Search results for "Cytomegalovirus infections"

showing 10 items of 153 documents

Comparison of the new Abbott Real Time CMV assay and the Abbott CMV PCR Kit for the quantitation of plasma cytomegalovirus DNAemia

2012

CMV DNA loads measured by the new Abbott RealTime CMV PCR were significantly higher than those quantitated by the Abbott CMV PCR kit (approximately 1 log(10)), and provided a better estimate of the actual CMV load present in plasma specimens as inferred by the use of the WHO standard.

Microbiology (medical)business.industryCongenital cytomegalovirus infectionCytomegalovirusvirus diseasesGeneral Medicinemedicine.diseasePolymerase Chain ReactionVirologylaw.inventionInfectious DiseasesReal-time polymerase chain reactionlawCytomegalovirus InfectionsDNA ViralLinear ModelsmedicineHumansCytomegalovirus infectionsDna viralbusinessPolymerase chain reactionDiagnostic Microbiology and Infectious Disease
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Tetraspanins in infections by human cytomegalo- and papillomaviruses

2017

Members of the tetraspanin family have been identified as essential cellular membrane proteins in infectious diseases by nearly all types of pathogens. The present review highlights recently published data on the role of tetraspanin CD151, CD81, and CD63 and their interaction partners in host cell entry by human cytomegalo- and human papillomaviruses. Moreover, we discuss a model for tetraspanin assembly into trafficking platforms at the plasma membrane. These platforms might persist during intracellular viral trafficking.

Models Molecular0301 basic medicineCellular membraneTetraspaninsCytomegalovirusTetraspanin 24BiologyEndocytosismedicine.disease_causeBiochemistryTetraspanin 28Viral Proteins03 medical and health sciencesTetraspaninmedicineHumansPapillomaviridaeCD151Tetraspanin 30Cell MembranePapillomavirus InfectionsCytomegalovirusVirus InternalizationVirologyCell biology030104 developmental biologyCytomegalovirus Infectionsembryonic structuresIntracellularCD81Biochemical Society Transactions
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Adoptive CD8 T Cell Control of Pathogens Cannot Be Improved by Combining Protective Epitope Specificities

2008

Adoptive transfer of CD8 T cells has the potential to cure infectious or malignant diseases that are refractory to conventional chemotherapy. A practically important but still unanswered question is whether mixtures of protective CD8 T cells with different epitope specificities mediate more efficient effector cell functions than do the monospecific individual CD8 T cell populations. In this study, we have addressed this issue for models of viral and bacterial infection. CD8 T cell-mediated cytotoxicity in vitro and protection in vivo were assessed to test whether CD8 T cell lines cooperate in target cell lysis and control of infection, respectively. Our data clearly show that mixtures of cy…

MuromegalovirusAdoptive cell transferT cellEpitopes T-LymphocyteBacteremiaStreptamerCD8-Positive T-LymphocytesBiologyEpitopeMicemedicineAnimalsImmunology and AllergyCytotoxic T cellViremiaAntigen-presenting cellT lymphocyteAdoptive TransferListeria monocytogenesVirologyDisease Models AnimalInfectious Diseasesmedicine.anatomical_structureCytomegalovirus InfectionsImmunologyFemaleCD8The Journal of Infectious Diseases
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In vivo impact of cytomegalovirus evasion of CD8 T-cell immunity: Facts and thoughts based on murine models

2010

Cytomegaloviruses (CMVs) co-exist with their respective host species and have evolved to avoid their elimination by the hosts' immune effector mechanisms and to persist in a non-replicative state, known as viral latency. There is evidence to suggest that latency is nevertheless a highly dynamic condition during which episodes of viral gene desilencing, which can be viewed as incomplete reactivations, cause intermittent antigenic activity that stimulates CD8 memory-effector T cells and drives their clonal expansion. These T cells are supposed to terminate reactivation before completion of the productive viral cycle. In this view, CMVs do not "evade" their respective host's immune response bu…

MuromegalovirusCancer ResearchT cellAntigen presentationReceptors Antigen T-CellCytomegalovirusCD8-Positive T-LymphocytesBiologyMiceImmune systemAntigenVirologyVirus latencymedicineAntigenic variationAnimalsCytotoxic T cellViral InterferenceImmune EvasionAntigen PresentationHistocompatibility Antigens Class IHerpesviridae Infectionsmedicine.diseaseVirologyVirus LatencyDisease Models AnimalInfectious Diseasesmedicine.anatomical_structureCytomegalovirus InfectionsImmunologyVirus ActivationVirus Research
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Cytomegalovirus Encodes a Positive Regulator of Antigen Presentation

