Search results for "Dendritic cell"

showing 10 items of 447 documents

Plasmacytoid dendritic cells promote systemic sclerosis with a key role for TLR8

2018

Systemic sclerosis (SSc) is a multisystem life-threatening fibrosing disorder that lacks effective treatment. The link between the inflammation observed in organs such as the skin and profibrotic mechanisms is not well understood. The plasmacytoid dendritic cell (pDC) is a key cell type mediating Toll-like receptor (TLR)-induced inflammation in autoimmune disease patients, including lupus and skin diseases with interface dermatitis. However, the role of pDCs in fibrosis is less clear. We show that pDCs infiltrate the skin of SSc patients and are chronically activated, leading to secretion of interferon-α (IFN-α) and CXCL4, which are both hallmarks of the disease. We demonstrate that the s…

0301 basic medicineMaleInflammationPlasmacytoid dendritic cellPlatelet Factor 4SclerodermaArticlePathogenesis03 medical and health sciencesBleomycinMice0302 clinical medicineFibrosismedicineAnimalsHumansSkin030203 arthritis & rheumatologyAutoimmune diseaseSystemic lupus erythematosusScleroderma Systemicintegumentary systembusiness.industryMedicine (all)Interferon-alphahemic and immune systemsGeneral MedicineTLR7Dendritic CellsMiddle Agedmedicine.diseaseFibrosisDisease Models Animal030104 developmental biologyToll-Like Receptor 7Toll-Like Receptor 8ImmunologyFemalemedicine.symptombusinessSignal Transduction
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Vaccination with trifunctional nanoparticles that address CD8+ dendritic cells inhibits growth of established melanoma

2016

Aim: We wanted to assess the potency of a trifunctional nanoparticle (NP) that targeted and activated CD8+ dendritic cells (DC) and delivered an antigen to induce antitumor responses. Materials & methods: The DC targeting and activating properties of ferrous NPs conjugated with immunostimulatory CpG-oligonucleotides, anti-DEC205 antibody and ovalbumin (OVA) as a model antigen to induce antigen-specific T-cell responses and antitumor responses were analyzed. Results: OVA-loaded NP conjugated with immunostimulatory CpG-oligonucleotides and anti-DEC205 antibody efficiently targeted and activated CD8+ DC in vivo, and induced strong OVA-specific T-cell activation. Vaccination of B16/OVA tum…

0301 basic medicineMaterials sciencebiologyBiomedical EngineeringMedicine (miscellaneous)BioengineeringDendritic cellDevelopmentMolecular biology03 medical and health sciencesCTL*Ovalbumin030104 developmental biology0302 clinical medicineAntigenIn vivoCancer researchbiology.proteinGeneral Materials ScienceAntibodyNanocarriersCD8030215 immunologyNanomedicine
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Myeloid cells as orchestrators of the tumor microenvironment: novel targets for nanoparticular cancer therapy.

2016

Macrophages, myeloid-derived suppressor cells and tolerogenic dendritic cells are central players of a heterogeneous myeloid cell population, with the ability to suppress innate and adaptive immune responses and thus to promote tumor growth. Their influx and local proliferation are mainly induced by the cancers themselves, and their numbers in the tumor microenvironment and the peripheral blood correlate with decreased survival. Therapeutic targeting these innate immune cells, either aiming at their elimination or polarization toward tumor suppressive cells is an attractive novel approach to control tumor progression and block metastasis. We review the current understanding of cancer immun…

0301 basic medicineMyeloidPolymersmedicine.medical_treatmentPopulationBiomedical EngineeringMedicine (miscellaneous)BioengineeringDevelopmentBiology03 medical and health sciences0302 clinical medicineImmune systemNeoplasmsmedicineTumor MicroenvironmentAnimalsHumansGeneral Materials ScienceMyeloid CellsRNA Small InterferingeducationCancer immunologyeducation.field_of_studyTumor microenvironmentDrug CarriersInnate immune systemMacrophagesMyeloid-Derived Suppressor CellsImmunotherapyDendritic CellsImmunity Innate030104 developmental biologymedicine.anatomical_structureTumor progression030220 oncology & carcinogenesisImmunologyNanoparticlesImmunotherapyNanomedicine (London, England)
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Unusual presentation of blastic plasmacytoid dendritic cell neoplasm: Pitfalls in other hematolymphoid neoplasms

2020

Abstract Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is a rare CD4+/CD56+ hematological malignancy with aggressive clinical course and poor prognosis. Histologically, BPDCN is characterized by a diffuse, monomorphous infiltration of cutaneous, subcutaneous, and sometimes other tissues such as lymph nodes and bone marrow, by medium-sized neoplastic cells with blastoid morphology. Typically, there is absence of lymphocytic infiltrate. Diagnosis relies on immunophenotypic expression of CD4, CD56, and the more specific markers of plasmacytoid dendritic cells CD123, CD303/BDCA2, and TCL1. We report a case of a 57-year-old man who presented a 4 cm-long solitary, erythemateous lesion on t…

