Search results for "DISEASES"

showing 10 items of 20537 documents

Cardiac regenerative capacity is age- and disease-dependent in childhood heart disease

2018

Objective We sought to define the intrinsic stem cell capacity in pediatric heart lesions, and the effects of diagnosis and of age, in order to inform evidence-based use of potential autologous stem cell sources for regenerative medicine therapy. Methods Ventricular explants derived from patients with hypoplastic left heart syndrome (HLHS), tetralogy of Fallot (TF), dilated cardiomyopathy (DCM) and ventricular septal defect (VSD) were analyzed following standard in vitro culture conditions, which yielded cardiospheres (C-spheres), indicative of endogenous stem cell capacity. C-sphere counts generated per 5 mm3 tissue explant and the presence of cardiac progenitor cells were correlated to pa…

0301 basic medicineHeart Septal Defects VentricularAgingHeart diseaseCell TransplantationCardiovascular Proceduresmedicine.medical_treatmentCardiomyopathylcsh:Medicine030204 cardiovascular system & hematologyBiochemistryHypoplastic left heart syndromeTissue Culture TechniquesElectrocardiography0302 clinical medicineAnimal CellsHeart RegenerationHypoplastic Left Heart SyndromeNeurobiology of Disease and RegenerationMedicine and Health SciencesMorphogenesisBlood and Lymphatic System ProceduresMyocytes CardiacChildlcsh:ScienceCells CulturedTetralogy of FallotMultidisciplinaryStem CellsStem Cell TherapyDilated cardiomyopathyHeartStem-cell therapyCardiac Transplantationmedicine.anatomical_structureNeurologyChild PreschoolCardiologyTetralogy of Fallotcardiovascular systemStem cellCellular TypesAnatomyResearch ArticleCardiomyopathy Dilatedmedicine.medical_specialtyAdolescentHeart VentriclesSurgical and Invasive Medical Procedures03 medical and health sciencesInternal medicinemedicineHumansRegenerationVimentincardiovascular diseasesClinical GeneticsTransplantationbusiness.industrylcsh:RInfant NewbornCorrectionInfantBiology and Life SciencesProteinsMesenchymal Stem CellsCell BiologyOrgan Transplantationmedicine.diseaseCytoskeletal Proteins030104 developmental biologyVentricleCardiovascular Anatomylcsh:QbusinessOrganism DevelopmentDevelopmental BiologyStem Cell TransplantationPLoS ONE
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Environmental Factors Such as Noise and Air Pollution and Vascular Disease

2020

Significance: According to the World Health Organization, noncommunicable diseases are the globally leading cause of mortality. Recent Advances: About 71% of 56 million deaths that occurred worldwide are due to noncommunicable cardiovascular risk factors, including tobacco smoking, unhealthy diets, lack of physical activity, overweight, arterial hypertension, diabetes, and hypercholesterolemia, which can be either avoided or substantially reduced. Critical Issues: Thus, it is estimated that 80% of premature heart disease, stroke, and diabetes can be prevented. More recent evidence indicates that environmental stressors such as noise and air pollution contribute significantly to the global b…

0301 basic medicineHeart diseasePhysiologyClinical BiochemistryDiseaseOverweightRisk AssessmentBiochemistry03 medical and health sciencesRisk FactorsAir PollutionDiabetes mellitusEnvironmental healthmedicineAnimalsHumansEndotheliumVascular DiseasesEndothelial dysfunctionMolecular BiologyGeneral Environmental Science030102 biochemistry & molecular biologybusiness.industryNoise pollutionVascular diseaseStressorEnvironmental ExposureCell Biologymedicine.diseaseOxidative Stress030104 developmental biologyCardiovascular DiseasesGeneral Earth and Planetary SciencesDisease Susceptibilitymedicine.symptomNoisebusinessAntioxidants & Redox Signaling
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Disentangling the effect of host genetics and gut microbiota on resistance to an intestinal parasite

