Search results for "EFFECTOR"

showing 10 items of 217 documents

Kinetic models for nucleocytoplasmic transport of messenger RNA

1995

Abstract Much is known about the mechanism by which mRNAs cross the nuclear envelope (the translocation stage of nucleocytoplasmic transport), but far less is known about the preceding (intranuclear migration/release) and succeeding (cytoplasmic binding) stages. Therefore, existing information suffices for articulating detailed kinetic models of translocation, but not models for the overall mRNA transport process. In this paper, we show that simple kinetic models of translocation can (i) accommodate date about nucleocytoplasmic distributions of endogenous transcripts; (ii) predict the overall effects on these distributions of effectors such as insulin and epidermal growth factor; (iii) thro…

Cell NucleusStatistics and ProbabilityCytoplasmMessenger RNAModels GeneticGeneral Immunology and MicrobiologyMechanism (biology)EffectorApplied MathematicsChromosomal translocationGeneral MedicineBiologyTranslocation GeneticGeneral Biochemistry Genetics and Molecular BiologyCell biologyKineticsBiochemistryNucleocytoplasmic TransportEpidermal growth factorCytoplasmModeling and SimulationAnimalsMRNA transportRNA MessengerGeneral Agricultural and Biological SciencesJournal of Theoretical Biology
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NKG2D induces Mcl-1 expression and mediates survival of CD8 memory T cell precursors via phosphatidylinositol 3-kinase.

2013

Abstract Memory formation of activated CD8 T cells is the result of a specific combination of signals that promote long-term survival and inhibit differentiation into effector cells. Much is known about initial cues that drive memory formation, but it is poorly understood which signals are essential during the intermediate stages before terminal differentiation. NKG2D is an activating coreceptor on Ag-experienced CD8 T cells that promotes effector cell functions. Its role in memory formation is currently unknown. In this study, we show that NKG2D controls formation of CD8 memory T cells by promoting survival of precursor cells. We demonstrate that NKG2D enhances IL-15–mediated PI3K signalin…

Cell SurvivalImmunologyCytomegalovirusBiologyCD8-Positive T-LymphocytesLymphocyte ActivationMiceMemory cellPrecursor cellmedicineImmunology and AllergyCytotoxic T cellAnimalsIL-2 receptorReceptors ImmunologicInterleukin-15Mice KnockoutPrecursor Cells T-LymphoidNK cells; NKG2D; CD8 T cellsEffectorCell DifferentiationNKG2DNKG2D; CD8 T cell memory; Mcl1; PI3KCell biologyMice Inbred C57BLmedicine.anatomical_structureProto-Oncogene Proteins c-bcl-2NK Cell Lectin-Like Receptor Subfamily KCytomegalovirus InfectionsMyeloid Cell Leukemia Sequence 1 ProteinPhosphatidylinositol 3-KinaseMemory T cellImmunologic MemoryCD8Signal TransductionJournal of immunology (Baltimore, Md. : 1950)
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Herpes virus entry mediator synergizes with Toll-like receptor mediated neutrophil inflammatory responses

2006

In microbial infections polymorphnuclear neutrophils (PMN) constitute a major part of the innate host defence, based upon their ability to rapidly accumulate in inflamed tissues and clear the site of infection from microbial pathogens by their potent effector mechanisms. The recently described transmembrane receptor herpes virus entry mediator (HVEM) is a member of the tumour necrosis factor receptor super family and is expressed on many haematopoietic cells, including T cells, B cells, natural killer cells, monocytes and PMN. Interaction of HVEM with the natural ligand LIGHT on T cells has a costimulatory effect, and increases the bactericidal activity of PMN. To further characterize the f…

Cell SurvivalNeutrophilsImmunologyInflammationBiologyLigandsCell DegranulationNeutrophil ActivationPhagocytosismedicineHumansImmunology and AllergyOpsoninCells CulturedRespiratory BurstToll-like receptorInnate immune systemEffectorInterleukin-8Toll-Like ReceptorsDegranulationOriginal ArticlesAcquired immune systemRespiratory burstCell biologyImmunologyInflammation Mediatorsmedicine.symptomReceptors Tumor Necrosis Factor Member 14Immunology
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In vitro effects of aminobisphosphonates on Vgamma9Vdelta2 T cell activation and differentiation.

