Search results for "Digestive System"

showing 10 items of 1747 documents

Different mechanisms of the inhibition of the transient outward current in rat ventricular myocytes.

1994

The mechanism of drug-induced inhibition of the transient outward current, Ito, has been investigated in rat ventricular myocytes using the whole cell patch clamp technique. Ito was activated by 300 ms depolarizing voltage clamp steps in 10 mV increments from −50 mV up to +40 mV. At +40 mV, Ito peaked after about 3 ms, and the time course of inactivation was appropriately described by two time constants, τfast = 17 ms and τslow = 203 ms. Verapamil, quinidine sulfate and nifedipine preferentially depressed Ito at the end of the 300 ms depolarizing voltage clamp step; the inactivation of Ito was accelerated by all drugs, whereas peak Ito was less affected. The time course of drug action at +4…

MalePotassium ChannelsVoltage clampHeart VentriclesPharmacologydigestive systemMembrane PotentialsRats Sprague-Dawleychemistry.chemical_compoundQuinidine SulfateNifedipinemedicineAnimalsVentricular FunctionPatch clampCells CulturedPharmacologyMembrane potentialCardiac transient outward potassium currentMyocardiumHeartGeneral MedicineTetraethylammonium chlorideRatsElectrophysiologychemistryBiophysicsVerapamilmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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KRAS and BRAF mutations in circulating tumour DNA from locally advanced rectal cancer.

2018

Abstract There are limited data on circulating, cell-free, tumour (ct)DNA analysis in locally advanced rectal cancer (LARC). Digital droplet (dd)PCR was used to investigate KRAS/BRAF mutations in ctDNA from baseline blood samples of 97 LARC patients who were treated with CAPOX followed by chemoradiotherapy, surgery and adjuvant CAPOX ± cetuximab in a randomised phase II trial. KRAS mutation in G12D, G12V or G13D was detected in the ctDNA of 43% and 35% of patients with tumours that were mutant and wild-type for these hotspot mutations, respectively, according to standard PCR-based analyses on tissue. The detection rate in the ctDNA of 10 patients with less common mutations was 50%. In 26 ca…

MaleProto-Oncogene Proteins B-rafOrganoplatinum CompoundsScienceCetuximabArticleCirculating Tumor DNAProto-Oncogene Proteins p21(ras)SDG 3 - Good Health and Well-beingAntineoplastic Combined Chemotherapy ProtocolsJournal ArticleHumansneoplasmsCapecitabineDigestive System Surgical ProceduresAgedRectal NeoplasmsQRChemoradiotherapy AdjuvantMiddle AgedPrognosisdigestive system diseasesOxaliplatinTreatment OutcomeMutation/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingMedicineFemaleScientific reports
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TA-MUC1 epitope in non-small cell lung cancer

2007

MUC1 (CD227), an established tumor marker, is expressed on glandular epithelia and on epithelial tumors. Tumor MUC1 differs from normal MUC1 by modified glycan side chains. Recently, a novel carbohydrate-induced conformational tumor-associated MUC1 epitope (TA-MUC1) was described, whose clinical relevance in lung cancer is not known. Eighty-five paraffin embedded tissue sections of non-small cell lung cancer (NSCLC) patients (73% male; mean age 64+/-9 years) were stained with the monoclonal antibody PankoMab (against TA-MUC1) and compared with the established antibodies E29 and 214D4 regarding prognostic relevance. TA-MUC1 is virtually absent in bronchial epithelium. As shown by multivariat…

MalePulmonary and Respiratory MedicineCancer ResearchPathologymedicine.medical_specialtyLung Neoplasmsmedicine.drug_classMonoclonal antibodydigestive systemAntibodiesEpitopeEpitopesCarcinoma Non-Small-Cell LungCarcinomamedicineHumansskin and connective tissue diseasesLung cancerneoplasmsLymph nodeMUC1AgedTumor markerbiologybusiness.industryMucin-1Antibodies MonoclonalMiddle AgedPrognosismedicine.diseaseImmunohistochemistrybiological factorsdigestive system diseasesTreatment Outcomemedicine.anatomical_structureOncologyLymphatic Metastasisbiology.proteinFemaleAntibodybusinessLung Cancer
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MUC1 intracellular bioactivation mediates lung fibrosis

2019

BackgroundSerum KL6/mucin 1 (MUC1) has been identified as a potential biomarker in idiopathic pulmonary fibrosis (IPF), but the role of MUC1 intracellular bioactivation in IPF is unknown.ObjectiveTo characterise MUC1 intracellular bioactivation in IPF.Methods and resultsThe expression and phosphorylation of Thr41 and Tyr46 on the intracellular MUC1-cytoplasmic tail (CT) was increased in patients with IPF (n=22) compared with healthy subjects (n=21) and localised to fibroblasts and hyperplastic alveolar type II cells. Transforming growth factor (TGF)-β1 phosphorylated SMAD3 and thereby increased the phosphorylation of MUC1-CT Thr41 and Tyr46 in lung fibroblasts and alveolar type II cells, ac…