2019

11 pages; International audience; Resistance to infection is a multifactorial trait, and recent work has suggested that the gut microbiota can also contribute to resistance. Here, we performed a fecal microbiota transplant to disentangle the contribution of the gut microbiota and host genetics as drivers of resistance to the intestinal nematode Heligmosomoides polygyrus. We transplanted the microbiota of a strain of mice (SJL), resistant to H. polygyrus, into a susceptible strain (CBA) and vice-versa. We predicted that if the microbiota shapes resistance to H. polygyrus, the FMT should reverse the pattern of resistance between the two host strains. The two host strains had different microbi…

0301 basic medicineHeligmosomoides polygyrusGut floramedicine.disease_causeFecal microbiota transplant0302 clinical medicinefluids and secretionsMESH: Fecal Microbiota TransplantationParasite hostingColonizationMESH: AnimalsMESH: Strongylida InfectionsDisease ResistanceGeneticsNematospiroides dubiusbiology[SDV.BA]Life Sciences [q-bio]/Animal biologyFecal Microbiota Transplantation3. Good healthInfectious DiseasesMESH: Nematospiroides dubiusGenetic Background030231 tropical medicineIntestinal parasiteHeterologousMice Inbred StrainsMESH: Disease ResistanceMESH: Host-Parasite InteractionsMESH: Mice Inbred Strainsdigestive systemMESH: Gastrointestinal MicrobiomeHost-Parasite Interactions03 medical and health sciencesImmunityparasitic diseasesmedicineAnimals[SDV.MP.PAR]Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyStrongylida InfectionsHost (biology)ImmunityLife history traitsMESH: Genetic Backgroundbiology.organism_classificationGastrointestinal MicrobiomeDisease Models Animalstomatognathic diseases030104 developmental biologyParasitologyHeligmosomoides polygyrusMESH: Disease Models Animal[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Gut-microbiota-derived extracellular vesicles: Overlooked mediators in host–helminth interactions?

2021

Helminth infections impact the composition of the mammalian gut microbiota; however, the mechanisms underpinning these interactions are, thus far, unknown. In this article, we propose that microbiota-derived extracellular vesicles might represent key players in host-helminth-microbiome crosstalk, and outline future directions to elucidate their role(s) in host-parasite relationships.

0301 basic medicineHelminth infectionsHost (biology)030231 tropical medicineHelminthiasisBiologyGut florabiology.organism_classificationdigestive systemExtracellular vesiclesGastrointestinal MicrobiomeHost-Parasite InteractionsCell biologyExtracellular Vesicles03 medical and health sciencesCrosstalk (biology)fluids and secretions030104 developmental biology0302 clinical medicineInfectious DiseasesHelminthsparasitic diseasesAnimalsHumansParasitologyTrends in Parasitology
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Th2 and Th1 Responses: Clear and Hidden Sides of Immunity Against Intestinal Helminths.

2017

Intestinal helminthiases affect millions of people worldwide, mainly in developing regions, where they cause a significant negative impact on human health and socioeconomic growth of affected populations. However, intestinal helminthiases are still among the most neglected tropical diseases. Protective immunity against intestinal helminths is associated with development of type 2 responses. Nevertheless, in some host–intestinal helminth combinations, local Th1 responses are initiated, inducing chronicity. The usage of helminth–mouse models is useful for elucidating the mechanisms behind the initiation of each type of response. Herein, the current knowledge on these topics is reviewed, payin…

0301 basic medicineHelminthiasisHelminthiasisBiologyHost-Parasite Interactions03 medical and health sciencesHuman health0302 clinical medicineImmune systemTh2 CellsImmunityHelminthsmedicineHelminthsAnimalsHumansDeveloping regionsIntestinal Diseases ParasiticTh1 Cellsmedicine.diseaseImmunity Humoral030104 developmental biologyInfectious DiseasesImmunologyNeglected tropical diseasesParasitologyTh1 response030215 immunologyTrends in parasitology
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Classic Models for New Perspectives: Delving into Helminth–Microbiota–Immune System Interactions