2006

ABSTRACT Murine cytomegalovirus encodes three regulators of antigen presentation to antiviral CD8 T cells. According to current paradigms, all three regulators are committed to the inhibition of the presentation of antigenic peptides. Whereas m152/gp40 catalyzes the retention of peptide-loaded major histocompatibility complex (MHC) class I molecules in a cis -Golgi compartment, m06/gp48 binds stably to class I molecules and directs them into the cellular cargo-sorting pathway of lysosomal degradation. Regulator m04/gp34 also binds stably to class I molecules, but unlike m152 and m06, it does not downmodulate MHC class I cell surface expression. It has entered the literature as a direct inhi…

MuromegalovirusImmunologyAntigen presentationRegulatorCD8-Positive T-LymphocytesVirus ReplicationMajor histocompatibility complexMicrobiologyMiceViral ProteinsMuromegalovirusAntigenVirologyMHC class IAnimalsHumansCytotoxic T cellAntigens ViralCells CulturedGlycoproteinsAntigen PresentationMice Inbred BALB CMembrane GlycoproteinsbiologyAntigen processingHistocompatibility Antigens Class IH-2 AntigensFibroblastsEmbryo Mammalianbiology.organism_classificationAdoptive TransferMolecular biologyMice Inbred C57BLInsect ScienceCytomegalovirus Infectionsbiology.proteinPathogenesis and ImmunityFemaleCarrier ProteinsPeptidesT-Lymphocytes CytotoxicJournal of Virology
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Tumor Control in a Model of Bone Marrow Transplantation and Acute Liver-Infiltrating B-Cell Lymphoma: an Unpredicted Novel Function of Cytomegalovirus

2002

ABSTRACTTumor relapse and cytomegalovirus (CMV) infection are major concerns in the therapy of hematopoietic malignancies by bone marrow transplantation (BMT). Little attention so far has been given to a possible pathogenetic interplay between CMV and lymphomas. CMV inhibits stem cell engraftment and hematopoietic reconstitution. Thus, by causing maintenance of bone marrow aplasia and immunodeficiency, CMV could promote tumor relapse. Alternatively, CMV could aid tumor remission. One might think of cytopathogenic infection of tumor cells, induction of apoptosis or inhibitory cytokines, interference with tumor cell extravasation or tumor vascularization, or bystander stimulation of an antitu…

MuromegalovirusLymphoma B-CellCD30ImmunologyBone Marrow AplasiaBiologyMicrobiologyMiceImmune systemhemic and lymphatic diseasesVirologyTumor Cells CulturedmedicineAnimalsCytotoxic T cellB-cell lymphomaBone Marrow TransplantationMice Inbred BALB CTumor Necrosis Factor-alphamedicine.diseaseLymphomaDisease Models AnimalHaematopoiesisLiverInsect ScienceCytomegalovirus InfectionsImmunologyPathogenesis and ImmunityStem cellJournal of Virology
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Cytomegalovirus DNAemia and risk of mortality in allogeneic hematopoietic stem cell transplantation: Analysis from the Spanish Hematopoietic Transpla…

2020

The net impact of cytomegalovirus (CMV) DNAemia on overall mortality (OM) and nonrelapse mortality (NRM) following allogeneic hematopoietic stem cell transplantation (allo-HSCT) remains a matter of debate. This was a retrospective, multicenter, noninterventional study finally including 749 patients. CMV DNA monitoring was conducted by real-time polymerase chain reaction (PCR) assays. Clinical outcomes of interest were OM and NRM through day 365 after allo-HSCT. The cumulative incidence of CMV DNAemia in this cohort was 52.6%. A total of 306 out of 382 patients with CMV DNAemia received preemptive antiviral therapy (PET). PET use for CMV DNAemia, but not the occurrence of CMV DNAemia, taken …

Oncologymedicine.medical_specialtybone marrowinfection and infectious agents - viralmedicine.medical_treatmentCongenital cytomegalovirus infectionCytomegaloviruscomplicationHematopoietic stem cell transplantation030230 surgerylaw.inventionCell therapy03 medical and health sciences0302 clinical medicinelawInternal medicinehemic and lymphatic diseasesRisk of mortalityImmunology and AllergyMedicineHumansTransplantation HomologousPharmacology (medical)Cumulative incidencePolymerase chain reactionRetrospective StudiesTransplantationbusiness.industryHematopoietic Stem Cell Transplantationvirus diseasesmedicine.diseasepracticeTransplantationinfectious infection and infectious agents - viral: Cytomegalovirus (CMV) [bone marrow/hematopoietic stem cell transplantation clinical research/practice complication]infectiousclinical researchCohortCytomegalovirus InfectionsDNA Viralhematopoietic stem cell transplantationCytomegalovirus (CMV)business
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Rapidly progressive organising pneumonia associated with cytomegalovirus infection in a patient with psoriasis.