MalePulmonary and Respiratory MedicineCellRisk AssessmentTransforming Growth Factor beta1BleomycinMice03 medical and health sciencesIdiopathic pulmonary fibrosis0302 clinical medicinemedicineAnimalsHumansGene silencingMolecular Targeted TherapyRNA MessengerSmad3 ProteinFibroblastneoplasmsCells CulturedMUC1030304 developmental biologyMice Knockout0303 health sciencesbusiness.industryBiopsy NeedleMucin-1Fibroblastsmedicine.diseaseImmunohistochemistryIdiopathic Pulmonary Fibrosisdigestive system diseasesDisease Models Animalmedicine.anatomical_structureGene Expression Regulation030228 respiratory systemCancer researchFemalebusinessMyofibroblastIntracellularSignal TransductionTransforming growth factorThorax
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Expression of KIT (CD117) in Biphasic Pulmonary Blastoma. Novel Data on Histogenesis

2003

Biphasic pulmonary blastoma (BPB) is a rare primary neoplasm of the lung and its histogenesis is still uncertain. It has been proposed that BPB is derived from mesoderm or endoderm. Others suggested an origin from a single pluripotential cell. We present a case of a BPB with emphasis on expression of the stem cell factor receptor KIT (CD117). We describe a 61-year-old male patient with a BPB of the upper right lobe. Immunohistochemical analysis was performed using a panel of several antibodies including anti-CD117. Strong cytoplasmic expression of CD117 was found in the epithelium (cytokeratin-positive) as well as in the spindle cells (cytokeratin-negative). Expression of CD117 in both mese…

MalePulmonary and Respiratory MedicineMesodermPathologymedicine.medical_specialtyLung NeoplasmsStromal cellbiologyCD117Biphasic Pulmonary BlastomaMesenchymal stem cellStem cell factorMiddle AgedHistogenesisImmunohistochemistrydigestive system diseasesProto-Oncogene Proteins c-kitmedicine.anatomical_structurebiology.proteinmedicineHumansEndodermLungLung
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The density and type of MECA-79-positive high endothelial venules correlate with lymphocytic infiltration and tumour regression in primary cutaneous …

2013

Aims Tumour-infiltrating lymphocytes have prognostic value in malignant melanoma. High endothelial venules (HEVs) are specialized vessels present in lymph nodes and tertiary lymphoid organs. CCL19, CCL21 and CCR7 regulate lymphocyte migration through HEVs. The aim of our study was to correlate HEV density in cutaneous primary and metastatic malignant melanomas with clinicopathological parameters, and with CCL19, CCL21 and CCR7 mRNA expression. Methods and results High endothelial venule density was evaluated by immunohistochemistry with a specific antibody, MECA-79, and chemokine expression was evaluated by real-time PCR. MECA-79-positive vessels, covered by cuboidal (C-HEV) or flat (F-HEV)…

MaleReceptors CCR7Pathologymedicine.medical_specialtySkin NeoplasmsHistologyEndotheliumvirusesHigh endothelial venulesC-C chemokine receptor type 7BiologyPathology and Forensic MedicineLymphocytes Tumor-InfiltratingmedicineHumansRNA MessengerRNA NeoplasmMelanomaLymphatic VesselsRetrospective StudiesChemokine CCL21MelanomaMembrane Proteinsvirus diseasesGeneral MedicineMiddle AgedPrognosismedicine.diseaseImmunohistochemistrydigestive system diseasesLymphatic systemmedicine.anatomical_structureAntigens SurfaceCutaneous melanomaChemokine CCL19ImmunohistochemistryFemaleCCL21Histopathology
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Dissemination of hepatocellular carcinoma is mediated via chemokine receptor CXCR4

2006

In different tumour entities, expression of the chemokine receptor 4 (CXCR4) has been linked to tumour dissemination and poor prognosis. Therefore, we evaluated, if the expression of CXCR4 exerts similar effects in human hepatocellular carcinoma (HCC). Expression analysis and functional assays were performed in vitro to elucidate the impact of CXCL12 on human hepatoma cells lines. In addition, expression of CXCR4 was evaluated in 39 patients with HCC semiquantitatively and correlated with both, tumour and patients characteristics. Human HCC and hepatoma cell lines displayed variable intensities of CXCR4 expression. Loss of p53 function did not impact on CXCR4 expression. Exposure to CXCL12 …

