Search results for "Desmin"

showing 10 items of 77 documents

Regeneration of lingual musculature in rats using myoblasts over porcine bladder acellular matrix

2020

To use tissue engineering muscle repair (TEMR) for regenerating the lingual musculature of hemiglossectomized rats using neonatal myoblasts (NM) on porcine acellular urinary bladder matrix (AUBM).The study used 80 male rats. A volumetric muscle loss (VML) injury was created on the left side of the tongue. The rats were randomized into four groups: Group 1 (AUBM + myoblasts); Group 2 (AUBM); Group 3 (myoblasts); and Group 4 (control). NM were obtained from neonatal rats. The animals were weighed on day 0 and just before euthanasia. Five rats in each group were euthanized at days 2, 14, 28, and 42; the tongues were prepared for morphometric analysis, postoperative left hemitongue weight, and …

MaleSwineUrinary BladderMatrix (biology)Myoblasts03 medical and health sciences0302 clinical medicineTongueTissue engineeringTonguemedicineAnimalsRegenerationMyocyteGeneral DentistryUrinary bladderTissue Engineeringbusiness.industryRegeneration (biology)030206 dentistryAnatomymusculoskeletal systemRatsmedicine.anatomical_structureOtorhinolaryngology030220 oncology & carcinogenesisImmunohistochemistryDesminbusinessOral Diseases
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Controlled intermittent shortening contractions of a muscle-tendon complex: muscle fibre damage and effects on force transmission from a single head …

2005

This study was performed to examine effects of prolonged (3 h) intermittent shortening (amplitude 2 mm) contractions (muscles were excited maximally) of head III of rat extensor digitorum longus muscle (EDL III) on indices of muscle damage and on force transmission within the intact anterior crural compartment. Three hours after the EDL III exercise, muscle fibre damage, as assessed by immunohistochemical staining of structural proteins (i.e. dystrophin, desmin, titin, laminin-2), was found in EDL, tibialis anterior (TA) and extensor hallucis longus (EHL) muscles. The damaged muscle fibres were not uniformly distributed throughout the muscle cross-sections, but were located predominantly ne…

MaleTime Factorsanimal structuresPhysiologyMolecular Sequence DataMuscle Fibers SkeletalVesicular Transport ProteinsMuscle ProteinsBiochemistryDesminExtensor digitorum longus muscleTendonsSDG 3 - Good Health and Well-beingmedicineAnimalsRNA MessengerMuscle fibreRats WistarMuscle SkeletalbiologyChemistryReverse Transcriptase Polymerase Chain ReactionMETIS-229512Compartment (ship)Gene Expression Profilingmusculoskeletal neural and ocular physiologyMembrane ProteinsCell BiologyAnatomyOrgan Sizemusculoskeletal systemTendonRatsSlack lengthmedicine.anatomical_structureIR-104003Models Animalbiology.protein/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingTitinDesminStress MechanicalDystrophintissuesMuscle Contraction
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Cytogenetic study of a spindle-cell rhabdomyosarcoma of the parotid gland.

1999

The cytogenetic analysis of a spindle-cell rhabdomyosarcoma of the parotid gland in a 6-year-old boy is reported. The tumor cells showed an abnormal karyotype with a hypotriploid modal chromosome number and clonal structural rearrangements affecting chromosomes 1, 8, 12, 21, and 22. The tumor karyotype was: 59, XY, -1, -3, -4, -5, -6, +8, +8, +del(8)(q22q24), -9, -10, del(12)(q13), -15, -16, -17, -18, der(21)t(12;21)(p11;p11), -22, der(22)t(1;22)(q12;p11).

Malecongenital hereditary and neonatal diseases and abnormalitiesCancer Researchmedicine.medical_specialtyPathologyTransplantation HeterologousMice NudeBiologyMyosinsDesminMiceRhabdomyosarcomaGeneticsmedicineAnimalsChromosomes HumanHumansVimentinRhabdomyosarcomaSpindle cell rhabdomyosarcomaChildMolecular BiologyChromosome AberrationsMyoglobinCytogeneticsKaryotypeModal Chromosome NumberAnatomymedicine.diseaseImmunohistochemistryActinsParotid glandParotid Neoplasmsmedicine.anatomical_structureKaryotypingImmunohistochemistrySarcomaNeoplasm TransplantationCancer genetics and cytogenetics
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Prophylactic Implantable Cardioverter Defibrillator Placement in a Sporadic Desmin Related Myopathy and Cardiomyopathy

2004

Desminopathy is a neuromuscular disorder associated with the accumulation of the protein desmin. This article reports a case of a man with a mutation in the desmin gene suffering from cardiomyopathy and skeletal myopathy. This patient underwent implantable cardioverter defibrillator (ICD) implantation for prognostic considerations and subsequently developed a sustained ventricular tachycardia (SVT). While nonsustained VTs (NSVT) have previously been reported, this is the first time that a SVT could be seen in a patient with this disease.

