arrest in vitro with a …

Colorectal cancerColorectal NeoplasmCriptoMiceIntercellular Signaling Peptides and ProteinTumor Cells CulturedRegulation of gene expressionCulturedstem cell; CRIPTO 1GPI-Linked ProteinCell biologyNeoplasm ProteinsTumor CellsGene Expression Regulation NeoplasticGenes srcNeoplastic Stem CellsIntercellular Signaling Peptides and ProteinsFemaleStem cellColorectal NeoplasmsHumanSignal Transductioncolorectal cancerBiologyGPI-Linked ProteinsAnimals; Colorectal Neoplasms; Female; GPI-Linked Proteins; Gene Expression Regulation Neoplastic; Genes src; Humans; Intercellular Signaling Peptides and Proteins; Mice; Neoplasm Proteins; Neoplastic Stem Cells; Proto-Oncogene Proteins c-akt; Signal Transduction; Spheroids Cellular; Tumor Cells Cultured; Cell Biology; Molecular BiologyNeoplasm ProteinCancer stem cellSettore MED/04 - PATOLOGIA GENERALESpheroids CellularmedicineGene silencingAnimalsHumansClonogenic assayProtein kinase BMolecular BiologysrcOriginal PaperNeoplasticAnimalCell Biologymedicine.diseaseGene Expression RegulationGenesNeoplastic Stem CellCellularSpheroidsanimals; colorectal neoplasms; female; GPI-linked proteins; gene expression regulation; neoplastic; genes src; humans; intercellular signaling peptides and proteins; mice; neoplasm proteins; neoplastic stem cells; proto-oncogene proteins c-akt; signal transduction; spheroids; cellular; tumor cells; culturedAnimals; Colorectal Neoplasms; Female; GPI-Linked Proteins; Gene Expression Regulation Neoplastic; Genes src; Humans; Intercellular Signaling Peptides and Proteins; Mice; Neoplasm Proteins; Neoplastic Stem Cells; Proto-Oncogene Proteins c-akt; Signal Transduction; Spheroids Cellular; Tumor Cells Cultured; Molecular Biology; Cell BiologyProto-Oncogene Proteins c-akt
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