Search results for "Autoimmunity"

showing 10 items of 349 documents

Association between hypovitaminosis D and systemic sclerosis: True or fake?

2016

Abstract Background Vitamin D insufficiency/deficiency is considered a major factor triggering and enhancing several autoimmune disorders; hypovitaminosis D has been reported to be common in Systemic Sclerosis (SSc). Previous studies assessing vitamin D insufficiency/deficiency in SSc have been reviewed, and the relation with pathogenesis and clinical features has been examined. Content Eligibility criteria were: reporting measurement of Vitamin D serum levels in all participants and evaluating adult onset-SSc individuals as patients group. Results: The association between clinical features and low hormone levels is controversial. Manifold data have shown vitamin D insufficiency/deficiency …

0301 basic medicinemedicine.medical_specialtyClinical BiochemistryVitamin D Systemic Sclerosis Hypovitaminosis D AutoimmunityDiseasemedicine.disease_causeBiochemistryCalcitriol receptorAutoimmunityPathogenesis03 medical and health sciences0302 clinical medicineHypovitaminosisDLCOInternal medicinemedicineVitamin D and neurologyHumansVitamin D030203 arthritis & rheumatologyScleroderma Systemicbusiness.industryBiochemistry (medical)General MedicineVitamin D Deficiency030104 developmental biologyImmunologybusinessHormone
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2020

The brain ventricles are part of the fluid compartments bridging the CNS with the periphery. Using MRI, we previously observed a pronounced increase in ventricle volume (VV) in the experimental autoimmune encephalomyelitis (EAE) model of multiple sclerosis (MS). Here, we examined VV changes in EAE and MS patients in longitudinal studies with frequent serial MRI scans. EAE mice underwent serial MRI for up to 2 months, with gadolinium contrast as a proxy of inflammation, confirmed by histopathology. We performed a time-series analysis of clinical and MRI data from a prior clinical trial in which RRMS patients underwent monthly MRI scans over 1 year. VV increased dramatically during preonset E…

0301 basic medicinemedicine.medical_specialtyPathologybusiness.industryMultiple sclerosisExperimental autoimmune encephalomyelitisGeneral MedicineFluid compartmentsmedicine.diseasemedicine.disease_causeAutoimmunity03 medical and health sciences030104 developmental biology0302 clinical medicinemedicine.anatomical_structureAtrophyNeuroimagingVentricle030220 oncology & carcinogenesismedicineHistopathologybusinessJCI Insight
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Autoantibodies in Spondyloarthritis, Focusing on Anti-CD74 Antibodies

2019

Spondyloarthritis (SpA) is an inflammatory rheumatic disease with diverse clinical presentation. The diagnosis of SpA remains a big challenge in daily clinical practice because of the limitation in specific biomarkers of SpA, more biomarkers are still needed for SpA diagnosis and disease activity monitoring. In the past, SpA was considered predominantly as auto-inflammatory disease vs. autoimmune disease. However, in recent years several researches demonstrated a broad autoantibody response in SpA patients. Study also indicated that mice lack of ZAP70 in T cell develop SpA featured inflammation. These studies indicated the autoimmune features of SpA and gave rise to the potential use of aut…

0301 basic medicinemusculoskeletal diseaseslcsh:Immunologic diseases. AllergyCD74autoantibodiesdiagnosisImmunologyAutoimmunityDiseaseAutoantigensAutoimmune DiseasesPathogenesis03 medical and health sciences0302 clinical medicineHypothesis and TheorySpondylarthritismedicineImmunology and AllergyHumansHeat-Shock ProteinsAutoimmune diseasebiologybusiness.industryChinese patientsAutoantibodyHistocompatibility Antigens Class IIspondyloarthritismedicine.diseaseClinical PracticeAntigens Differentiation B-LymphocyteProtein Phosphatase 2Cstomatognathic diseases030104 developmental biology14-3-3 ProteinsROC CurveImmunologybiology.proteinBiomarker (medicine)Antibodybusinessbeta 2-Microglobulinlcsh:RC581-607Biomarkers030215 immunologyanti-CD74 autoantibodyFrontiers in Immunology
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Is COVID-19 a proteiform disease inducing also molecular mimicry phenomena?

2020

2019-20 coronavirus outbreakCoronavirus disease 2019 (COVID-19)Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2)Pneumonia ViralAutoimmunityDiseaseComorbiditymedicine.disease_causeAntibodies ViralBiochemistryBetacoronavirusmedicineHumansViral immunologyPandemicsbusiness.industrySARS-CoV-2Molecular MimicryCOVID-19Endothelial CellsCovid 19Cell BiologyMolecular mimicryAcute DiseasebusinessCoronavirus InfectionsNeuroscience
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Is molecular mimicry the culprit in the autoimmune haemolytic anaemia affecting patients with COVID‐19?

2020

2019-20 coronavirus outbreakCoronavirus disease 2019 (COVID-19)autoantibodiesPneumonia Viralmedicine.disease_causeCulpritAutoimmunityBetacoronavirusCOVID‐19Correspondenceankirin 1PandemicHumansMedicineAnemia Hemolytic AutoimmuneChildPandemicsBetacoronavirubiologyCoronavirus InfectionSARS-CoV-2business.industryautoimmunityMolecular MimicryAutoantibodyCOVID-19Hematologyautoantibodiebiology.organism_classificationVirologyMolecular mimicryAnemia Hemolytic AutoimmuneCoronavirus InfectionsbusinessBetacoronavirusHumansevere acute respiratory syndrome coronavirus 2British Journal of Haematology
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Does dietary salt induce autoimmunity?

