Search results for "FGF-2"

showing 10 items of 23 documents

Fibroblast growth factor-2 and its receptor expression in proliferating precursor cells of the subventricular zone in the adult rat brain

2008

Several findings have suggested the existence in the subventricular zone (SVZ) from sagittal sections of adult rat brain of a trophic mechanism, mediated by fibroblast growth factor-2 (FGF-2) and its multiple high-affinity FGF receptors (FGFRs), regulating neurogenesis mainly by controlling precursor cell proliferation. However, no clear data are available on the expression of FGF-2 and FGFRs in proliferating precursor cells of the SVZ. To address these questions we examined FGF-2 mRNA and its FGFR mRNA expression in proliferating precursor cells of the SVZ by using a double labeling procedure, combining in situ hybridization for FGF-2 and its FGFR mRNA with immunohistochemistry for bromode…

Maleanimal diseasesReceptor expressionGene ExpressionFGF-2Subventricular zoneSVZBiologySettore BIO/09 - FisiologiaCerebral Ventricleschemistry.chemical_compoundPrecursor cellmedicineAnimalsReceptor Fibroblast Growth Factor Type 3RNA MessengerReceptor Fibroblast Growth Factor Type 1Rats WistarReceptor Fibroblast Growth Factor Type 2BrdUCell ProliferationFGF receptorGeneral NeuroscienceFibroblast growth factor receptor 1NeurogenesisBrainPrecursor cellsFibroblast growth factor receptor 4RatsCell biologyAdult Stem CellsFGF receptorsmedicine.anatomical_structureBromodeoxyuridinenervous systemchemistryFibroblast growth factor receptorFibroblast Growth Factor 2NeuroscienceBromodeoxyuridineBrdU; FGF receptors; FGF-2Neuroscience Letters
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Nicotine-induced fibroblast growth factor-2 restores the age-related decline of precursor cell proliferation in the subventricular zone of rat brain.

2007

Precursor cell proliferation is present in the subventricular zone (SVZ) of the lateral ventricles and the subgranular zone (SGZ) of the dentate gyrus of the hippocampus of adult rat and persists during aging although at reduced levels. Previous studies have shown that acute intermittent nicotine treatment significantly increases fibroblast growth factor-2 (FGF-2) expression in several brain regions of aged rats. The aim of the present investigation was to test the hypothesis that nicotine-induced expression of FGF-2 may restore the age-related decline of precursor cell proliferation. It was first demonstrated that nicotine treatment increases both mRNA and protein FGF-2 in the SVZ of aged …

Malemedicine.medical_specialtyAgingNicotineBasic fibroblast growth factorSubventricular zoneCell CountNerve Tissue ProteinsBiologyFibroblast growth factorSettore BIO/09 - FisiologiaAntibodiesSubgranular zoneNestinchemistry.chemical_compoundIntermediate Filament ProteinsInternal medicinePrecursor cellLateral VentriclesGlial Fibrillary Acidic ProteinmedicineAnimalsNicotinic AgonistsRats WistarMolecular BiologyCell ProliferationAnalysis of VarianceCell growthGeneral NeuroscienceDentate gyrusFibroblast growth factor receptor 1BrainPrecursor proliferationFGF-2 FGFR1 SVZ Nicotine AgedRatsAdult Stem CellsEndocrinologymedicine.anatomical_structurenervous systemchemistryBromodeoxyuridineGene Expression RegulationFibroblast Growth Factor 2Neurology (clinical)Developmental BiologyBrain research
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Acute intermittent nicotine treatment produces a reduction in the total number of FGF-2 immunoreactive astroglial cells in the substantia nigra of th…

2004

To understand the morphological substrate of the nicotine effect on nigral FGF-2 expression, a stereological analysis of FGF-2 immunoreactive neuronal and glial profiles has been performed in the substantia nigra of the rat after acute intermittent nicotine treatment. The major finding of this paper is the demonstration that this type of nicotine treatment produces a significant reduction in the total number of nuclear FGF-2 immunoreactive astroglial profiles in the substantia nigra. A parallel analysis of nigral FGF-1 and FGF-5 immunoreactivities showed no effect of this type of nicotine treatment. The results may be explained by an inhibition of FGF-2 synthesis in a subpopulation of nigra…