2018

Whilst a wealth of data indicate that infections by gastrointestinal helminths are accompanied by significant alterations in the composition of the vertebrate gut flora, little is known of the immune-molecular mechanisms that regulate host-parasite-microbiota interactions. 'Traditional' experimental models of gastrointestinal helminthiases, in which the role(s) of each of the components of this triad can be tested, provide an opportunity to advance research in this area. In this article, we propose the Echinostoma caproni-mouse system as a potentially useful tool for studies of the role of the host gut microbiota in preventing pathology and inducing parasite clearance via interleukin (IL)-2…

0301 basic medicineHelminthiasisHelminthiasisInterleukinBiologyGut floramedicine.diseasebiology.organism_classificationModels BiologicalHost-Parasite Interactions03 medical and health sciences030104 developmental biologyInfectious DiseasesImmune systemImmunological FactorsImmunityImmunologymedicineAnimalsHumansHelminthsParasitologyGut homeostasisTrends in Parasitology
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Genetic characterization of influenza viruses from influenza-related hospital admissions in the St. Petersburg and Valencia sites of the Global Influ…

2016

Abstract Background Continuous surveillance for genetic changes in circulating influenza viruses is needed to guide influenza prevention and control. Objectives To compare intra-seasonal influenza genetic diversity of hemagglutinin in influenza A strains isolated from influenza hospital admissions collected at two distinct sites during the same season. Study design Comparative phylogenetic analysis of full-length hemagglutinin genes from 77 isolated influenza A viruses from the St. Petersburg, Russian Federation and Valencia, Spain sites of the Global Influenza Hospital Surveillance Network (GIHSN) during the 2013/14 season. Results We found significant variability in A(H3N2) and A(H1N1)pdm…

0301 basic medicineHemagglutinin glycoproteinsMalevirusesHemagglutinin Glycoproteins Influenza Virusmedicine.disease_causeGlobal HealthRussia0302 clinical medicineGenomic sequencingInfluenza A Virus H1N1 SubtypeInfluenza A virusH3N2 subtype030212 general & internal medicineCladeChildPhylogenyAged 80 and overPhylogenetic treevirus diseasesMiddle AgedInfectious DiseasesInfluenza A virusChild PreschoolEpidemiological MonitoringRNA ViralFemaleSeasonsAdultAdolescentHemagglutinin (influenza)Genome ViralBiologyH5N1 genetic structure03 medical and health sciencesYoung AdultVirologyInfluenza preventionGenetic variationInfluenza HumanmedicineHumansAgedGenetic diversityInfluenza A Virus H3N2 SubtypeGenetic VariationInfantSequence Analysis DNAVirology030104 developmental biologyH1N1 subtypeSpainbiology.proteinInfluenza virusJournal of Clinical Virology
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Paroxysmal nocturnal haemoglobinuria: When delay in diagnosis and long therapy occurs

2017

Paroxysmal nocturnal hemoglobinuria (PNH) is a rare clonal disorder characterized by hemolytic anemia, bone marrow failure and thrombosis, caused by a somaticmutation in PIG-A gene that results in theabsence of CD55 and CD59, two important complement regulatory proteins. In thispaper, a case of PNH is retrospectively examined looking for clinical and laboratory features, and the entire course of the disease from the onset of the symptoms isdescribed, together with an adequate follow-up over a 7-years treatment period. Inthis case, the not specificity and the limited clinical relevance of the symptoms led to adelay in diagnosis. After thrombosis, Eculizumab therapy has been shown to be effec…