2013

Two recent publications suggest that dietary salt may polarize TH17 cells and therefore increase the risk of developing autoimmune disease. Where low salt diets can readily be tested for their therapeutic effects in autoimmune disease, more work is needed to connect dietary salts with the development of immunopathology.

610 Medicine & healthBiology10263 Institute of Experimental Immunologymedicine.disease_causeImmediate early proteinAutoimmunity1307 Cell Biology03 medical and health sciences0302 clinical medicineLow-salt dietsImmunopathology1312 Molecular BiologymedicineMolecular Biology030304 developmental biologyAutoimmune disease0303 health sciencesCell Biologymedicine.diseaseResearch Highlight3. Good healthImmunology570 Life sciences; biologyInterleukin 17030215 immunologyDietary saltCell research
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Synthetic Polyclonal-Derived CDR Peptides as an Innovative Strategy in Glaucoma Therapy

2019

The pathogenesis of glaucoma is strongly associated with the occurrence of autoimmune-mediated loss of retinal ganglion cells (RGCs) and additionally, recent evidence shows that specific antibody-derived signature peptides are significantly differentially expressed in sera of primary-open angle glaucoma patients (POAG) compared to healthy controls. Synthetically antibody-derived peptides can modulate various effector functions of the immune system and act as antimicrobial or antiviral molecules. In an ex vivo adolescent glaucoma model, this study, for the first time, demonstrates that polyclonal-derived complementarity-determining regions (CDRs) can significantly increase the survival rate …

<i>Sus scrofa domestica</i>lcsh:MedicineRetinal ganglionEpitopeArticleSus scrofa domestica03 medical and health scienceschemistry.chemical_compound0302 clinical medicineImmune systemMedicine030304 developmental biology0303 health sciencesHTRA2synthetic CDR peptidesbusiness.industrylcsh:RautoimmunityRetinalGeneral MedicineProtein ubiquitinationCell biologyglaucomachemistryneuroprotectionSignal transductionbusinessVDAC2030217 neurology & neurosurgeryEx vivoJournal of Clinical Medicine
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Controversies on the role of Th17 in cancer: a TGF-β-dependent immunosuppressive activity?

2012

The immune system has important roles in limiting the spread of cancer and shaping the tumor microenvironment. Although the contributions of T helper 17 (Th17) cells (a subtype of CD4(+) T lymphocytes) to autoimmunity and allergy response are well known, their roles in cancer remain ambiguous. Despite adoptive transfer studies indicating that mouse Th17 cells support anticancer immunity, the Th17 cells that naturally infiltrate experimental tumors appear to have a tumor-promoting effect. These contradictory properties can be related to the high degree of plasticity inherent in Th17 cells and their capacity to differentiate into tumoricidal Th1-like cells. Mouse Th17 cells induced by transfo…

Adoptive cell transferAngiogenesisAntigen-Presenting Cellschemical and pharmacologic phenomenaBiologymedicine.disease_causeAutoimmunityMice03 medical and health sciences0302 clinical medicineImmune systemAntigenAntigens CDTransforming Growth Factor betaImmunityNeoplasmsImmune TolerancemedicineAnimals5'-NucleotidaseMolecular Biology030304 developmental biologyImmunity Cellular0303 health sciencesTumor microenvironmentNeovascularization PathologicApyraseModels ImmunologicalCell DifferentiationTh1 Cells3. Good health030220 oncology & carcinogenesisImmunologyCancer researchTh17 CellsMolecular MedicineTransforming growth factorTrends in Molecular Medicine
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Research in practice: Regulatory T cells - targets for therapeutic approaches?

2010

• regulatory T cells • tolerance • signal transduction • autoimmunity • allergies • cancer Summary Regulatory T cells (Tregs) are essential for induction and maintenance of immunological tolerance. They contribute to prevention of autoimmunity by control and modulation of immune responses. The prevalence of autoimmune diseases, chronic infections, cancer and allergies has markedly increased in the last decades. In additions the treatment of these disorders is often unsatisfactory so that improvements are needed. This has stimulated intensive research in the biology of Tregs. Recent studies revealed that naturally occurring CD4 + CD25 + Tregs (nTregs) and induced Tregs (iTregs) are critical …

Adoptive cell transferEffectorCancerhemic and immune systemschemical and pharmacologic phenomenaDermatologyBiologymedicine.disease_causemedicine.diseaselaw.inventionAutoimmunityImmune systemlawImmunologymedicineSuppressorIL-2 receptorSignal transductionJDDG: Journal der Deutschen Dermatologischen Gesellschaft
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CD4+CD25+ Regulatory T Cells and TGF-Beta in Mucosal Inflammation

2008

Transforming growth factor-beta (TGF-beta) is an anti-inflammatory cytokine which plays a key role in the maintenance of the immune system homeostasis. Indeed the abrogation of the TGF-beta signaling in immune cells leads to autoimmunity and inflammation in several organs including the gut. TGF-beta acts at multiple levels to maintain the immune system in check. However, TGF-beta has been recently shown to play a key role in the peripheral generation and function of CD4+CD25+ regulatory T cells, a subset of suppressive lymphocytes involved in the control of effector T cell activation and proliferation. Consistently abrogation of Tregs maturation as observed in different systems leads to a p…

Adoptive cell transfermedicine.medical_treatmentT cellInflammationBiologymedicine.disease_causeCell biologyAutoimmunityImmune systemCytokinemedicine.anatomical_structureTGF beta signaling pathwaymedicineIL-2 receptormedicine.symptom
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