2016

A 63-year-old woman experienced progressive respiratory distress and psoriatic plaques. The radiographic images showed diffuse interstitial infiltrates. The surgical open lung biopsy revealed an obliteration of the alveolar spaces by plugs of connective tissue distributed within the terminal bronchioles, alveolar ducts and spaces. No relevant cause was determined, and she was diagnosed with idiopathic organising pneumonia. The patient was discharged with oral glucocorticosteroid and supplemental oxygen therapy. One month later, the patient’s pulmonary status had progressively worsened, and she was re-admitted. She required higher oxygen concentrations and mechanical ventilation. Pharmacol…

Pulmonary and Respiratory MedicineGanciclovirPathologymedicine.medical_specialtyCyclophosphamidemedicine.medical_treatmentCongenital cytomegalovirus infectionlcsh:MedicineConnective tissueInterstitial disease connective diseaseSettore MED/10 - Malattie Dell'Apparato RespiratorioPsoriasismedicineHumansPsoriasiscytomegalovirus infectionInterstitial pneumoniaMechanical ventilationRespiratory distressbusiness.industrylcsh:RMiddle Agedmedicine.diseaseRegimenmedicine.anatomical_structureCryptogenic Organizing PneumoniaCytomegalovirus InfectionsFemaleCardiology and Cardiovascular Medicinebusinessmedicine.drugMonaldi archives for chest disease = Archivio Monaldi per le malattie del torace
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Exogenous introduction of an immunodominant peptide from the non-structural IE1 protein of human cytomegalovirus into the MHC class I presentation pa…

2008

Exogenous introduction of particle-associated proteins of human cytomegalovirus (HCMV) into the major histocompatibility complex (MHC) class I presentation pathway by subviral dense bodies (DB) is an effective way to sensitize cells against CD8 T-cell (CTL) recognition and killing. Consequently, these particles have been proposed as a platform for vaccine development. We have developed a strategy to refine the antigenic composition of DB. For proof of principle, an HCMV recombinant (RV-VM3) was generated that encoded the immunodominant CTL determinant IE1TMY from the IE1 protein in fusion with the major constituent of DB, the tegument protein pp65. To generate RV-VM3, a bacterial artificial…

Recombinant Fusion ProteinsvirusesCytomegalovirusImmunodominanceMajor histocompatibility complexImmediate-Early Proteinslaw.inventionViral ProteinsAntigenlawVirologyMHC class IHumansAntigen PresentationbiologyHistocompatibility Antigens Class IVirionvirus diseasesViral VaccinesGenetic TherapyFusion proteinVirologyPeptide FragmentsCTL*Cytomegalovirus Infectionsbiology.proteinRecombinant DNACD8T-Lymphocytes CytotoxicJournal of General Virology
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Evidence against a key role for transforming growth factor-beta1 in cytomegalovirus-induced bone marrow aplasia.

1998

During immunodeficiency after sublethal haematoablative treatment, cytomegalovirus (CMV) infection interferes with haematopoietic reconstitution and can cause lethal bone marrow (BM) aplasia. The in vivo model of murine CMV infection has identified the BM stroma as the principal target site of CMV in the haematopoietic cord. The infected cell type is the reticular stromal cell which forms the stromal network and produces essential haemopoietins, such as stem-cell factor (SCF). The expression of SCF was found to be reduced in the infected stroma, but the stromal network was not disrupted and the number of infected stromal cells was too low to explain the functional deficiency. These facts ca…

Stromal cellmedicine.medical_treatmentCytomegalovirusGene ExpressionBone Marrow CellsBone Marrow AplasiaCD8-Positive T-LymphocytesKidneyVirus ReplicationMiceTransforming Growth Factor betaVirologymedicineAnimalsCytotoxic T cellBone Marrow DiseasesBone Marrow TransplantationMice Inbred BALB CbiologyTransforming growth factor betaVirologyHematopoiesisHaematopoiesisCytokinemedicine.anatomical_structureLiverCytomegalovirus Infectionsbiology.proteinFemaleImmunotherapyBone marrowStromal CellsTransforming growth factorJournal of General Virology
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