Malemedicine.medical_specialtyAdolescentmedicine.medical_treatmentCardiomyopathyDiseaseDesminInternal medicineHumansMedicineDESMIN-RELATED MYOPATHYbusiness.industryImplantable Cardioverter-Defibrillator PlacementGeneral MedicineImplantable cardioverter-defibrillatormedicine.diseaseSkeletal myopathyDefibrillators ImplantableSustained ventricular tachycardiaMutationTachycardia VentricularCardiologyDesminCardiomyopathiesCardiology and Cardiovascular MedicinebusinessMyopathies Structural CongenitalPacing and Clinical Electrophysiology
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Arrhythmic sudden cardiac death in a 3-year-old child with intimal fibroplasia of coronary arteries, aorta, and its branches

2001

Abstract We report an unusual case of “arrhythmic” sudden cardiac death in a 3-year-old child who died of ischemic myocardial lesions as a result of intimal fibroplasia of the coronary arteries. Also affected were the aorta and its major branches, whereas renal and mesenteric arteries, celiac trunk, and systemic veins were normal. Histopathologic examination showed severe concentric thickening of intima because of a proliferation of spindle-shaped cells (mesenchymal cells) set in an abundant extracellular matrix. In some vascular segments the intima was densely fibrotic and hyalinized. No significant inflammation, foam cells, cholesterol clefts, or other evidence of atheroma were present. T…

Malemedicine.medical_specialtyHeart VentriclesFibromuscular dysplasiaPathology and Forensic MedicineSudden cardiac deathPolypsAdventitiaInternal medicinemedicine.arterymedicineFibromuscular DysplasiaHumansVimentinMesenteric arteriesAortaAortabusiness.industryMyocardiumArrhythmias CardiacGeneral Medicinemedicine.diseaseCoronary VesselsImmunohistochemistryActinsCoronary arteriesDeath Sudden CardiacAtheromamedicine.anatomical_structureChild Preschoolcardiovascular systemCardiologyDesminTunica IntimaCardiology and Cardiovascular MedicinebusinessCardiovascular Pathology
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The disruption of myofibre structures in rat skeletal muscle after forced lengthening contractions.

1998

Specific antibodies against structural proteins (actin, desmin, dystrophin, fibronectin) of muscle fibres were used to study the effect of forced lengthening contractions on muscle microarchitecture. Tibialis anterior (TA) muscle of male Wistar rats were subjected to 240 forced lengthening contractions. At consecutive time points (0, and 6 h, 2, 4, and 7 days) after stimulation, the TA muscle was excised for biochemical and histological assays. Beta-Glucuronidase activity, a quantitative indicator of muscle damage, showed increased values 2-7 days after the lengthening, peaking on day 4 (11.7-fold increase). A typical course of histopathological changes (myofibre swelling, necrosis and rege…

Malemedicine.medical_specialtyNecrosisPhysiologyClinical BiochemistryDesminDystrophinMyofibrilsPhysiology (medical)Internal medicinemedicineMyocyteAnimalsRats WistarIntermediate filamentMuscle SkeletalActinGlucuronidasebiologyChemistrySkeletal muscleImmunohistochemistryActinsElectric StimulationFibronectinsRatsEnzyme ActivationEndocrinologymedicine.anatomical_structurebiology.proteinDesminStress Mechanicalmedicine.symptomMyofibrilDystrophinMuscle ContractionPflugers Archiv : European journal of physiology
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Cell type heterogeneity of intermediate filament expression in epithelia of the human pituitary gland.

1989

In the present study we have localized immunohistochemically the intermediate filament proteins of the human pituitary gland (adenohypophysis, pars intermedia and pars tuberalis) by an indirect immunoperoxidase technique or by double immunofluorescence methods and analysed the individual cytokeratin polypeptides using two-dimensional gel electrophoresis. We found that the expression of cytokeratins in different epithelial cells of the human anterior pituitary gland was heterogeneous. Whereas the endocrine cells only expressed cytokeratins 8 and 18, the folliculo-stellate cells exhibited a reactivity for cytokeratins 7, 8, 18 and 19 as well as for GFAP and vimentin. The squamous epithelial c…

Malemedicine.medical_specialtyPituitary glandHistologyVimentinmacromolecular substancesEpitheliumDesminCytokeratinIntermediate Filament ProteinsInternal medicineGlial Fibrillary Acidic ProteinmedicineIntermediate Filament ProteinHumansVimentinIntermediate filamentMolecular BiologybiologyPars intermediaEpithelial CellsCell BiologyGeneral MedicineMolecular biologyImmunohistochemistryMedical Laboratory Technologymedicine.anatomical_structureEndocrinologyPituitary Glandbiology.proteinKeratinsFemalePars tuberalisAnatomyGeneral Agricultural and Biological SciencesEndocrine glandHistochemistry
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The Role of Immunohistochemistry in Rhabdomyosarcoma Diagnosis Using Tissue Microarray Technology and a Xenograft Model

