Search results for "Dative"

showing 10 items of 2381 documents

Acute effects on the human EEG after an external exposure to 200 ppm methanol

2001

Objectives: Even low concentrations of organic solvents may cause acute effects on the human central nervous system. The German MAK (threshold limit value) of methanol is 200 ppm. The aim of this study was to investigate whether acute exposure to 200 ppm methanol causes adverse effects, measured by EEG, and moreover, whether it is possible to differentiate between sedative and excitatory effects with this method. Methods: Twelve healthy subjects were exposed for 4 h to 200 ppm and to 20 ppm (control) in an exposure chamber in a cross-over design. The EEG was recorded before (reference) and at the end of each exposure with, the subject's eyes closed and opened and during a choice reaction te…

AdultCentral Nervous SystemMaleAcute effectsmedicine.drug_classThreshold limit valueElectroencephalographychemistry.chemical_compoundReference ValuesHumansMedicineSingle-Blind MethodThreshold Limit ValuesInhalation ExposureCross-Over Studiesmedicine.diagnostic_testInhalationbusiness.industryMethanolPublic Health Environmental and Occupational HealthElectroencephalographychemistrySedativeAnesthesiaToxicityExposure chamberMethanolbusinessInternational Archives of Occupational and Environmental Health
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Acute, subchronic and discontinuation effects of zopiclone on sleep EEG and nocturnal melatonin secretion

1996

Zopiclone is a new short half-life cyclopyrrolone hypnotic agent acting at the GABA-benzodiazepine receptor complex. In order to characterize its pharmacological profile, the effects of 7.5 mg zopiclone on nocturnal melatonin secretion were investigated under polysomnographic control in 11 healthy subjects following acute and subchronic administration as well as after abrupt discontinuation of the drug. No effect of zopiclone on the melatonin plasma levels could be observed. Regarding both total melatonin production and the temporal pattern of melatonin secretion during the night, there was no difference between placebo baseline condition, acute and subchronic administration, and discontinu…

AdultDrugReceptor complexmedicine.medical_specialtyTime Factorsmedicine.drug_classmedia_common.quotation_subjectPharmacologyPlaceboPiperazinesHypnoticMelatoninInternal medicinemedicineHumansHypnotics and SedativesPharmacology (medical)ChildBiological PsychiatryMelatoninmedia_commonPharmacologyZopicloneElectroencephalographyDiscontinuationPsychiatry and Mental healthEndocrinologyNeurologyPharmacodynamicsNeurology (clinical)SleepPsychologyAzabicyclo Compoundsmedicine.drugEuropean Neuropsychopharmacology
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Erythrocyte Membrane Phosphatidylserine Exposure in Obesity

2008

It has been suggested that increased erythrocyte membrane phosphatidylserine (PS) exposure could contribute to hypercoagulability and hemorheological disturbances in obesity. The aim of our study was to evaluate PS exposure in obese patients and in a control group and to correlate this with hemorheological properties, i.e., erythrocyte aggregability (EA) and deformability, and to evaluate the effect of weight loss on these parameters. An anthropometric and analytical evaluation was performed at baseline and after 3 months on a diet (very low-calorie diet for 4 weeks and low-calorie diet for 2 months) on 49 severe or morbid obese patients (37 women, 12 men) and 55 healthy volunteers (39 wome…

AdultErythrocyte AggregationMalemedicine.medical_specialtyAdolescentEndocrinology Diabetes and MetabolismMedicine (miscellaneous)Phosphatidylserinesmedicine.disease_causeErythrocyte aggregationFlow cytometrychemistry.chemical_compoundYoung AdultEndocrinologyWeight lossInternal medicineErythrocyte DeformabilityMalondialdehydeWeight LossMedicineErythrocyte deformabilityHumansObesityProtein PrecursorsNutrition and Dieteticsmedicine.diagnostic_testbusiness.industryErythrocyte MembranePhosphatidylserineMiddle AgedMalondialdehydePathophysiologyPeptide FragmentsOxidative StressEndocrinologychemistryCase-Control StudiesImmunologyFemaleProthrombinmedicine.symptombusinessOxidative stress
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Potential effects of age-associated oxidative stress on mammalian oocytes/embryos

1996

This bioessay aims to explain the different effects of maternal ageing and postovulatory oocyte ageing on mammalian oocytes/embryos under the scope of 'the oxygen radical-mitochondrial injury hypothesis of ageing'. This hypothesis assumes a key role in the senescent process of oxygen radical damage to mitochondrial DNA, proteins and lipids. It is proposed that a decrease in intracellular ATP concentrations and glutathione (GSH)/glutathione disulphide (GSSG) ratio together with a concomitant increase in cytosolic Ca2+ are major factors causing the observed detrimental effects of ageing on cytoskeletal fibres, fertilization and embryo development.