0301 basic medicineHemolytic anemiaPediatricsmedicine.medical_specialtyrenal failureParoxysmal nocturnal haemoglobinuriaparoxysmal nocturnal hemoglobinuriaCase ReportDiseaseCD5903 medical and health sciencesthrombotic eventshemic and lymphatic diseasesMedicineClinical significancebusiness.industrylcsh:RC633-647.5Bone marrow failureHematologylcsh:Diseases of the blood and blood-forming organsEculizumabEculizumabmedicine.diseaseThrombosisparoxysmal nocturnal hemoglo-binuria thrombotic events renal failure Eculizumab030104 developmental biologyParoxysmal nocturnal hemoglobinuriabusinessmedicine.drug
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Rab33B Controls Hepatitis B Virus Assembly by Regulating Core Membrane Association and Nucleocapsid Processing

2017

Many viruses take advantage of cellular trafficking machineries to assemble and release new infectious particles. Using RNA interference (RNAi), we demonstrate that the Golgi/autophagosome-associated Rab33B is required for hepatitis B virus (HBV) propagation in hepatoma cell lines. While Rab33B is dispensable for the secretion of HBV subviral envelope particles, its knockdown reduced the virus yield to 20% and inhibited nucleocapsid (NC) formation and/or NC trafficking. The overexpression of a GDP-restricted Rab33B mutant phenocopied the effect of deficit Rab33B, indicating that Rab33B-specific effector proteins may be involved. Moreover, we found that HBV replication enhanced Rab33B expres…

0301 basic medicineHepatitis B virusBiologymedicine.disease_causeVirusArticleCell LineCell membraneRab33B03 medical and health sciencesnucleocapsid assemblyTranscription (biology)RNA interferenceVirologymedicineHumansSecretionNucleocapsidcore/capsid membrane associationHepatitis B virus030102 biochemistry & molecular biologyEffectorVirus AssemblyCell MembraneVirologyHepatitis B Core Antigenshepatitis B virus; Rab GTPase; Rab33B; core/capsid membrane association; nucleocapsid assembly; virus traffickingTransport proteinProtein Transport030104 developmental biologyInfectious Diseasesmedicine.anatomical_structurevirus traffickingrab GTP-Binding ProteinsHost-Pathogen InteractionsHepatocytesRab GTPaseViruses; Volume 9; Issue 6; Pages: 157
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Failure on voxilaprevir, velpatasvir, sofosbuvir and efficacy of rescue therapy

2021

Background & Aims There are limited data on patients with chronic HCV infection in whom combination voxilaprevir (VOX), velpatasvir (VEL), sofosbuvir (SOF) retreatment fails. Thus, we aimed to assess treatment failure and rescue treatment options in these patients. Methods Samples from 40 patients with HCV genotypes (GT) 1-4 in whom VOX/VEL/SOF retreatment failed were collected within the European Resistance Study Group. Population-based resistance analyses were conducted and clinical parameters and retreatment efficacies were evaluated retrospectively in 22 patients. Results Most VOX/VEL/SOF failure patients were infected with HCV GT3a (n = 18, 45%) or GT1a (n = 11, 28%) and had cirrhosis …

0301 basic medicineHepatitis C Virusmedicine.medical_specialtySofosbuvirVoxilaprevirPopulationresistance-associated substitutionsDirect-acting antiviralVoxilaprevir/velpatasvir/sofosbuvir.GastroenterologySettore MED/07Telaprevir03 medical and health scienceschemistry.chemical_compound0302 clinical medicineVoxilaprevir/Velpatasvir/SofosbuvirInternal medicineBoceprevirRescue therapymedicineResistance-associated substitutioneducationdirect-acting antiviralsDAAeducation.field_of_studyHepatologybusiness.industryvirus diseasesGlecaprevirDAA; HCV; Hepatitis C Virus; Voxilaprevir/Velpatasvir/Sofosbuvir; direct-acting antivirals; rescue therapy; resistance-associated substitutionsdigestive system diseasesPibrentasvirRegimen030104 developmental biologychemistryHCV030211 gastroenterology & hepatologyHepatitis C virubusinessmedicine.drugJournal of Hepatology
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