2006

In this study we have evaluated the in vitro effects of four different aminobisphosphonates, alendronate, risedronate, neridronate and zoledronate, on Vγ9Vδ2 T cell activation and differentiation. All tested aminobisphosphonates induce an IL-2-dependent activation and expansion of Vγ9Vδ2 T lymphocytes in primary PBMC cultures of healthy donors. Most notably, they also determine a different distribution of Vγ9Vδ2 T cell subsets, with decrease of Tnaive and TCM cells and increase of TEM and TEMRA Vγ9Vδ2 cells, indicating that in vitro treatment with aminobisphosphonates induces Vγ9Vδ2 T lymphocytes to differentiate towards an effector/cytotoxic phenotype. Accordingly, Vγ9Vδ2 T lymphocytes cu…

Cell SurvivalT cellT-LymphocytesImmunologyLymphocyte ActivationPeripheral blood mononuclear cellMonocytesFlow cytometry03 medical and health sciencesInterferon-gamma0302 clinical medicinemedicineImmunology and AllergyCytotoxic T cellHumansCytotoxicityImage CytometryPharmacologymedicine.diagnostic_testBone Density Conservation AgentsDiphosphonatesChemistryEffectorCell DifferentiationFlow CytometryPhenotypeIn vitromedicine.anatomical_structure030220 oncology & carcinogenesisCancer research030215 immunologyInternational journal of immunopathology and pharmacology
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Induction of secondary cytotoxic T-lymphocytes in vitro does not require cell proliferation.

1976

SummaryUsing a mouse in vitro allograft model, evidence has been obtained that, in contrast to the accepted view, the generation of cytotoxic effector function in T-lymphocytes does not necessarily require cell division.

Cell divisionCell growthEffectorT-LymphocytesMice Inbred StrainsBiologymedicine.diseaseCytotoxicity Tests ImmunologicGeneral Biochemistry Genetics and Molecular BiologyIn vitroMitomycinsTissue cultureMiceHistocompatibility AntigensImmunologyCancer researchmedicineNeoplasmCytotoxic T cellAnimalsImmunologic MemoryFunction (biology)Cell DivisionProceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.)
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A natural-like synthetic small molecule impairs bcr-abl signaling cascades and induces megakaryocyte differentiation in erythroleukemia cells

2013

Over the past years, we synthesized a series of new molecules that are hybrids of spirocyclic ketones as complexity-bearing cores with bi- and ter-phenyls as privileged fragments. Some of these newly-shaped small molecules showed antiproliferative, pro-apoptotic and differentiating activity in leukemia cell lines. In the present study, to investigate more in depth the mechanisms of action of these molecules, the protein expression profiles of K562 cells treated with or without the compounds IND_S1, MEL_T1, IND_S7 and MEL_S3 were analyzed using two-dimensional gel electrophoresis coupled with mass spectrometry. Proteome comparisons revealed several differentially expressed proteins, mainly r…

Cell signalingProteomeMegakaryocyte differentiationCellular differentiationFusion Proteins bcr-abllcsh:MedicineBiologyProteomicsSmall Molecule Librariesbi- and ter-phenylsantiproliferative pro-apoptotic differentiating activity leukemiaMolecular Cell BiologyChemical BiologyBiomarkers TumorCluster AnalysisHumansnetwork analysiRNA Messengerlcsh:ScienceBiologyCell ShapeMultidisciplinaryGene Expression Regulation LeukemicEffectorSystems Biologylcsh:RleukemiaReproducibility of ResultsHNF4-alphaHematologyMolecular biologyNeoplasm ProteinsChemistrycell differentiationSpectrometry Mass Matrix-Assisted Laser Desorption-IonizationMultivariate AnalysisProteomeMedicineEGR1PROTEOMICSlcsh:QLeukemia Erythroblastic AcuteMedicinal ChemistrySignal transductionK562 CellsMegakaryocytesResearch ArticleSignal TransductionK562 cells
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Notch in T Cell Differentiation: All Things Considered.