2015

Rhabdomyosarcomas (RMS) may resemble other non-myogenic sarcomas and malignant rhabdoid tumor (MRT). Alveolar rhabdomyosarcoma (ARMS) often harbors a typical translocation, but embryonal rhabdomyosarcoma (ERMS) lacks any specific rearrangement. Histopathology is not always sufficient for an unequivocal diagnosis, necessitating ancillary studies, including immunohistochemistry (IHC). Sixteen genetically tested RMS and two MRT were xenografted and followed in successive passages. Tissue microarrays were constructed including samples from original and xenograft tumors. Desmin, myogenin, CK, EMA, INI1, LSD1, AP2 beta, fibrillin-2, HMGA2, nestin, and SIRT1 were tested using immunohistochemical s…

Malemusculoskeletal diseasesmedicine.medical_specialtyPathologygenetic structuresMice NudeBiologyPathology and Forensic MedicineDiagnosis DifferentialMiceRhabdomyosarcomaBiomarkers TumormedicineAnimalsHumansRhabdomyosarcomaRhabdoid TumorTissue microarraytissue microarraysGeneral MedicineNestinmedicine.diseasemusculoskeletal systemImmunohistochemistryDisease Models AnimalxenograftsTissue Array AnalysisPediatrics Perinatology and Child HealthimmunohistochemistryAlveolar rhabdomyosarcomaCancer researchHeterograftsImmunohistochemistryHistopathologyDesminEmbryonal rhabdomyosarcomarhabdomyosarcoma
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Immunocytochemical typification of mesothelial cells in effusions: in vivo and in vitro models.

1994

We have performed immunocytochemical, immunoelectron microscopy, Western blot, and culture techniques using monoclonal antibodies against cytokeratin, vimentin, and desmin on 17 benign and 20 malignant effusions of pleural and ascitic origin. Triple coexpression of these three antigens was observed in benign reactive mesothelial cells as well as in one case of mesothelioma. All metastatic adenocarcinoma cells were consistently negative to desmin and positive to cytokeratin and vimentin. Present results were helpful to distinguish reactive and malignant mesothelioma from metastatic carcinoma cells in effusions.

MesotheliomaPathologymedicine.medical_specialtyHistologyImmunoelectron microscopyBlotting WesternVimentinmacromolecular substancesBiologyAdenocarcinomaModels BiologicalEpitheliumPathology and Forensic MedicineMetastatic carcinomaDesminDiagnosis DifferentialImmunoenzyme TechniquesPleural diseaseCytokeratinmedicineAscitic FluidHumansVimentinCells CulturedGeneral Medicinemedicine.diseasePleural Effusionbiology.proteinAdenocarcinomaKeratinsDesminFemaleMesothelial CellFollow-Up StudiesDiagnostic cytopathology
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Atrial natriuretic peptide and CD34 overexpression in human idiopathic dilated cardiomyopathies.

2007

Idiopathic dilated cardiomyopathy (IDCM) is a primary myocardial disease of unknown cause characterized by ventricular chamber enlargement with impaired contractile function. In familial forms of IDCM, mutations of genes coding for cytoskeletal proteins related to force transmission, such as dystrophin, cardiac actin, desmin, and delta-sarcoglycan, have been identified. Here, we report the data of a retrospective investigation carried out to evaluate the expression of atrial natriuretic peptide (ANP), CD34, troponin T and nestin in the myocardium of patients affected with IDCM. Formalin-fixed and paraffin-embedded consecutive tissue sections from the ventricular wall of 10 human normal hear…

Microbiology (medical)ventricular myocytesCardiomyopathy Dilatedmedicine.medical_specialtyHeart VentriclesCardiomyopathyAntigens CD34Nerve Tissue ProteinsANP; CD34; nestin; troponin T; endothelial cells; ventricular myocytesPathology and Forensic MedicineNestinAtrial natriuretic peptideIntermediate Filament ProteinsTroponin TAntigens CDReference ValuesInternal medicineIdiopathic dilated cardiomyopathymedicineImmunology and AllergyHumansTroponin Tbiologybusiness.industryDilated cardiomyopathyGeneral MedicineNestinmedicine.diseaseTroponinImmunohistochemistryCardiologybiology.proteinendothelial cellDesminCD34AutopsybusinessANPAtrial Natriuretic FactorBiomarkersAPMIS : acta pathologica, microbiologica, et immunologica Scandinavica
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