MaleReceptors CXCR4Cancer ResearchChemokinePathologymedicine.medical_specialtyCarcinoma HepatocellularActive Transport Cell NucleusliverSensitivity and SpecificityCXCR4MetastasisChemokine receptorhepatocellularCell MovementPredictive Value of TestsTumor Cells CulturedCarcinomamedicinemetastasisHumansNeoplasm InvasivenessReceptorMolecular DiagnosticsCell ProliferationCXCR4biologychemokineLiver NeoplasmsMiddle AgedFlow Cytometrymedicine.diseaseImmunohistochemistryChemokine CXCL12digestive system diseasesSurvival RateOncologyHepatocellular carcinomaDisease ProgressionCancer researchbiology.proteinImmunohistochemistryFemaleChemokines CXCBritish Journal of Cancer
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Gut bacterial ClpB-like gene function is associated with decreased body weight and a characteristic microbiota profile

2020

[Background]: The chaperone ClpB, a bacterial protein, is a conformational antigen-mimetic of α-melanocyte-stimulating hormone (α-MSH) implicated in body weight regulation in mice. We here investigated the potential associations of gut bacterial ClpB-like gene function with obesity status and gut microbiota in humans.

MaleRikenellaceaeGut floraPrevotellaceaemedicine.disease_causeFecesMice0302 clinical medicineOverweight personsClostridiaceae2. Zero hunger0303 health sciencesIntestins -- MalaltiesEndopeptidase ClpFecal Microbiota TransplantationMiddle AgedPersones obeses3. Good healthIntestins -- Microbiologialcsh:QR100-130FemaleIntestines -- DiseasesAdultMicrobiology (medical)medicine.medical_specialtyFirmicutesBiologyIntestines -- Microbiologydigestive systemMicrobiologylcsh:Microbial ecology03 medical and health sciencesInternal medicinemedicineAnimalsHumansMicrobiomeObesityBacterial gene functionEscherichia coliAged030304 developmental biologyBacteriaResearchBacterial gene function ; Body weight regulation ; Microbiome ; Obesitybiology.organism_classificationGastrointestinal MicrobiomeMice Inbred C57BLCross-Sectional StudiesEndocrinologyCase-Control StudiesBody weight regulationMicrobiomeCLPB030217 neurology & neurosurgery
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Association analyses identify 31 new risk loci for colorectal cancer susceptibility

2019

Colorectal cancer (CRC) is a leading cause of cancer-related death worldwide, and has a strong heritable basis. We report a genome-wide association analysis of 34,627 CRC cases and 71,379 controls of European ancestry that identifies SNPs at 31 new CRC risk loci. We also identify eight independent risk SNPs at the new and previously reported European CRC loci, and a further nine CRC SNPs at loci previously only identified in Asian populations. We use in situ promoter capture Hi-C (CHi-C), gene expression, and in silico annotation methods to identify likely target genes of CRC SNPs. Whilst these new SNP associations implicate target genes that are enriched for known CRC pathways such as Wnt …

MaleScienceInheritance Patternscancer genetics/Datasets as Topiccolorectal cancerGenome-wide association studiesPolymorphism Single NucleotideArticleWhite PeopleAsian PeopleRisk FactorsCancer genomicsHumansGenetic Predisposition to Diseaselcsh:ScienceCancer geneticsneoplasmscancer genomicsQgenomiikkaMiddle AgedColorectal cancerdigestive system diseasesperäsuolisyöpäsyöpägeenitGenetic LociCase-Control Studiesgenome-wide association studieslcsh:QsyöpätauditFemaleColorectal NeoplasmsGenome-Wide Association StudyNature Communications
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Hepatitis B virus reactivation and alemtuzumab therapy

2005

Reactivation of hepatitis B virus infection in subjects receiving cytotoxic treatment for heamatological malignancies occurs in 21–53% of chronic HBsAg carriers and in an unknown number of HBsAg negative subjects harbouring occult HBV infection. Immmunotherapy with alemtuzumab, a humanized monoclonal antibody against CD52 epitopes on lymphocytes cells produces deep immunosuppression. We describe two subjects with chronic lymphocytic leukaemia and occult HBV infection who developed a virological and biochemical flare of hepatitis B following immunotherapy with alemtuzumab. One of them developed full blown hepatitis with seroreversion from anti-HBs to HBsAg after four weeks of alemtuzumab the…

MaleSettore MED/07 - Microbiologia E Microbiologia ClinicaHepatitis B virusHBsAgCD52Antibodies Neoplasmmedicine.medical_treatmenthepatitis B viruAntibodies Monoclonal Humanizedmedicine.disease_causeCampath-1HSettore MED/15 - Malattie Del SanguemedicineHumansAlemtuzumabImmunosuppression TherapyHepatitisHepatitis B virusSettore MED/12 - GastroenterologiaHepatitis B Surface Antigensbusiness.industryacute hepatitiAntibodies Monoclonalvirus diseasesLamivudineImmunosuppressionHematologyGeneral MedicineMiddle AgedHepatitis BHepatitis Bmedicine.diseaseLeukemia Lymphocytic Chronic B-Celldigestive system diseasesLamivudineDNA ViralImmunologyReverse Transcriptase InhibitorsAlemtuzumabFemaleVirus Activationbusinesschronic lymphocytic leukaemiamedicine.drugEuropean Journal of Haematology
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