2015

Differentiation of naive T cells into effector cells is required for optimal protection against different classes of microbial pathogen and for the development of immune memory. Recent findings have revealed important roles for the Notch signaling pathway in T cell differentiation into all known effector subsets, raising the question of how this pathway controls such diverse differentiation programs. Studies in preclinical models support the therapeutic potential of manipulating the Notch pathway to alleviate immune pathology, highlighting the importance of understanding the mechanisms through which Notch regulates T cell differentiation and function. We review these findings here, and outl…

Cell typeReceptors NotchEffectorT cellT-LymphocytesImmunologyNotch signaling pathwayCell DifferentiationBiologyLymphocyte ActivationImmune systemmedicine.anatomical_structureT cell differentiationImmunologymedicineImmunology and AllergyAnimalsHumansReceptorFunction (biology)Signal TransductionTrends in immunology
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Research in practice: Different dendritic cell types in skin with various functions - important implications for intradermal vaccines

2011

Summary It was long believed that epidermal Langerhans cells (LC) are responsible for the initiation of cellular immunity. Only recently it has been shown that in skin alone 5 different subtypes of dendritic cells (DC) can be identified. Among these, LC, but also two Langerin-expressing dermal DC populations and two more Langerin-negative DC subtypes exist. Novel findings in the model disease leishmaniasis, as well as evidence from research in contact hypersensitivity, have revealed that activation of LC in skin leads to induction of regulatory, immunosuppressive T cells, whereas the other skin DC subtypes stimulate effector T cells. Thus, when producing vaccines designed for intradermal us…

Cellular immunityintegumentary systembusiness.industryEffectorIntradermal useContact hypersensitivityLeishmaniasisDermatologyDendritic cellmedicine.diseaseModel diseaseImmunologymedicinebusinessJDDG: Journal der Deutschen Dermatologischen Gesellschaft
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IL-17 and related cytokines involved in the pathology and immunotherapy of multiple sclerosis: Current and future developments.

2014

Multiple sclerosis (MS), an autoimmune neurological disorder, is driven by self-reactive T helper (Th) cells. Research on the role of Th17 lymphocytes in MS pathogenesis has made significant progress in identifying various immunological as well as environmental factors that induce the differentiation and expansion of these cells, different subsets of Th17 cells with varying degrees of pathogenicity, and the role of the secreted effector cytokines. While approved therapies for MS offer significant benefit to patients, there remain unmet needs. Ongoing clinical trials aim to translate the advanced knowledge of Th17 cytokines to improved therapies. This review discusses the current status and …

Central Nervous SystemPathologymedicine.medical_specialtyEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisEndocrinology Diabetes and Metabolismmedicine.medical_treatmentImmunologyAutoimmunityNeurological disorderGeneral Biochemistry Genetics and Molecular BiologyUnmet needsPathogenesisMicemedicineImmunology and AllergyAnimalsHumansEffectorbusiness.industryMultiple sclerosisInterleukin-17Cell DifferentiationImmunotherapyInterferon-betamedicine.diseaseClinical trialImmunologyTh17 CellsInterleukin 17ImmunotherapyInflammation MediatorsbusinessCytokinegrowth factor reviews
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Similar Camp Transfer Of Naturally Occurring Regulatory T Cells More Effectively Suppresses Effector Functions Of Th1 Compared To Th2 Cells

2011

ChemistryIL-2 receptorEffector functionsCell biologyB36. AIRWAY IMMUNE MECHANISMS AND INFLAMMATION: ANIMAL MODELS
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