AdultFetal ProteinsEmbryologyBiologymedicine.disease_causeDNA MitochondrialCongenital AbnormalitiesMicechemistry.chemical_compoundAdenosine TriphosphateNeoplasmsGeneticsmedicineAnimalsHumansMolecular BiologyCellular SenescenceCytoskeletonMammalsEgg ProteinsEmbryogenesisObstetrics and GynecologyEmbryoCell BiologyGlutathioneEmbryo MammalianOocyteGlutathioneCell biologyOxidative StressCytosolFertilitymedicine.anatomical_structureReproductive MedicineBiochemistrychemistryAgeingFertilizationOocytesReactive Oxygen SpeciesOxidation-ReductionIntracellularOxidative stressMaternal AgeDevelopmental BiologyMolecular Human Reproduction
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Differences in the behavior of advanced glycation end products and advanced oxidation protein products in patients with allergic rhinitis

2013

BACKGROUND: The presence of oxidative stress in patients with asthma is well documented; however, the role of oxidative stress in allergic rhinitis has received less attention, although it is likely to be similar to that observed in patients with asthma. Advanced glycation end products (AGEs) and advanced oxidation protein products (AOPPs) are compounds formed by the transformation of macromolecules, including proteins, which can serve as densitometric markers of oxidative stress and inflammation in several diseases. OBJECTIVE: The aim of this study was to investigate the role of AGEs and AOPPs as new markers of oxidative stress and inflammation in patients affected by allergic rhinitis. ME…

AdultGlycation End Products AdvancedMaleRhinitis Allergic PerennialSettore MED/09 - Medicina InternaAllergyAllergic rhinitisAllergic rhinitiHumansAdvanced glycation end productsAgedPeroxidaseAdvanced oxidation protein productImmunoglobulin EMiddle AgedAdvanced glycation end products.; Advanced oxidation protein products; Allergic rhinitis; Oxidative stress.; AllergyRhinitis AllergicOxidative StressSpectrometry FluorescenceAdvanced Oxidation Protein ProductsSpectrophotometryCase-Control StudiesOxidative stress: AllergyFemaleAdvanced glycation end productOxidation-ReductionBiomarkers
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Lipid peroxidation products and antioxidants in human disease.

1998

Lipid peroxidation (LPO) is a free radical-related process that in biologic systems may occur under enzymatic control, e.g., for the generation of lipid-derived inflammatory mediators, or nonenzymatically. This latter form is associated mostly with cellular damage as a result of oxidative stress, which also involves cellular antioxidants in this process. This article focuses on the relevance of two LPO products, malondialdehyde (MDA) and 4-hydroxynonenal (HNE), to the pathophysiology of human disease. The former has been studied in human serum samples of hepatitis C virus-infected adults and human immunodeficiency virus-infected children. In these two cases it is shown that the specific ass…

AdultHealth Toxicology and MutagenesisHIV InfectionsPharmacologyBiologymedicine.disease_causeAntioxidantsLipid peroxidationMicechemistry.chemical_compoundMalondialdehydemedicineAnimalsHumansDiseaseChildchemistry.chemical_classificationAldehydesGlutathione PeroxidaseGlutathione peroxidasePublic Health Environmental and Occupational HealthGlutathioneMalondialdehydeHepatitis CIn vitroPathophysiologyRatsOxidative StressEnzymechemistryImmunologyBlood VesselsLipid PeroxidationOxidative stressResearch ArticleEnvironmental Health Perspectives
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Sprint and endurance training in relation to redox balance, inflammatory status and biomarkers of aging in master athletes

2019

Purpose: Studies have shown a positive influence of intense athletic training on several biomarkers of aging, but it remains unclear whether this influence is dependent of exercise-training-mode. This study compared redox balance, cytokine levels and biomarkers of aging between master sprinters and endurance athletes, as well as in young and middle-aged individuals as controls. Methods: Participants were male master sprinters (SA, 50±8.9yrs; n=13) and endurance runners (EA, 53±8.2yrs; n=18) with remarkable athletic experience (∼25yrs of practice), besides untrained young (YC, 22.7±3.9yrs; n=17) and age-matched controls (MC, 45.5±9.8yrs; n=12). Anamnesis, anthropometrics, biomarkers of aging…

AdultMale0301 basic medicineAgingCancer Researchmedicine.medical_specialtyPhysiologymedicine.medical_treatmentClinical BiochemistryInflammation030204 cardiovascular system & hematologymedicine.disease_causeBiochemistryRunningYoung Adult03 medical and health sciences0302 clinical medicineBiomarkers of agingEndurance trainingInternal medicineHumansMedicineKlothoAgedInflammationbiologyAthletesbusiness.industryMiddle Agedbiology.organism_classificationEndurance TrainingFibroblast Growth Factor-23Cross-Sectional Studies030104 developmental biologyEndocrinologyCytokineSprintAthletesmedicine.symptombusinessOxidation-ReductionBiomarkersOxidative stressNitric Oxide
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Assessment of the cytotoxic potential of an aqueous-ethanolic extract from Thalassia testudinum angiosperm marine grown in the Caribbean Sea