Malemedicine.medical_specialtyNicotineCentral nervous systemFGF-2Substantia nigraStereologyFGF-1Cell CountBiologyFibroblast growth factornicotine; FGF-2; stereology; immunoreactivity; substantia nigra; FGF-1; FGF-5FGF-5NicotineRats Sprague-DawleyStereotaxic TechniquesInternal medicinemedicineAnimalsGeneral NeuroscienceAlkaloidImmunohistochemistryRatsSubstantia Nigramedicine.anatomical_structureEndocrinologyNicotinic agonistnervous systemsubstantia nigraAstrocytesStereotaxic techniquestereologyFibroblast Growth Factor 2immunoreactivitymedicine.drugnicotineNeuroscience letters
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An asymmetric electrospun membrane for the controlled release of ciprofloxacin and FGF-2: Evaluation of antimicrobial and chemoattractant properties.

2021

Here, an asymmetric double-layer membrane has been designed and fabricated by electrospinning as a tool for a potential wound healing application. A hydrophobic layer has been produced by using a polyurethane-polycaprolactone (PU-PCL) copolymer and loaded with the antibacterial ciprofloxacin whereas an ion responsive hydrophilic layer has been produced by using an octyl derivative of gellan gum (GG-C8) and polyvinyl alcohol (PVA) and loaded with the growth factor FGF-2. This study investigated how the properties of this asymmetric membrane loaded with actives, were influenced by the ionotropic crosslinking of the hydrophilic layer. In particular, the treatment in DPBS and the crosslinking i…

Materials sciencePolyurethanesNanofibersBioengineeringmacromolecular substances02 engineering and technologyChemotaxis (FGF-2)Antimicrobial activity (CPX); Chemotaxis (FGF-2); Double layer electrospun membrane; Gellan gum alkyl-derivative; Polyurethanes010402 general chemistry01 natural sciencesPolyvinyl alcoholGellan gum alkyl-derivativeBiomaterialschemistry.chemical_compoundAnti-Infective AgentsCiprofloxacinCopolymerDouble layer electrospun membraneChemotactic Factorstechnology industry and agriculture021001 nanoscience & nanotechnologyAntimicrobialControlled releaseBandagesGellan gumElectrospinning0104 chemical sciencesAnti-Bacterial AgentsAntimicrobial activity (CPX)MembranechemistryMechanics of MaterialsSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoDelayed-Action PreparationsBiophysicsFibroblast Growth Factor 20210 nano-technologyLayer (electronics)Materials scienceengineering. C, Materials for biological applications
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Neurotrophic systems promoting neurogenesis in the adult rat brain

2008

Neurogenesis neurotrophic factors FGF-2Settore BIO/09 - Fisiologia
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CLUSTERING OF SPECIFIC MOLECULES IN SHED VESICLES.

2005

In several tumor cell lines serum addition causes release of vesicles that bud from the cell surface and can be purified from cell conditionated media. These vesicles are known to be involved in cell migration and tumor progression. We recently demonstrated that FGF-2, a growth factor devoid of the classical signaling sequence, is secreted as a component of these vesicles. In order to analyze how molecules are clustered in shed vesicles we followed their intracellular movements by immunofluorescence techniques. The role of cytoskeletal components was analyzed using molecules such as paclitaxol, nocodazole, colchicin and cytochalasin which destabilize their organization. In the absence of se…

SHED VESICLES FGF-2 SphK.
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Nicotine-induced FGF-2 mRNA in rat brain is preserved during aging

2004

Indirect trophic actions of nicotine on brain during aging are suggested from observations describing nicotine as a cognitive enhancer, increasing vigilance and improving learning and memory, and both in vitro and in vivo models have demonstrated neuroprotective effects of nAChR agonists. Previously, we have reported that an acute intermittent (-)nicotine treatment significantly increases fibroblast growth factor-2 (FGF-2) mRNA and protein in several brain regions of rat brain. The present study was designed to analyse if nicotine-induced FGF-2 expression in the rat brain was preserved during aging. Using in situ hybridization and quantitative RNase protection assay the present paper report…