0301 basic medicinePathologymedicine.medical_specialtyMyeloidMYCASXL1BlastoidPathology and Forensic MedicineLymphocytic Infiltrate03 medical and health sciences0302 clinical medicineBlastic plasmacytoid dendritic cell neoplasmT-cells infiltrationlcsh:PathologyMedicineTET2biologybusiness.industryETV6medicine.diseasebiology.organism_classificationLymphomaETV6030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisInterleukin-3 receptorBone marrowbusinessInfiltration (medical)lcsh:RB1-214Human Pathology: Case Reports
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In Situ Activation of Pituitary-Infiltrating T Lymphocytes in Autoimmune Hypophysitis

2017

AbstractAutoimmune hypophysitis (AH) is a chronic inflammatory disease characterized by infiltration of T and B lymphocytes in the pituitary gland. The mechanisms through which infiltrating lymphocytes cause disease remain unknown. Using a mouse model of AH we assessed whether T lymphocytes undergo activation in the pituitary gland. Infiltrating T cells co-localized with dendritic cells in the pituitary and produced increased levels of interferon-γ and interleukin-17 upon stimulation in vitro. Assessing proliferation of CD3- and B220-postive lymphocytes by double immunohistochemistry (PCNA-staining) and flow cytometry (BrdU incorporation) revealed that a discrete proportion of infiltrating …

0301 basic medicinePathologymedicine.medical_specialtyPituitary glandT-LymphocytesT cellCD3Lymphocyte ActivationArticleImmunophenotypingFlow cytometryMice03 medical and health sciences0302 clinical medicineImmunophenotypingParenchymamedicineAnimalsAutoimmune HypophysitisLymphocyte CountCell ProliferationB-LymphocytesMultidisciplinarymedicine.diagnostic_testbiologybusiness.industryDendritic Cellsmedicine.diseaseImmunohistochemistry3. Good healthDisease Models Animal030104 developmental biologymedicine.anatomical_structurePituitary Gland030220 oncology & carcinogenesisbiology.proteinAutoimmune hypophysitisCytokinesFemaleInflammation MediatorsbusinessInfiltration (medical)BiomarkersScientific Reports
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Parasites Dampen Dendritic Cell Activation to Ensure Their Survival

2017

Dendritic cells (DCs) are critical for induction of protective immunity against Leishmania major. However, DC activation occurs only several weeks after parasite transmission. Parasites synthesize a macrophage-inducible C-type lectin (Mincle) ligand. Engagement of Mincle by the parasite ligand dampens DC activation, thus delaying induction of interferon-γ-producing T cells responsible for parasite eradication.

0301 basic medicineProtective immunityLigandLectinmacromolecular substancesDendritic cellBiologybiology.organism_classificationCell biology03 medical and health sciences030104 developmental biologyInfectious Diseasesbiology.proteinParasite hostingParasitologyParasite transmissionLeishmania majorTrends in Parasitology
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Down-Regulation of CD62L Shedding in T Cells by CD39+ Regulatory T Cells Leads to Defective Sensitization in Contact Hypersensitivity Reactions

2016

Injection of regulatory T cells (Tregs) followed by sensitization with 2,4,6-trinitrochlorobenzene induced a transient increase in size and cellularity of skin-draining lymph nodes (LNs) in mice. This led us to hypothesize that Tregs may affect the trafficking of T cells from and to peripheral LNs. Two to three hours after sensitization, we found fewer CD8+ T cells expressing CD62L in LNs compared with untreated controls. Injection of wild-type Tregs prevented this down-regulation of CD62L. In contrast, Tregs devoid of the adenosine triphosphate (ATP)-degrading ecto-enzyme CD39 were unable to do so. As for the mechanism of CD62L regulation, we found that ATP, which is released in skin upon …

0301 basic medicineRegulatory T cellBlotting WesternMedizinDown-Regulationchemical and pharmacologic phenomenaDermatologyT-Lymphocytes RegulatoryBiochemistryArticleMice03 medical and health scienceschemistry.chemical_compoundAdenosine Triphosphate0302 clinical medicineImmune systemDownregulation and upregulationAntigenAntigens CDReference ValuesmedicineAnimalsImmunologic FactorsL-SelectinMolecular BiologyCells CulturedSensitizationintegumentary systemChemistryApyrasehemic and immune systemsCell BiologyDendritic cellFlow CytometryCell biologyMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureEpidermal CellsDermatitis Allergic ContactImmunologyImmunizationLymph NodesEpidermisAdenosine triphosphateCD8030215 immunologyJournal of Investigative Dermatology
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Enterococcus hirae and Barnesiella intestinihominis Facilitate Cyclophosphamide-Induced Therapeutic Immunomodulatory Effects.