2018

Abstract Objectives Reported antioxidant, anti-inflammatory and neuroprotective properties for one aqueous-ethanolic extract from Thalassia testudinum which grows in the Caribbean Sea compelled us to explore about extract cytotoxic effects. Methods Cell viability was assayed on tumour (HepG2, PC12, Caco-2 and 4T1) and non-tumour (VERO, 3T3, CHO, MCDK and BHK2) cell lines. The extract effects upon primary cultures of rat and human hepatocytes and human lymphocytes were assayed. Key findings The extract exhibited cytotoxicity against cancer cells compared to normal cells, and the IC50 values were 102 μg/ml for HepG2, 135 μg/ml for PC12, 165 μg/ml for Caco-2 and 129 μg/ml for 4T1 cells after 4…

AdultMale0301 basic medicineCell SurvivalDNA damagePrimary Cell CulturePharmaceutical ScienceHydrocharitaceaePC12 CellsRats Sprague-DawleyInhibitory Concentration 5003 medical and health sciencesNeoplasmsAnimalsHumansCytotoxic T cellLymphocytesViability assayCytotoxicityPharmacologyDose-Response Relationship DrugEthanolbiologyPlant ExtractsChemistryPharmacology. TherapyWaterHep G2 Cellsbiology.organism_classificationAntineoplastic Agents PhytogenicMolecular biologyRatsComet assay030104 developmental biologyCaribbean RegionCell cultureThalassia testudinumCancer cellSolventsCaco-2 CellsThalassia testudinum DNA damage cytotoxicity oxidative stressJournal of pharmacy and pharmacology
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Assessing the risk of cytomegalovirus DNAaemia in allogeneic stem cell transplant recipients by monitoring oxidative-stress markers in plasma

2017

The level of antioxidants, such as thiol-containing tripeptide glutathione (GSH), in cytomegalovirus (CMV)-infected cells is notably increased. We previously showed that GSH levels in plasma, as measured by untargeted 1H nuclear magnetic resonance, are higher in allogeneic stem cell transplant (allo-SCT) recipients who subsequently develop CMV viraemia. We hypothesized that the net level of oxidative-stress markers present in plasma may be reduced in patients who develop CMV DNAaemia compared to those who do not. We serially monitored the levels of malondialdehyde (MDA) and carbonylated proteins (CPs) early after allo-SCT and assessed whether they could predict the occurrence of CMV DNAaemi…

AdultMale0301 basic medicineCongenital cytomegalovirus infectionCytomegalovirusBiologymedicine.disease_causeAntioxidantsProtein Carbonylation03 medical and health scienceschemistry.chemical_compound0302 clinical medicineMalondialdehydeVirologymedicineHumansViremiaAgedArea under the curvevirus diseasesGlutathioneMiddle AgedViral LoadMalondialdehydemedicine.diseaseGlutathioneVirologyConfidence intervalOxidative Stress030104 developmental biologyROC CurvechemistryHematologic Neoplasms030220 oncology & carcinogenesisCytomegalovirus InfectionsDNA ViralImmunologyBiomarker (medicine)FemaleStem cellOxidative stressStem Cell TransplantationJournal of General Virology
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Blood levels of nitric oxide and DNA breaks assayed in whole blood and isolated peripheral blood mononucleated cells in patients with multiple sclero…

2019

Abstract Oxidative stress, especially overproduction of nitric oxide (NO), is considered to be one of the crucial factors in the pathogenesis of multifactorial multiple sclerosis (MS). DNA breaks could be one of the consequences of oxidative stress; however, data on DNA breakage in MS are very few and contradictory. There are no data on direct measurements of NO production in the blood of MS patients. The goal of this study was to determine the level of single-stranded DNA breaks in whole blood or isolated peripheral blood mononuclear cells (PBMNCs) by means of alkaline single cell gel electrophoresis (comet assay) and to evaluate production of NO in the human blood by applying electron par…

AdultMale0301 basic medicineMultiple SclerosisDNA damageHealth Toxicology and Mutagenesis010501 environmental sciencesNitric Oxidemedicine.disease_cause01 natural sciencesPeripheral blood mononuclear cellNitric oxide03 medical and health scienceschemistry.chemical_compoundGeneticsmedicineHumansDNA Breaks Single-StrandedAged0105 earth and related environmental sciencesWhole bloodGel electrophoresisChemistryMultiple sclerosisElectron Spin Resonance SpectroscopyMiddle Agedmedicine.diseaseMolecular biologyComet assay030104 developmental biologyLeukocytes MononuclearFemaleComet AssaySingle-Cell AnalysisOxidative stressDNA DamageMutation Research/Genetic Toxicology and Environmental Mutagenesis
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