SenescenceNicotineAgingmedicine.medical_specialtyCentral nervous systemStimulationIn situ hybridizationBiologynAChRNeuroprotectionNicotine treatmentStriatumNicotineHippocampuInternal medicineGliamedicineSubstantia nigraAnimalsTissue DistributionRNA MessengerAcetylcholine receptorGeneral NeuroscienceBrainNeuronFGF-2 expression; nicotine treatment; hippocampus; substantia nigra; striatum; brain; neurons; glia; nAChR; neurotrophism; neuroprotectionNeuroprotectionRatsmedicine.anatomical_structureEndocrinologyGene Expression RegulationFGF-2 expressionNeurotrophismFibroblast Growth Factor 2Neurology (clinical)NeuronGeriatrics and GerontologyDevelopmental Biologymedicine.drug
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Shedding of Membrane Vesicles Mediates Fibroblast Growth Factor-2 Release from Cells

2003

Fibroblast growth factor-2 (FGF-2), a polypeptide with regulatory activity on cell growth and differentiation, lacks a conventional secretory signal sequence, and its mechanism of release from cells remains unclear. We characterized the role of extracellular vesicle shedding in FGF-2 release. Viable cells released membrane vesicles in the presence of serum. However, in serum-free medium vesicle shedding was dramatically down-regulated, and the cells did not release FGF-2 activity into their conditioned medium. Addition of serum to serum-starved cells rapidly induced intracellular FGF-2 clustering under the plasma membrane and into granules that colocalized with patches of the cell membrane …

SerumFGF-2 extracellular vesiclesBiologyFibroblast growth factorBiochemistryCulture Media Serum-FreeSettore MED/13 - EndocrinologiaCell Line; Tumor; Endothelial Cells; Fibroblast Growth Factor 2; Secretory VesiclesCell LineCell membraneSettore BIO/13 - Biologia ApplicataCell Line TumorSettore BIO/10 - BiochimicamedicineHumansProtein IsoformsFibroblastMolecular BiologyTumorSecretory VesiclesVesicleCell MembraneEndothelial CellsCell BiologyExtracellular vesicleSecretory VesicleCell biologyKineticsmedicine.anatomical_structureMicroscopy FluorescenceCell cultureFibroblast Growth Factor 2IntracellularJournal of Biological Chemistry
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NEURONS PRODUCE FGF-2 AND VEGF SECRETE THEM AT LEST IN PART BY SHEDDING EXTRACELLULAR VESCICLES

2007

Abstract We previously found that neurons are able to affect the ability of brain capillary endothelial cells to form in vitro a monolayer with properties resembling the blood-brain barrier. We then looked, by immunofluorescence and western analysis, for factors, produced by neurons, with the potential to influence growth and differentiation of endothelial cells. In the present paper, we report that neurons produce both vascular endothelial growth factor and fibroblast growth factor 2, two well-known angiogenic factors. More interestingly, we gained evidence that both factors are released by neurons, at least in part, by shedding of extracellular vesicles, that contain β1 integrin, a membra…

Vascular Endothelial Growth Factor AFGF-2BiologyFibroblast growth factorchemistry.chemical_compoundsheddingNeurofilament ProteinsGlial Fibrillary Acidic ProteinExtracellularAnimalsSecretionRats WistarCells CulturedNeuronsVesicleIntegrin beta1Secretory VesiclesCell BiologyArticlesVEGFTransport proteinCell biologyRatsVascular endothelial growth factorVascular endothelial growth factor AProtein TransportMembrane proteinchemistryAstrocytesMolecular Medicineneurons vesicles fibroblastic growth factor-2 vascular endothelial growth factorCamptothecinFibroblast Growth Factor 2Extracellular Spaceextracellular vesicles
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Cellule staminali A6 di topo producono vescicole che contengono HSP70 e FGF-2

2006

Vescicole HSP70. FGF-2
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