2016

International audience; The efficacy of the anti-cancer immunomodulatory agent cyclophosphamide (CTX) relies on intestinal bacteria. How and which relevant bacterial species are involved in tumor immunosurveillance, and their mechanism of action are unclear. Here, we identified two bacterial species, Enterococcus hirae and Barnesiella intestinihominis that are involved during CTX therapy. Whereas E. hirae translocated from the small intestine to secondary lymphoid organs and increased the intratumoral CD8/ Treg ratio, B. intestinihominis accumulated in the colon and promoted the infiltration of IFN-gamma-producing gamma delta Tau cells in cancer lesions. The immune sensor, NOD2, limited CTX…

0301 basic medicineRichnessNod2 Signaling Adaptor Proteinmedicine.disease_causeMice0302 clinical medicineEnterococcus hiraeNOD2NeoplasmsIntestine Small[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyImmunology and AllergyGut MicrobiotaCancerbiology3. Good healthImmunosurveillanceInfectious Diseases030220 oncology & carcinogenesisBarnesiella intestinihominis[SDV.IMM]Life Sciences [q-bio]/ImmunologyImmunotherapymedicine.symptomInfectionmedicine.drugCyclophosphamideColonImmunologyTranslocationInflammation03 medical and health sciencesInterferon-gammaImmune systemMonitoring ImmunologicmedicineAnimalsImmunologic FactorsCyclophosphamideInflammationEnterococcus hiraeAntitumor ImmunityBacteriaDendritic CellsTh1 Cellsmedicine.diseasebiology.organism_classificationMice Inbred C57BL030104 developmental biologyIntestinal MicrobiotaImmunologyOvarian cancerImmunologic MemoryImmunity
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Nutritional control of IL-23/Th17-mediated autoimmune disease through HO-1/STAT3 activation

2017

AbstractThe nutritional curcumin (CUR) is beneficial in cell-mediated autoimmune diseases. The molecular mechanisms underlying this food-mediated silencing of inflammatory immune responses are poorly understood. By investigating antigen-specific immune responses we found that dietary CUR impairs the differentiation of Th1/Th17 cells in vivo during encephalomyelitis and instead promoted Th2 cells. In contrast, feeding CUR had no inhibitory effect on ovalbumin-induced airway inflammation. Mechanistically, we found that CUR induces an anti-inflammatory phenotype in dendritic cells (DC) with enhanced STAT3 phosphorylation and suppressed expression of Il12b and Il23a. On the molecular level CUR …

0301 basic medicineSTAT3 Transcription FactorCurcuminEncephalomyelitis Autoimmune ExperimentalOvalbuminEncephalomyelitisInterleukin-23ArticleAutoimmune Diseases03 medical and health sciencesMiceImmune systemTh2 CellsmedicineInterleukin 23Gene silencingAnimalsPhosphorylationSTAT3Autoimmune diseaseInflammationImmunity CellularMultidisciplinarybiologyChemistryMembrane ProteinsDendritic Cellsmedicine.diseaseCell biologyHeme oxygenase030104 developmental biologybiology.proteinPhosphorylationTh17 CellsHeme Oxygenase-1Scientific Reports
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SPARC regulation of PMN clearance protects from pristane-induced lupus and rheumatoid arthritis

2021

Summary The secreted protein acidic and rich in cysteine (SPARC) is a matricellular protein with unexpected immunosuppressive function in myeloid cells. We investigated the role of SPARC in autoimmunity using the pristane-induced model of lupus that, in mice, mimics human systemic lupus erythematosus (SLE). Sparc−/− mice developed earlier and more severe renal disease, multi-organ parenchymal damage, and arthritis than the wild-type counterpart. Sparc+/- heterozygous mice showed an intermediate phenotype suggesting Sparc gene dosage in autoimmune-related events. Mechanistically, reduced Sparc expression in neutrophils blocks their clearance by macrophages, through defective delivery of don'…

0301 basic medicineScienceImmunologyArthritis02 engineering and technologySettore MED/08 - Anatomia Patologicamedicine.disease_causeArticleAutoimmunityPathogenesis03 medical and health sciencesmedicineSettore MED/05 - Patologia ClinicaMacrophageMolecular physiologyMultidisciplinarySystemic lupus erythematosusbusiness.industryQMatricellular proteinDendritic cell021001 nanoscience & nanotechnologymedicine.diseaseBiological sciences Immunology Molecular physiologyBiological sciences030104 developmental biologyRheumatoid arthritisCancer research0210 nano-technologybusinessiScience
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